In the last two decades few topics in philosophy of science have received as much attention as mechanistic explanation. A significant motivation for these accounts is that scientists frequently use the term “mechanism” in their explanations of biological phenomena. While scientists appeal to a variety of causal concepts in their explanations, many philosophers argue or assume that all of these concepts are well understood with the single notion of mechanism. This reveals a significant problem with mainstream mechanistic (...) accounts– although philosophers use the term “mechanism” interchangeably with other causal concepts, this is not something that scientists always do. This paper analyses two causal concepts in biology–the notions of “mechanism” and “pathway”–and how they figure in biological explanation. I argue that these concepts have unique features, that they are associated with distinct strategies of causal investigation, and that they figure in importantly different types of explanation. (shrink)

Though common sense says that causes must temporally precede their effects, the hugely influential interventionist account of causation makes no reference to temporal precedence. Does common sense lead us astray? In this paper, I evaluate the power of the commonsense assumption from within the interventionist approach to causal modeling. I first argue that if causes temporally precede their effects, then one need not consider the outcomes of interventions in order to infer causal relevance, and that one can instead (...) use temporal and probabilistic information to infer exactly when X is causally relevant to Y in each of the senses captured by Woodward’s interventionist treatment. Then, I consider the upshot of these findings for causal decision theory, and argue that the commonsense assumption is especially powerful when an agent seeks to determine whether so-called “dominance reasoning” is applicable. (shrink)

Though causality seems to have a natural place in chemical thought, the analysis of the underlying causal concepts requires attention to two different research styles. In Part One I attempt a classification and critical analysis of several philosophical accounts of causal concepts which appear to be very diverse. I summarize this diversity which ranges from causality as displayed in regular concomitances of types of events to causality as the activity of agents. Part Two is concerned with (...) the analysis of contrasting chemical discourses, comparing the classical atomist style of Boyle, and Lavoisier and von Liebig with the later energeticist style of van’t Hoff and Hinshelwood. In detail different clusters of causal concepts can be abstracted from these discursive styles, yet they all approximate the Realist format for causal discourse. By way of summary I make an attempt to map the vernacular distinctions of Part Two onto the philosophical territory of Part One. The argument is rounded off with a brief analysis of a chemical publication of 2008. (shrink)

In this paper I analyze the process by which modelers in systems biology arrive at an adequate representation of the biological structures thought to underlie data gathered from high-throughput experiments. Contrary to views that causal claims and explanations are rare in systems biology, I argue that in many studies of gene regulatory networks modelers aim at a representation of causal structure. In addressing modeling challenges, they draw on assumptions informed by theory and pragmatic considerations in a manner that (...) is guided by an interventionist conception of causal structure. While doubts have been raised about the applicability of this notion of causality to complex biological systems, it is here seen to be an adequate guide to inquiry. (shrink)

The concept of scaffold is widespread in science and increasingly common in evolutionary biology. While this concept figures in causal explanations, it is not clear what scaffolds are and what role they play in those explanations. Here we present evolutionary scaffolding explanation as a distinct type of explanatory strategy, distinguishing it from other types of evolutionary explanation. By doing so, we clarify the meaning of “scaffold” as a causal concept and its potential contribution to accounts of evolutionary novelty (...) and major transitions. (shrink)

