What is the nature of causation? How is causation linked with explanation? And can there be an adequate theory of explanation? These questions and many others are addressed in this unified and rigorous examination of the philosophical problems surrounding causation, laws and explanation. Part 1 of this book explores Hume's views on causation, theories of singular causation, and counterfactual and mechanistic approaches. Part 2 considers the regularity view of laws and laws as relations among (...) universals, as well as recent alternative approaches to laws. Part 3 examines the issues arising from deductive-nomological explanation, statistical explanation, the explanation of laws and the metaphysics of explanation. Accessible to readers of all levels, this book provides an excellent introduction to one of the most enduring problems of philosophy. (shrink)

Causation is important. It is, as Hume said, the cement of the universe, and lies at the heart of our conceptual structure. Causation is one of the most fundamental tools we have for organizing our apprehension of the external world and ourselves. But philosophers' disagreement about the correct interpretation of causation is as limitless as their agreement about its importance. The history of attempts to elucidate the nature of this concept and to situate it with respect to (...) other fundamental concepts is almost as long as the history of philosophy itself. In this first English translation of Causalite; et lois de la nature Max Kistler seeks to reconstruct a unified concept of causation that is general enough to adequately deal with both elementary physical processes and the macroscopic level of phenomena we encounter in everyday life. It will be of great interest to philosophers of science and metaphysics; and also to students and scholars of philosophy of mind where concepts of causation and law play a prominent role. (shrink)

This is the first English translation of _Causalite´ et Lois de La Nature,_ and is an important contribution to the theory of causation_._ Max Kistler reconstructs a unified concept of causation that is general enough to adequately deal with both elementary physical processes, and the macroscopic level of phenomena we encounter in everyday life. This book will be of great interest to philosophers of science and metaphysics, and also to students and scholars of philosophy of mind where concepts of (...) causation and law play a prominent role. (shrink)

The article examines a recent interventionist account of causation by Ross, in which electronic configurations of atoms are considered to be the cause of chemical behavior. More specifically I respond to the claim that a change in electronic configuration of an atom, such as occurs in the artificial synthesis of elements, causes a change in the behavior of the atom in question. I argue that chemical behavior is governed as much by the nuclear charge of an atom as it (...) is by its electronic structure. It is suggested that an adequate analysis requires attention to the dynamical interactions between nuclear charges and those of electrons, as typically carried out through the application of the Schrödinger equation. It is concluded that electronic configurations can only be said be causal in a weak sense that is somewhat analogous to the causal arguments that are invoked in folk physics. (shrink)

In The Biopsychosocial Model of Health and Disease, Derek Bolton and Grant Gillett argue that a defensible updated version of the biopsychosocial model requires a metaphysically adequate account of disease causation that can accommodate biological, psychological, and social factors. This present paper offers a philosophical critique of their account of biopsychosocial causation. I argue that their account relies on claims about the normativity and the semantic content of biological information that are metaphysically contentious. Moreover, I suggest that (...) these claims are unnecessary for a defence of biopsychosocial causation, as the roles of multiple and diverse factors in disease causation can be readily accommodated by a more widely accepted and less metaphysically contentious account of causation. I then raise the more general concern that they are misdiagnosing the problem with the traditional version of the biopsychosocial model. The challenge when developing an explanatorily valuable version of the biopsychosocial model, I argue, is not so much providing an adequate account of biopsychosocial causation, but providing an adequate account of causal selection. Finally, I consider how this problem may be solved to arrive at a more explanatorily valuable and clinically useful version of the biopsychosocial model. (shrink)

While it is generally agreed that the concept of homology refers to individuated traits that have been inherited from common ancestry, we still lack an adequate account of trait individuation or inheritance. Here I propose that we utilize a counterfactual criterion of causation to link each trait with a developmental-causal (DC) gene. A DC gene is made up of the genetic information (which might or might not be physically contiguous in the genome) that is needed for the production (...) of the organismic attributes that comprise the trait. I argue that individuated traits—phenes—correspond to organismic features that are caused by DC genes. Using such an approach, we can define a DC map, which shows the relations between each pair of phenes and provides a succinct summary of genotype-phenotype relationships and phenotypic complexity. Phenes in parents and offspring are judged to be homologous if their DC genes are composed of orthologous genetic factors. When comparing more distantly related organisms, traits are homologous when linked by a chain of parent-offspring homologs along the path of ancestry that links the two organisms. There are three possible ways to deal with the potential for multiple equivalent DC genes: maximal, minimal, and consensus homology. Whereas maximal homology has limited utility, the other two approaches have value and can help to guide research at the intersection of evolution and development. (shrink)

