Hume thought that if you believed in powers, you believed in necessary connections in nature. He was then able to argue that there were none such because anything could follow anything else. But Hume wrong-footed his opponents. A power does not necessitate its manifestations: rather, it disposes towards them in a way that is less than necessary but more than purely contingent. -/- In this paper a dispositional theory of causation is offered. Causes dispose towards their effects and often (...) produce them. But a set of causes, even though they may succeed in producing an effect, cannot necessitate it since the effect could have been counteracted by some additional power. This would require a separation of our concepts of causal production and causal necessitation. The most conspicuous cases of causation are those where powers accumulate and pass a requisite threshold for an effect to occur. -/- We develop a model for representing powers as constituent vectors within an n-dimensional quality space, where composition of causes appears as vector addition. Even our resultant vector, however, has to be understood as having dispositional force only. This model throws new light on causal modality and cases of prevention, causation by absence and probabilistic causation. (shrink)

permits a sound and rigorously definable notion of ‘originating cause’ or causa causans—a type of transition event—of an outcome event. Mackie has famously suggested that causes form a family of ‘inus’ conditions, where an inus condition is ‘an insufficient but non-redundant part of an unnecessary but sufficient condition’. In this essay the needed concepts of BST theory are developed in detail, and it is then proved that the causae causantes of a given outcome event have exactly the structure of a (...) set of Mackie inus conditions. The proof requires the assumption that there is no EPR-like ‘funny business’. This seems enough to constitute a theory of ‘causation’ in at least one of its many senses. Introduction The cement of the universe Preliminaries 3.1 First definitions and postulates 3.2 Ontology: propositions 3.3 Ontology: initial events 3.4 Ontology: outcome events 3.5 Ontology: transition events 3.6 Propositional language applied to events Causae causantes 4.1 Causae causantes are basic primary transition events 4.2 Causae causantes of an outcome chain 4.3 No funny business Causae causantes and inns and inus conditions 5.1 Inns conditions of outcome chains: not quite 5.2 Inns conditions of outcome chains 5.3 Inns conditions of scattered outcome events 5.4 Inus conditions for disjunctive outcome events 5.5 Inns and inus conditions of transition events Counterfactual conditionals Appendix: Tense and modal connectives in BST. (shrink)

Metaphysicians of causation have long debated the existence of primitive causal modalities (e.g., powers), with reductionists and realists taking opposing stances. However, little attention has been given to the legitimacy of the metaphysical question itself, despite our longstanding awareness of Rudolf Carnap’s critique of metaphysics. This article develops a (broadly) Carnapian-pragmatist approach to causation as an alternative to existing metaphysical approaches. Within this pragmatist approach, metaphysical questions about causation are reinterpreted as practical questions about the choice of (...) causal frameworks. To motivate and justify this new approach, I argue that, in emphasizing the priority of ontology over methodology, metaphysical approaches to causation fail to adequately capture the interplay between causal ontology and causal methodology in scientific practice. In contrast, the Carnapian approach provides a more appealing alternative that emphasizes the mutual dependence and “balance” between the two in an ongoing process of scientific inquiry. I use the recent controversy over “What counts as a cause” in statistical causal inference as a case study to demonstrate how the Carnapian approach can help us better understand the role of ontological issues in methodological practices. (shrink)

This Open Access book (see link to Taylor & Francis below) critically examines the recent discussions of powers and powers-based accounts of causation. The author then develops an original view of powers-based causation that aims to be compatible with the theories and findings of natural science. Recently, there has been a dramatic revival of realist approaches to properties and causation, which focus on the relevance of Aristotelian metaphysics and the notion of powers for a scientifically informed view (...) of causation. In this book, R.D. Ingthorsson argues that one central feature of powers-based accounts of causation is arguably incompatible with what is today recognised as fact in the sciences, notably that all interactions are thoroughly reciprocal. Ingthorsson’s powerful particulars view of powers-based causation accommodates for the reciprocity of interactions. It also draws out the consequences of that view for the issue of causal necessity and offers a way to understand the constitution and persistence of compound objects as causal phenomena. Furthermore, Ingthorsson argues that compound entities, so understood, are just as much processes as they are substances. A Powerful Particulars View of Causation will be of great interest to scholars and advanced students working in metaphysics, philosophy of science, and neo-Aristotelian philosophy, while also being accessible for a general audience. (shrink)