Volition and Allied Causal Concepts is a work of aetiology and metapsychology. Aetiology is the branch of philosophy and logic devoted to the study of causality (the cause-effect relation) in all its forms; and metapsychology is the study of the basic concepts common to all psychological discourse, most of which are causal. Volition (or free will) is to be distinguished from causation and natural spontaneity. The latter categories, i.e. deterministic causality and its negation, have been treated (...) in a separate work, The Logic of Causation. Volition may be characterized as personal causality, a relation between an agent (the self or soul) and his actions (acts of will). Unlike causation, this relation cannot be entirely defined using conditional (if–then) propositions. Although we can say that the agent is a sine qua non of his actions, we cannot say that the agent is invariably (in all or specific circumstances) followed by his actions. It appears that both an act of will and its negation remain possible to a soul in any given set of circumstances. This defines freedom of the will, and implies the responsibility of the agent for his actions. Introspection provides knowledge of particular acts of will. The existence of freewill implies a distinction between necessary causation (determinism independent of volition) and inertial causation (determinism, except when some contrary will interferes). An act of will occurs on a spiritual plane. It may have natural (mental or physical) consequences; those that inevitably follow it may be regarded as directly willed, whereas those that vary according to circumstances must be considered indirectly willed. Volition presupposes some degree of consciousness. So-called involuntary acts of will involve a minimum of attention, whereas mindful acts are fully conscious. Even pure whim involves intention. Most volitions moreover involve valuation, some sort of projection of goals, deliberation on means, choice and decision. To judge responsibility, various distinctions are called for, like that between intentional, incidental and accidental consequences. Volitional action can be affected through the terms and conditions of the world surrounding its agent, but also more intimately through the influence of concrete or abstract aspects of that world that the subject has cognized. The causal concept of influence, and its implication of cognition (of inner or outer information, including emotions), are crucial to measuring the effort involved in volition. Influences make willing easier or harder, yet do not curtail its essential freedom. All the causal concepts used in psychological explanation – affections, appetites, instincts, habits, obsessions, compulsions, urges and impulses – can be elucidated thanks to this important finding. Much of human (and animal) behavior can thus be both acknowledged as volitional and as variously influenced. Volition and Allied Causal Concepts is a work of ambitious scope, intent on finally resolving philosophical and logical issues that have always impeded progress in psychology. It clarifies the structure and workings of the psyche, facilitating hygienic and therapeutic endeavors. The relation between volition and physical laws is discussed, as is the place of volition in biology. Concepts used in biology, analogous to that of purpose, are incidentally analyzed. Theological issues are also dealt with, as are some topics in ethics and law. (shrink)

Albert Einstein's attitude towards quantum mechanics—and statistical physics in general—was a puzzle to many of his contemporaries, and has remained a puzzle to the present. Though he made many significant contributions to statistical physics, he continually refused to regard that branch of science as fundamental. The present essay demonstrates that his attitude towards statistical physics was formed during his earliest investigations—between 1901 and 1903. In particular, it is shown that in Einstein's view, statistical laws are based upon non-statistical assumptions. This (...) view influenced much of his later thinking. A few illustrations are offered, concluding with a discussion of his opposition to quantum mechanics. (shrink)

A major debate in the philosophy of biology centers on the question of how we should understand the causal structure of natural selection. This debate is polarized into the causal and statistical positions. The main arguments from the statistical side are that a causal construal of the theory of natural selection's central concept, fitness, either (i) leads to inaccurate predictions about population dynamics, or (ii) leads to an incoherent set of causal commitments. In this essay, I (...) argue that neither the predictive inaccuracy nor the incoherency arguments successfully undermine the causal account of fitness. 1 Introduction2 The Importance of Trait Fitness3 Trait Fitness is not a Silver Bullet4 The Fundamental Incoherency Argument5 Car Racing and Trait Fitness Reversals6 Population Subdivisions and Evolution7 The STP and the Argument for the Incoherency of the Causal Account of Fitness8 Conclusions. (shrink)

A major debate in the philosophy of biology centers on the question of how we should understand the causal structure of natural selection. This debate is polarized into the causal and statistical positions. The main arguments from the statistical side are that a causal construal of the theory of natural selection's central concept, fitness, either (i) leads to inaccurate predictions about population dynamics, or (ii) leads to an incoherent set of causal commitments. In this essay, I (...) argue that neither the predictive inaccuracy nor the incoherency arguments successfully undermine the causal account of fitness. 1 Introduction2 The Importance of Trait Fitness3 Trait Fitness is not a Silver Bullet4 The Fundamental Incoherency Argument5 Car Racing and Trait Fitness Reversals6 Population Subdivisions and Evolution7 The STP and the Argument for the Incoherency of the Causal Account of Fitness8 Conclusions. (shrink)

