Causal exclusion arguments are taken to threaten the autonomy of the special sciences, and the causal efficacy of mental properties. A recent line of response to these arguments has appealed to “independently plausible” and “well grounded” theories of causation to rebut key premises. In this paper I consider two papers which proceed in this vein and show that they share a common feature: they both require causes to be proportional to their effects. I argue that this feature is a bug, (...) and one that generalises: any attempt to rescue the autonomy of the special sciences, or the efficacy of the mental, from exclusion worries had better not look to proportionality for help. (shrink)

This paper, like its companion explores some ways in which, on the one hand, normative theorizing about causation and causal reasoning and, on the other, empirical psychological investigations into causal cognition can be mutually illuminating. The paper carries out this exploration in connection with a variety of topics—the role of information about the presence of a “physical connection” between cause and effect in causal judgment, the role of “proportionality” in choosing the appropriate “level” of explanation, and the role of mechanism (...) information in causal judgment. (shrink)

The principle of proportionate causality is often cited as a cause for concern that Thomistic metaphysics may be irreconcilable with a theory of biological evolution. St. Thomas does hold that for the generation of what he calls perfect animals, a generator of the same species is required. This study clarifies what the proportionate causes of generated organisms are for Thomas, examining his views about spontaneous generation, reproductive generation, and hybridization, while also articulating the roles of both the (...) heavenly bodies and their separate movers as universal causes of generation. This study establishes that Thomas’s assertion of the need for a univocal generator for perfect animals is grounded not in the principle of proportionate causality, but rather in physical and biological doctrines received from Aristotle and in a causal principle that seems reconcilable with biological evolution, namely, that a remote universal cause requires more mediating causes to produce more powerful effects. (shrink)

A much-discussed idea in the causation literature is that it is preferable to invoke causes which are proportional to—neither too general nor too specific for—the effect. This article presents various ways of understanding this idea. In what sense are such causal claims ‘preferable’? And what is it for one event to be ‘proportional’ to another? In a companion article, ‘Proportionality in Causation, Part II: Applications and Challenges’, I discuss the principal applications of the resulting theories of proportionality, and the challenges (...) they face. (shrink)

It seems intuitive to think that if you contribute more to an outcome, you should be more morally responsible for it. Some philosophers think this is correct. They accept the thesis that ceteris paribus one's degree of moral responsibility for an outcome is proportionate to one's degree of causal contribution to that outcome. Yet, what the degree of causal contribution amounts to remains unclear in the literature. Hence, the underlying idea in this thesis remains equally unclear. In this article, (...) I will consider various plausible criteria for measuring degrees of causal contribution. After each of these criteria, I will show that this thesis entails implausible results. I will also show that there are other plausible theoretical options that can account for the kind of cases that motivate this thesis. I will conclude that we should reject this thesis. (shrink)

The consensus is that repeatable artworks cannot be identified with particular material individuals. A perennial temptation is to identify them with types, broadly construed. Such identification, however, faces the so-called “Creation Problem.” This problem stems from the fact that, on the one hand, it seems reasonable to accept the claims that (1) repeatable artworks are types, (2) types cannot be created, and (3) repeatable artworks are created, but, on the other hand, these claims are mutually inconsistent. A possible solution to (...) the Creation Problem is to argue that claim (2) can be rejected because (a) the only motivation for it is that a type, being abstract, cannot stand in causal relations, but (b) this motivation is ungrounded, since types can, in fact, stand in such relations. Clearly, in order for this solution to be successful, it is necessary to substantiate the possibility of types to be causally efficacious. In this essay, I examine an attempt to do this with the help of Yablo’s principle of proportionality, which has been undertaken by Walters (2013) and, more recently, Juvshik (2018). Although the argument they advance may seem to provide strong support for the causal efficacy of types, I think it actually fails to do this. To explain why this is so, I first show that this argument commits us to the existence of widespread causal overdetermination involving types and then argue that this commitment is both epistemically and ontologically problematic. (shrink)

In ‘Proportionality in Causation, Part I: Theories’, I presented various ways of understanding the idea that causes which are ‘proportional’ to their effects are in some sense preferable. In this companion article, I discuss the principal applications of the resulting theories of proportionality, and the challenges they face.