Agent causation presupposes that actions are behaviors under the causal control of the agent’s mental states, its beliefs and desires. Here the idea of conscious causation in causal explanations of actions is examined, specifically, actions said to be the result of conscious efforts. Causal–functionalist theories of consciousness purport to be naturalistic accounts of the causal efficacy of consciousness. Flanagan argues that his causal–functionalist theory of consciousness satisfies naturalistic constraints on causation and that his causal efficacy thesis is (...) compatible with results of Libet’s experiments on conscious causation. First, the notions of conscious effort and conscious causation are analyzed with respect to the project of naturalizing the mind, that is, the attempt to assimilate folk-psychological explanation to the causal model of explanation in the natural sciences. It is argued that a serious obstacle for any naturalist program is that mental states are individuated by their semantic content, not the mechanistic, physical properties of their neural state instantiations. In particular, it is argued that explanation by reference to mental state content yields not a causal but an interpretive or rationalizing account of action in which the question of causal efficacy is irrelevant. Then, Flanagan’s causal–functionalist theory of consciousness is critically assessed; specifically his interpretations of Libet’s negative experimental results on the causal efficacy of consciousness are diagnosed and disputed. It is contended that Flanagan misinterprets the results of Libet’s consciousness experiments and that his functionalist concept of consciousness fails to yield an adequate explanation of the alleged causal efficacy of consciousness. Finally, his thesis is countered with other experimental results that appear to favor an epiphenomenalist view over the causal efficacy account of consciousness. (shrink)

In this paper I argue that many scholars involved in the contemporary free will debates have underappreciated the philosophical appeal of agent causation because the resources of contemporary emergentism have not been adequately introduced into the discussion. Whereas I agree that agent causation’s main problem has to do with its intelligibility, particularly with respect to the issue of how substances can be causally relevant, I argue that the notion of substance causation can be clearly articulated from an (...) emergentist framework. According to my proposal, a free agent is _a causally powerful substance that emerges in an anomic way from her constitutive mental events_, downwardly constraining, selecting and, in this way, having control on them. As we shall see, this particular concept of agent causation not only makes sense of the deep insight behind agent libertarianism, but it also provides us with the resources to solve some of the main objections that have been raised against it. It is true that here I cannot develop a complete defense of the evidential credentials of emergentism. Still, even if the considerations that follow do not serve to convince detractors of agent causal libertarian accounts of free will, they do suggest that libertarian agent causation is more promising than is typically acknowledged. (shrink)

The so-called problem of mental causation as discussed in the recent literature raises three central challenges for an adequate solution from a physicalist perspective: the threat of epiphenomenalism, the problem of externalism (or the difficulty in accounting for the causal efficacy of extrinsic mental properties) and the problem of causal exclusion (or the threat of over determination). We wish to account for mental causationas a real phenomenon within a physicalistic framework without accepting epiphenomenalism or overdetermination. The key ideas (...) of our proposal are an internal realism of causation combined with a relative notion of individuating events. We are arguing?contra Davidson?tha there is no absolute notion of events (neither as types nor as tokens) but rather one which is relative to explanatory interests and our intuitions concerning a relevant spatial and temporal overlap. Furthermore, we are presupposing a metaphysics of internal realism: We can only characterize entities by means of concepts produced within our epistemological framework. Physical concepts and mental concepts crossclassify the world as it is. Relying on this framework we try to explain how mental causation can be adequately described: Although mental concepts are not reducible to physical concepts and mental event-tokens may be different from "underlying" physical event-tokens, mental events are real phenomena that are realized by physical phenomena in special context conditions. (shrink)

Metaphysicians of causation have long debated the existence of primitive causal modalities (e.g., powers), with reductionists and realists taking opposing stances. However, little attention has been given to the legitimacy of the metaphysical question itself, despite our longstanding awareness of Rudolf Carnap’s critique of metaphysics. This article develops a (broadly) Carnapian-pragmatist approach to causation as an alternative to existing metaphysical approaches. Within this pragmatist approach, metaphysical questions about causation are reinterpreted as practical questions about the choice of (...) causal frameworks. To motivate and justify this new approach, I argue that, in emphasizing the priority of ontology over methodology, metaphysical approaches to causation fail to adequately capture the interplay between causal ontology and causal methodology in scientific practice. In contrast, the Carnapian approach provides a more appealing alternative that emphasizes the mutual dependence and “balance” between the two in an ongoing process of scientific inquiry. I use the recent controversy over “What counts as a cause” in statistical causal inference as a case study to demonstrate how the Carnapian approach can help us better understand the role of ontological issues in methodological practices. (shrink)

Among the questions that any adequate account of the nature of causation must answer, the following are especially crucial. First, how are causal states affairs – including both causal laws and causal relations between events, – related to noncausal states and affairs? Second, which are more basic – causal laws or causal relations? Third, how should the direction of causation be defined? Since David Hume’s time, reductionist answers have held sway, and philosophers have generally maintained that all (...) causal relations between events are logically supervenient upon causal laws and noncausal states affairs, that causal laws are supervenient upon noncausal facts, and that the direction of causation is definable in noncausal terms. Since the 1970s, however, anti-reductionists have challenged all of these reductionist claims. Among the reductionist views that are examined are, first, reductionist views that attempt to analyze causation in terms of nomological relations; second, counterfactual approaches to causation; and third, probabilistic approaches. Realist approaches to causation are then described, and the issue of singularist approaches to causation and causal laws, and of reductionism and the direction of causation are then considered. (shrink)