This paper argues that both teleological and causal concepts are required for explanations of intentional actions. It argues against ‘causalism’, the idea that action explanations are essentially causal. This requires analyzing Mele’s Q-Signals-from-Mars argument that having a purpose and behaving so as to achieve it aren’t sufficient to explain an intentional action. Though Mele’s example shows that external causal interference can defeat the claim that an intentional action has been performed, this is consistent with teleological (...) class='Hi'>concepts being required (even if not sufficient) for action explanation. Mele’s example would work even if causalism were true. But causalism is false. Causalism depends on the idea that ‘agents always do what they want’ can be understood as saying agents have mental states, desires, that cause their behavior. But intentional actions involve what agents want only in the sense that actions have purposes, which are not mental states and cannot be the causes of actions. To perform an intentional action is to pursue some purpose in some way. This paper argues that neither the reference to the purpose that explains why the action was performed, nor the causal account of how this purpose was pursued, can be eliminated. (shrink)

How are causal judgements such as 'The ice on the road caused the traffic accident' connected with counterfactual judgements such as 'If there had not been any ice on the road, the traffic accident would not have happened'? This volume throws new light on this question by uniting, for the first time, psychological and philosophical approaches to causation and counterfactuals. Traditionally, philosophers have primarily been interested in connections between causal and counterfactual claims on the level of meaning or (...) truth-conditions. More recently, however, they have also increasingly turned their attention to psychological connections between causal and counterfactual understanding or reasoning. At the same time, there has been a surge in interest in empirical work on causal and counterfactual cognition amongst developmental, cognitive, and social psychologists--much of it inspired by work in philosophy. In this volume, twelve original contributions from leading philosophers and psychologists explore in detail what bearing empirical findings might have on philosophical concerns about counterfactuals and causation, and how, in turn, work in philosophy might help clarify the issues at stake in empirical work on the cognitive underpinnings of, and relationships between, causal and counterfactual thought. (shrink)

We start this paper by arguing that causality should, in analogy with force in Newtonian physics, be understood as a theoretical concept that is not explicated by a single definition, but by the axioms of a theory. Such an understanding of causality implicitly underlies the well-known theory of causal nets and has been explicitly promoted by Glymour. In this paper we investigate the explanatory warrant and empirical content of TCN. We sketch how the assumption of directed cause–effect relations can (...) be philosophically justified by an inference to the best explanation. We then ask whether the explanations provided by TCN are merely post-facto or have independently testable empirical content. To answer this question we develop a fine-grained axiomatization of TCN, including a distinction of different kinds of faithfulness. A number of theorems show that although the core axioms of TCN are empirically empty, extended versions of TCN have successively increasing empirical content. (shrink)

Abstract1. Introduction2. Causality in 19th and Early 20th Century Medicine3. A Lakatosian Approach to the History of Medicine.

Causality in 19th and Early 20th Century Medicine 3 A Lakatosian Approach to the History of Medicine.

This paper proposes a possible reconstruction and philosophical-logical clarification of Gödel's idea of causality as the philosophical fundamental concept. The results are based on Gödel's published and non-published texts (including Max Phil notebooks), and are established on the ground of interconnections of Gödel's dispersed remarks on causality, as well as on the ground of his general philosophical views. The paper is logically informal but is connected with already achieved results in the formalization of a causal account of Gödel's onto-theological (...) theory. Gödel's main causal concepts are analysed (will, force, enjoyment, God, time and space, life, form, matter). Special attention is paid to a possible causal account of some of Gödel's logical concepts (assertion, privation, affirmation, negation, whole, part, general, particular, subject, predicate, necessary, possible, implication), as well as of logical antinomies. The problem of mechanical and non-mechanical procedures in the work with and on concepts is addressed in terms of Gödel's causal view. (shrink)

How feasible are conceptual engineering projects of social concepts that aim for the engineered concept to be widely adopted in ordinary everyday life? Predominant frameworks on the psychology of concepts that shape work on stereotyping, bias, and machine learning have grim implications for the prospects of conceptual engineers: conceptual engineering efforts are ineffective in promoting certain social-conceptual changes. Specifically, since conceptual components that give rise to problematic social stereotypes are sensitive to statistical structures of the environment, purely conceptual (...) change won’t be possible without corresponding world change. This tradition, however, tends to ignore that concepts don’t only encode statistical, but also causal information. Paying attention to this feature of concepts, I argue, shows that conceptual engineering is not only possible. There is an imperative to conceptually-engineer. (shrink)

