Ecological fitness has been suggested to provide a unifying definition of fitness. However, a metric for this notion of fitness was in most cases unavailable except by proxy with differential reproductive success. In this article, I show how differential persistence of lineages can be used as a way to assess ecological fitness. This view is inspired by a better understanding of the evolution of some clonal plants, colonial organisms, and ecosystems. Differential persistence shows the limitation of an ensemblist noncausal understanding (...) of evolution. Causal explanations are necessary to understand the evolution by natural selection of these biological systems. †To contact the author write to: Department of Philosophy, University of Montreal, P.O. Box 6128, Station Centre‐Ville, Montreal, Quebec, H3C 3J7 Canada; e‐mail: [email protected]. (shrink)

In this work we try to study theories of causation based upon causal processes and causal interactions in the context of classical and quantum physics. Our central aim is to find out whether such causal theories are compatible with the world picture suggested by contemporary theories of physics. In the first part, we review, compare and try to place among more general taxonomical schemes, the causal theories by Russell (the causal lines approach), Reichenbach (mark (...) method, probabilistic causality and the principle of common cause), Salmon (mark method, structure, invariant and conserved quantities approaches) and Dowe (conserved quantity theory). In the second part of the work, we deal with some problems that process theories of causation face whenever one tries to define, rigorously, causal concepts (e.g. “causal process” or “conjunctive common cause”) in the context of classical relativistic physical theories (van Dam – Wigner particle theory, the free Klein-Gordon field, classical electromagnetic theory) as well as quantum theories (algebraic relativistic quantum field theory). In this vein, special attention has been given to the locality problem in physical theories as it emerges from the structure of local classical the quantum theories (semantic locality), the relation between global and local conditions for physical systems (global and local determinism) as well as the problem of action at a distance, which is of central importance for the theories of causation under examination (Bell’s inequalities, EPR paradox, Reeh-Schlieder theorem and local independence conditions). We argue for three different theses: first, there are physical theories in the context of which it is impossible even to formulate the necessary physical hypotheses that allow us to define basic causal concepts and causal principles; second, there are physical theories which allow us to formulate these hypotheses only ad hoc; finally, in some cases it is necessary to revise basic causal principles and concepts bequeathed by the philosophical tradition. (shrink)

Abstract In this paper I consider an easier-to-read and improved to a certain extent version of the causal chance-based analysis of counterfactuals that I proposed and argued for in my A Theory of Counterfactuals. Sections 2, 3 and 4 form Part I: In it, I survey the analysis of the core counterfactuals (in which, very roughly, the antecedent is compatible with history prior to it). In section 2 I go through the three main aspects of this analysis, which are (...) the following. First, it is a causal analysis, in that it requires that intermediate events to which the antecedent event is not a cause be preserved in the main truth-condition schema. Second, it highlights the central notion to the semantics of counterfactuals on the account presented here -- the notion of the counterfactual probability of a given counterfactual, which is the probability of the consequent given the following: the antecedent, the prior history, and the preserved intermediate events. Third, it considers the truth conditions for counterfactuals of this sort as consisting in this counterfactual probability being higher than a threshold. In section 3, I re-formulate the analysis of preservational counterfactuals in terms of the notion of being a cause, which ends up being quite compact. In section 4 I illustrate this analysis by showing how it handles two examples that have been considered puzzling – Morgenbesser's counterfactual and Edgington's counterfactual. Sections 5 and on constitute Part II: Its main initial thrust is provided in section 5, where I present the main lines of the extension of the theory from the core counterfactuals (analyzed in part I) to counterfactuals (roughly) whose antecedents are not compatible with their prior history. In this part II, I elaborate on counterfactuals that don't belong to the core, and more specifically on so-called reconstructional counterfactuals (as opposed to the preservational counterfactuals, which constitute the core counterfactual-type). The heart of the analysis is formulated in terms of processes leading to the antecedent (event/state), and more specifically in terms of processes likely to have led to the antecedent, a notion which is analyzed entirely in terms of chance. It covers so-called reconstructional counterfactuals as opposed to the core, so-called preservational counterfactuals, which are analyzed in sections 2 and 3 of part I. The counterfactual probability of such reconstructional counterfactuals is determined via the probability of possible processes leading to the antecedent weighed, primarily and roughly, by the conditional probability of the antecedent given such process: The counterfactual probability is thus, very roughly, a weighted sum for all processes most likely to have led to the antecedent, diverging at a fixed time. In section 6 I explain and elaborate further on the main points in section 5. In section 7 I illustrate the reconstructional analysis. I specify counterfactuals which are so-called process-pointers, since their consequent specifies stages in processes likely to have led to their antecedent. I argue that so-called backtracking counterfactuals are process-pointers counterfactuals, which fit into the reconstructional analysis, and do not call for a separate reading. I then illustrate cases where a speaker unwittingly employs a certain counterfactual while charitably construable as intending to assert (or ‘having in mind’) another. Here I also cover the issue of how to construe what one can take as back-tracking counterfactuals, or counterfactuals of the reconstructional sort, and more specifically, which divergence point they should be taken as alluding to (prior to which the history is held fixed). Some such cases also give rise to what one can take as a dual reading of a counterfactual between preservational and reconstructional readings. Such cases may yield an ambiguity, where in many cases one construal is dominant. In section 8 I illustrate the analysis by applying it to the famous Bizet-Verdi counterfactuals. This detailed analysis of counterfactuals (designed for the indeterministic case) has three main distinctive elements: its being chance-based, its causal aspect, and the use it makes of processes most likely to have led to the antecedent-event. This analysis is couched in a very different conceptual base from, and is an alternative account to, analyses in terms of the standard notion of closeness or distance of possible worlds, which is the main feature of the Stalnaker-Lewis-type analyses of counterfactuals. This notion of closeness or distance plays no role whatsoever in the analysis presented here. (This notion of closeness has been left open by Stalnaker, and to significant extent also by Lewis's second account.) . (shrink)

