Becoming productive with grammatical categories is a gradual process in children's language development. Here, we investigated this transition process by focusing on Turkish causatives. Previous research examining spontaneous and elicited production of Turkish causatives with familiar verbs attested the onset and early stages of productivity at ages 2 to 3 (Aksu-Koç & Slobin, 1985; Nakipoğlu, Uzundag, & Sarıgül, 2021). So far, however, we know very little about children's understanding of causatives with novel verbs. In the present study, (...) we asked: (a) When does the generalization of causative morphology in a novel context emerge? and (b) What role does child-directed input play in this development? To answer the first question, we conducted comprehension-judgment experiments with children aged 2;6–6;11 using pseudo-verbs (Study 1 & 2). Results showed that children preferred the Turkish causative suffix -DIr over an unrelated or no suffix to denote caused events earliest at age 4;10. To answer the second question, we analyzed child-directed speech from a longitudinal corpus of Turkish language acquisition (Study 3). Results showed that when addressing children younger than age 3, caregivers used the -DIr suffix with little variation considering the overall variability of verbs they could utter. Overall, these findings suggest that productivity with morphological causatives in a novel context emerges in a later stage of acquisition. This later development might partly be accounted for by the insufficient variation of morphological causatives in the early input. (shrink)

Grammatical theory has long wrestled with the fact that causative constructions exhibit properties of both single words and complex phrases. However, as Paul Kiparsky has observed, the distribution of such properties of causatives is not arbitrary: ‘construal’ phenomena such as honorification, anaphor and pronominal binding, and quantifier ‘floating’ typically behave as they would if causatives were syntactically complex, embedding constructions; whereas case marking, agreement and word order phenomena all point to the analysis of causatives as single lexical (...) items.1 Although an analysis of causatives in terms of complex syntactic structures has frequently been adopted in an attempt to simplify the mapping to semantic structure, we believe that motivating syntactic structure based on perceived semantics is questionable because in general a syntax/semantics homomorphism cannot be maintained without vitiating syntactic theory (Miller 1991). Instead, we sketch a strictly lexical theory of Japanese causatives that deals with the evidence offered for a complex phrasal analysis. Such an analysis makes the phonology, morphology and syntax parallel, while a mismatch occurs with the semantics. The conclusions we will reach are given in (1). (shrink)

Rousseau is considered to be at once the most modern political thinker of the 18th century and the most ancient in his allegiance to classical republicanism. These essays address the place of liberty in his moral and political philosophy, and the origins, meaning, strength, weakness and significance of his argument.

In his 2000 book, From Chaos to Coercion: Detention and the Control of Tuberculosis, Richard Coker makes a number of important observations and arguments regarding the use of coercive public health measures in response to infectious disease threats. In particular, Coker argues that we have a tendency to neglect public health threats and then demand immediate action, which can leave policymakers with fewer effective options and may require (or may be perceived as requiring) more aggressive, coercive measures to achieve public (...) health goals. While Coker makes a convincing case as to why we should find it ethically problematic when governments find themselves in this position and resort to coercion, left outstanding is the question of whether this should preclude governments and health authorities from using coercion if and when they do find themselves in this position. In this paper, I argue that, while we should consider it ethically objectionable when governments resort to coercion because they have neglected a public health threat, its causes, and other possible responses to that threat, this should not then necessarily rule out the use of coercion in such circumstances; that there are ethically objectionable antecedents for why coercion is being considered should not necessarily or automatically cause us to think coercion in such cases cannot be justified. I address an objection to this argument and draw several conclusions about how governments’ use of coercion in public health should be evaluated. (shrink)

There are two main formulations of a key causal principle in the Cartesian a priori philosophical system: one, present in Meditation III, says that the cause of the representational content of an idea must be situated at the same or higher level in ontology than the level at which the object represented is situated, the other, present in the axioms section of the Second Replies, says that the cause must contain the same property as is represented by the idea. This (...) central chapter defends four main contentions. The same‐property formulation is basic in Descartes's system. The notion of causality in the basic causal principle does not represent a spatio temporally extended natural process but a form of intentional explanation. When point is combined with the interpretation of the rule of truth offered in Ch. 2, the rule of truth and the basic causal principle prove to be equivalent. Finally, in light of, there is one main pattern of inference in Cartesian epistemology taking the rule of truth/causal principle as its major premise and underlying all of Descartes arguments from my ideas to the existence of things outside my ideas, including the proof of my own existence, the proof of the existence of God in Meditations III and V and the proof of the existence of the external world in Meditation VI and the Principles of Philosophy II,1. (shrink)