Although there has been much recent discussion on mechanisms in philosophy of science and social theory, no shared understanding of the crucial concept itself has emerged. In this paper, a distinction between two core concepts of mechanism is made on the basis that the concepts correspond to two different research strategies: the concept of mechanism as a componential causal system is associated with the heuristic of functional decomposition and spatial localization and the concept of mechanism as an (...) abstract form of interaction is associated with the strategy of abstraction and simple models. The causal facts assumed and the theoretical consequences entailed by an explanation with a given mechanism differ according to which concept of mechanism is in use. Research strategies associated with mechanism concepts also involve characteristic biases that should be taken into account when using them, especially in new areas of application. (shrink)

The recent literature on causality has seen the introduction of several distinctions within causality, which are thought to be important for understanding the widespread scientific practice of focusing causal explanations on a subset of the factors that are causally relevant for a phenomenon. Concepts used to draw such distinctions include, among others, stability, specificity, proportionality, or actual-difference making. In this contribution, I propose a new distinction that picks out an explanatorily salient class of causes in biological systems. Some (...) select causes in complex biological systems, I argue, have the property of enabling coherent causal control of these systems. Examples of such control variables include hormones and other signaling molecules, e.g., TOR (target of rapamycin), morphogens or the products of homeotic selector genes in embryonic pattern formation. I propose an analysis of this notion based on concepts borrowed from causal graph theory. (shrink)

ABSTRACT There is a disagreement over how to understand Nietzsche's view of science. According to what I call the Negative View, Nietzsche thinks science should be reconceived or superseded by another discourse, such as art, because it is nihilistic. By contrast, what I call the Positive View holds that Nietzsche does not think science is nihilistic, so he denies that it should be reinterpreted or overcome. Interestingly, defenders of each position can appeal to Nietzsche's understanding of naturalism to support their (...) interpretation. I argue that Nietzsche embraces a social constructivist conception of causality that renders his naturalism incompatible with the views of naturalism attributed to him by the two dominant readings. (shrink)

The kernel of this volume is an English translation of Jan Łukasiewicz’s classic work on the concept of cause (1906). It is the starting point for analytical considerations on causality of two generations of philosophers belonging to the tradition of the Lvov-Warsaw School.

I propose a new account of homology, according to which homology is a correspondence of developmental mechanisms due to common ancestry, formally defined as an isomorphism of causal graphs over lineages. The semiformal definition highlights the role of homology as a higher-order principle unifying evolutionary models and also provides definite meanings to concepts like constraints, evolvability, and novelty. The novel interpretation of homology suggests a broad perspective that accommodates evolutionary developmental biology and traditional population genetics as distinct but (...) complementary approaches to understand evolution, prompting further empirical and theoretical research. (shrink)

The doctrine of the immaculate conception, which is a dogma binding on all Roman Catholics and also held by members of some other Christian denominations, holds that Mary the mother of Jesus Christ was conceived without the stain of original sin as a result of the redeeming effects of Christ’s later life, passion, death, and resurrection. In this paper I argue first that, even on an orthodox reading of this doctrine, the immaculate conception seems to result in a kind of (...) causal time loop. I then consider several common philosophical objections to causal time loops, showing how each is either not a serious problem for causal time loops in general or is not a serious problem for the immaculate conception time loop in particular because of some particular features of that particular loop. The upshot of this discussion is that it shows that anyone who is committed to the dogma of the immaculate conception is also committed to the possibility, and, indeed, the actuality, of at least one causal time loop, but also that this is no reason to reject the dogma, since all of the major worries for causal time loops can be resolved in one way or another. (shrink)

The present paper deals with the tools that can be used to represent causation and to reason about it and, specifically, with their diversity. It focuses on so-called “causal probabilities”—that is, probabilities of effects given one of their causes—and critically surveys a recent paper in which Joyce argues that the values of these probabilities do not depend on one’s conception of causation. I first establish a stronger independence claim: I show that the very definition of causal probabilities is (...) independent of one’s conception of causation. Second, I investigate whether causal probabilities indeed take the same values under their different possible definitions. (shrink)