There are several supposedly lethal objections to the view that causation is essentially proportional. The first targets an account of proportionality in terms of causal models, pointing out that proportionality is too easily satisfied in causal model accounts of causation through manipulation of the range of values that a variable can take (Franklin-Hall, 2016). The second argues that proportionality legitimizes only the most general things as causes, and proportionality thereby contravenes causal intuitions (Bontly, 2005; Franklin-Hall, 2016; McDonnell, 2018, 2017; Weslake, (...) 2013). The final, and perhaps most intractable, objection holds that proportionality counter-intuitively legitimizes disjunctive causes (Shapiro and Sober, 2012; Weslake, 2017; Woodward, 2018). This paper provides a unified response to these objections, which is best formulated in a causal model framework. I first articulate two independently plausible principles of variable selection – exclusivity and exhaustivity. I then show how the adoption of these principles responds to Franklin-Hall’s objection, and dissolves the remaining two. (shrink)

This paper is concerned with the content of the causal proportionality constraint. It investigates two general versions of the constraint, namely “horizontal” and “vertical” proportionality. Moreover, it discusses whether proportionality is considered an ontic or an epistemic, i.e. explanatory, constraint on causation in the context of some of the most prominent theories of causation. The following main claims are defended: (1) The horizontal (HP) and the vertical version (VP) of the proportionality constraint are logically independent. (2) HP is implied by (...) some prominent theories of causation, not by others. (3) None of the discussed popular theories of causation contradicts either HP or VP. (4) HP and VP are not ontic or epistemic principles as such; rather, whether they are ontic or epistemic depends on the theories chosen plus background assumptions about the existence of higher-level causes and their non-identity to lower-level ones. (shrink)

Proportionality—the idea that causes are neither too general nor too specific for their effects—seems to recommend implausibly disjunctive causes (McGrath, 1998 ; Shapiro & Sober, 2012 ; Franklin-Hall, 2016 ). I argue that this problem should be avoided by appeal to the notion of cohesion. I propose an account of cohesion in terms of the similarity structure of property-spaces, argue that it is not objectionably mysterious, and that alternative approaches—based on naturalness, interventionism, and contrastivism—are inadequate without appeal to it. In (...) an appendix, I show how my proposal can be perspicuously formalized by adapting structural equation models. (shrink)

According to the so-called 'proportionality principle', causes should be proportional to their effects: they should be both enough and not too much for the occurrence of their effects. This principle is the subject of an ongoing debate. On the one hand, many maintain that it is required to address the problem of causal exclusion and take it to capture a crucial aspect of causation. On the other hand, many object that it renders accounts of causation implausibly restrictive and often reject (...) the principle wholesale. I argue that there is exaggeration on both sides. While one half of the principle is overly demanding, the other half is unobjectionable. And while the unobjectionable half does not block exclusion arguments on its own, it provides a nuanced picture of higher-level causation, fits with recent developments in philosophy of causation, and motivates adjustments to standard difference-making accounts of causation. I conclude that at least half of the proportionality principle is worth taking seriously. (shrink)

Supporters of the autonomy of higher-level causation (or explanation) often appeal to proportionality, arguing that higher-level causes are more proportional than their lower-level realizers. Recently, measures based on information theory and causal modeling have been proposed that allow one to shed new light on proportionality and the related notion of specificity. In this paper we apply ideas from this literature to the issue of higher vs. lower-level causation (and explanation). Surprisingly, proportionality turns out to be irrelevant for the question of (...) whether higher-level causes (or explanations) can be autonomous; specificity is a much more informative notion for this purpose. (shrink)

A cause is proportional to its effect when, roughly speaking, it is at the right level of detail. There is a lively debate about whether proportionality is a necessary condition for causation. One of the main arguments against a proportionality constraint on causation is that many ordinary and seemingly perfectly acceptable causal claims cite causes that are not proportional to their effects. In this paper, I suggest that proponents of a proportionality constraint can respond to this objection by developing an (...) idea that is present in Yablo’s early work on proportionality, but which has strangely been ignored by both Yablo and others in the subsequent debate. My suggestion is that proportionality—and, indeed, causation itself—is relative to a domain of events. At the metaphysical level, this means that the causal relation has an extra relatum—namely, a domain of events. At the level of language, it introduces a new way in which causal claims are context-sensitive: what is expressed by a causal claim depends on the contextually relevant domain of events. As I argue, this suggestion allows us to accommodate the truth of ordinary causal claims while extending the explanatory benefits of a proportionality constraint. (shrink)

Lynch et al. (Biol Philos 34:62, 2019) employ stability, specificity, and proportionality as criteria for evaluating microbiome causal explanations. Although these causal characteristics signify relevant differences between causal roles, I suggest that they should not be used as general criteria for strong or good causal explanations.