The evolutionary emergence of biological processes in organisms with inner, qualitative aspects has not been explained in any sufficient way by neurobiology, nor by the traditional neo-Darwinian paradigm — natural selection would appear to work just as well on insentient zombies (with the right behavioral input-output relations) as on real sentient animals. In consciousness studies one talks about the ‘hard problem’ of qualia. In this paper I sketch a set of principles about sign action, causality and emergent evolution. On (...) the basis of these principles, I characterize a concept of cause that would allow for a naturalistic explanation of the origin of consciousness. The suggested account of causation also turns the ‘hard problem’ of qualia into the easier problem of relating experimental biology to experiential biology. (shrink)

Concerning any object of philosophical analysis, we can ask several questions, including the two posed in the title of this paper. Despite difficulties in formulating a precise criterion to distinguish causal processes from pseudoprocesses, and causal interactions from mere spatiotemporal intersections, I argue that Salmon answered the first of these questions with extraordinary clarity. The second question, by contrast, has received very little attention. I will present two problems: in the first, it seems that Salmon has provided (...) exactly the conceptual resources needed to solve the problem; in the second, it is difficult to see how causal processes and interactions may be used to shed any light. In general, the way to carry Salmon's program forward will be to demonstrate that these resources can be made to do real philosophical work. (shrink)

I offer an alternative interpretation of Van Fraassen's influential arguments against causal realism in quantum mechanics. These arguments provide in fact a good guide to the different causal models available for the Einstein-Podolsky-Rosen correlations, which in turn shed light on the nature of quantum propensities.

My earlier attempt to develop a trope nominalist account of the relation between tropes and causal processes. In accordance with weak dispositional essentialism (Hendry & Rowbottom 2009), I remain uncommitted to full-blown necessity of causal functional laws. Instead, the existence of tropes falling under a determinable and certain kind of causal processes guarantee that corresponding functional laws do not have falsifying instances.