People generally accept that there is causation by omission—that the omission of some events cause some related events. But this acceptance elicits the selection problem, or the difficulty of explaining the selection of a particular omissive cause or class of causes from the causal conditions. Some theorists contend that dependence theories of causation cannot resolve this problem. In this paper, we argue that the appeal to norms adequately resolves the selection problem for dependence theories, and we provide novel experimental evidence (...) for it. (shrink)

This chapter explores some issues having to do with the structure of the evidential reasoning we use to infer causal and lawful claims. It is argued that such reasoning always makes use of prior, causally, or nomologically committed information, thus undercutting various views that attempt to reduce causal and lawful claims to claims about regularities. A non-reductive account of laws and causes built around the notion of invariance is advanced as an alternative.

Paul Pietroski presents an original philosophical theory of actions and their mental causes. We often act for reasons: we deliberate and choose among options, based on our beliefs and desires. However, bodily motions always have biochemical causes, so it can seem that thinking and acting are biochemical processes. Pietroski argues that thoughts and deeds are in fact distinct from, though dependent on, underlying biochemical processes within persons.

Hunting Causes and Using Them argues that causation is not one thing, as commonly assumed, but many. There is a huge variety of causal relations, each with different characterizing features, different methods for discovery and different uses to which it can be put. In this collection of new and previously published essays, Nancy Cartwright provides a critical survey of philosophical and economic literature on causality, with a special focus on the currently fashionable Bayes-nets and invariance methods - and it exposes (...) a huge gap in that literature. Almost every account treats either exclusively how to hunt causes or how to use them. But where is the bridge between? It's no good knowing how to warrant a causal claim if we don't know what we can do with that claim once we have it. This book will interest philosophers, economists and social scientists. (shrink)

It may seem obvious that causing disability in another person is morally problematic in a way that removing or preventing a disability is not. This suggests that there is a moral asymmetry between causing disability and causing non-disability. This chapter investigates whether there are any differences between these two types of actions that might explain the existence of a general moral asymmetry. After setting aside the possibility that having a disability is almost always bad or harmful for a person (a (...) view that we have critiqued at length elsewhere), seven putative differences are considered. Ultimately, it is concluded that none of these seven factors can ground a general moral asymmetry between causing disability and causing non-disability, though each factor can provide some moral reason to avoid causing disability in certain particular cases. (shrink)

Since the appearance of a widely influential book, Self-Knowledge and Self-ldentity, Sydney Shoemaker has continued to work on a series of interrelated issues in the philosophy of mind and metaphysics. This volume contains a collection of the most important essays he has published since then. The topics that he deals with here include, among others, the nature of personal and other forms of identity, the relation of time to change, the nature of properties and causality and the relation between the (...) two, dualism and immortality, and the nature of mental states. All the essays show the same care and precision in argument as the earlier book, but they also reveal a substantial shift in Professor Shoemaker's position to a form of materialism. In fact, a number of papers together constitute what is probably the most subtle and rigourous defence yet of a sophisticated functionalism in the account of the mind. (shrink)