In his recent article, ``Self-Consciousness'’, George Bealer has set outa novel and interesting argument against functionalism in the philosophyof mind. I shall attempt to show, however, that Bealer's argument cannotbe sustained.In arguing for this conclusion, I shall be defending three main theses.The first is connected with the problem of defining theoreticalpredicates that occur in theories where the following two features arepresent: first, the theoretical predicate in question occurswithin both extensional and non-extensional contexts; secondly, thetheory in question asserts that the relevant (...) theoretical states enterinto causal relations. What I shall argue is that a Ramsey-styleapproach to the definition of such theoretical terms requires twodistinct quantifiers: one which ranges over concepts, and theother which ranges over properties in the world.My second thesis is a corollary: since the theories on whichBealer is focusing have both of the features just mentioned, and sincethe method that he employs to define theoretical terms in his argumentagainst functionalism does not involve both quantifiers that range overproperties and quantifiers that range over concepts, that method isunsound.My final thesis is that when a sound method is used, Bealer's argumentagainst functionalism no longer goes through.The structure of my discussion is as follows. I begin by setting out twoarguments – the one, a condensed version of Bealer's argument, andthe other, an argument that parallels Bealer's argument very closely.The parallel argument leads to a conclusion, however, that, rather thanbeing merely somewhat surprising, seems very implausible indeed. Forwhat the second argument establishes, if sound, is that there can betheoretical terms that apply to objects by virtue of their first-orderphysical properties, but whose meaning cannot be defined via aRamsey-style approach.Having set out the two parallel arguments, I then go on to focus uponthe second, to determine what is wrong with it. My diagnosis will bethat the problem with the argument arises from the fact that it involvesdefining a theoretical term that occurs both inside and outside ofopaque contexts, for the method employed fails to take into account thefact that the types of entities that are involved in the relevanttruthmakers are different when a sentence occurs within an extensionalcontext from those involved when a sentence occurs within anon-extensional context.I then go on to discuss how one should define a theoretical term thatoccurs within such theories, and I argue that in such a case one needstwo quantifiers, ranging over different types of entities – on theone hand, over properties and relations, and the other, over concepts. Ithen show that, when such an approach is followed, the argument inquestion collapses.I then turn to Bealer's argument against functionalism, and I show,first, that precisely the same method of defining theoretical terms canbe applied there, and, secondly, that, when this is done, it turns outthat that argument is also unsound.Next, I consider two responses that Bealer might make to my argument,and I argue that those responses would not succeed.Finally, I conclude by asking exactly where the problem lies in the caseof Bealer's argument. My answer will be that it is not simply the factthat one is dealing with a theoretical term that occurs in bothextensional and non-extensional contexts. It is rather the combinationof that feature together with the fact that the theory in questionasserts that the relevant type of theoretical state enters into causalrelations. For the first of these features means that the Ramseysentence for the theory must involve quantification over concepts, whilethe presence of the second feature means that the Ramsey sentence mustinvolve quantification over properties in the world, and so no attemptto offer a Ramsey-style account of the meaning of the relevanttheoretical term can succeed unless one employs both quantification overconcepts and quantification over properties. Bealer, however, in hisargument against functionalism, uses a method of defining theoreticalterms that does not involve both types of quantification, and it isprecisely because of this that his argument does not in the end succeed. (shrink)

There is an assumption common in the philosophy of mind literature that kinds in our sciences—or causal kinds, at least—are individuated by the causal powers that objects have in virtue of the properties they instantiate. While this assumption might not be problematic by itself, some authors take the assumption to mean that falling under a kind and instantiating a property amount to the same thing. I call this assumption the “Property-Kind Individuation Principle”. A problem with this principle arises (...) because there are cases where we can sort objects by their possession of common causal powers, and yet those objects do not intuitively form a causal kind. In this short note, I discuss why the Property-Kind Individuation Principle is thus not a warranted metaphysical assumption. (shrink)

This commentary argues that one specific but central concept in Lewis's theory, circular causality, is fundamentally flawed and should be discarded – first, because it does not make theoretical sense, and, second, because it leads to problems in practice, such as confounding the interaction between different systems with the relationship between different levels of analysis of a single system.