In an influential article published in 1982, Bas Van Fraassen developed an argument against causal realism on the basis of an analysis of the Einstein-Podolsky-Rosen correlations of quantum mechanics. Several philosophers of science and experts in causal inference -including some causal realists like Wesley Salmon- have accepted Van Fraassen’s argument, interpreting it as a proof that the quantum correlations cannot be given any causal model. In this paper I argue that Van Fraassen’s article can also be (...) interpreted as a good guide to the different causal models available for the EPR correlations, and their relative virtues. These models in turn give us insight into some of the unusual features that quantum propensicies might have. (shrink)

Scepticism concerning the idea of causation being linked to contingent chance-raising is articulated in Beebee’s challenging chapter. She suggests that none of these approaches will avoid the consequence that spraying defoliant on a weed is a cause of the weed’s subsequent health. We will always be able to abstract away enough of the healthy plant processes so all that’s left is the causal chain involving defoliation and health. In those circumstances, there will be contingent chance-raising. Beebee’s conclusion is (...) that we should reject the idea of contingent chance-raising and just accept that all causation involves chance-raising. This involves the reclassification of some intuitive cases of causation as causal processes without causation but rather hindering (a distinctive kind of process). It seems clear from this discussion and from the brief earlier remarks about the status of prevention that the classification of types of causal processes and the characterization of their link to causation are matters of some importance. (shrink)

In this paper, we attempt to explicate Salmon’s idea of a causal process, as defined in terms of the mark method, in the context of C*-dynamical systems. We prove two propositions, one establishing mark manifestation infinitely many times along a given interval of the process, and, a second one, which establishes continuous manifestation of mark with the exception of a countable number of isolated points. Furthermore, we discuss how these results can be implemented in the context of the Haag–Araki (...) theories of relativistic quantum fields on Minkowski spacetime. (shrink)

In a well-cited book chapter, ecologist Jared Diamond characterizes three main types of experiment performed in community ecology: laboratory experiment, field experiment, and natural experiment. Diamond argues that each form of experiment has strengths and weaknesses, with respect to, for example, realism or the ability to follow a causal trajectory. But does Diamond’s typology exhaust the available kinds of cause-finding practices? Some social scientists have characterized something they call “causal process tracing.” Is this a fourth type of experiment (...) or something else? I examine Diamond’s typology and causal process tracing in the context of a case study concerning the dynamics of wolf and deer populations on the Kaibab Plateau in the 1920s, a case that has been used as a canonical example of a trophic cascade by ecologists but which has also been subject to controversy. I argue that ecologists have profitably deployed causal process tracing together with other types of experiment to help settle questions of causality in this case. It remains to be seen how widespread the use of causal process tracing outside of the social sciences is (or could be), but there are some potentially promising applications, particularly with respect to questions about specific causal sequences. (shrink)

Wesley Salmon has developed a theory of causation which makes use of the concepts of a "causal process" and a "causal interaction." Roughly, a causal process is a process which transmits its own structure, and a causal interaction is an intersection of processes which transforms the character of these processes. The cause-effect relation is analyzed as a causal interaction followed by a causal process which terminates in a further causal interaction. In (...) this paper I present a series of problem cases which run "counter" to Salmon's account. (shrink)

Wesley Salmon in Scientific Explanation and the Causal Structure of the World offers a causal account of scientific explanation (Salmon 1984). In this context he develops an analysis of the concept of causality, an analysis which marks a break with various contemporary theories of causation including the Neo-Humean view, counterfactual theory, and manipulability theory. His analysis also goes well beyond the confines of probabilistic accounts such as that developed by Suppes. Salmon attempts to explicate the concept of causation (...) by way of two concepts, that of a causal process and that of a causal interaction, which, he defines with some precision and care. In this paper I will present a series of problem cases which run contrary to Salmon’s line of analysis.In developing his analysis of causation Salmon makes use of the notion of a “process”. (shrink)

This paper examines a promising probabilistic theory of singular causation developed by David Lewis. I argue that Lewis' theory must be made more sophisticated to deal with certain counterexamples involving pre-emption. These counterexamples appear to show that in the usual case singular causation requires an unbroken causal process to link cause with effect. I propose a new probabilistic account of singular causation, within the framework developed by Lewis, which captures this intuition.