It seems to be generally accepted that (a) counterfactual conditionals are to be analysed in terms of possible worlds and inter-world relations of similarity and (b) causation is conceptually prior to counterfactuals. I argue here that both (a) and (b) are false. The argument against (a) is not a general metaphysical or epistemological one but simply that, structurally speaking, possible worlds theories are wrong: this is revealed when we try to extend them to cover the case of probabilistic counterfactuals. (...) Indeed a type of counterfactual probability exists which cannot be expressed in possible worlds terms at all. The argument against (b) emerges when we look at the form of an adequate account of both probabilistic and non-probabilistic counterfactuals. I do this by sketching and defending an approach to counterfactuals that, first, invoke a generalized notion of cause as primitive and, secondly, is algorithmic in form: counterfactuals are evaluated algorithmically in terms of other counterfactuals, without vicious circularity. Structures like possible worlds do not play a role either in general truth-conditions or in evaluation. They are simply the wrong sorts of structures. (shrink)

Mental causation is the causation of physical effects by mental causes. The paradigm case of mental causation is the causation of someone’s bodily movement by a mental state or event of hers. The belief that mental causation exists is deeply rooted in common sense. It seems uncontroversial to say, for instance, that a sudden pain caused Jones to wince, or that Smith’s thirst caused him to have a drink. Nevertheless, explaining how the mind can have (...) physical effects has proven a challenge for philosophers of mind. For physical effects already have physical causes, which threatens the claim that they also have mental causes. The problem is most pressing for positions according to which the mind is not itself physical. However, recent decades have also seen a debate over whether the view that the mind is physical can adequately explain mental causation. (shrink)

What exactly do social scientists and biologists say when they make causal claims? This question is one of the central puzzles in philosophy of science. Alexander Reutlinger sets out to answer this question. He aims to provide a theory of causation in the special sciences (that is, a theory causation in the social sciences, the biological sciences and other higher-level sciences). According one recent prominent view, causation is that causation is intimately tied to manipulability and the (...) possibility of intervene. Reutlinger's main negative target is to argue interventionist account of causation is not adequate. Where do interventionist accounts go wrong? Reutlinger argues that the central concept of the interventionist theories – that is, the very concept of an intervention – is tremendously problematic. Reutlinger's main positive claim consists in replacing the interventionist approach by an alternative explication of causation in the special sciences, the comparative variability theory of causation. This alternative preserves many insights of the interventionist account without a commitment to the claim that causation and interventions are intimately tied together. (shrink)

ABSTRACTOaksford and Chater critiqued the logic programming approach to nonmonotonicity and proposed that a Bayesian probabilistic approach to conditional reasoning provided a more empirically adequate theory. The current paper is a reply to Stenning and van Lambalgen's rejoinder to this earlier paper entitled ‘Logic programming, probability, and two-system accounts of reasoning: a rejoinder to Oaksford and Chater’ in Thinking and Reasoning. It is argued that causation is basic in human cognition and that explaining how abnormality lists are created (...) in LP requires causal models. Each specific rejoinder to the original critique is then addressed. While many areas of agreement are identified, with respect to the key differences, it is concluded the current evidence favours the Bayesian approach, at least for the moment. (shrink)

It is commonplace to distinguish between propositional justification (having good reasons for believing p) and doxastic justification (believing p on the basis of those good reasons).One necessary requirement for bridging the gap between S’s merely having propositional justification that p and S’s having doxastic justification that p is that S base her belief that p on her reasons (propositional justification).A plausible suggestion for what it takes for S’s belief to be based on her reasons is that her reasons must contribute (...) causally to S’s having that belief. Though this suggestion is plausible, causal accounts of the basing relation that have been proposed have not fared well. In particular, cases involving causal deviancy and cases involving over-determination have posed serious problems for causal accounts of the basing relation. Although previous causal accounts of the basing relation seem to fall before these problems, it is possible to construct an acceptable causal account of the basing relation. That is, it is possible to construct a causal account of the basing relation that not only fits our intuitions about doxastic justification in general, but also is not susceptible to the problems posed by causal deviancy and causal over-determination. The interventionist account of causation provides the tools for constructing such an account. My aim is to make use of the insights of the interventionist account of causation to develop and defend an adequate causal account of the basing relation. (shrink)

Fundamental physics makes no clear use of causal notions; it uses laws that operate in relevant respects in both temporal directions and that relate whole systems across times. But by relating causation to evidence, we can explain how causation fits in to a physical picture of the world and explain its temporal asymmetry. This paper takes up a deliberative approach to causation, according to which causal relations correspond to the evidential relations we need when we decide on (...) one thing in order to achieve another. Tamsin's taking her umbrella is a cause of her staying dry, for example, if and only if her deciding to take her umbrella for the sake of staying dry is adequate grounds for believing she'll stay dry. This correspondence explains why causation matters: knowledge of causal structure helps us make decisions that are evidence of outcomes we seek. The account also explains why we can control the future and not the past, and why causes come before their effects. When agents properly deliberate, their decisions can never count as evidence for any outcomes they may seek in the past. From this it follows that causal relations don't run backwards. This deliberative asymmetry is itself traced back to asymmetries of evidence and entropy, providing a new way of deriving causal asymmetry from temporally symmetric laws. (shrink)