In their paper "How Fundamental Physics represents Causality", Andreas Bartels and Daniel Wohlfarth maintain that there is place for causality in General Relativity. Their argument contains two steps: First they show that there are time-asymmetric models in General Relativity, then they claim to derive that two events are causally connected if and only if there is a time-asymmetric energy flow from one event to the other. In our comment we first give a short summary of their paper followed by a (...) section introducing and pondering different conceptions of causation since Bartels and Wohlfarth don’t explicitly declare which notion of causation they build on in the paper. In order to analyze their argument in detail we formalize their crucial step in logical terms. This helps to pose the question whether their proposed derivation is not just a definition in a more precise way. (shrink)

Extended Abstract This book introduces and develops a new, universal method of the scientific comprehension of reality providing the objective, ...

A recent cover of the German news magazine Der Spiegel announced: “Victory over Genes. Smarter, healthier, happier: How we can outwit our genome” (2010). The magazine’s article, instead, emphasizes the importance of epigenetics. According to Florian Maderspacher (2010), who reprinted the cover in his editorial in Current Biology, the relief or “schadenfreude” about the apparent victory over genes—which the cover, the article, and commentaries to it reveal—is, in part, a German phenomenon. It echoes “a latent anti-scientific attitude in parts of (...) the German public,” which for historic reasons is particularly strong with regard to genetics: “After all, the idea that people’s properties—good or bad—are determined by .. (shrink)

Pharmacogenetics and pharmacogenomics are related facets of cutting edge therapeutic research in a field that relates pharmacological properties to the genetic characteristics of human beings. An optimistic interpretation suggests that “One-Size-Fits-All” therapeutics, whose effects can only be predicted in probabilistic terms, will give way eventually to individual tailor-made therapies with entirely predictable properties in each patient. Yet the concept of anticipating individual pharmacotherapeutic response appears to disregard some of the fundamental limitations of causal understanding in the biological world of (...) structure–action relationships. It is not simply a problem of confronting complexity – which might be overcome in time with advances in biotechnology and computing power – but the inescapable paradox of the genetic characteristics of individuals within the human species. The aleatory process of evolution ensures that with the exception of monozygotic twins, every human being is genetically unique. At the same time there appears to be more in common than divides. Pharmacogenomics provides a climate in which to explore and better understand the variable causal relationships between genetic element, protein and therapeutic or toxic actions, but can only do so in abstraction. Thus␣predictive power may be enhanced but is ultimately constrained by insuperable probabilistic considerations. (shrink)

Carey argues that the aspects of categorization that are diagnostic of deep conceptual structure and, by extension, narrow conceptual content, must be distinguished from those aspects that are incidental to categorization tasks. For natural kind concepts, discriminating between these two types of processes is complicated by the role of explanatory stance and the causal history of features in determining category structure.

Arguably no concept is more fundamental to science than that of causality, for investigations into cases of existence, persistence, and change in the natural world are largely investigations into the causes of these phenomena. Yet the metaphysics and epistemology of causality remain unclear. For example, the ontological categories of the causal relata have been taken to be objects (Hume 1739), events (Davidson 1967), properties (Armstrong 1978), processes (Salmon 1984), variables (Hitchcock 1993), and facts (Mellor 1995). (For convenience, causes and (...) effects will usually be understood as events in what follows.) Complicating matters, causal relations may be singular (Socrates’s drinking hemlock caused Socrates’s death) or general (Drinking hemlock causes death); hence the relata might be tokens (e.g., instances of properties) or types (e.g., types of events) of the category in question. Other questions up for grabs are: Are singular causes metaphysically and/or epistemologically prior to general causes or vice versa (or neither)? What grounds the intuitive asymmetry of the causal relation? Are macro-causal relations reducible to micro-causal relations? And perhaps most importantly: Are causal facts (e.g., the holding of causal relations) reducible to non-causal facts (e.g., the holding of certain spatiotemporal relations)? (shrink)