The neutral and nearly neutral theories of molecular evolution are sometimes characterized as theories about drift alone, where drift is described solely as an outcome, rather than a process. We argue, however, that both selection and drift, as causal processes, are integral parts of both theories. However, the nearly neutral theory explicitly recognizes alleles and/or molecular substitutions that, while engaging in weakly selected causal processes, exhibit outcomes thought to be characteristic of random drift. A narrow focus (...) on outcomes obscures the significant role of weakly selected causal processes in the nearly neutral theory. (shrink)

Causal claims in physics may have two familiar kinds of support: theoretical and experimental. This paper claims that a rigorous mathematical derivation in a realistic model is necessary, though not sufficient, for full theoretical support. The support is not provided by the derivation itself; but rather it comes from a detailed back-tracing through the derivation, matching the mathematical dependencies, point by point, with details of the causal story. This back-tracing is not enough to pick out the correct (...) class='Hi'>causal story, however; a good deal of background causal knowledge is required as well. These claims are illustrated by a detailed example of what causes the Lamb dip in gas lasers. (shrink)

Presentism is the view that only present temporal entities (tenselessly) exist. A widely-discussed problem for presentism concerns causation and, more specifically, the supposed cross-temporally relational character of it. I think that the best reply to this problem can already be found in the literature on temporal ontology: it consists, roughly, in showing that (at least) some of the main approaches to causation can be rephrased so as to avoid commitment to any cross-temporal relation, including the causal relation itself. The (...) main purpose of this paper is to extend this reply to the process view, an approach to causation that has not been considered within this debate until now. I shall do this by taking into account Dowe’s conserved quantity theory—a recent and prominent theory of this sort— and employing it as a proxy for the other major process theories of causation. In dealing with Dowe’s process theory of causation, however, two additional problems must be faced: one concerns the four-dimensional spacetime framework on which its formulation relies; the other concerns the very notion of causal process (and the companion notion of causal interaction). While the presentistic account of Dowe’s theory (and, virtually, of the process view of causation in general) put forth in this paper is intended primarily as a contribution to the mentioned paraphrase- based enterprise of reconciliation between presentism and causation, I shall also offer some reasons for presentists to prefer the process view of causation to the other views of causation that have already been reconciled with presentism. (shrink)

According to the theory developed here, we may trace out the processes emanating from a cause in such a way that any consequence lying along one of these processes counts as an effect of the cause. This theory gives intuitive verdicts in a diverse range of problem cases from the literature. Its claims about causation will never be retracted when we include additional variables in our model. And it validates some plausible principles about causation, including Sartorio's ‘Causes as (...) Difference Makers’ principle and Hitchcock's ‘Principle of Sufficient Reason’. (shrink)

Jazmín Cevasco y Paul van den Broek Universidad de Buenos Aires y Consejo Nacional de Investigaciones Científicas … Read More →.

There is a widespread belief that the so-called process theories of causation developed by Wesley Salmon and Phil Dowe have given us an original account of what causation really is. In this paper, I show that this is a misconception. The notion of “causal process” does not offer us a new ontological account of causation. I make this argument by explicating the implicit ontological commitments in Salmon and Dowe’s theories. From this, it is clear that Salmon’s Mark Transmission Theory (...) collapses to a counterfactual theory of causation, while the Conserved Quantity Theory collapses to David Fair’s phsyicalist reduction of causation. (shrink)

I argue that Binkley et al. use causal process tracing in conjunction with a natural trajectory experiment and two natural snapshot experiments in their re-examination of the Kaibab. This shows that Aldo Leopold may have been right about trophic cascade in the Kaibab in the 1920s, i.e., that there are good reasons to think that a loss of predators led to a deer irruption which decreased aspen recruitment. Using the different cause-finding practices in combination can strengthen causal inferences (...) and mitigate the shortcomings that each practice has. (shrink)