It is commonplace to distinguish between propositional justification (having good reasons for believing p) and doxastic justification (believing p on the basis of those good reasons).One necessary requirement for bridging the gap between S’s merely having propositional justification that p and S’s having doxastic justification that p is that S base her belief that p on her reasons (propositional justification).A plausible suggestion for what it takes for S’s belief to be based on her reasons is that her reasons must contribute (...) causally to S’s having that belief. Though this suggestion is plausible, causal accounts of the basing relation that have been proposed have not fared well. In particular, cases involving causal deviancy and cases involving over-determination have posed serious problems for causal accounts of the basing relation. Although previous causal accounts of the basing relation seem to fall before these problems, it is possible to construct an acceptable causal account of the basing relation. That is, it is possible to construct a causal account of the basing relation that not only fits our intuitions about doxastic justification in general, but also is not susceptible to the problems posed by causal deviancy and causal over-determination. The interventionist account of causation provides the tools for constructing such an account. My aim is to make use of the insights of the interventionist account of causation to develop and defend an adequate causal account of the basing relation. (shrink)

The counterfactual analysis of causation has focused on one particular counterfactual conditional, taking as its starting-point the suggestion that C causes E iff (C E). In this paper, some consequences are explored of reversing this counterfactual, and developing an account starting with the idea that C causes E iff (E C). This suggestion is discussed in relation to the problem of pre-emption. It is found that the 'reversed' counterfactual analysis can handle even the most difficult cases of pre-emption with (...) only minimal complications. The paper closes with a discussion of the wider philosophical implications of developing a reversed counterfactual analysis, especially concerning the differentiation of causes from causal conditions, causation by absences, and the extent to which causes suffice for their effects. (shrink)

What is the connection between causation and responsibility? Is there a best way to theorize philosophically about causation? Which factors determine and influence what we judge to be the cause of something? Bringing together interdisciplinary research from experimental philosophy, traditional philosophy and psychology, this collection showcases the most recent developments and approaches to questions about causation. Chapters discuss the diverse theoretical ramifications of empirical findings in experimental philosophy of causation, providing a comprehensive survey of key issues (...) such as the perception and learning of causal relations, omission, normative considerations, mechanism, voluntariness and legal theories of causation. With novel contributions from both experts and rising stars, Advances in Experimental Philosophy of Causation demonstrates the value of empirical work and opens new domains of inquiry at the cutting edge of the field. (shrink)

The basic idea of counterfactual theories of causation is that the meaning of causal claims can be explained in terms of counterfactual conditionals of the form “If A had not occurred, C would not have occurred”. While counterfactual analyses have been given of type-causal concepts, most counterfactual analyses have focused on singular causal or token-causal claims of the form “event c caused event e”. Analyses of token-causation have become popular in the last thirty years, especially since the (...) class='Hi'>development in the 1970's of possible world semantics for counterfactuals. The best known counterfactual analysis of causation is David Lewis's (1973b) theory. However, intense discussion over thirty years has cast doubt on the adequacy of any simple analysis of singular causation in terms of counterfactuals. Recent years have seen a proliferation of different refinements of the basic idea to achieve a closer match with commonsense judgements about causation. (shrink)

The starting point in the development of probabilistic analyses of token causation has usually been the naïve intuition that, in some relevant sense, a cause raises the probability of its effect. But there are well-known examples both of non-probability-raising causation and of probability-raising non-causation. Sophisticated extant probabilistic analyses treat many such cases correctly, but only at the cost of excluding the possibilities of direct non-probability-raising causation, failures of causal transitivity, action-at-a-distance, prevention, and causation by (...) absence and omission. I show that an examination of the structure of these problem cases suggests a different treatment, one which avoids the costs of extant probabilistic analyses. (shrink)

The thesis is defended that the theories of causation, time and space, and levels of reality are mutually interrelated in such a way that the difficulties internal to theories of causation and to theories of space and time can be understood better, and perhaps dealt with, in the categorial context furnished by the theory of the levels of reality. The structural condition for this development to be possible is that the first two theories be opportunely generalized.

