In this paper, we argue that, omissions are not events or actions, but rather fact-like entities, and that, insofar as only events and actions can be causes, omissions cannot be causes. Nevertheless, since omissions can, and often do, play a role in the explanations of events, their place in such explanations must be found; and an attempt to find such a place is made.

This item has been retired at the request of its author.

Folk theories—untutored people’s (often implicit) theories about various features of the world—have been fashionable objects of inquiry in psychology for almost two decades now (e.g., Hirschfeld and Gelman 1994), and more recently they have been of interest in experimental philosophy (Nichols 2004). Folk theories of psy- chology, physics, biology, and ethics have all come under investigation. Folk meta- physics, however, has not been as extensively studied. That so little is known about folk metaphysics is unfortunate for (at least) two reasons. (...) First, folk metaphysics is almost certainly implicit, and it is likely to be our default way of thinking about metaphysical problems. Moreover, one’s metaphysical commitments can have pro- found consequences—in scientific, religious, and ethical contexts, for example. Thus, folk metaphysics ought to be dragged out into the open and exposed to criticism. As Peirce eloquently remarked (1994, 1.129; see also 1994, 7.579). (shrink)

Butterfield's (1992a,b,c) claim of the equivalence of absence of Lewisian probabilistic counterfactual causality (LC) to Hellman's stochastic Einstein locality (SEL) is questioned. Butterfield's assumption on which the proof of his claim is based would suffice to prove that SEL implies absence of LC also for appropriately given versions of these notions in algebraic quantum field theory, but the assumption is not an admissible one. The conclusion must be that the relation of SEL and absence of LC is open, and that (...) they may be independent. (shrink)

The physical and/or intrinsic connection approach to causation has become prominent in the recent literature, with Salmon, Dowe, Menzies, and Armstrong among its leading proponents. I show that there is a type of causation, causation by disconnection, with no physical or intrinsic connection between cause and effect. Only Hume-style conditions approaches and hybrid conditions-connections approaches appear to be able to handle causation by disconnection. Some Hume-style, extrinsic, absence-relating, necessary and/or sufficient condition component of the causal relation (...) proves to be needed. (shrink)

For the framework of event causation—i.e. the framework according to which causation is a relation between events—absences or omissions pose a problem. Absences, it is generally agreed, are not events; so, under the framework of event causation, they cannot be causally related. But, as a matter of fact, absences are often taken to be causes or effects. The problem of absence causation is thus how to make sense of causation that apparently involves (...) absences as causes or effects. In an influential paper, Helen Beebee offers a partial solution to the problem by giving an account of causation by absence. I argue that Beebee's account can be extended to cover causation of absence as well. More importantly, I argue that the extended Beebeeian account calls for a major modification to David Lewis's theory of causal explanation, usually taken as standard. Compared to the standard theory, the.. (shrink)

In this paper, I defend a counterfactual approach to causation by absences from some recent criticisms due to Sartorio.

Several theories of causation reject causation of or by absences. They thereby clash with much of what we think and say about what causes what. This paper examines a way in which one kind of theory, causal dispositionalism, can be modified so as to accept absence causation, while still retaining a fundamental commitment of dispositionalism. The proposal adopts parts of a strategy described by David Lewis. The paper concludes with a brief discussion of the problem of (...) the proliferation of causes. (shrink)

If _presentism_ is true, then no wholly non-present events exist. If _absence orthodoxy_ is true, then no absences exist. I discuss a well-known causal argument against presentism, and develop a very similar argument against absence orthodoxy. I argue that solutions to the argument against absence orthodoxy can be adopted by the presentist as solutions to the argument against presentism. The upshot is that if the argument against absence orthodoxy fails, then so does the argument against presentism.