The principle that causes always render their effects more likely is fundamental to the enterprise of reducing facts of causation to facts about (objective) chances. This reductionist enterprise faces famous difficulties in accommodating common-sense intuitions about causal processes, if it insists on cashing out causal processes in terms of streams of events in which every event that belongs to the stream is a cause of the adjoining event downstream of it. I shall propose modifications to this (...) way of cashing out causal processes, still well within the reductionist faith. These modifications will allow the reductionist to handle processes successfully, on the assumption that the reductionist proposal is itself otherwise satisfactory. I shall then argue that the reductionist enterprise lies squarely behind the Theory of Relativity, and so has all the confirmatory weight of Relativity behind it. However this is not all good news for reductionists. For throughout I shall simply assume that the reductionist proposal, to the effect that causes are just chance-raisers, is correct. And I shall sidestep problems with that proposal as such. And so I shall show that, if in the end we find the reductionist proposal unsatisfactory, it cannot be on grounds of its treatment of causal processes as such. Thus, while I shall argue that causal processes pose no extra trouble for reductionists, I shall be making a case that all the action between reductionists and their opponents should be focused upon the proposal to reduce the two-term causal relation itself to relations amongst probabilities. (shrink)

In this paper I question Devitt and Sterelny’s proposal that reference borrowing is a causal process and that the reference borrower is ignorant about the referent.I argue that borrowers need to have some true beliefs about the referent. If so, reference borrowing involves a causal chain of communication together with some associated description. The conclusion is that what is needed for reference borrowing of other kind terms is also needed for the natural kind terms. There is no need (...) to posit a difference between the two. Thus as you cannot refer to daggers by ‘dagger’ unless you realize that they are edged weapons, so you cannot refer to tigers by ‘tiger’ unless you realize that they are animals of a certain sort. The theory of reference borrowing that we need here in both cases seems to be descriptive-causal and not only causal.If the traditional views of borrowing have demanded too much of individual speakers, the causal picture surely demands too little. Mere causal connection with some antecedent tradition of name use does not suffice for the preservation of reference. (shrink)

In this paper I question Devitt and Sterelny’s proposal that reference borrowing is a causal process and that the reference borrower is ignorant about the referent.I argue that borrowers need to have some true beliefs about the referent. If so, reference borrowing involves a causal chain of communication together with some associated description. The conclusion is that what is needed for reference borrowing of other kind terms is also needed for the natural kind terms. There is no need (...) to posit a difference between the two. Thus as you cannot refer to daggers by ‘dagger’ unless you realize that they are edged weapons, so you cannot refer to tigers by ‘tiger’ unless you realize that they are animals of a certain sort. The theory of reference borrowing that we need here in both cases seems to be descriptive-causal and not only causal.If the traditional views of borrowing have demanded too much of individual speakers, the causal picture surely demands too little. Mere causal connection with some antecedent tradition of name use does not suffice for the preservation of reference. (shrink)

Recent discussions in the philosophy of biology have brought into question some fundamental assumptions regarding evolutionary processes, natural selection in particular. Some authors argue that natural selection is nothing but a population-level, statistical consequence of lower-level events (Matthen and Ariew [2002]; Walsh et al. [2002]). On this view, natural selection itself does not involve forces. Other authors reject this purely statistical, population-level account for an individual-level, causal account of natural selection (Bouchard and Rosenberg [2004]). I argue that each (...) of these positions is right in one way, but wrong in another; natural selection indeed takes place at the level of populations, but it is a causal process nonetheless. (shrink)

In biomedical measurement, biomarkers are used to achieve reliable prediction of, and useful causal information about patient outcomes while minimizing complexity of measurement, resources, and invasiveness. A biomarker is an assayable metric that discloses the status of a biological process of interest, be it normative, pathophysiological, or in response to intervention. The greatest utility from biomarkers comes from their ability to help clinicians (and researchers) make and evaluate clinical decisions. In this paper we discuss a specific methodological use of (...) clinical biomarkers in pharmacological measurement: Some biomarkers, called ‘surrogate markers’, are used to substitute for a clinically meaningful endpoint corresponding to events and their penultimate risk factors. We confront the reliability of clinical biomarkers that are used to gather information about clinically meaningful endpoints. Our aim is to present a systematic methodology for assessing the reliability of multiple surrogate markers (and biomarkers in general). To do this we draw upon the robustness analysis literature in the philosophy of science and the empirical use of clinical biomarkers. After introducing robustness analysis we present two problems with biomarkers in relation to reliability. Next, we propose an intervention-based robustness methodology for organizing the reliability of biomarkers in general. We propose three relevant conditions for a robust methodology for biomarkers: (R1) Intervention-based demonstration of partial independence of modes: In biomarkers partial independence can be demonstrated through exogenous interventions that modify a process some number of “steps” removed from each of the markers. (R2) Comparison of diverging and converging results across biomarkers: By systematically comparing partially-independent biomarkers we can track under what conditions markers fail to converge in results, and under which conditions they successfully converge. (R3) Information within the context of theory: Through a systematic cross-comparison of the markers we can make causal conclusions as well as eliminate competing theories. We apply our robust methodology to currently developing Alzheimer’s research to show its usefulness for making causal conclusions. (shrink)