This critical notice highlights the important contributions that Eric Watkins's writings have made to our understanding of theories about causation developed in eighteenth-century German philosophy and by Kant in particular. Watkins provides a convincing argument that central to Kant's theory of causation is the notion of a real ground or causal power that is non-Humean (since it doesn't reduce to regularities or counterfactual dependencies among events or states) and non-Leibnizean because it doesn't reduce to logical or conceptual relations. (...) However, we raise questions about Watkins's more specific claims that Kant completely rejects a model on which the first relatum of a phenomenal causal relation is an event and that he maintains that real grounds are metaphysically and not just epistemically indeterminate. -/- . (shrink)

In an attempt to improve upon Alexander Pruss’s work (The principle of sufficient reason: A reassessment, pp. 240–248, 2006), I (Weaver, Synthese 184(3):299–317, 2012) have argued that if all purely contingent events could be caused and something like a Lewisian analysis of causation is true (per, Lewis’s, Causation as influence, reprinted in: Collins, Hall and paul. Causation and counterfactuals, 2004), then all purely contingent events have causes. I dubbed the derivation of the universality of causation the (...) “Lewisian argument”. The Lewisian argument assumed not a few controversial metaphysical theses, particularly essentialism, an incommunicable-property view of essences (per Plantinga’s, Actualism and possible worlds, reprinted in: Davidson (ed.) Essays in the metaphysics of modality, 2003), and the idea that counterfactual dependence is necessary for causation. There are, of course, substantial objections to such theses. While I think a fight against objections to the Lewisian argument can be won, I develop, in what follows, a much more intuitive argument for the universality of causation which takes as its inspiration a result from Frederic B. Fitch’s work (J Symb Logic 28(2):135–142, 1963) [with credit to who we now know was Alonzo church’s, Referee Reports on Fitch’s Definition of value, in: (Salerno (ed.), New essays on the knowability paradox, 2009)] that if all truths are such that they are knowable, then (counter-intuitively) all truths are known. The resulting Church–Fitch proof for the universality of causation is preferable to the Lewisian argument since it rests upon far weaker formal and metaphysical assumptions than those of the Lewisian argument. (shrink)