A statement of the form ‘C caused E’ obeys the requirement of proportionality precisely when C says no more than what is necessary to bring about E. The thesis that causal statements must obey this requirement might be given a semantic or a pragmatic justification. We use the idea that causal claims are contrastive to criticize both.

If counterfactual dependence is sufficient for causation and if omissions can be causes, then all events have many more causes than common sense tends to recognize. This problem is standardly addressed by appeal to pragmatics. However, Carolina Sartorio [2010] has recently raised what I shall argue is a more interesting problem concerning omissions for counterfactual theories of causation—more interesting because it demands a more subtle pragmatic solution. I discuss the relationship between the idea that causes are proportional to their effects, (...) the idea that causation is contrastive, and the question of the dimensions along which causal explanations should be evaluated with respect to one another. (shrink)

Stephen Yablo’s notion of proportionality, despite controversies surrounding it, has played a significant role in philosophical discussions of mental causation and of high-level causation more generally. In particular, it is invoked in James Woodward’s interventionist account of high-level causation and explanation, and is implicit in a novel approach to constructing variables for causal modeling in the machine learning literature, known as causal feature learning (CFL). In this article, we articulate an account of proportionality inspired by both Yablo’s account of proportionality (...) and the CFL account of variable construction. The resulting account has at least three merits. First, it illuminates an important feature of the notion of proportionality, when it is adapted to a probabilistic and interventionist framework. The feature is that at the center of the notion of proportionality lies the concept of “determinate intervention effects.” Second, it makes manifest a virtue of (common types of) high-level causal/explanatory statements over low-level ones, when relevant intervention effects are determinate. Third, it overcomes a limitation of the CFL framework and thereby also addresses a challenge to interventionist accounts of high-level causation. (shrink)

The recent literature on causality has seen the introduction of several distinctions within causality, which are thought to be important for understanding the widespread scientific practice of focusing causal explanations on a subset of the factors that are causally relevant for a phenomenon. Concepts used to draw such distinctions include, among others, stability, specificity, proportionality, or actual-difference making. In this contribution, I propose a new distinction that picks out an explanatorily salient class of causes in biological systems. Some (...) select causes in complex biological systems, I argue, have the property of enabling coherent causal control of these systems. Examples of such control variables include hormones and other signaling molecules, e.g., TOR (target of rapamycin), morphogens or the products of homeotic selector genes in embryonic pattern formation. I propose an analysis of this notion based on concepts borrowed from causal graph theory. (shrink)

This paper reexamines the case for a proportionality constraint on causation. The general idea behind the proportionality constraint is that causes need the right amount of detail. The cause needs to be detailed enough to be sufficient for the effect yet general enough to be fully relevant to the effect. The case for the proportionality constraint mainly rests on some examples. Suppose we are searching for the cause of an injury: “being hit by a red bus” is too detailed, “being (...) hit” isn't detailed enough, but “being hit by a bus” is about right. This sort of example has undeniable intuitive appeal. However, this intuitive appeal needs to be examined with more care before jumping to conclusions about the metaphysics of causation and the mereology of causal relata. Here, I reexamine the case for a proportionality constraint on causation and compare several pragmatic explanations of our intuitions about Yablo’s examples, in particular a pragmatic explanation that notices the loose use in our naming of causal relata and a pragmatic explanation that appeals to a contrastivist account of causation. (shrink)

A considerable number of philosophers are attracted to what might be called ‘causal type-abstractionism’ – the view that photographs, symphonies, models of cars, novels, flags, and other multiply i...