Dowe (1996) argues that the success of the backward causation hypothesis in quantum mechanics would provide strong support for a version of Reichenbach's account of the direction of causal processes, which takes the direction of causation to rest on the fork asymmetry. He also criticises my perspectival account of the direction of causation, which takes causal asymmetry to be a projection of our own temporal asymmetry as agents. In this reply I take issue with Dowe's argument at (...) three main points: his claim that the backward causation hypothesis in QM is incompatible with my perspectival approach to the direction of causation; his defence of the fork asymmetry approach against a general criticism of mine based on the time-symmetry of microphysics; and his application of his preferred account of the direction of causal processes to the relevant cases in QM. (shrink)

This paper considers how 5- to 11-year-olds’ verbal reasoning about the causality underlying extended, dynamic natural processes links to various facets of their statistical thinking. Such continuous processes typically do not provide perceptually distinct causes and effect, and previous work suggests that spatial–temporal analysis, the ability to analyze spatial configurations that change over time, is a crucial predictor of reasoning about causal mechanism in such situations. Work in the Humean tradition to causality has long emphasized on the (...) importance of statistical thinking for inferring causal links between distinct cause and effect events, but here we assess whether this is also viable for causal thinking about continuous processes. Controlling for verbal and non-verbal ability, two studies administered a battery of covariation, probability, spatial–temporal, and causal measures. Results indicated that spatial–temporal analysis was the best predictor of causal thinking across both studies, but statistical thinking supported and informed spatial–temporal analysis: covariation assessment potentially assists with the identification of variables, while simple probability judgment potentially assists with thinking about unseen mechanisms. We conclude that the ability to find out patterns in data is even more widely important for causal analysis than commonly assumed, from childhood, having a role to play not just when causally linking already distinct events but also when analyzing the causal process underlying extended dynamic events without perceptually distinct components. (shrink)

argues that the success of the backward causation hypothesis in quantum mechanics would provide strong support for a version of Reichenbach's account of the direction of causal processes, which takes the direction of causation to rest on the fork asymmetry. He also criticises my perspectival account of the direction of causation, which takes causal asymmetry to be a projection of our own temporal asymmetry as agents. In this reply I take issue with Dowe's argument at three main (...) points: his claim that the backward causation hypothesis in QM is incompatible with my perspectival approach to the direction of causation; his defence of the fork asymmetry approach against a general criticism of mine based on the time-symmetry of microphysics; and his application of his preferred account of the direction of causal processes to the relevant cases in QM. (shrink)

Minimally, causal realism (as understood here) is the view that accounts of causation in terms of mere, regular or probabilistic conjunction are unsatisfactory, and that causal phenomena are correctly associated with some form of de re necessity. Classic arguments, however, some of which date back to Sextus Empiricus and have appeared many times since, including famously in Russell, suggest that the very notion of causal realism is incoherent. In this paper I argue that if such objections seem (...) compelling, it is only because everyday expressions concerning causal phenomena are misleading with respect to certain metaphysical details. These expressions generally make reference to the relations of events or states of affairs, but ignore or obscure the role played by causal properties. I argue that on a proposed alternative, an analysis in terms of causal processes, more refined descriptions of causal phenomena escape the charge of incoherence. Causal necessity is here located in the relations of causal properties. I distinguish this view from the recent process theories of Salmon and Dowe, which are disinterested in causal realism. (shrink)