The Logic of Causation: Definition, Induction and Deduction of Deterministic Causality is a treatise of formal logic and of aetiology. It is an original and wide-ranging investigation of the definition of causation (deterministic causality) in all its forms, and of the deduction and induction of such forms. The work was carried out in three phases over a dozen years (1998-2010), each phase introducing more sophisticated methods than the previous to solve outstanding problems. This study was intended as part (...) of a larger work on causal logic, which additionally treats volition and allied cause-effect relations (2004). The Logic of Causation deals with the main technicalities relating to reasoning about causation. Once all the deductive characteristics of causation in all its forms have been treated, and we have gained an understanding as to how it is induced, we are able to discuss more intelligently its epistemological and ontological status. In this context, past theories of causation are reviewed and evaluated (although some of the issues involved here can only be fully dealt with in a larger perspective, taking volition and other aspects of causality into consideration, as done in Volition and Allied Causal Concepts). Phase I: Macroanalysis. Starting with the paradigm of causation, its most obvious and strongest form, we can by abstraction of its defining components distinguish four genera of causation, or generic determinations, namely: complete, partial, necessary and contingent causation. When these genera and their negations are combined together in every which way, and tested for consistency, it is found that only four species of causation, or specific determinations, remain conceivable. The concept of causation thus gives rise to a number of positive and negative propositional forms, which can be studied in detail with relative ease because they are compounds of conjunctive and conditional propositions whose properties are already well known to logicians. The logical relations (oppositions) between the various determinations (and their negations) are investigated, as well as their respective implications (eductions). Thereafter, their interactions (in syllogistic reasoning) are treated in the most rigorous manner. The main question we try to answer here is: is (or when is) the cause of a cause of something itself a cause of that thing, and if so to what degree? The figures and moods of positive causative syllogism are listed exhaustively; and the resulting arguments validated or invalidated, as the case may be. In this context, a general and sure method of evaluation called ‘matricial analysis’ (macroanalysis) is introduced. Because this (initial) method is cumbersome, it is used as little as possible – the remaining cases being evaluated by means of reduction. Phase II: Microanalysis. Seeing various difficulties encountered in the first phase, and the fact that some issues were left unresolved in it, a more precise method is developed in the second phase, capable of systematically answering most outstanding questions. This improved matricial analysis (microanalysis) is based on tabular prediction of all logically conceivable combinations and permutations of conjunctions between two or more items and their negations (grand matrices). Each such possible combination is called a ‘modus’ and is assigned a permanent number within the framework concerned (for 2, 3, or more items). This allows us to identify each distinct (causative or other, positive or negative) propositional form with a number of alternative moduses. This technique greatly facilitates all work with causative and related forms, allowing us to systematically consider their eductions, oppositions, and syllogistic combinations. In fact, it constitutes a most radical approach not only to causative propositions and their derivatives, but perhaps more importantly to their constituent conditional propositions. Moreover, it is not limited to logical conditioning and causation, but is equally applicable to other modes of modality, including extensional, natural, temporal and spatial conditioning and causation. From the results obtained, we are able to settle with formal certainty most of the historically controversial issues relating to causation. Phase III: Software Assisted Analysis. The approach in the second phase was very ‘manual’ and time consuming; the third phase is intended to ‘mechanize’ much of the work involved by means of spreadsheets (to begin with). This increases reliability of calculations (though no errors were found, in fact) – but also allows for a wider scope. Indeed, we are now able to produce a larger, 4-item grand matrix, and on its basis find the moduses of causative and other forms needed to investigate 4-item syllogism. As well, now each modus can be interpreted with greater precision and causation can be more precisely defined and treated. In this latest phase, the research is brought to a successful finish! Its main ambition, to obtain a complete and reliable listing of all 3-item and 4-item causative syllogisms, being truly fulfilled. This was made technically feasible, in spite of limitations in computer software and hardware, by cutting up problems into smaller pieces. For every mood of the syllogism, it was thus possible to scan for conclusions ‘mechanically’ (using spreadsheets), testing all forms of causative and preventive conclusions. Until now, this job could only be done ‘manually’, and therefore not exhaustively and with certainty. It took over 72’000 pages of spreadsheets to generate the sought for conclusions. This is a historic breakthrough for causal logic and logic in general. Of course, not all conceivable issues are resolved. There is still some work that needs doing, notably with regard to 5-item causative syllogism. But what has been achieved solves the core problem. The method for the resolution of all outstanding issues has definitely now been found and proven. The only obstacle to solving most of them is the amount of labor needed to produce the remaining (less important) tables. As for 5-item syllogism, bigger computer resources are also needed. (shrink)

This book presents a history of the volitional theory of causation—the philosophical proposal that volition, or will, of the same or broadly the same stamp as that which we experience in our own deliberate and voluntary doings, should be taken as the basis for all causality. Few today know much about the volitional theory of causation, and even fewer have given it any serious attention. But if current opinion regards this suggestion as an unusual one, of minor importance, (...) the historical record shows otherwise, revealing that it is a theory which has been proposed and developed again and again throughout the modern era. Its obscurity is only a recent phenomenon. Starting at the beginning of the Early Modern period and progressing right up modern times, the historical discussion takes in both supporters and critics, as well as both famous and less well-known figures, to tell the story of a long-running debate which contemporary history of philosophy has forgotten. (shrink)

This article presents Roman Ingarden’s theory of causation, as developed in volume III of The Controversy about the Existence of the World, and defends analternative which uses some important insights of Ingarden. It rejects Ingarden’s claim that a cause is simultaneous with its effect and that a cause necessitates its effect. It uses Ingarden’s notion of ‘inclinations’ and accepts Ingarden’s claim that an event cannot necessitate a later event.