Causal theories of action, perception and knowledge are each beset by problems of so-called ‘deviant’ causal chains. For each such theory, counterexamples are formed using odd or co-incidental causal chains to establish that the theory is committed to unpalatable claims about some intentional action, about a case of veridical perception or about the acquisition of genuine knowledge. In this paper I will argue that three well-known examples of a deviant causal chain have something in common: they each violate Yablos proportionality (...) constraint on causation. I will argue that this constraint provides the key to saving causal theories from deviant chains. (shrink)

Human microbiome research makes causal connections between entire microbial communities and a wide array of traits that range from physiological diseases to psychological states. To evaluate these causal claims, we first examine a well-known single-microbe causal explanation: of Helicobacter pylori causing ulcers. This apparently straightforward causal explanation is not so simple, however. It does not achieve a key explanatory standard in microbiology, of Koch’s postulates, which rely on manipulations of single-microorganism cultures to infer causal relationships to disease. When Koch’s postulates (...) are framed by an interventionist causal framework, it is clearer what the H. pylori explanation achieves and where its explanatory strengths lie. After assessing this ‘simple’, single-microbe case, we apply the interventionist framework to two key areas of microbiome research, in which obesity and mental health states are purportedly explained by microbiomes. Despite the experimental data available, interventionist criteria for explanation show that many of the causal claims generated by microbiome research are weak or misleading. We focus on the stability, specificity and proportionality of proposed microbiome causal explanations, and evaluate how effectively these dimensions of causal explanation are achieved in some promising avenues of research. We suggest some conceptual and explanatory strategies to improve how causal claims about microbiomes are made. (shrink)

Many philosophers have raised difficulties for any attempt to proportion punishment severity to crime seriousness. One reason for this may be that offering a full theory of proportionality is simply too ambitious. I suggest a more modest project: setting a lower bound on proportionate punishment. That is, I suggest a metric to measure when punishment is not disproportionately severe. I claim that punishment is not disproportionately severe if it imposes costs on a criminal wrongdoer which are no greater than (...) the costs which they intentionally caused to others. I flesh out the implications of this Lower bound by discussing how to measure the costs of crime. Methodologically, I claim that different costs should be compared by considering preferences. Substantively, I claim that many proportionality judgements undercount the costs of crime by focusing only on the marginal and not the average cost. I suggest that we may hold defendants causally responsible for their contribution to the costs of that type of crime. (shrink)

Human microbiome research makes causal connections between entire microbial communities and a wide array of traits that range from physiological diseases to psychological states. To evaluate these causal claims, we first examine a well-known single-microbe causal explanation: of Helicobacter pylori causing ulcers. This apparently straightforward causal explanation is not so simple, however. It does not achieve a key explanatory standard in microbiology, of Koch’s postulates, which rely on manipulations of single-microorganism cultures to infer causal relationships to disease. When Koch’s postulates (...) are framed by an interventionist causal framework, it is clearer what the H. pylori explanation achieves and where its explanatory strengths lie. After assessing this ‘simple’, single-microbe case, we apply the interventionist framework to two key areas of microbiome research, in which obesity and mental health states are purportedly explained by microbiomes. Despite the experimental data available, interventionist criteria for explanation show that many of the causal claims generated by microbiome research are weak or misleading. We focus on the stability, specificity and proportionality of proposed microbiome causal explanations, and evaluate how effectively these dimensions of causal explanation are achieved in some promising avenues of research. We suggest some conceptual and explanatory strategies to improve how causal claims about microbiomes are made. (shrink)

It is commonly assumed that causation is transitive and in this paper I aim to reconcile this widely-held assumption with apparent evidence to the contrary. I will discuss a familiar approach to certain well-known counterexamples, before introducing a more resistant sort of case of my own. I will then offer a novel solution, based on Yablo’s proportionality principle, that succeeds in even these more resistant cases. There is a catch, however. Either proportionality is a constraint on which causal claims are (...) true, and the solution works, or it is not and causation is not transitive after all. I will argue that the first horn has unacceptable consequences and should be rejected, but that the second horn is less costly than it might initially appear. (shrink)