In this paper we provide a general account of the causal models which attempt to provide a solution to the famous measurement problem of Quantum Mechanics. We will argue that—leaving aside instrumentalism which restricts the physical meaning of QM to the algorithmic prediction of measurement outcomes—the many interpretations which can be found in the literature can be distinguished through the way they model the measurement process, either in terms of the efficient cause or in terms of the final cause. (...) We will discuss and analyze why both, ‘final cause’ and ‘efficient cause’ models, face severe difficulties to solve the measurement problem. In contradistinction to these schemes we will present a new model based on the immanent cause which, we will argue, provides an intuitive understanding of the measurement process in QM. (shrink)

Studies exploring how students learn and understand science processes such as diffusion and natural selection typically find that students provide misconceived explanations of how the patterns of such processes arise (such as why giraffes’ necks get longer over generations, or how ink dropped into water appears to “flow”). Instead of explaining the patterns of these processes as emerging from the collective interactions of all the agents (e.g., both the water and the ink molecules), students often explain the (...) pattern as being caused by controlling agents with intentional goals, as well as express a variety of many other misconceived notions. In this article, we provide a hypothesis for what constitutes a misconceived explanation; why misconceived explanations are so prevalent, robust, and resistant to instruction; and offer one approach of how they may be overcome. In particular, we hypothesize that students misunderstand many science processes because they rely on a generalized version of narrative schemas and scripts (referred to here as a Direct-causal Schema) to interpret them. For science processes that are sequential and stage-like, such as cycles of moon, circulation of blood, stages of mitosis, and photosynthesis, a Direct-causal Schema is adequate for correct understanding. However, for science processes that are non-sequential (or emergent), such as diffusion, natural selection, osmosis, and heat flow, using a Direct Schema to understand these processes will lead to robust misconceptions. Instead, a different type of general schema may be required to interpret non-sequential processes, which we refer to as an Emergent-causal Schema. We propose that students lack this Emergent Schema and teaching it to them may help them learn and understand emergent kinds of science processes such as diffusion. Our study found that directly teaching students this Emergent Schema led to increased learning of the process of diffusion. This article presents a fine-grained characterization of each type of Schema, our instructional intervention, the successes we have achieved, and the lessons we have learned. (shrink)

How do we make causal judgments? Many studies have demonstrated that people are capable causal reasoners, achieving success on tasks from reasoning to categorization to interventions. However, less is known about the mental processes used to achieve such sophisticated judgments. We propose a new process model—the mutation sampler—that models causal judgments as based on a sample of possible states of the causal system generated using the Metropolis–Hastings sampling algorithm. Across a diverse array of tasks and (...) conditions encompassing over 1,700 participants, we found that our model provided a consistently closer fit to participant judgments than standard causal graphical models. In particular, we found that the biases introduced by mutation sampling accounted for people's consistent, predictable errors that the normative model by definition could not. Moreover, using a novel experimental methodology, we found that those biases appeared in the samples that participants explicitly judged to be representative of a causal system. We conclude by advocating sampling methods as plausible process‐level accounts of the computations specified by the causal graphical model framework and highlight opportunities for future research to identify not just what reasoners compute when drawing causal inferences, but also how they compute it. (shrink)

Process theories of causality seek to explicate causality as a property of individual causal processes. This paper examines the capacity of such theories to account for the asymmetry of causation. Three types of theories of asymmetry are discussed; the subjective, the temporal, and the physical, the third of these being the preferred approach. Asymmetric features of the world, namely the entropic and Kaon arrows, are considered as possible sources of causal asymmetry and a physical theory of asymmetry (...) is subsequently developed with special reference to the questions of objectivity and backwards causation. (shrink)

Processes produce changes: rivers erode their banks and thunderstorms cause floods. If I am right that organisms are a kind of process, then the causally efficacious behaviours of organisms are also examples of processes producing change. In this paper I shall try to articulate a view of how we should think of causation within a broadly processual ontology of the living world. Specifically, I shall argue that causation, at least in a central class of cases, is the interaction (...) of processes, that such causation is the exercise of a capacity inherent in that process and, negatively, that causation should not be understood as the instantiation of universal laws. The approach I describe has substantial similarities with the process causality articulated by Wesley Salmon and Phil Dowe for physical causation, making it plausible that the basic approach can be applied equally to the non-living world. It is an approach that builds at crucial points on the criticisms of determinism and universal causality famously articulated by Elizabeth Anscombe. (shrink)