In The Secret Connexion1 Galen Strawson argues against the traditional interpretation of Hume, according to which Hume’s theory of meaning leads him to a regularity theory of causation. In actual fact, says Strawson, ‘Hume believes firmly in some sort of natural necessity’ (p. 277). What Hume denied was that we are aware of causal connections outrunning regular succession, and that we have a ‘positively or descriptively contentful conception’ of such powers (p. 283); he did not deny that there are (...) such powers, or that they are what we are talking about when we talk about causation. Strawson has four central lines of argument. His ‘most direct evidence’ (p. 2) against a regularity interpretation consists of (1) passages where Hume refers to hidden powers underlying the regularities of which we are aware. Strawson’s broader motivations for rejecting the traditional interpretation are (2) that the regularity theory is in itself quite absurd, and (3) that it is incompatible with Hume’s ‘non-committal scepticism’. And the method which he uses to defend his interpretation against pressure from the theory of ideas is (4) to develop some comments of Hume’s on ‘relative’ ideas into something like a further theory of content to supplement the theory of ideas. Strawson develops almost the strongest case I can imagine for his claims. I shall try to explain why he leaves me unconvinced. (shrink)

Causation is important when considering how an organism maintains health, why disease arises in a healthy person, and how one may intervene to change the course of a disease. This paper explores the form of causative relationships in health, disease and intervention, with particular regard to the pathological and biopsychosocial models. Consistent with the philosophical view of dispositionalism, we believe that objects are the fundamental relata of causation. By accepting the broad scope of the biopsychosocial model, we argue (...) that psychological and social constructs be considered objects. We think that this 'biopsychosocial dispositionalism' offers the flexibility required to describe causation throughout health, disease and intervention pathways. When constructing mechanistic chains to describe causative pathways, we argue that an object will causally connect with others through actions; transfers of energy from one object to another, initiated by the manifestation of one or more dispositional property. Finally, our analysis of causative interactions utilises the concept that a common form of interaction exists between disease and intervention pathways. This common form will always be an object, but the mode of interaction will vary with each disease. We describe how intervention may act through objects being shared between converging mechanistic chains, or through the removal and/or insertion of objects in such chains. We believe that this analysis provides novel insight to the forms of causative transactions that can occur. In addition, we hope that the findings of this analysis represent the first step towards developing a framework for appraising the composition of mechanistic theories. (shrink)

In this book, J. L. Mackie makes a careful study of several philosophical issues involved in his account of causation. Mackie follows Hume's distinction between causation as a concept and causation as it is ‘in the objects’ and attempts to provide an account of both aspects. Mackie examines the treatment of causation by philosophers such as Hume, Kant, Mill, Russell, Ducasse, Kneale, Hart and Honore, and von Wright. Mackie's own account involves an analysis of causal statements (...) in terms of counterfactual conditionals though these are judged to be incapable of giving a complete account of causation. Mackie argues that regularity theory too can only offer an incomplete picture of the nature of causation. In the course of his analysis, Mackie critically examines the account of causation offered by Kant, as well as the contemporary Kantian approaches offered by philosophers such as Bennett and Strawson. Also addressed are issues such as the direction of causation, the relation of statistical laws and functional laws, the role of causal statements in legal contexts, and the understanding of causes both as ‘facts’ and ‘events’. Throughout the discussion of these topics, Mackie develops his own complex account of the nature of causation, finally bringing his analysis to bear in regard to the topic of teleology and the question of whether final causes can be justifiably reduced to efficient causes. (shrink)

A regularity theory of causation analyses type-level causation in terms of Boolean difference-making. The essential ingredient that helps this theoretical framework overcome the problems of Hume’s and Mill’s classical accounts is a principle of non-redundancy: only Boolean dependency structures from which no elements can be eliminated track causation. The first part of this paper argues that the recent regularity theoretic literature has not consistently implemented this principle, for it disregarded an important type of redundancies: structural redundancies. Moreover, (...) it is shown that a regularity theory needs to be underwritten by a hitherto neglected metaphysical background assumption stipulating that the world's causal makeup is not ambiguous. Against that background, the second part then develops a new regularity theory that does justice to all types of redundancies and, thereby, provides the first all-inclusive notion of Boolean difference-making. (shrink)