What precisely does aristotle mean when he asserts that something is (or comes to be) "for" "the" "sake" "of" something? I suggest that the answer to this question may be found by examining aristotle's position on the problem of reduction in biology, As it arises within his own scientific "and" "philosophical" context. I discuss the role of the concepts of "nature" and "potential" in aristotelian scientific explanation, And reformulate the reduction problem in that light. I answer the main question (...) by establishing that aristotle holds an "irreducibility" thesis in regard to the generation and development of a living organism, And that this thesis is the core of his conception of final causality. I conclude by arguing that aristotle's teleology is fundamentally "empirical" in character, And not an a priori doctrine brought "to" the study of nature. (shrink)

Norton has recently argued that causation is merely a useful folk concept and that it fails to hold for some simple systems even in the supposed paradigm case of a causal physical theory – namely Newtonian mechanics. The purpose of this article is to argue against this devaluation of causality in physics. My main argument is that Norton’s alleged counterexample to causality within standard Newtonian physics fails to obey what I shall call the causal core of Newtonian mechanics. (...) In particular, I argue, Norton’s example is not in conformity with Newton’s first law. Moreover, Norton’s reformulation of this first law seems insufficient as a replacement for the original version since the notion of inertial frames in the resulting reformulated theory lacks a physical justification, and since an intelligible notion of time in Newtonian mechanics appears to be closely tied to Newton’s first law in its standard form. I will finally suggest how, given a plausible relationist account of time, the causal core of Newtonian mechanics may play a central role also in relativity and quantum theory. (shrink)

Our encounters with the physical world are filled with miraculous puzzles-wind appears from somewhere, heavy objects float on oceans, yet smaller objects go to the bottom of our water-filled buckets. As adults, instead of confronting a whole world, we are reduced to driving from one parking garage to another. The Child's Conception of Physical Causality, part of the very beginning of the ground-breaking work of the Swiss naturalist Jean Piaget, is filled with creative experimental ideas for probing the most sophisticated (...) ways of thinking in children. The strength of Piaget's research is evident in this collection of empirical data, systematically organized by tasks that illuminate how things work. Piaget's data are remarkably rich. In his new introduction, Jaan Valsiner observes that Piaget had no grand theoretical aims, yet the book's simple power cannot be ignored. Piaget's great contribution to developmental psychology was his "clinical method"-a tactic that integrated relevant aspects of naturalistic experiment, interview, and observation. Through this systematic inquiry, we gain insight into children's thinking. Reading Piaget will encourage the contemporary reader to think about the unity of psychological phenomena and their theoretical underpinnings. His wealth of creative experimental ideas probes into the most sophisticated ways of thinking in children. Technologies change, yet the creative curiosity of children remains basically unhindered by the consumer society. Piaget's data preserve the reality of the original phenomena. As such, this work will provide a wealth of information for developmental psychologists and those involved in the field of experimental science. Jean Piaget is known for investigations of thought processes. He was professor at Geneva University and director of the International Center for Epistemology. He is the author of The Language and Thought of the Child, Judgment and Reasoning in the Child, The Origin of Intelligence in Children, and The Early Growth of Logic in the Child. Jaan Valsiner is professor of psychology at Clark University, and a recognized authority on the life and work of Piaget. (shrink)

This paper is concerned with the content of the causal proportionality constraint. It investigates two general versions of the constraint, namely “horizontal” and “vertical” proportionality. Moreover, it discusses whether proportionality is considered an ontic or an epistemic, i.e. explanatory, constraint on causation in the context of some of the most prominent theories of causation. The following main claims are defended: (1) The horizontal (HP) and the vertical version (VP) of the proportionality constraint are logically independent. (2) HP is implied (...) by some prominent theories of causation, not by others. (3) None of the discussed popular theories of causation contradicts either HP or VP. (4) HP and VP are not ontic or epistemic principles as such; rather, whether they are ontic or epistemic depends on the theories chosen plus background assumptions about the existence of higher-level causes and their non-identity to lower-level ones. (shrink)