Human microbiome research makes causal connections between entire microbial communities and a wide array of traits that range from physiological diseases to psychological states. To evaluate these causal claims, we first examine a well-known single-microbe causal explanation: of Helicobacter pylori causing ulcers. This apparently straightforward causal explanation is not so simple, however. It does not achieve a key explanatory standard in microbiology, of Koch’s postulates, which rely on manipulations of single-microorganism cultures to infer causal relationships to disease. When Koch’s postulates (...) are framed by an interventionist causal framework, it is clearer what the H. pylori explanation achieves and where its explanatory strengths lie. After assessing this ‘simple’, single-microbe case, we apply the interventionist framework to two key areas of microbiome research, in which obesity and mental health states are purportedly explained by microbiomes. Despite the experimental data available, interventionist criteria for explanation show that many of the causal claims generated by microbiome research are weak or misleading. We focus on the stability, specificity and proportionality of proposed microbiome causal explanations, and evaluate how effectively these dimensions of causal explanation are achieved in some promising avenues of research. We suggest some conceptual and explanatory strategies to improve how causal claims about microbiomes are made. (shrink)

The interventionist account of causation offers a criterion to distinguish causes from non-causes. It also aims at defining various desirable properties of causal relationships, such as specificity, proportionality and stability. Here we apply an information-theoretic approach to these properties. We show that the interventionist criterion of causation is formally equivalent to non-zero specificity, and that there are natural, information-theoretic ways to explicate the distinction between potential and actual causal influence. We explicate the idea that the description of causes should be (...) proportional to that of their effects. Then we draw a distinction between two ideas in the existing literature, the range of invariance of a causal relationship and its stability. The range of invariance is related to specificity and range of causal values. Stability concerns the effect of additional variables on the relationship between some focal pair of cause and effect variables. We show how to distinguish and measure the direct influence of background variables on the effect variable, and their influence on the relationship between the focal cause and the effect variable. Finally, we discuss the limitations of the information-theoretic approach, and offer prospects for complementary approaches. (shrink)

This paper responds to recent criticisms of the idea that true causal claims, satisfying a minimal “interventionist” criterion for causation, can differ in the extent to which they satisfy other conditions—called stability and proportionality—that are relevant to their use in explanatory theorizing. It reformulates the notion of proportionality so as to avoid problems with previous formulations. It also introduces the notion of conditional independence or irrelevance, which I claim is central to understanding the respects and the extent to which upper (...) level explanations can be “autonomous”. (shrink)

This paper argues that according to Adam Smith the workings of (anything but extremely simple) sympathetic judgment (s) presuppose and crucially depend on counterfactual causal reasoning in the sympathetic process. In particular it argues for four related claims: (i) that according to Smith that the sympathetic process depends on a type of causal reasoning that goes well beyond the kind of simulationist theory standardly attributed to him; (ii) that the Smithian imagination in the sympathetic process works by way of counterfactual (...) reasoning and that even the feelings we ought to feel as a consequence of the sympathetic process need not be actual, but counterfactual; (iii) that Smithian agents are non-trivially understood as belonging to the causal order of nature; (iv) that Smitian judgments of propriety are intrinsically judgments about the proportionality of causal relations. (shrink)

This dissertation begins by addressing the question of when a causal model is apt for deciding questions of actual causation with respect to some target situation. I first provide relevant background about causal models, explain what makes them promising as a tool for analyzing actual causation, and motivate the need for a theory of aptness as part of such an analysis (Chapter 1). I then define what it is for a model on a given interpretation to be accurate of, that (...) is, say only true things about, some target situation. This involves a systematization of various representational principles mentioned and/or discussed throughout the literature into a method of interpretation, which I propose be taken as standard (Chapter 2). Next, I explain and address two reasons for which accuracy as I’ve defined it is insufficient for aptness. The first reason – already discussed in the literature – is the problem of structural isomorphs. In response, I propose the aptness condition of Explicit Partial Mediation (Chapter 3). The second reason – which has yet to be noticed – is the problem of the indeterminacy of accuracy. As I demonstrate, a model is accurate of a target situation only relative to a set of background possibilities – what I call a modal profile. It follows that a model represents a situation only relative to some modal profile or other. I go on to discuss the ramifications of this observation for a theory of actual causation in terms of models. I argue that the relativity be taken at face value and built into our metaphysical account of causation, resulting in a view that I call causal relativism (Chapter 4). I explore one advantage of this view in detail: that the resulting account can defend the principle of strong proportionality against several objections (Chapter 5). Finally, I apply the earlier discussion of aptness to attempts to provide a semantics of counterfactuals in terms of causal models – an interventionist semantics. I show how just as a similarity semantics relies on an opaque notion of similarity, an interventionist semantics relies on an analogous notion of aptness. The challenge of articulating aptness thus undermines the claim that an interventionist semantics avoids representational problems inherent in a similarity semantics (Chapter 6). I close with a recap and suggestions for future research (Chapter 7). -/- . (shrink)