What does it mean to ‘calculate’—today? The pause introduced by the dash in this question marks the inescapable necessity of historicizing the problem of calculation. In his provocative essay, ‘Toward a Critique of the Ineffectual: Heidegger’s Reading of Aristotle and the Construction of an Action without Ends’, Dimitris Vardoulakis proposes a philosophical and political programme in order to counter the negative effects of ‘Heidegger’s mistake’ (the conflation of causality and instrumentality through a mistranslation of Aristotle) that has led to the (...) theoretical fetishization of the ‘ineffectual’ (action without ends) in continental philosophy. In his conclusion, Vardoulakis advocates for a return to a politics and ethics of practical judgment that avoids the twin pitfalls of asserting either the impossibility or the absolute possibility of calculation. As he puts it, we need to acknowledge today ‘the exigency to calculate in the face of the provisional ends of instrumentality, or to perform the judgment that the Greeks called phronesis’ [2022: 244]. This is why we should keep in mind that Heidegger’s critique of technology in the 1950s was articulated in the context of the early Cold War, atomic warfare, and the rise of cybernetics. But, as many have recently argued, in the age of so-called ‘Big Data’, neoliberal rationality has increasingly replaced the epistemology of ‘causality’ with epistemologies of ‘correlation’: the algorithmic calculation of probabilistic relations between clusters of variables (based on information harvested by digital technologies designed for the total surveillance of human life). As Chris Anderson notoriously announced in his 2008 editorial, ‘The End of Theory’, ‘correlation supersedes causation, and science can advance even without coherent models, unified theories, or really any mechanistic explanation at all’. He calls this epistemology ‘dimensionally agnostic statistics’ (the amount of data available can no longer be visualized in its totality). This worldview, however, functions as a basic attack on one of the fundamental (and still often repeated) ground-truths of modern science: correlation is not causation. As Anderson puts it: ‘There is now a better way. Petabytes allow us to say: “Correlation is enough”’. In other words, the stakes of Vardoulakis’s argument must be tested against the rise of this neoliberal elimination of causality from corporate epistemologies that has led to an inversion of the terms of the Heideggerian critique of technology: as it turns out, technology has produced an ideology of pure instrumentality without causation. Today, there are only ends without causes. (shrink)

In Causes, Laws, and Free Will, Kadri Vihvelin argues that we can have free will even if everything we do is predictable given the laws of nature and the past. The belief that determinism robs us of free will springs from mistaken beliefs about the metaphysics of causation, the nature of laws, and the logic of counterfactuals.

A discussion of Aristotle’s thought on determinism and culpability, Necessity, Cause, and Blame also reveals Richard Sorabji’s own philosophical commitments. He makes the original argument here that Aristotle separates the notions of necessity and cause, rejecting both the idea that all events are necessarily determined as well as the idea that a non-necessitated event must also be non-caused. In support of this argument, Sorabji engages in a wide-ranging discussion of explanation, time, free will, essence, and purpose in nature. He also (...) provides historical perspective, arguing that these problems remain intimately bound up with modern controversies. “ Necessity, Cause and Blame would be counted by all as one of Sorabji’s finest. The book is essential for philosophers—both specialists on the Greeks and modern thinkers about free will—and also compelling for non-specialists.”—Martha Nussbaum “Original and important . . . The book relates Aristotle’s discussions to both the contemporary debates on determinism and causation and the ancient ones. It is especially detailed on Stoic arguments about necessity . . . and on the social and legal background to Aristotle’s thought.”— Choice “It is difficult to convey the extraordinary richness of this book. . . . A Greekless philosopher could read it with pleasure . . . At the same time, its learning and scholarship are enormous.”—G. E. M. Anscombe, Times Literary Supplement. (shrink)

Michael T. Ferejohn presents a new analysis of Aristotle's theory of explanation and scientific knowledge, in the context of its Socratic roots. Ferejohn shows how Aristotle resolves the tension between his commitment to the formal-case model of explanation and his recognition of the role of efficient causes in explaining natural phenomena.

Within evolutionary biology a distinction is frequently made between proximate and ultimate causes. One apparently plausible interpretation of this dichotomy is that proximate causes concern processes occurring during the life of an organism while ultimate causes refer to those processes (particularly natural selection) that shaped its genome. But ultimate causes are not sought through historical investigations of an organisms lineage. Rather, explanations referring to ultimate causes typically emerge from functional analyses. But these functional analyses do not identify causes of any (...) kind, much less ultimate ones. So-called ultimate explanations are not about causes in any sense resembling those of proximate explanations. The attitude, implicit in the term ultimate cause, that these functional analyses are somehow superordinate to those involving proximate causes is unfounded. Ultimate causes are neither ultimate nor causes. (shrink)

We propose a new definition of actual causes, using structural equations to model counterfactuals. We show that the definition yields a plausible and elegant account of causation that handles well examples which have caused problems for other definitions and resolves major difficultiesn in the traditional account.