Available from UMI in association with The British Library. Requires signed TDF. ;I deal with two themes: the idea that an account of thought should be given by giving an account of the ascription of thoughts by a radical interpreter--which I call interpretationism; and the idea that psychological concepts like action and perception are essentially causal. It has often been thought that these two themes conflict; or at least, that if they can co-exist, then they must be kept separate, and (...) associated with different perspectives on behaviour. But is that right? I discuss the character of interpretationism and the nature of causal theories, and examine the extent to which the two themes are compatible. ;Chapter I explains and argues for an interpretationist approach, distinguishing between different interpretationist views, and defending interpretationism against various common objections. ;Chapter II examines the links between interpretationism and the anomalousness of the mental. I discuss a number of different arguments and distinguish a range of theses about anomalousness and indeterminacy which they may support. ;Chapter III discusses the motivation for causal theories. I explain why it matters that we should give causal theories within an interpretationist perspective rather than treating the two themes as insights of different perspectives; and I examine the difficulties of doing so. ;Chapter IV considers the causal theory of vision. I argue against the view that the interpretationist's conception of experience undermines the causal theory. ;Chapter V considers the case of action. I develop a substantive causal theory of action which responds to the following argument: that if we accept the interpretationist's claim that the mental is anomalous, then we must concede that mental properties cannot be causally relevant. ;From the particular cases, and on general grounds, I conclude that it is possible to adopt an interpretationist perspective which simultaneously sees the mental in causal terms; and that such a perspective is essential for an adequate understanding of the mind. (shrink)

Several authors have recently claimed that the notorious causal exclusion problem, according to which higher-level causes are threatened with causal pre-emption by lower-level causes, can be avoided if causal relevance is understood in terms of Woodward's interventionist account of causation. They argue that if causal relevance is defined in interventionist terms, there are cases where only higher-level properties, but not the lower-level properties underlying them, qualify as causes of a certain effect. In this article, I show that the line (...) of reasoning supposed to establish this claim does not succeed and that interventionism is not better capable of dealing with higher-level causal claims than other accounts of causation. According to Woodward, higher-level causal claims are nonetheless more adequate than lower-level ones if they describe a realization-independent dependency relationship and, hence, meet the requirement that causes should be proportional to their effects. I argue, however, that combining interventionism with proportionality considerations raises difficulties and that, therefore, Woodward's account does not vindicate higher-level causation. (shrink)

Yablo suggests that we can understand the possibility of mental causation by supposing that mental properties determine physical properties, in the classic sense of determination according to which red determines scarlet. Determinates and their determinables do not compete for causal relevance, so if mental and physical properties are related as determinable and determinates, they should not compete for causal relevance either. I argue that this solution won''t work. I first construct a more adequate account of determination than that (...) provided by Yablo. I then consider two common accounts of the mental, token identity theories and dispositional theories, and argue that on neither do mental and physical properties satisfy the requirements for determination. (shrink)

I offer a brief review of, and critical response to, Neil Levy’s fascinating recent book Hard Luck, where he argues that no one is ever free or morally responsible not because of determinism or indeterminism, but because of luck. Two of Levy’s central arguments in defending his free will nihilism concern the nature and role of explanation in a theory of moral responsibility and the nature of akrasia. With respect to explanation, Levy argues that an adequate theory of moral (...) responsibility must be able provide contrastive explanations of why an agent performs one action rather than another, and that libertarians lack the resources to provide such explanations. With respect to akrasia, Levy argues that it is impossible to be directly morally responsible for akratic actions. In response I argue that any sense of contrastive explanation that can reasonably be thought to be a requirement on an adequate theory of moral responsibility is a sense that agent-causal libertarians can secure. I then argue that the agent-causal theory of free will offers an alternative and attractive understanding of motivation and self-control that makes it plausible to think that we can be morally responsible for akratic actions. (shrink)

This paper defends and develops an argument against epiphenomenalism, broadly construed. I argue first for a definition of epiphenomenalism which includes ‘non-reductive’ materialism as well as classical dualistic epiphenomenalism. I then present an argument that if epiphenomenalism were true it would be impossible to know about or even refer to our conscious states -- and therefore impossible even to formulate epiphenomenalism. David Chalmers has defended epiphenomenalism against such arguments; I consider this defence and attempt to show that it fails. I (...) conclude that an adequate account of mental causation requires us to abandon the principle of the causal closure of the physical, and attempt to rebut charges that it would be ‘unscientific’ to do so. (shrink)