Sydney Shoemaker has attempted to save mental causation by a new account of realization. As Brian McLaughlin argues convincingly, the account has to face two major problems. First, realization does not guarantee entailment. So even if mental properties are realized by physical properties, they need not be entailed by them. This is the first, rather general metaphysical problem. A second problem, which relates more directly to mental causation is that Shoemaker must appeal to some kind of proportionality as a constraint (...) on causation in order to avoid redundant mental causation. I argue that, in addition, a “piling problem” arises, since causal powers seem to be bestowed twice. Then, I try to sketch an alternative view of the relation between causal powers and properties—a reductionist view—which fares better on some accounts. But it may have to face another and, perhaps, serious problem, the “problem of the natural unity of properties”. Finally, I will pose a question about the relation between causal powers and causation. (shrink)

This paper responds to criticisms of my account of the role of proportionality and stability in causal reasoning. It reformulates the notion of proportionality in response to objections.

objects are standardly taken to be causally inert, but this claim is rarely explicitly argued for. In the context of his platonism about musical works, in order for musical works to be audible, Julian Dodd argues that abstracta are causally efficacious in virtue of their concrete tokens participating in events. I attempt to provide a principled argument for the causal inertness of abstracta by first rejecting Dodd’s arguments from events, and then extending and generalizing the causal exclusion argument to the (...) abstract/concrete distinction. For reasons of parsimony, if concrete tokens or instantiations of abstract objects account for all causal work, then there is no reason to attribute causal efficacy to abstracta, and thus reason to maintain their causal inertness. I then consider how one of the main arguments against causal exclusion, namely Stephen Yablo’s notion of “proportionality”, could be modified to support the causal efficacy of abstracta. I argue that from a few simple premises Yablo’s account in fact supports their causal inertness. Having a principled reason for the causal inertness of abstracta appears to entail that the musical platonist must admit that we never literally hear the musical work, but only its performances. I sketch a solution to this problem available to Dodd, so that the musical platonist can maintain that musical works are abstract objects and are causally inert while retaining their audibility. (shrink)

Causalists about explanation claim that to explain an event is to provide information about the causal history of that event. Some causalists also endorse a proportionality claim, namely that one explanation is better than another insofar as it provides a greater amount of causal information. In this chapter I consider various challenges to these causalist claims. There is a common and influential formulation of the causalist requirement – the ‘Causal Process Requirement’ – that does appear vulnerable to these anti-causalist challenges, (...) but I argue that they do not give us reason to reject causalism entirely. Instead, these challenges lead us to articulate the causalist requirement in an alternative way. This alternative articulation incorporates some of the important anti-causalist insights without abandoning the explanatory necessity of causal information. For example, proponents of the ‘equilibrium challenge’ argue that the best available explanations of the behaviour of certain dynamical systems do not appear to provide any causal information. I respond that, contrary to appearances, these equilibrium explanations are fundamentally causal, and I provide a formulation of the causalist thesis that is immune to the equilibrium challenge. I then show how this formulation is also immune to the ‘epistemic challenge’ – thus vindicating (a properly formulated version of) the causalist thesis. (shrink)

The claim that happiness and virtue ought to be proportionate to one another has often been expressed in the idea of a future world of divine justice, despite many moral difficulties with this idea. This paper argues that human efforts to enact such a proportionment are, ironically, justified by the same reasons that make the idea of divine justice seem so problematic. Moralists have often regarded our frailty and fallibility as reasons for abstaining from the judgment of others; and (...) doubts about our deserving some proportionment of happiness or unhappiness often arise insofar as virtue and vice may be explained on a causal basis. This paper argues that our fallibility and our susceptibility to social influence render judgment and response indispensable, because – given these characteristics – our actions and responses decide the morality that we actually share with one another. In this situation, to ‘judge not’ is to abandon the field to those with no such scruples. (shrink)