We propose a new definition of actual causes, using structural equations to model counterfactuals. We show that the definition yields a plausible and elegant account of causation that handles well examples which have caused problems for other definitions and resolves major difficulties in the traditional account. 1. Introduction2. Causal models: a review2.1Causal models2.2Syntax and semantics3. The definition of cause4. Examples5. A more refined definition6. DiscussionAAppendix: Some Technical IssuesA.1The active causal processA.2A closer look at AC2(b)A.3Causality with infinitely many variablesA.4Causality in nonrecursive (...) models. (shrink)

A condition is formulated in terms of the probabilities of two pairs of correlated events in a classical probability space which is necessary for the two correlations to have a single (Reichenbachian) common-cause and it is shown that there exists pairs of correlated events probabilities of which violate the necessary condition. It is concluded that different correlations do not in general have a common common-cause. It is also shown that this conclusion remains valid even if one weakens slightly Reichenbach's definition (...) of common-cause. The significance of the difference between common-causes and common common-causes is emphasized from the perspective of Reichenbach's Common Cause Principle. (shrink)

I argue that the criterion of just cause is not independent of proportionality and other valid jus ad bellum criteria. One cannot know whether there is a just cause without knowing whether the other (valid) criteria (apart from ‘right intention’) are satisfied. The advantage of this account is that it is applicable to all wars, even to wars where nobody will be killed or where the enemy has not committed a rights violation but can be justifiably warred against anyway. This (...) account also avoids the inefficiency of having proportionality considerations come up at two different points: in a separate criterion of just cause and in the criterion of proportionality proper. ‘Right intention’, the subjective element of the justification of a war, on the other hand, is not to be subsumed under the criterion of just cause: there can be a just cause without anybody knowing it. Conversely, however, the subjective element requires that those responsible for waging the war do know that the justifying objective conditions are fulfilled. This is in one sense more demanding than traditional just war theory; in another sense, however, it is less demanding: nobody needs to intend to fight for a ‘just aim’. (shrink)

ABSTRACT The underlying cause of the financial meltdown was much more mundane than a “crisis of capitalism”: The real origins lay in mostly obscure housing, tax, and regulatory policies of the U.S. government. The Community Reinvestment Act, the affordable‐housing “mission” of Fannie Mae and Freddie Mac, penalty‐free refinancing of home loans, penalty‐free defaults on home loans, tax preferences for home‐equity borrowing, and reduced capital requirements for banks that held mortgages and mortgage‐backed securities combined with each other to create the incentives (...) for both subprime lending and the housing bubble that eventually led to the financial collapse. (shrink)

This is an expanded edition of Sydney Shoemaker's seminal collection of his work on interrelated issues in the philosophy of mind and metaphysics. Reproducing all of the original papers, many of which are now regarded as classics, and including four papers published since the first edition appeared in 1984, Identity, Cause, and Mind's reappearance will be warmly welcomed by philosophers and students alike.

We propose a new definition of actual causes, using structural equations to model counterfactuals. We show that the definition yields a plausible and elegant account of causation that handles well examples which have caused problems for other definitions and resolves major difficulties in the traditional account.

Is the common cause principle merely one of a set of useful heuristics for discovering causal relations, or is it rather a piece of heavy duty metaphysics, capable of grounding the direction of causation itself? Since the principle was introduced in Reichenbach’s groundbreaking work The Direction of Time (1956), there have been a series of attempts to pursue the latter program—to take the probabilistic relationships constitutive of the principle of the common cause and use them to ground the direction of (...) causation. These attempts have not all explicitly appealed to the principle as originally formulated; it has also appeared in the guise of independence conditions, counterfactual overdetermination, and, in the causal modelling literature, as the causal markov condition. In this paper, I identify a set of difficulties for grounding the asymmetry of causation on the principle and its descendents. The first difficulty, concerning what I call the vertical placement of causation, consists of a tension between considerations that drive towards the macroscopic scale, and considerations that drive towards the microscopic scale—the worry is that these considerations cannot both be comfortably accommodated. The second difficulty consists of a novel potential counterexample to the principle based on the familiar Einstein Podolsky Rosen (EPR) correlations in quantum mechanics. (shrink)