Several philosophers have embraced the view that high-level events—events like Zimbabwe's monetary policy and its hyper-inflation—are causally related if their corresponding low-level, fundamental physical events are causally related. I dub the view which denies this without denying that high-level events are ever causally related causal emergentism. Several extant philosophical theories of causality entail causal emergentism, while others are inconsistent with the thesis. I illustrate this with David Lewis's two theories of causation, one of which entails causal emergentism, the other (...) of which entails its negation. I then argue for causal emergentism on the grounds that it provides the only adequate means of squaring the apparent plenitude of causal relations between low-level events with the apparent scarcity of causal relations between high-level events. This tension between the apparent abundance of low-level causation and the apparent scarcity of high-level causation has been noted before. However, it has been thought that various theses about the semantics or the pragmatics of causal claims could be used to ameliorate the tension without going in for causal emergentism. I argue that none of the suggested semantic or pragmatic strategies meet with success, and recommend emergentist theories of causality in their stead. As Lewis's 1973 account illustrates, causal emergentism is consistent with the thesis that all facts reduce to microphysical facts. (shrink)

A biblical approach to reconciling God’s sovereignty with creaturely responsibility should avoid the extremes of global occasionalism and completely autonomous creatures. This paper evaluates the standard intermediary solutions offered by conservationists and concurrentists. It argues that while each contributes insights which a satisfactory account should retain, none is fully adequate. Even Leibniz’s sophisticated response, which accounts for providence, miracles, and moral responsibility, unacceptably abridges creaturely power to implement decisions. My alternative proposal seeks to explain how creatures can retain full (...) responsibility for their actions even though God reserves the right to override creaturely power with His greater power. (shrink)

The aim of this paper is to examine the notion of social mechanisms by comparison with the notions of evolutionary and physical mechanisms. It is argued that social mechanisms are based on trends, and not lawlike regularities, so that social mechanisms are different from mechanisms in the natural sciences. Taking as an example of social causation the abolition of the slave trade, the paper argues that social mechanisms should be incorporated in Weber’s wider notion of adequate causation (...) in order to achieve their explanatory purpose. (shrink)

SummaryBecause it is not generally appreciated that Hume's analysis of the causal tie as radically contingent or ‘irrational’ is bound up with his specialised theory of cognition, its historical position is widely misconceived. Even a rationalist like Spinoza would agree that if, as Hume maintains, the causal tie holds between items each of which is‘ adequately’ grasped independently of the other, i.e. between what Spinoza calls ‘substances’, then the tie is indeed irrational. Also, Kant does not attempt to show that (...) the causal tie, as Hume sees it, is not irrational. His anti‐Humean result is based on the prior denial Humean causal fundaments are available in experiential cognition as we know it.RésuméLa position historique de Hume est généralement mal comprise parce qu'on ne voit pas que son analyse de la relation causale comme étant radicalement contingente ou »irrationnelle« est liée à sa théorie particulière de la connaissance. Même un rationaliste tel que Spinoza accorderait que la causalité est en effet irrationnelle si elle relie, ainsi que le prétend Hume, deux choses saisies indépendamment, c'est‐à‐dire deux substances selon Spinoza. Même Kant ne cherche pas à démontrer que le lien causal tel que Hume le voit n'est pas irrationnel. Son résultat anti‐humien est basé sur la négation préalable que le fondement de la causalité puisse être tiré de la connaissance expérimentale.ZusammenfassungWeil man im allgemeinen nicht berilcksichtigt, dass Humes Analyse der kausalen Verknüpfung als einer radikal kontingenten oder »irrationalen« Relation mit seiner besonderen erkennt‐nistheoretischen Position verbunden ist, wird deren historische Bedeutung oft missverstanden. Sogar ein Rationalist wie Spinoza würde zugestehen, dass die Verknüpfung irrational sein muss, wenn sie zwischen Substanten irn Sinne Spinozas, d.h. zwischen Dingen bestehen soll, die unabhängig voneinander adäquat erfasst werden, wie Hume das behauptet. Auch Kant versucht nicht zu zeigen, dass eine Kausalverknüpfung, so wie Hume sie auffasst, nicht irrational sei. Denn er bestreitet ja gerade, dass kausale Grundlagen auf Grund gewöhnlicher Erfahrung überhaupt gefunden werden können. (shrink)

Theistic religious believers should be concerned that the God they worship is not an idol. Conceptions of God thus need to be judged according to criteria of religious adequacy that are implicit in the ‘God-role’—that is, the way the concept of God properly functions in the conceptual economy and form of life of theistic believers. I argue that the conception of God as ‘omniGod’—an immaterial personal creator with the omni-properties—may reasonably be judged inadequate, at any rate from the perspective of (...) a relationship ethics based on the Christian revelation that God is Love. I go on to suggest that a conception of God as the power of love within the natural universe might prove more adequate, with God’s role as creator understood in terms of final rather than efficient causation. (shrink)