Much contemporary debate on the nature of mechanisms centers on the issue of modulating negative causes. One type of negative causability, which I refer to as “causation by absence,” appears difficult to incorporate into modern accounts of mechanistic explanation. This paper argues that a recent attempt to resolve this problem, proposed by Benjamin Barros, requires improvement as it overlooks the fact that not all absences qualify as sources of mechanism failure. I suggest that there are a number of (...) additional types of effects caused by absences that need to be incorporated to account for the diversity of causal connections in the biological sciences. Furthermore, it is argued that recognizing natural variability in mechanisms, such as attenuation, leads to some interesting line-drawing issues for contemporary philosophy of mechanisms. (shrink)

Much contemporary debate on the nature of mechanisms centers on the issue of modulating negative causes. One type of negative causability, which I refer to as "causation by absence," appears difficult to incorporate into modern accounts of mechanistic explanation. This paper argues that a recent attempt to resolve this problem, proposed by Benjamin Barros, requires improvement as it overlooks the fact that not all absences qualify as sources of mechanism failure. I suggest that there are a number of (...) additional types of effects caused by absences that need to be incorporated to account for the diversity of causal connections in the biological sciences. Furthermore, it is argued that recognizing natural variability in mechanisms, such as attenuation, leads to some interesting line-drawing issues for contemporary philosophy of mechanisms. (shrink)

Causation is at once familiar and mysterious. Neither common sense nor extensive philosophical debate has led us to anything like agreement on the correct analysis of the concept of causation, or an account of the metaphysical nature of the causal relation. Causation: A User's Guide cuts a clear path through this confusing but vital landscape. L. A. Paul and Ned Hall guide the reader through the most important philosophical treatments of causation, negotiating the terrain by taking (...) a set of examples as landmarks. They clarify the central themes of the debate about causation, and cover questions about causation involving omissions or absences, preemption and other species of redundant causation, and the possibility that causation is not transitive. Along the way, Paul and Hall examine several contemporary proposals for analyzing the nature of causation and assess their merits and overall methodological cogency.The book is designed to be of value both to trained specialists and those coming to the problem of causation for the first time. It provides the reader with a broad and sophisticated view of the metaphysics of the causal relation. (shrink)

Hume thought that if you believed in powers, you believed in necessary connections in nature. He was then able to argue that there were none such because anything could follow anything else. But Hume wrong-footed his opponents. A power does not necessitate its manifestations: rather, it disposes towards them in a way that is less than necessary but more than purely contingent. -/- In this paper a dispositional theory of causation is offered. Causes dispose towards their effects and often (...) produce them. But a set of causes, even though they may succeed in producing an effect, cannot necessitate it since the effect could have been counteracted by some additional power. This would require a separation of our concepts of causal production and causal necessitation. The most conspicuous cases of causation are those where powers accumulate and pass a requisite threshold for an effect to occur. -/- We develop a model for representing powers as constituent vectors within an n-dimensional quality space, where composition of causes appears as vector addition. Even our resultant vector, however, has to be understood as having dispositional force only. This model throws new light on causal modality and cases of prevention, causation by absence and probabilistic causation. (shrink)

In “A Subjectivist’s Guide to Objective Chance,” David Lewis says that he is “led to wonder whether anyone but a subjectivist is in a position to understand objective chance.” The present essay aims to motivate this same Lewisean attitude, and a similar degree of modest subjectivism, with respect to objective causation. The essay begins with Newcomb problems, which turn on an apparent tension between two principles of choice: roughly, a principle sensitive to the causal features of the relevant situation, (...) and a principle sensitive only to evidential factors. Two-boxers give priority to causal beliefs, and one-boxers to evidential beliefs. The essay notes that a similar issue can arise when the modality in question is chance, rather than causation. In this case, the conflict is between decision rules based on credences guided solely by chances, and rules based on credences guided by other sorts of probabilistic evidence. Far from excluding cases of the latter kind, Lewis’s Principal Principle explicitly allows for them, in the form of the caveat that credences should follow beliefs about chances only in the absence of “inadmissible evidence.” The essay then exhibits a tension in Lewis’s views on these two matters, by presenting a class of decision problems—some of them themselves Newcomb problems—in which Lewis’s view of the relevance of inadmissible evidence seems in tension with his causal decision theory. It offers a diagnosis for this dilemma and proposes a remedy, based on an extension of a proposal due to Ned Hall and others from the case of chance to that of causation. The remedy suggests a new view of the relation between causal decision theory and evidential decision theory, namely, that they stand to each other much as chance stands to credence, being objective and subjective faces of the same practical coin. This has much the same metaphysical benefits as Lewis’s own view of chance and also throws interesting new light on Newcomb problems, providing an irenic resolution of the apparent disagreement between causal and evidential decision rules. (shrink)