Causation is important when considering: how an organism maintains health; why disease arises in a healthy person; and, how one may intervene to change the course of a disease. This paper explores the form of causative relationships in health, disease and intervention, with particular regard to the pathological and biopsychosocial models. Consistent with the philosophical view of dispositionalism, we believe that objects are the fundamental relata of causation. By accepting the broad scope of the biopsychosocial model, we argue (...) that psychological and social constructs be considered objects. We think that this ‘biopsychosocial dispositionalism’ offers the flexibility required to describe causation throughout health, disease and intervention pathways. When constructing mechanistic chains to describe causative pathways, we argue that an object will causally connect with others through actions; transfers of energy from one object to another, initiated by the manifestation of one or more dispositional property. Finally, our analysis of causative interactions utilises the concept that a common form of interaction exists between disease and intervention pathways. This common form will always be an object, but the mode of interaction will vary with each disease. We describe how intervention may act through objects being shared between converging mechanistic chains, or through the removal and/or insertion of objects in such chains. We believe that this analysis provides novel insight to the forms of causative transactions that can occur. In addition, we hope that the findings of this analysis represent the first step towards developing a framework for appraising the composition of mechanistic theories. (shrink)

Peter Menzies has developed a novel version of the exclusion principle that he claims to be compatible with the possibility of mental causation. Menzies proposes to frame the exclusion principle in terms of a difference-making account of causation, understood in counterfactual terms. His new exclusion principle appears in two formulations: upwards exclusion — which is the familiar case in which a realizing event causally excludes the event that it realizes — and, more interestingly, downward exclusion, in which an (...) event causally excludes its realizer. This paper shows that one consequence of Menzies’s proposed solution to the problem of mental causation is a ubiquitous violation of the principle of closure — a fact that forces him into a trilemma to which we see no satisfactory response. (shrink)

Before the story can be told, however, some stage-setting is necessary. First, it is important to be clear about the most basic doctrines of Pre-established Harmony, Occasionalism, and Physical Influx. Physical Influx asserts intersubstantial causation amongst finite substances. For instance, when I appear to kick a ball, I really am the cause of the ball's motion. Pre-established Harmony denies intersubstantial causation, but affirms intrasubstantial causation. According to Pre-established Harmony, then, I am not the cause of the ball's (...) motion, but rather the ball is simply causing itself to move. Occasionalism, like Pre-established Harmony, denies intersubstantial causation, but, unlike Pre-established Harmony, it denies intrasubstantial causation as well. Occasionalism typically asserts that God alone, that is, an infinite substance, is the cause of all changes, and thus of the ball's motion. Though there are other ways of defining these causal theories, this way has the advantage that the causal theories form an exhaustive disjunction. (shrink)

Leading philosophers & psychologists offer an assessment of the commonsense view that perceptual experience is an immediate awareness of mind-independent objects. They examine the nature of perception, its role in the acquisition of knowledge, the role of causation in perception, & how perceptual understanding develops in humans.

Tort law depends on three key concepts: causation, responsibility, and fault. However, I argue that the three key concepts are neither necessary, nor sufficient, for tort.

A regularity theory of causation analyses type-level causation in terms of Boolean difference-making. The essential ingredient that helps this theoretical framework overcome the problems of Hume’s and Mill’s classical accounts is a principle of non-redundancy: only Boolean dependency structures from which no elements can be eliminated track causation. The first part of this article argues that the recent regularity-theoretic literature has not consistently implemented this principle, for it disregarded an important type of redundancies: structural redundancies. Moreover, it (...) is shown that a regularity theory needs to be underwritten by a hitherto neglected metaphysical background assumption stipulating that the world’s causal makeup is not ambiguous. Against that background, the second part then develops a new regularity theory that does justice to all types of redundancies and, thereby, provides the first all-inclusive notion of Boolean difference-making. (shrink)

The experimental and highly regarded Gifford Lectures at Edinburgh University was endowed in the late nineteenth century. Over the years, participants have including many leading representatives of religion, science, and philosophy. This series has as its subject, The Development of Mind. First published in 1972, the series continues to attract widespread interest. In this volume, contributors argue about the mind from diverse analytical standpoints. The focus of the series remains the relationship between religion, science, and philosophy. This volume attempts (...) to achieve a comprehensive view of the subject of mind. The mental development of children in the light of modern psychology is discussed, and the distinction between "how" and "why" questions is put forward with clarity. The development of mind is further contrasted with the evolution of embryos in the young. The mind is considered as a capacity for intellectual activity, and as a multi-purpose program. Goal-directed behavior and language development are given importance, and issues of cosmic purpose, and the how and why of evolution, are never far from the surface of the argument. The lecturers know their opposition and their positions, and the cut-and-thrust of the discussion has much acumen and wit to it. Issues ranging from the impact of mind on theories of religion, causation, and rational will are examined in an informal, yet compelling, manner. (shrink)