Several authors have argued that act–utilitarianism cannot provide an adequate critique of buying meat because a single meat purchase will not actually cause more farm animals to be raised or slaughtered. Thus, regardless of whether or not the production of meat is inhumane to animals, someone who buys meat is doing nothing wrong. This argument fails to show that meat purchases are morally permissible, however, because it assumes that act–utilitarians would use actual utility in their decision to buy or (...) not to buy meat. I show that act–utilitarians cannot use actual utility as a decision procedure and must instead use expected utility to prescribe or proscribe actions. I then demonstrate how expected utility can be applied to cases of contributory causation, where many people seem morally responsible for causing something to happen. Buying meat is one case of contributory causation where the probability of any single individual's affecting meat production is slight, but the expected disutility of affecting that production is substantial. Thus, in its expected utility form, act–utilitarianism defeats the ‘causal inefficacy’ defence of buying meat [1]. (shrink)

Christopher Franklin argues that the hard luck view, which I have recently defended, is misnamed: the arguments turn on absence of control and not on luck. He also argues that my objections to agent-causal libertarianism depend on a demand, for a contrastive explanation that guarantees the choice the agent makes, which would be question-begging in the dialectical context. In response to the first objection, I argue that though Franklin may be right that it is absence of control that matters to (...) freedom and not luck, we often are unable to discern whether an agent lacks sufficient control without asking whether the event is lucky for the agent; hence we cannot dispense with worries about luck in favor of concern about control. In response to the second worry, I argue that though Franklin is right in claiming that there is a perfectly good explanation regarding how the agent-causal power is exercised, it is not an adequate explanation given the context, because the same kind of explanation is available on event-causal theories. Agent-causal libertarians aim to avoid the luck objection which they acknowledge is powerful against event-causal libertarians; if it is true that they can offer nothing more than the latter kinds of theories in the way of control over actions, they fail in their aim. (shrink)

This text focuses on the theory of popular politics constructed within the context of analytical Marxism, and asks if rational choice theory provides an adequate basis for explaining patterns of social, political and economic behaviour in traditional China.

In this paper I wish to argue that counterfactual analyses of causation are inadequate. I believe the counterfactuals that are involved in counterfactual analyses of causation are often false, and thus the theories do not provide an adequate account of causation. This is demonstrated by the presentation of a counterexample to the counterfactual analyses of causation. I then present a unified theory of causation that is based upon probability and counterfactuals. This theory accounts for (...) both deterministic and indeterministic causation, and is not subject to many of the traditional problems facing theories of causation. (shrink)

Nonreductive physicalism provides an appealing solution to the nature of mental properties. But its success as a theory of mental properties has been called into doubt by claims that it cannot adequately handle the problems of mental causation, as it leads either to epiphenomenalism or to thoroughgoing overdetermination. I argue that these apparent problems for the nonreductivist are based in fundamental confusion about causation and explanation. I distinguish two different types of explanation and two different relations to which (...) they appeal: causation and determination. I argue that these types of explanation do not compete with one another, nor do these relations jointly result in overdetermination. In closing I develop a nonreductivist solution to mental causation which avoids both the hazards of epiphenomenalism and of overdetermination and so demonstrates a way to save nonreductive physicalism from the problems of mental causation. (shrink)

Philosophers of science today by and large reject the cataclysmic and irrational interpretation of the scientific enterprise claimed by Kuhn. Many computational models have been implemented to rationally study the conceptual change in science. In this recent tradition a key role is played by the concept of abduction as a mechanism by which new explanatory hypotheses are introduced. Nevertheless some problems in describing the most interesting abductive issues rise from the classical computational approach. It describes a cognitive process (and so (...) abduction) by the manipulation of internal symbolic representations of external world. This view assumes a discrete set of representations fixed in discrete time jumps, and cannot adequately account for the issue of anticipation and causation of a new hypothesis. An integration of the traditional computational view with some ideas developed inside the so-called dynamical approach can suggest some important insights. The concept of attractor is very significant. It permits a description of the abductive generation of new hypotheses in terms of a catastrophic rearrangement of the parameters responsible for the behavior of the system. (shrink)

Nonreductive physicalism provides an appealing solution to the nature of mental properties. But its success as a theory of mental properties has been called into doubt by claims that it cannot adequately handle the problems of mental causation, as it leads either to epiphenomenalism or to thoroughgoing overdetermination. I argue that these apparent problems for the nonreductivist are based in fundamental confusion about causation and explanation. I distinguish two different types of explanation and two different relations to which (...) they appeal: causation and determination. I argue that these types of explanation do not compete with one another, nor do these relations jointly result in overdetermination. In closing I develop a nonreductivist solution to mental causation which avoids both the hazards of epiphenomenalism and of overdetermination and so demonstrates a way to save nonreductive physicalism from the problems of mental causation. (shrink)

During the past decades several philosophers of science and social scientists have been interested in the problems of causation. Recently attention has been given to probabilistic causation in dichotomous causal systems. The paper uses the basic features of probabilistic causation to argue that the causal modeling approaches developed by such researchers as Blalock (1964) and Duncan (1975) can provide, when an additional assumption is added, adequate qualitative measures of one variableś causal influence upon another. Finally, some (...) of the difficulties and issues involved in developing adequate quantitative measures are discussed, and it is concluded that the causal modeling measures cannot provide adequate quantitative measures. (shrink)