Carolina Sartorio argues that only the actual causes of our behaviour matter to our freedom. The key, she claims, lies in a correct understanding of the role played by causation in a view of that kind. Causation has some important features that make it a responsibility-grounding relation, and this contributes to the success of the view. Also, when agents act freely, the actual causes are richer than they appear to be at first sight; in particular, they reflect the (...) agents' sensitivity to reasons, where this includes both the existence of actual reasons and the absence of other reasons. So acting freely requires more causes and quite complex causes, as opposed to fewer causes and simpler causes, and is compatible with those causes being deterministic. The book connects two different debates, the one on causation and the one on the problem of free will, in new and illuminating ways. (shrink)

The starting point in the development of probabilistic analyses of token causation has usually been the naïve intuition that, in some relevant sense, a cause raises the probability of its effect. But there are well-known examples both of non-probability-raising causation and of probability-raising non-causation. Sophisticated extant probabilistic analyses treat many such cases correctly, but only at the cost of excluding the possibilities of direct non-probability-raising causation, failures of causal transitivity, action-at-a-distance, prevention, and causation by absence (...) and omission. I show that an examination of the structure of these problem cases suggests a different treatment, one which avoids the costs of extant probabilistic analyses. (shrink)

The counterfactual analysis of causation has focused on one particular counterfactual conditional, taking as its starting-point the suggestion that C causes E iff (C E). In this paper, some consequences are explored of reversing this counterfactual, and developing an account starting with the idea that C causes E iff (E C). This suggestion is discussed in relation to the problem of pre-emption. It is found that the 'reversed' counterfactual analysis can handle even the most difficult cases of pre-emption with (...) only minimal complications. The paper closes with a discussion of the wider philosophical implications of developing a reversed counterfactual analysis, especially concerning the differentiation of causes from causal conditions, causation by absences, and the extent to which causes suffice for their effects. (shrink)

This is a brief philosophical introduction to, and an annotated translation of, the section on absence from Śālikanātha’s Pramāṇapārāyaṇa (Study of the Instruments of Knowledge), a foundational work of Prābhākara epistemology. In this section, which focuses on the epistemology of absence, Śālikanātha argues against the Bhāṭṭa view that there is a sui generis instrument of knowledge (pramāṇa) by which we learn of absence (abhāva). He does so by arguing for a subjective reductionist thesis about absence, according to which the absence (...) of a perceivable (dṛśya) object at a locus is identical with a positive state of awareness (buddhi) whose content includes the locus but not the perceivable object. If correct, Śālikanātha argues, we should therefore learn of absence in the same ways we acquire self-knowledge more generally. While developing his reductionism about absence, Śālikanātha responds to a range of concerns, including the objection that his view cannot explain causation by absence. (shrink)

In this dissertation, I develop and defend a model of causation that allows for dualist mental causation in worlds where the physical domain is physically complete. In Part I, I present the dualist ontology that will be assumed throughout the thesis and identify two challenges for models of mental causation within such an ontology: the exclusion worry and the common cause worry. I also argue that a proper response to these challenges requires a thoroughly lightweight account of (...) causation, i.e. an account that allows for causes to be metaphysically distinct from the phenomena that produce or physically necessitate their effects. In Part II, I critically evaluate contemporary responses to these challenges from the philosophical literature. In particular, I discuss List and Stoljar’s criticism of exclusion worries, Kroedel’s alternative dualist ontology, concerns about the notion of causal sufficiency, and Lowe’s models of dualist mental causation. I argue that none of these proposals provide independent motivation for a thoroughly lightweight account of causation and therefore leave room for improvement. In the first four chapters of Part III, I develop a thoroughly lightweight model of causation, which builds on interventionist approaches to causation. First, I explain how so-called ‘holding fixed’-requirements in standard interventionist accounts stand in the way of dualist mental causation. I then argue that interventionist accounts should impose a robustness condition on causal correlations and that, with this condition in place, the ‘holding fixed’-requirements can be weakened such that they do allow for dualist mental causation. I dub the interventionist model with such weakened ‘holding fixed’-requirements ‘insensitive interventionism’, argue that it can counter the exclusion worry as well as the common cause worry, and explain under which circumstances it would predict there to be dualist mental causation. Importantly, these circumstances might, for all we know, hold in the actual world. In the final three chapters of Part III, I defend insensitive interventionism against some objections. I consider the objection that causation must be productive, the objection that causes must physically necessitate their effects, and the objection that insensitive interventionism is too permissive. I respond by drawing from the literature on causation by absences and on the relation between causation and fundamental physics. Overall, insensitive interventionism performs as well as standard interventionist accounts. I conclude that insensitive interventionism is a credible model of causation. (shrink)