This article suggests a revision of the theory of causal nets. In section 1 we introduce an axiomatization of TCN based on a realistic understanding. It is shown that the causal Markov condition entails three independent principles. In section 2 we analyze indeterministic decay as the major counterexample to one of these principles: screening off by common causes. We call SCC-violating common causes interactive causes. In section 3 we develop a revised version of TCN, called TCN*, which accounts for interactive (...) causes. It is shown that there are interactive causal models that admit of no faithful noninteractive reconstruction. (shrink)

This paper is about the history of a question in ancient Greek philosophy and medicine: what holds the parts of a whole together? The idea that there is a single cause responsible for cohesion is usually associated with the Stoics. They refer to it as the synectic cause (αἴτιον συνεκτικόν), a term variously translated as ‘cohesive cause,’ ‘containing cause’ or ‘sustaining cause.’ The Stoics, however, are neither the first nor the only thinkers to raise this question or to propose a (...) single answer. Many earlier thinkers offer their own candidates for what actively binds parts together, with differing implications not only for why we are wholes rather than heaps, but also why our bodies inevitably become diseased and fall apart. This paper assembles, up to the time of the Stoics, one part of the history of such a cause: what is called ‘the synechon’ (τὸ συνέχον) – that which holds things together. Starting with our earliest evidence from Anaximenes (sixth century BCE), the paper looks at different candidates and especially the models and metaphors for thinking about causes of cohesion which were proposed by different philosophers and doctors including Empedocles, early Greek doctors, Diogenes of Apollonia, Plato and Aristotle. My goal is to explore why these candidates and models were proposed and how later philosophical objections to them led to changes in how causes of cohesion were understood. (shrink)

There is an initial presumption against disjunctive causes. First of all, for some people causation is a relation between events. But, arguably, there are no disjunctive events, since events are particulars and thus they have spatiotemporal locations, while it is unclear what the spatiotemporal location of a disjunctive event could be.1 More importantly, even if one believes that entities like facts can enter in causal relations, and even if there are disjunctive facts, it is still hard to see how disjunctive (...) facts could be causes. Imagine, for instance, the following scenario. I have a gun filled with red paint and another gun filled with blue paint, and I fire both guns at my neighbor’s white wall. A moment later, there is a graffiti on the wall and my neighbor notifies the police. He would have done so regardless of the graffiti’s color, since all he cares about is the existence of a graffiti on his wall. Is it plausible to claim that a disjunctive fact is a cause of his notifying the police? In particular, is it plausible to claim that he notified the police because I fired the red-paint gun or the blue-paint gun (the thought being that my firing paint of either color would have sufficed)? It seems not. The police was notified because of the actual graffiti on the wall, and the actual graffiti on the wall is made of a certain pattern of colored patches. Imagine, that, as it turns out, there are patches of both colors on the wall. Then it seems that both my firing the red-paint gun and my firing the blue-paint gun were causes of my neighbor’s notifying the police. In other words, my firing the red-paint gun and my firing the blue-paint gun jointly caused the outcome: each of them was a contributory cause of the outcome’s occurrence. On the other hand, imagine that there are only patches of one color on the wall. Then it seems that my firing only one of the guns was a cause. Either way, the disjunction fails to be a cause: either my firing the red-paint gun was a cause, or my firing the blue-paint gun was a cause, or they were both causes, but their disjunction was not.. (shrink)

Biologists studying complex causal systems typically identify some factors as causes and treat other factors as background conditions. For example, when geneticists explain biological phenomena, they often foreground genes and relegate the cellular milieu to the background. But factors in the milieu are as causally necessary as genes for the production of phenotypic traits, even traits at the molecular level such as amino acid sequences. Gene-centered biology has been criticized on the grounds that because there is parity among causes, the (...) “privileging” of genes reflects a reductionist bias, not an ontological difference. The idea that there is an ontological parity among causes is related to a philosophical puzzle identified by John Stuart Mill: what, other than our interests or biases, could possibly justify identifying some causes as the actual or operative ones, and other causes as mere background? The aim of this paper is to solve this conceptual puzzle and to explain why there is not an ontological parity among genes and the other factors. It turns out that solving this puzzle helps answer a seemingly unrelated philosophical question: what kind of causal generality matters in biology? (shrink)