During the past decades several philosophers of science and social scientists have been interested in the problems of causation. Recently attention has been given to probabilistic causation in dichotomous causal systems. The paper uses the basic features of probabilistic causation to argue that the causal modeling approaches developed by such researchers as Blalock and Duncan can provide, when an additional assumption is added, adequate qualitative measures of one variableś causal influence upon another. Finally, some of the (...) difficulties and issues involved in developing adequate quantitative measures are discussed, and it is concluded that the causal modeling measures cannot provide adequate quantitative measures. (shrink)

This volume brings together a number of previously unpublished essays that will advance the reader's philosophical understanding of specific aspects of causation, agency and moral responsibility. These are deeply intertwined notions, and a large proportion of the volume is taken up by papers that shed light on their mutual connections or defend certain claims concerning them. The volume investigates several important questions, including: Can causation be perceived? If it can, can it be perceived in any way other than (...) visually? Can the interventionist theory developed by James Woodward offer an adequate account of causation? Is a causal relation a necessary condition for moral responsibility? Can there be a responsibility difference without a causal difference? Are causal ascriptions based on the more primitive language game of blame ascriptions? What can be learnt from the analogy between causal interference and unbreakable processes, on one side, and motivational interferences and "unbreakable" resolutions, on the other side? How can humans be routinely considered responsible for non-deliberated omissions? Should the connection between moral responsibility and the epistemic conditions usually required for moral responsibility to be obtained be weakened? What is the connection between awareness of one's doing and intentional action? These essays constitute a valid contribution towards the discovery of reasonable answers to such deep questions as the metaphysics of causal relations, the epistemology of causal explanations, the interventionist theory of causation, and the relationship among causality and moral responsibility, willpower, agency and intentional action. (shrink)

The conceptual relation between objective becoming and the direction of time is explored by discussing an ontologically asymmetric notion of causation. It is claimed that such a notion, in terms of which Stein defined objective becoming in Minkowski spacetime, has either a purely metaphysical status or is reducible to physical concepts. In the former case, it is adequate for Stein's purpose but irrelevant to physical theories. In the latter, the causal asymmetry can be related to irreversible physical processes (...) only in an extrinsic way. This dilemma creates additional difficulties both to a unified theory of the direction of time and to the project of making room for becoming in the physical world. (shrink)

In the first ever book-length treatment of David Hume’s philosophy of action, Constantine Sandis brings together seemingly disparate aspects of Hume’s work to present an understanding of human action that is much richer than previously assumed. Sandis showcases Hume’s interconnected views on action and its causes by situating them within a wider vision of our human understanding of personal identity, causation, freedom, historical explanation, and morality. In so doing, he also relates key aspects of the emerging picture to contemporary (...) concerns within the philosophy of action and moral psychology, including debates between Humeans and anti-Humeans about both 'motivating' and 'normative' reasons. Character and Causation takes the form of a series of essays which collectively argue that Hume’s overall project proceeds by way of a soft conceptual revisionism that emerges from his Copy Principle. This involves re-calibrating our _philosophical_ ideas of all that agency involves to fit scheme that more readily matches the range of impressions that human beings actually have. On such a reading, once we rid ourselves of a _certain_ kind of metaphysical ambition we are left with perfectly adequate account of how it is that people can act in character, freely, and for good reasons. The resulting picture is one that both unifies Hume's practical and theoretical philosophy and radically transforms contemporary philosophy of action for the better. (shrink)

Hume’s criticisms of divine causation are insufficient because he does not respond to important philosophical positions that are defended by those whom he closely read. Hume’s arguments might work against the background of a Cartesian definition of body, or a Malebranchian conception of causation, or some defenses of occasionalism. At least, I will not here argue that they succeed or fail against those targets. Instead, I will lay out two major deficiencies in his arguments against divine causation. (...) I call these “deficiencies” because Hume does not adequately address live positions. This does not mean, of course, that there are not problems with these views or that Hume could not have given strong arguments against them. Rather, Hume’s arguments, which can seem comprehensive to the twenty-first century reader, are in fact not so. For the deficiencies discussed in this essay, I point to writers from Hume’s near context (many of whom we know he read carefully) who held the views not discussed, and I provide reasons why Hume seems not to have entertained these possibilities. I won’t distinguish between accidentally overlooking and actively ignoring. By drawing attention to these three deficiencies, I have two goals. The first is to demonstrate the diversity of seventeenth and early eighteenth century views on divine causation, especially among philosophers in England, Scotland, and Ireland, who are often ignored by philosophers today who, like Hume, focus on continental Cartesians while ignoring British dualists and vitalists. The second goal is to make today’s readers aware of shortcomings in Hume’s arguments to encourage productively contextual readings of Hume and his contemporaries. (shrink)