I focus on token, deterministic causal claims as they feature in causal explanations. Adequately handling absences is difficult for most causal theories, including theories of causal explanation. Yet so is adequately handling cases of late preemption. The best account of absence-causal claims as they appear in causal explanations is Jonathan Schaffer's quaternary, contrastive account. Yet Schaffer's account cannot handle preemption. The account that best handles late preemption is James Woodward's interventionist account. Yet Woodward's account is inadequate when it comes (...) to absences. I propose an account that handles both absences and preemption by transposing Schaffer's account into an interventionist framework. (shrink)

In this article, I begin by giving a brief history of melanoma causation. I then discuss the current manner in which malignant melanoma is classified. In general, these systems of classification do not take account of the manner of tumour causation. Instead, they are based on phenomenological features of the tumour, such as size, spread, and morphology. I go on to suggest that misclassification of melanoma is a major problem in clinical practice. I therefore outline an alternative means (...) of classifying these tumours based on causal factors. By analogy with similar systems that have recently emerged for other cancers, I suggest that this causal classification is likely to be both workable and helpful, even in the absence of a full causal-mechanistic understanding of the aetiology of the tumour. (shrink)

Analyses of singular causation often make use of the idea that a cause increases the probability of its effect. Of particular salience in such accounts are the values of the probability function of the effect, conditional on the presence and absence of the putative cause, analysed around the times of the events in question: causes are characterized by the effect’s probability function being greater when conditionalized upon them. Put this way, it becomes clearer that the ‘behaviour’ of probability functions (...) in small intervals about the times in question ought to be of concern. In this article, I make an extended case that causal theorists employing the ‘probability raising’ idea should pay attention to the continuity question. Specifically, if the probability functions are ‘jumping about’ in ways typical of discontinuous functions, then the stability of the relevant probability increase is called into question. The rub, however, is that sweeping requirements for either continuity or discontinuity are problematic and, as I argue, this constitutes a ‘continuity bind’. Hence more subtle considerations and constraints are needed, two of which I consider: utilizing discontinuous first derivatives of continuous probability functions, and abandoning point probability for imprecise probability. _1_ Introduction _2_ Probability Trajectories and Continuity _2.1_ Probability trajectories _2.2_ Causation as discontinuous jumps _2.3_ Against systematic discontinuity _3_ Broader Discontinuity Concerns _4_ The Continuity Bind _4.1_ Retaining continuity with discontinuous first derivatives _4.2_ Imprecise probability trajectories _5_ Concluding Remarks Appendix. (shrink)

In this article I review the core elements of Carolina Sartorio’s actual causal sequence account of free will and moral responsibility, and propose two revisions. First, I suggest replacing the contested notion of absence causation by the relatively uncontroversial notion of causal explanation by absences. Second, I propose retaining explanation by unreduced dispositions, of which Sartorio appears to be wary. I then set out a response to her critical treatment of manipulation arguments against compatibilism. Lastly, I point out (...) that Sartorio’s reasons-sensitivity condition on moral responsibility is amenable to a conception of moral responsibility that, unlike the one she endorses, dispenses with basic desert. (shrink)

Moral and political philosophers no longer condemn harm inflicted on nonhuman animals as self-evidently as they did when animal welfare and animal rights advocacy was at the forefront in the 1980s, and sentience, suffering, species-typical behavior, and personhood were the basic concepts of the discussion. The article shows this by comparing the determination with which societies seek responsibility for human harm to the relative indifference with which law and morality react to nonhuman harm. When harm is inflicted on humans, policies (...) concerning negligence and duty of care and principles such as the ‘but for’ rule and the doctrine of double effect are easily introduced. When harm is inflicted on nonhumans, this does not happen, at least not any more. As an explanation for the changed situation, the article offers a shift in discussion and its basic terminology. Simple ethical considerations supported the case for nonhuman animals, but many philosophers moved on to debate different views on political justice instead. This allowed the creation of many conflicting views that are justifiable on their own presuppositions. In the absence of a shared foundation, this fragments the discussion, focuses it on humans, and ignores or marginalizes nonhuman animals. (shrink)

But if there aren’t, then ‘they’ are not caused by anything and do not cause anything. That certainly appears to be false, however. John’s absence from our party might have been caused by his having fallen ill, and might cause a commotion. Dick’s not eating his soup might have been caused by his having fallen ill, and might cause a commotion.