Do I cause global warming, climate change and their related harms when I go for a leisure drive with my gas-guzzling car? The current verdict seems to be that I do not; the emissions produced by my drive are much too insignificant to make a difference for the occurrence of global warming and its related harms. I argue that our verdict on this issue depends on what we mean by ‘causation’. If we for instance assume a simple counterfactual analysis of (...) causation according to which ‘C causes E’ means ‘if C had not occurred, E would not have occurred’, we must conclude that a single drive does not cause global warming. However, this analysis of causation is well-known for giving counterintuitive results in some important cases. If we instead adopt Lewis’s analysis of causation, it turns out that it is indeterminate whether I cause global warming when I go for a single drive. Still, in contexts where we seek to control or understand global warming, there is a pressure to adopt a more fragile view of this event. When we adopt such a view, it turns out that a single drive does cause global warming. This means that we cannot like Sinnott-Armstrong and Kingston and Sinnott-Armstrong reject the idea that I should refrain from going for a leisure drive simply because such a drive does not cause global warming. (shrink)

Aristotle’s typical procedure is to identify something's four causes. Intentional action has typically been treated as an exception: most think that Aristotle has the standard causalist account, according to which an intentional action is a bodily movement efficiently caused by an attitude of the appropriate sort. I show that action is not an exception to Aristotle’s typical procedure: he has the resources to specify four causes of action, and thus to articulate a powerful theory of action unlike any other on (...) offer. (shrink)

A just cause for war is a type of wrong that may make those responsible for it morally liable to military attack as a means of preventing or rectifying it. This claim has implications that conflict with assumptions of the current theory of just war.

When is a cause of a cause of an effect also a cause of that effect? The right answer is either Sometimes or Always . In favour of Always , transitivity is considered by some to be necessary for distinguishing causes from redundant non-causal events. Moreover transitivity may be motivated by an interest in an unselective notion of causation, untroubled by principles of invidious discrimination. And causal relations appear to add up like transitive relations, so that the obtaining of the (...) overarching relation is not independent of the obtaining of the intermediaries. On the other hand, in favour of Sometimes , often we seem not to treat events that are very spatiotemporally remote from an effect as its causes, even when connected to the effect in question by a chain of counterfactual or chance-raising dependence. Moreover cases of double prevention provide counterexamples to causal transitivity even over short chains. According to the argument of this paper, causation is non-transitive. Transitizing causation provides no viable account of causal redundancy. An unselective approach to causation may motivate resisting the distance counterexamples to transitivity, but it does not help with double prevention, and even makes it more intractable. The strongest point in favour of transitivity is the adding up of causal relations, and this is the point that extant non-transitizing analyses have not adequately addressed. I propose a necessary condition on causation that explains the adding up phenomenon. In doing so it also provides a unifying explanation of distance and double prevention counterexamples to transitivity. (shrink)

We propose new definitions of (causal) explanation, using structural equations to model counterfactuals. The definition is based on the notion of actual cause, as defined and motivated in a companion article. Essentially, an explanation is a fact that is not known for certain but, if found to be true, would constitute an actual cause of the fact to be explained, regardless of the agent's initial uncertainty. We show that the definition handles well a number of problematic examples from the literature. (...) 1. Introduction2. Causal models and the definition of actual causality: a review2.1Causal models2.2Syntax and semantics2.3The definition of cause3. Explanation: the basic definition4. Partial explanations and explanatory power5. The general definition6. DiscussionAppendix: the formal definition of causality. (shrink)

When two rocks shatter the window at once, what causes the window to shatter? Is the throwing of each individual rock a cause of the window shattering, or are the throwings only causes collectively? This question bears on the analysis of causation, and the metaphysics of macro-causation. I argue that the throwing of each individual rock is a cause of the window shattering, and generally that individual overdeterminers are causes.

Most people are skeptical of the claim that the expectation that a person would have a life that would be well worth living provides a reason to cause that person to exist. In this essay I argue that to cause such a person to exist would be to confer a benefit of a noncomparative kind and that there is a moral reason to bestow benefits of this kind. But this conclusion raises many problems, among which is that it must be (...) determined how the benefits conferred on people by causing them to exist weigh against comparable benefits conferred on existing people. In particular, might the reason to cause people to exist ever outweigh the reason to save the lives of existing people? (shrink)