If physical reality is nonseparable, as quantum mechanics suggests, then it may contain processes of a quite novel kind. Such nonseparable processes could connect space-like separated events without violating relativity theory or any defensible locality condition. Appeal to nonseparable processes could ground theoretical explanations of such otherwise puzzling phenomena as the two-slit experiment, and EPR- type correlations. We find such phenomena puzzling because they threaten cherished conceptions of how causes operate to produce their effects. But nonseparable (...) class='Hi'>processes offer us an alternative deal of natural order, conformity to which makes such phenomena seem quite normal and not at all unexpected. Attempts to answer the further question, as to whether an appeal to a nonseparable process provides a genuine "causal" explanation, have something to teach us about our concept of causation, but do not threaten to undermine the value of the explanation itself. (shrink)

Background: Causal reasoning as a way to make a diagnosis seems convincing. Modern medicine depends on the search for causes of disease and it seems fair to assert that such knowledge is employed in diagnosis. Causal reasoning as it has been presented neglects to some extent the conception of multifactorial disease causes. Goal: The purpose of this paper is to analyze aspects of causation relevant for discussing causal reasoning in a diagnostic context.

Philosophers of social science have emphasized mechanistic approaches to causal inquiry for some time now, showing why focusing on the mechanisms behind correlations is preferable to focusing on correlations alone (cf. Johnson 2006, Little 1991, Reiss 2007, 2009, Steel 2004, see also King, Keohane, and Verba 1994 for an example of purely correlational research). In Process Tracing: from Metaphor to Analytic Tool, political scientists Andrew Bennett and Jeffrey Checkel present a concrete method for finding evidence of causal mechanisms, (...) process tracing. Process tracers give evidence for causal relations in terms of the observable implications of the underlying causal mechanisms through which a putative cause affects some effect of interest. Such observable implications often take the form of a chain of events, or process, which connects cause and effect. Though Process Tracing contains applications of mechanistic reasoning unfamiliar to philosophers, and as such will be of interest to those working in the mechanist tradition, Bennett and Checkel’s own discussion of the philosophical foundations of process tracing is limited. The reader will have to delve deep into the volume’s contributed chapters for links between the literature on causation and this new method. (shrink)

Judea Pearl (2000) has recently advanced a theory of token causation using his structural equations approach. This paper examines some counterexamples to Pearl's theory, and argues that the theory can be modified in a natural way to overcome them.

This paper examines Wesley Salmon's "process" theory of causality, arguing in particular that there are four areas of inadequacy. These are that the theory is circular, that it is too vague at a crucial point, that statistical forks do not serve their intended purpose, and that Salmon has not adequately demonstrated that the theory avoids Hume's strictures about "hidden powers". A new theory is suggested, based on "conserved quantities", which fulfills Salmon's broad objectives, and which avoids the problems discussed.

Research has shown that it requires less time to process information that is part of an objective causal relation describing states of affairs in the world (She was out of breath because she was running), than information that is part of a subjective relation (She must have been in a hurry because she was running) expressing a claim or conclusion and a supporting argument. Representing subjectivity seems to require extra cognitive operations. In Mental Spaces Theory (MST; Fauconnier, Gilles. 1994. (...) Mental spaces: Aspects of meaning construction in natural language. Cambridge: MIT Press) the difference between these two relation types can be described in terms of an extra mental space in the discourse representation of subjective relations: representing the Subject of Consciousness (SoC). In processing terms, this might imply that the processing difference is not present if this SoC has already been established in the discourse. We tested this prediction in two eye tracking experiments. The results of Experiment 1 showed that signaling the subjectivity of the relation by introducing a subject of consciousness beforehand did not diminish the processing asymmetry compared to a neutral context. However, the relative complexity of subjective relations was diminished in the context of Free Indirect Speech (No! He was absolutely sure. There was no doubt about it. She was running so she was in hurry; Experiment 2). In terms of MST and the representation of subjectivity in general, this implies that not only creating a representation of a thinking subject, but also assigning a claim to this thinking subject requires extra processing effort. (shrink)