But if there aren’t, then ‘they’ are not caused by anything and do not cause anything. That certainly appears to be false, however. John’s absence from our party might have been caused by his having fallen ill, and might cause a commotion. Dick’s not eating his soup might have been caused by his having fallen ill, and might cause a commotion.

More than a century ago, Russell launched a forceful attack on causation, arguing not only that modern physics has no need for causal notions but also that our belief in causation is a relic of a pre-scientific view of the world. He thereby initiated a debate about the relations between physics and causation that remains very much alive today. While virtually everybody nowadays rejects Russell's causal eliminativism, many philosophers have been convinced by Russell that the fundamental physical (...) structure of our world doesn't contain causal relations. This raises the question of how to reconcile the central role of causal concepts in the special sciences and in common sense with the putative absence of causation in fundamental physics. (shrink)

Our approach aims at accounting for causal claims in terms of how the physical states of the underlying dynamical system evolve with time. Causal claims assert connections between two sets of physicals states—their truth depends on whether the two sets in question are genuinely connected by time evolution such that physical states from one set evolve with time into the states of the other set. We demonstrate the virtues of our approach by showing how it is able to account for (...) typical causes, causally relevant factors, being ‘the’ cause, and cases of overdetermination and causation by absences. (shrink)

Salmon has argued that science provides explanations by describing a causal nexus: For Salmon, this nexus is a network of processes and interactions. I argue that this picture of the causal nexus is insufficient for an account of scientific explanation: a taxonomy of causal relevance is also needed. ;Probabilistic theories of causation seem to provide such a taxonomy in their dichotomy between promoting and inhibiting causes. However, standard probabilistic theories are beset by a difficulty called the problem of disjunctive (...) factors. According to such theories, an effect is more probable in the presence of a cause than in its absence . But there are many ways in which a particular cause may come about, and many in which it may be absent; these ways need not confer the same probabilities upon the effect. Thus, there is no direct comparison of two probabilities to be made. My solution is to abandon the oversimplified dichotomy between promoting and inhibiting causes. Causal claims convey qualitative information about more complex probabilistic relationships. Interestingly, this picture of causation meshes naturally with extant theories of explanation. ;I show how this framework may be employed in resolving several outstanding problems in the theories of causation and explanation, such as the relationship between singular and general causal claims, and the role of contrastive stress in causal and explanatory claims. ;Finally, I show how my account of causal explanation can be connected with the semantic conception of scientific theories. According to this conception, a theory is characterized in terms of a class of mathematical models. I describe the relationship between these mathematical models and the probabilistic relationships that are described by causal explanations. These ideas are illustrated in an extended discussion of causation and explanation in evolutionary theory. (shrink)

The absence of a necessary connection in singular causation is a key step in the Humean argument against any form of necessity in causation. I argue that Hume's defence of this step is unsuccessful, and that the step could be skipped, accepting the possibility of necessary a posteriori truths. Still this does not suffice to guarantee a necessary connection in singular causation. Necessary a posteriori truths should be backed by necessary a priori truths. Thus, a main object (...) of this paper is to argue that an a priori philosophical concept of causality involves a necessary connection between its terms. (shrink)

A number of philosophers have argued that causation is not an objective feature of the microphysical world but rather is a perspectival phenomenon that holds only between “coarse-grained” entities such as those that figure in the special sciences. This view seems to pose a problem for arguments for physicalism that rely on the alleged causal completeness of physics. In this paper, I address this problem by arguing that the completeness of physics has two components, only one of which is (...) causal. These two components of completeness can be used in an argument for physicalism that is supported by strong inductive evidence even in the absence of microphysical causation. (shrink)