In this paper I start by briefly presenting an analysis of token cause and of token causal relevance that I developed elsewhere, and then apply it to the famous thirsty traveler riddle. One general outcome of the analysis of causal relevance employed here is that in preemption cases (early or late) the preempted cause is not a cause since it is causally irrelevant to the effect. I consider several variations of the thirsty traveler riddle. In the first variation the first (...) enemy emptied the canteen and the second enemy threw it away. On this variation, the act of neither enemy comes out, on the analysis employed here, as causally relevant to, and thus not as a cause of, the fact that the traveler died, but the conjunction of the two acts is a cause of it. This version is a case of mutual preemption. I argue that it has the same structure as the voting paradox, which thus has an analogous solution. In the standard version, in which the first enemy added poison to the water in the traveler's canteen, the act of the second enemy (who threw the canteen away) comes out, on the analysis used here, as causally relevant to and as a cause of the fact that the traveler died, but the act of the first enemy comes out as neither. I also make a comparison with Lewis' accounts, and discuss alternative treatments of the puzzle such as those of Hart and Honore and of Gavison, Margalit, and Ullmann-Margalit. (shrink)

Aristotle frequently describes essence as a “cause” or “explanation”, thus ascribing to essence some sort of causal or explanatory role. This explanatory role is often explicated by scholars in terms of essence “making the thing be what it is” or “making it the very thing that it is”. I argue that this is problematic, at least on the assumption that “making” expresses an explanatory relation, since it violates certain formal features of explanation. I then consider whether Aristotle is vulnerable to (...) this problem by examining the explanatory role of essence in Posterior Analytics and Metaphysics Z 17. (shrink)

Negative causation occurs when an absence serves as cause, effect, or causal intermediary. Negative causation is genuine causation, or so I shall argue. It involves no physical connection between cause and effect. Thus causes need not be physically connected to their effects.

Crosslinguistically, causative constructions conform to the following generalization: If the causal relation is syntactically concealed, then it is semantically direct. Concealed causatives span a wide syntactic spectrum, ranging from resultative complements in English to causative subjects in Miskitu. A unified type-driven theory is proposed which attributes the understood causal relation—and other elements of constructional meaning—to type lifting operations predictably licensed by type mismatch at LF. The proposal has far-reaching theoretical implications not only for the theory of compositionality and causation, (...) but also for the underlying theory of events, space, and time, in natural language discourse. (shrink)

The interventionist account of causation offers a criterion to distinguish causes from non-causes. It also aims at defining various desirable properties of causal relationships, such as specificity, proportionality and stability. Here we apply an information-theoretic approach to these properties. We show that the interventionist criterion of causation is formally equivalent to non-zero specificity, and that there are natural, information-theoretic ways to explicate the distinction between potential and actual causal influence. We explicate the idea that the description of causes should be (...) proportional to that of their effects. Then we draw a distinction between two ideas in the existing literature, the range of invariance of a causal relationship and its stability. The range of invariance is related to specificity and range of causal values. Stability concerns the effect of additional variables on the relationship between some focal pair of cause and effect variables. We show how to distinguish and measure the direct influence of background variables on the effect variable, and their influence on the relationship between the focal cause and the effect variable. Finally, we discuss the limitations of the information-theoretic approach, and offer prospects for complementary approaches. (shrink)

Many philosophers are persuaded by familiar arguments that free will is incompatible with causal determinism. Yet, notoriously, past attempts to articulate how the right type of indeterminism might secure the capacity for autonomous action have generally been regarded as either demonstrably inadequate or irremediably obscure. This volume gathers together the most significant recent discussions concerning the prospects for devising a satisfactory indeterministic account of freedom of action. These essays give greater precision to traditional formulations of the problems associated with indeterministic (...) accounts and to the range of theoretical avenues for pursuing resolutions. The first four essays set out different challenges (from both compatibilists and those skeptical of the possibility of free will) to the adequacy of standard indeterministic theories. The next seven essays meet one or more of these challenges. Each of the fundamental types of approach--simple indeterminism, causal indeterminism, and agent causation--is represented in these novel and sophisticated proposals. The collection finishes with two essays that debate whether compatibilism entails that freedom of choice is a comparatively rare phenomenon within an individual's life. Eloquently presenting some of the most compelling and accessible arguments surrounding this central philosophical issue, Agents, Causes, and Events makes a valuable contribution to courses in free will/action theory and metaphysics. (shrink)