It is shown that certain kinds of behavior, which hitherto were expected to be characteristic for classical gravity and quantum field theory in curved spacetime, as the infinite dimensional Bondi-Metzner-Sachs symmetry, holography on event horizons and an area proportionality of entropy, have in fact an unnoticed presence in Minkowski QFT.This casts new light on the fundamental question whether the volume proportionality of heat bath entropy and the (logarithmically corrected) dimensionless area law obeyed by localization-induced thermal behavior are different geometric parametrizations (...) which share a common primordial algebraic origin. Strong arguments are presented that these two different thermal manifestations can be directly related, this is in fact the main aim of this paper.It will be demonstrated that QFT beyond the Lagrangian quantization setting receives crucial new impulses from holography onto horizons.The present paper is part of a project aimed at elucidating the enormous physical range of “modular localization”. The latter does not only extend from standard Hamiltonian heat bath thermal states to thermal aspects of causal- or event-horizons addressed in this paper. It also includes the recent understanding of the crossing property of formfactors whose intriguing similarity with thermal properties was, although sometimes noticed, only sufficiently understood in the modular setting (Schroer in arXiv:0905.4006 (2009)). (shrink)

Intuitions play an important role in the debate on the causal status of high‐level properties. For instance, Kim has claimed that his “exclusion argument” relies on “a perfectly intuitive … understanding of the causal relation.” We report the results of three experiments examining whether laypeople really have the relevant intuitions. We find little support for Kim's view and the principles on which it relies. Instead, we find that laypeople are willing to count both a multiply realized property and its realizers (...) as causes, and regard the systematic overdetermination implied by this view as unproblematic. (shrink)

Lynch et al., in an article in this issue, argue that an entire microbiome is rarely, if ever, the right target of analysis for causal explanations in microbiome research. They argue, using interventionist criteria of proportionality, specificity and stability, for restricting causal claims to the smallest subset of microbes—a causal core—that generate the effect of interest. A further question remains: what kind of interactions generate a consortium of microbes that can operate as causal agents in this manner? Here I introduce (...) two possible kinds of such consortia: ‘trojan horses’ and ‘syntrophic’ individuals. (shrink)

Lynch et al., in an article in this issue, argue that an entire microbiome is rarely, if ever, the right target of analysis for causal explanations in microbiome research. They argue, using interventionist criteria of proportionality, specificity and stability, for restricting causal claims to the smallest subset of microbes—a causal core—that generate the effect of interest. A further question remains: what kind of interactions generate a consortium of microbes that can operate as causal agents in this manner? Here I introduce (...) two possible kinds of such consortia: ‘trojan horses’ and ‘syntrophic’ individuals. (shrink)

Much microbiota-gut-brain research focuses on the causal role of microbiomes as a whole, rather than their component parts: microbes. Hooks et al. find these whole-community explanations inadequate; however, they do not provide suggestions for better explanations. By appealing to proportionality – a criterion that can be used to develop more appropriate causal explanations – more accurate causal claims can be made.

This commentary focuses on the authors’ treatment of Koch’s postulates. It argues in favour of a modification of Koch’s postulates and their analysis in terms of necessary and sufficient conditions. This leads to a criticism of the authors’ treatment of the C. difficile case, and to query the need for the criteria of specificity and proportionality.

This commentary focuses on the authors’ treatment of Koch’s postulates. It argues in favour of a modification of Koch’s postulates and their analysis in terms of necessary and sufficient conditions. This leads to a criticism of the authors’ treatment of the C. difficile case, and to query the need for the criteria of specificity and proportionality.