We often explain by citing an absence or an omission. Apart from the problem of assigning a causal role to such apparently negative factors as absences and omissions, there is a puzzle as to why only some absences and omissions, out of indefinitely many, should figure in explanations. In this paper we solve this ’many absences problem’ by using the contrastive model of explanation. The contrastive model of explanation is developed by adapting Peter Lipton’s account. What initially (...) appears to be only a trivial amendment to Lipton’s Difference Condition enables us both to offer a much more satisfactory solution to the ’many absences problem’ than David Lewis did, and also to explain why explanation in terms of absences and omissions should be so common. (shrink)

Loewer (in: Physicalism and its discontents, 2001; Philos Phenomenol Res 65:655–663, 2002; in: Contemporary debates in philosophy of mind, 2007) has argued that the nonreductive physicalist should respond to the exclusion problem by endorsing the overdetermination entailed by their view. Kim’s (Physicalism, or something near enough, 2005; in: Contemporary debates in the philosophy of mind, 2007) argument against this reply is based on the premise that mental causation must be a productive relation in order to sustain human agency. In (...) this paper, I challenge the premise that mental causation is a productive relation by appealing to the underlying double prevention structure of the physiological mechanisms of human action. Since the causal pathways from an agent’s mental events to bodily movement involves an absence, mental causation is not productive. This places Kim in a troublesome dilemma in his debate with Loewer: either surrender mental causation or deny that causation is a productive relation. With the support offered for productive mental causation undermined, responses to the exclusion problem based on accepting overdetermination remain viable options for the nonreductive physicalist. (shrink)

_What Thomas Hobbes has to say of the nature of causation itself in_ _Entire Causes_ _and Their Only Possible Effects_ _is carried further in the first of the two excerpts here_ _-- although not at its start. His second subject in this imperfectly sequential piece of_ _writing is determinism itself -- a deterministic philosophy of mind. In the mind, as_ _elsewhere, each event has a 'necessary cause' -- a cause that necessitates the event._ _His third subject in the first (...) excerpt is freedom, this being voluntariness, and its_ _relation to the determinism. He gives a statement of what is now known as_ _Compatibilism -- roughly the doctrine that determinism and freedom properly_ _understood do not conflict with but are consistent with one another. We can be_ _entirely subject to determinism or 'necessity' and also be perfectly free. Certainly a_ _distinction between freedom as 'the absence of opposition', which can co-exist with_ _determinism, and some other kind of freedom, had been made before Hobbes. But it_ _will take a better historian than me to say if he was anticipated by someone else who_ _said that the particular freedom consistent with determinism is all that we can_ _properly mean by the term 'freedom'. Certainly he got in ahead of lovely_. (shrink)

_ Source: _Volume 10, Issue 3, pp 349 - 369 A theory of causation suitable for historiography must accommodate the many types of causal claims historians make. In this paper, I examine the advantages of applying D. K. Lewis’s counterfactual theory of causation to the philosophy of historiography. I contend that Lewis’s possible world semantics offers a superior framework for making sense of historical causation, and that it lays the foundation for historians to look at history as (...) causal series of events, remaining agnostic as to whether there may be historical regularities or laws. Lewis’s theory can also accommodate important notions often used by historians, such as absences as causes, historical necessity and contingency, and the role they play in the formulation of historical counterfactuals. (shrink)

Qiyās, or “correlational inference”, comprises a primary set of methodological tools recognized by a majority of premodern Sunnī jurists. Its elements, valid modes, and proper applications were the focus of continual argument and refinement. A particular area of debate was the methodology of determining or justifying the ʿilla: the legal cause giving rise to a ruling in God’s Law. This was most often discussed under the rubric of “the modes of causal justification”. Among these modes was the much debated test (...) of dawarān. In brief, proponents of dawarān employed it to justify claims that a property occasioned the ruling in an authoritative source-case. In concert with other considerations, the demonstrated co-presence and co-absence of property and ruling—that is, their concomitance “in existence” and “in nonexistence” — was taken as an indication that the property was the ruling’s ʿilla. Delving further into dawarān and causation, the current study interprets “in existence” and “in nonexistence” not as a kind of metaphor for true and false, but as an accurate terminology vis-à-vis the meaning of causality statements, fully compatible with dominant Islamicate views on causal agency. In brief, a deeper logical and linguistic analysis of the different existential modes of dawarān strongly suggests that we should distinguish property and ruling as types as opposed to tokens. Our reading of dawarān as shaped by a finer-grained structure not only allows us to identify the efficient occasioning process as a function which takes some particular token of the ʿilla and renders a token of the general ruling type, but it allows us to elucidate the role of taʿlīl in shaping an epistemological theory of argument to the best explanation: a sophisticated, premodern manifestation of abductive reasoning. (shrink)