In this paper, we advocate the idea that an adequate explanation of biological systems requires appealing to organizational closure as an emergent causal regime. We first develop a theoretical justification of emergence in terms of relatedness, by arguing that configurations, because of the relatedness among their constituents, possess ontologically irreducible properties, providing them with distinctive causal powers. We then focus on those emergent causal powers exerted as constraints, and we claim that biological systems crucially differ from other natural systems (...) in that they realize a closure of constraints, i.e. a second-order emergent regime of causation such that the constituents, each of them acting as a constraint, realize a mutual dependence among them, and are collectively able to self-maintain. Lastly, we claim that closure can be justifiably taken as an emergent regime of causation, without admitting that it inherently involves whole-parts causation, which would require to commit to stronger ontological and epistemological assumptions. (shrink)

I address an apparent conflict between Spinoza’s concepts of immanent causation and acting/doing [agere]. Spinoza apparently holds that an immanent cause undergoes [patitur] whatever it does. Yet according to his stated definition of acting and undergoing in the Ethics, this is impossible; to act is to be an adequate cause, while to undergo is to be merely a partial cause. Spinoza also seems committed to God’s being the adequate cause of all things, and, in a well-known passage, (...) appears to deny categorically that God is capable of undergoing. How then can God also be the immanent cause of all things, as Spinoza claims? On the basis of a close reading of the passage in question, I argue that Spinoza actually distinguishes between two senses of undergoing. An immanent cause undergoes not by being a partial cause but rather by being the metaphysical subject of its effect. While this sense of undergoing has its roots in scholasticism, Spinoza’s willingness to attribute such a capacity to undergo to God is idiosyncratic and reveals important ways in which his understanding of essence, perfection, and causation differs from the scholastic model. (shrink)

It has recently been objected that structural realism, in its various guises, is unable to adequately account for causal phenomena (see, for example, Psillos 2006). In this talk, I consider whether structural realism has the resources to address this objection.

Hume is famous for having introduced a radical theory of the nature of causation. To say that A causes B is just to say that A is constantly conjoined with B and that experience of the conjunction determines the mind to expect the one on the appearance of the other. It was this theory that awoke Kant from his dogmatic slumbers and established Hume as a founding figure of the various forms of positivism that emerged from the nineteenth century. (...) A. J. Ayer, for instance, records how the Vienna Circle in its manifesto of 1929 officially included Hume as a precursor of the movement: “Those who stand closest to the Vienna Circle in their general outlook are Hume and Mach. It is remarkable how much of the doctrine that is now thought to be especially characteristic of logical positivism was already stated, or at least foreshadowed by Hume.” But this interpretation is mistaken. Hume’s conception of causation is not positivistic but skeptical. Hume did present the above constant conjunction account of causation, but what is generally ignored is that he was dissatisfied and skeptical about it. In what follows I wish to explore the rationale of Hume’s skepticism about causation. I shall show that Hume understood the constant conjunction account to operate against the conceptual background of a theistic vision of the world. Hume does not think that any constant conjunction causal explanation is adequate because we do not and cannot understand the mind of God. (shrink)

Classical computational modellers of mind urge that the mind is something like a von Neumann computer operating over a system of symbols constituting a language of thought. Such an architecture, they argue, presents us with the best explanation of the compositionality, systematicity and productivity of thought. The language of thought hypothesis is supported by additional independent arguments made popular by Jerry Fodor. Paul Smolensky has developed a connectionist architecture he claims adequately explains compositionality, systematicity and productivity without positing any language (...) of thought, and without positing any operations over a set of symbols. This architecture encodes the information represented in linguistic trees without explicitly representing those trees or their constituents, and indeed without employing any representational vehicles with constituent structure. In a recent article, Fodor (1997; Connectionism and systematicity, Cognition , 62, 109-119) argues that Smolensky's proposal does not work. I defend Smolensky against Fodor's attack, and use this interchange as a vehicle for exploring and criticising the “Language of Thought” hypothesis more generally and the arguments Fodor adduces on its behalf. (shrink)

In this article, I defend my previous argument that natural divine causation suffers under the problem of causal overdetermination and that it cannot serve as a line of demarcation between theistic evolution (TE) and intelligent design (ID). I do this in light of Christoffer Skogholt's critique of my article. I argue that Skogholt underestimates the naturalistic ambitions of some current thinkers in TE and fails, therefore, to adequately respond to my main argument. I also outline how partial causation (...) better serves as a model for the relationship between God's providence and evolution. (shrink)

For many areas of philosophy, we lack an understanding of their developments between the fourteenth and seventeenth centuries. One such area is the development of the notion of final causation. The rejection of final causation is often described as one of the distinguishing hallmarks of so called Early Modern philosophy in relation to the Scholastic philosophical tradition. Our lack of understanding of the development of this notion in philosophy therefore impedes our ability to write an adequate history (...) of philosophy spanning these centuries. In this article, the notion of final causation as treated in the works of Chrysostom Javellus (1472–1538) and Francis Silvestri (of Ferrara) (1474–1526) is presented. It is argued that the treatment of final causation in these thinkers is already shaped by concerns regarding finality that we find in Early Modern philosophy. (shrink)