Theories of graded causation attract growing attention in the philosophical debate on causation. An important field of application is the controversial relationship between causation and moral responsibility. However, it is still unclear how exactly the notion of graded causation should be understood in the context of moral responsibility. One question is whether we should endorse a proportionality principle, according to which the degree of an agent’s moral responsibility is proportionate to their degree of causal contribution. A second question is (...) whether a theory of graded causation should measure closeness to necessity or closeness to sufficiency. In this paper, we argue that we should indeed endorse a proportionality principle and that this principle supports a notion of graded causation relying on closeness to sufficiency rather than closeness to necessity. Furthermore, we argue that this insight helps to provide a plausible analysis of the so-called ‘Moral Difference Puzzle’ recently described by Bernstein. (shrink)

The interventionist account of causal explanation, in the version presented by Jim Woodward, has been recently claimed capable of buttressing the widely felt—though poorly understood—hunch that high-level, relatively abstract explanations, of the sort provided by sciences like biology, psychology and economics, are in some cases explanatorily optimal. It is the aim of this paper to show that this is mistaken. Due to a lack of effective constraints on the causal variables at the heart of the interventionist causal-explanatory scheme, as presently (...) formulated it is either unable to prefer high-level explanations to low, or systematically overshoots, recommending explanations at so high of a level as to be virtually vacuous. (shrink)

ABSTRACT The exclusion problem is meant to show that non-reductive physicalism leads to epiphenomenalism: if mental properties are not identical with physical properties, then they are not causally efficacious. Defenders of a difference-making account of causation suggest that the exclusion problem can be solved because mental properties can be difference-making causes of physical effects. Here, we focus on what we dub an incompatibilist implementation of this general strategy and argue against it from a non-reductive physicalist perspective. Specifically, we argue that (...) incompatibilism undermines either the non-reductionist or the physicalist aspirations of non-reductive physicalism. (shrink)

The subset realization view proposes to solve the causal exclusion problem of non‐reductive mental instances by taking the mental instance as a part of its physical realizer. Many philosophers have argued that such a part‐whole relation will undermine physicalist realization because parts are ontologically prior to their wholes and the subset view is thus flawed. I argue that the relation that the subset view should propose is different from the ordinary part‐whole relation. What they should propose is another kind of (...) part‐whole relation under which parts are posterior to wholes. So, the subset view is not flawed in the aforementioned aspect. But discussion of the new part‐whole relation reveals that the subset view is in lack of a crucial argument against the exclusion reasoning, thus cannot solve the exclusion problem. Adopting the principle of causal proportionality (a route subset realizationists sometimes take) will not help the subset view either. So, my conclusion is, the subset view is yet to find a way to solve the exclusion problem. (shrink)

This work is a systematic investigation of a range of solutions offered today for the philosophical problem of mental causation. The premises constituting the problem are analyzed before a survey is developed of the most popular theories on mental causation. It is demonstrated in detail why most of these canonical solutions must be considered deficient. In a third part, the 'new compatibilist's' approach to mental causation is explored, which is characterized by assertion of a non-identity-but-non-distinctness principle. The last part aims (...) to offer an alternative solution to the problem. On the basis of a certain set of counterfactual conditionals, which are jointly taken to provide a definition of 'causal proportionality' that improves the existing definitions, it is shown that a specific, and hitherto widely neglected, version of causal overdeterminationism must be considered the most successful solution to the problem of mental causation. (shrink)

This paper attempts to elucidate three characteristics of causal relationships that are important in biological contexts. Stability has to do with whether a causal relationship continues to hold under changes in background conditions. Proportionality has to do with whether changes in the state of the cause “line up” in the right way with changes in the state of the effect and with whether the cause and effect are characterized in a way that contains irrelevant detail. Specificity is connected both to (...) David Lewis’ notion of “influence” and also with the extent to which a causal relation approximates to the ideal of one cause–one effect. Interrelations among these notions and their possible biological significance are also discussed. (shrink)

Does one’s causal responsibility increase the degree of one’s moral responsibility? The proponents of resultant moral luck hold that it does. Until quite recently, the causation literature has almost exclusively been interested in the binary question of whether one factor is a cause of an outcome. Naturally, the debate over resultant moral luck also revolved around this binary question. However, we have seen an increased interest in the question of degrees of causation in recent years. And some philosophers have already (...) explored various implications of a graded notion of causation on resultant moral luck. In this paper, I will do the same. But the implications that I will draw attention to are bad news for resultant moral luck. I will show that resultant moral luck entails some implausible results that leave resultant moral luck more indefensible than it was previously thought be. I will also show that what is typically taken to be the positive argument in favor of resultant moral luck fails. I will conclude that we should reject resultant moral luck. (NOTE: Email me for a copy.). (shrink)