It is widely held among philosophers that the conservation of energy is true and important, and widely held among philosophers of science that conservation laws and symmetries are tied together by Noether's first theorem. However, beneath the surface of such consensus lie two slights to Noether's first theorem. First, there is a 325+-year controversy about mind-body interaction in relation to the conservation of energy and momentum, with occasional reversals of opinion. The currently popular Leibnizian view, dominant since the late 19th (...) century, claims to find an objection to broadly Cartesian views in their implication of energy non-conservation. Here energy conservation is viewed as an oracle, an unchallengeable black box. But Noether's first theorem and its converse show that conservation and symmetry of the laws stand or fall together. Absent some basis for expecting conservation in brains that has a claim on the Cartesian, the objection is circular. An empirically based argument is possible, but is a different argument with little force except insofar as it is rooted in neuroscience. Second, General Relativity has a 100+-year-long controversy about whether gravitational energy exists and is objectively localized. The usual view is that gravitational energy exists but is not objectively localized, though some deny its existence. Without positive answers to both questions, generally applicable conservation laws do not exist: energy is not conserved. This conclusion is startling in itself and a problem for conserved quantity theories of causation. Yet Noether's first theorem applies to General Relativity, which has uncountably many symmetries of its laws and so has conservation laws, indeed uncountably many of them. Many authors downplay these laws due to their quirky properties; some authors even attempt to explain the laws' supposed nonexistence in terms of an absence of symmetries of the geometry, which is a distraction. Thus Noether's first theorem is widely ignored, left uninterpreted, or distorted in relation to General Relativity. Taking the theorem seriously seems possible, however, restoring the conservation of energy, or rather, energies. How do these controversies relate? One sometimes finds claims that General Relativity's supposed lack of conservation laws answers Leibniz on behalf of Descartes. Taking seriously the superabundance of formal conservation laws in General Relativity, however, suggests that General Relativity resists mind-to-body causation. This conclusion can be proven apart from interpretive controversies. The resistance is, however, finite and tends to be swamped by larger world-view considerations. (shrink)

Whether there exist causal relations between guns firing and people dying, between pedals pressed and cars accelerating, or between carbon dioxide emissions and global warming, is typically taken to be a mind-independent, objective, matter of fact. However, recent contributions to the literature on causation, in particular theories of contrastive causation and causal modelling, have undermined this central causal platitude by relativising causal facts to models or to interests. This thesis flies against the prevailing wind by arguing that we (...) must pay greater attention to which elements of our causal talk vary with context and which elements track genuine features of the world around us. I will argue that once these elements are teased apart we will be in a position to better understand some of the most persistent problems in the philosophy of causation: pre-emption cases, absence causation, failures of transitivity and overdetermination. The result is a naturalist account of causation, concordant with the contextual variability we find in our ordinary causal talk, and parsimonious with respect to the theoretical entities posited. (shrink)

Some omissions seem to be causes. For example, suppose Barry promises to water Alice’s plant, doesn’t water it, and that the plant then dries up and dies. Barry’s not watering the plant – his omitting to water the plant – caused its death. But there is reason to believe that if omissions are ever causes, then there is far more causation by omission than we ordinarily think. In other words, there is reason to think the following thesis true.

Open peer commentary on the article “Circularity and the Micro-Macro-Difference” by Manfred Füllsack. Upshot: We join Füllsack in his effort to untangle the concepts of circular causation, macro states, and observation by reanalyzing one of our own simulations in the light of these concepts. This simulation presents an example agent that keeps track of its own macro states. We examine how human observers can consider such an agent as an observing agent on its own.

This essay is concerned with concentrations of entities, which play an important—albeit often overlooked—role in scientific explanation. First, I discuss an example from molecular biology to show that concentrations can play an irreducible causal role. Second, I provide a preliminary philosophical analysis of this causal role, suggesting some implications for extant theories of causation. I conclude by introducing the concept of causation by concentration, a form of statistical causation whose widespread presence throughout the sciences has been unduly (...) neglected and which deserves to be studied in more depth. 1 Introduction2 Solving Lillie's Paradox: Lysogenic Induction in Phage λ3 Repressor Concentration and the Tuning of the Switch4 Concentration and Causality5 Preemption in Concentrations: Analysis and Implications6 Causation by Concentration: General Definition, Refinements, and Further Applications. (shrink)

This paper comments on Carolina Sartorio’s Causation and Free Will, challenging the non-modal conception of reasons-sensitivity that Sartorio advances.

In discussions on mental causation and externalism, it is often assumed that extrinsic, or relational, properties cannot have causal efficacy. In this paper I argue that this assumption is based on a category mistake, in that causal efficacy (dependence among events or states of affairs) is confused with causal influence (persistence of and interaction among objects). I then argue that relational properties are indeed causally efficacious, which I explain with the help of Dretske's notion of a 'structuring cause'.

Non-reductive Physicalism together with environment-dependence of content has been thought to be incompatible with the claim that beliefs are efficacious partly in virtue of their possession of content, that is, in virtue of their intentional properties. I argue that this is not so. First, I provide a general account of property causation. Then, I explain how, even given the truth of Non-reductive Physicalism and the environment-dependence of content, intentional properties will be efficacious according to this account. I go on (...) to relate my discussion to that concerning whether Anomalous Monism is committed to epiphenomenalism. I close by considering how my proposal suggests we should conceive of different levels of causation in a layered world. (shrink)

We present a probabilistic extension to active path analyses of token causation (Halpern & Pearl 2001, forthcoming; Hitchcock 2001). The extension uses the generalized notion of intervention presented in (Korb et al. 2004): we allow an intervention to set any probability distribution over the intervention variables, not just a single value. The resulting account can handle a wide range of examples. We do not claim the account is complete --- only that it fills an obvious gap in previous active-path (...) approaches. It still succumbs to recent counterexamples by Hiddleston (2005), because it does not explicitly consider causal processes. We claim three benefits: a detailed comparison of three active-path approaches, a probabilistic extension for each, and an algorithmic formulation. (shrink)

Does A cause B simply if A prevents what would have prevented B? Such a case is known as double prevention: where we have the prevention of a prevention. One theory of causation is that A causes B when B counterfactually depends on A and, as there is such a dependence, proponents of the view must rule that double prevention is causation.<br><br>However, if double prevention is causation, it means that causation can be an extrinsic matter, that (...) the cause and effect need not be connected by a continuous chain of events, that there can be causation by absence, and that there can be causation at a distance. All of these implications jar with strong intuitions we have about the nature of causation. There is, on the other hand, a theory of causation based on an ontology of real dispositions, where causation involves the passing around of powers. This theory in contrast entails that double prevention is not causation and, on this issue, it can claim a victory over the counterfactual dependence account. (shrink)

We present a probabilistic extension to active path analyses of token causation. The extension uses the generalized notion of intervention presented in : we allow an intervention to set any probability distribution over the intervention variables, not just a single value. The resulting account can handle a wide range of examples. We do not claim the account is complete --- only that it fills an obvious gap in previous active-path approaches. It still succumbs to recent counterexamples by Hiddleston, because (...) it does not explicitly consider causal processes. We claim three benefits: a detailed comparison of three active-path approaches, a probabilistic extension for each, and an algorithmic formulation. (shrink)

Studies performed in absence of gravitational constraint show that a living system is unable to choose between two different phenotypes, thus leading cells to segregate into different, alternative stable states. This finding demonstrates that the genotype does not determine by itself the phenotype but requires additional, physical constraints to finalize cell differentiation. Constraints belong to two classes: holonomic (independent of the system's dynamical states, as being established by the space‐time geometry of the field) and non‐holonomic (modified during those biological processes (...) to which they contribute in shaping). This latter kind of “constraints”, in which dynamics works on the constraint to recreate them, have emerged as critical determinants of self‐organizing systems, by manifesting a “closure of constraints.” Overall, the constraints act by harnessing the “randomness” represented by the simultaneous presence of equiprobable events restraining the system within one attractor. These results cast doubt on the mainstream scientific concept and call for a better understanding of causation in cell biology. (shrink)

This paper argues that several sorts of metaphysical and semantic indeterminacy afflict the causal relation. If, as it is plausible to hold, there is a relationship between causation and moral responsibility, then indeterminacy in the causal relation results in indeterminacy of moral responsibility more generally.