Philosophers of dispositionalism deny the Humean account of causality in terms of constant conjunction, contiguity, temporal priority and contingency. And some of them go further to explain the causal relation not between events or objects, but between properties, in terms of reciprocity, simultaneity, ubiquity, intentionality and holism. But their exposition seems to remain fragmented even though they try to make use of the notions of intentionality and holim. I would inquire reasons why it is piecemeal, by analysing that they employ (...) these notions of intentionality and holism which has been constructed within the dualistic tradition. If one wants to make the dispositional project to be powerful, she should look for the notions of intentionality and holism which are free of the dualistic traits and which have been constructed in the tradition of naturalism. The concept of integrationality may be a candidate for such a task. This conception was coined in Zhongyong under the name of cheng in that there is nothing without cheng, and was developed in one of Sok-hon Ham's thesis that any entity in the world contains "seed" within it to realize objectives shared by all involved. Humans are privileged and distinct from other natures, but there is continuity between them. An example of the integrationality applied to the case of humans would be that realization of myself and realizations of all others are one and not two. I would summarize notions of "cheng" and "seed" by offering the integrationality thesis, that is, that there is integrationality in any entity like dispositions such that the integrationality is a power to realize the embedded objective of it in the context where it interacts with all others. I would claim that integrationality is a metaphysical basis for a way of human thinking, by arguing that it is a necessary part of presupposition for our understanding of the world without which the world is not intelligible. The notion of integrationality is involved empirically as well as conceptually. Empirically, there are numerous reports from physics, biology and information science, which provide with a clue which may well be taken to show the ubiquitous integrationality. But some conceptual considerations would also indicate necessarily toward the same direction. These would include various theses of ontology, epistemology, disposition, modality, naturalized intentionality, fitting and topics like these. I will investigate some of them in order to justify my claim of this paper. (shrink)

This article attempts to develop the abandoned occasionalist model of causation into a credible present-day theory. If objects can never exhaust one another through their relations, it is hard to know how they can ever interact at all. This article handles the problem by dividing objects into two kinds: the real objects that emerge from Heidegger’s tool-analysis and the intentional objects of Husserl’s phenomenology. Each of these objects turns out to be split by an additional rift between the object (...) as an enduring unit and its plurality of traits. This explains Heidegger’s notorious ‘fourfold’ model of the thing. This article shows that Heidegger’s Geviert must be reinterpreted as a system of four tensions that can be identified as time, space, essence, and eidos. Time and space can no longer be left as peerless dimensions of the cosmos. Instead, they are shown to arise from the tensions between things and their qualities. And for this reason they are joined by essence (in the classical sense of the term) and eidos (in Husserl’s sense, not Plato’s) as two out of four basic features of the fabric of the world. (shrink)

Appraisal theories of emotion generally presuppose that emotions are “directed at” various items. They also hold that emotions have motivational properties. However, although it coheres well with their views, they have yet to seriously develop the idea that the function of emotional direction is to guide those properties. I argue that this “guidance hypothesis” can open up a promising new field of research in emotion theory. But I also argue that before appraisal theorists can take full advantage of it, they (...) must drop their further assumption that to determine an emotion's direction, one need only retrace the process that caused it. Contrary to this “retracing view,” I argue for an “independence thesis”: directed emotions are produced by two functionally independent sub-processes. The first, “affect-causation,” functions in part to produce a state with certain motivational properties given certain representations. The second, “affect-direction,” has the function of optimally guiding those motivational properties by associating them with representations that may properly be quite dissimilar from the causal ones. By provisionally adopting the independence thesis and empirically testing the guidance hypothesis, I argue that appraisal theorists stand a good chance of significantly increasing the explanatory power of their theories. (shrink)

Underdevelopment is the most controversial issue of our time. In a world which apparently exhibits so much power and yet does so little to drive it back, it represents the challenge par excellence. However, concerning this most pressing and controversial issue of underdevelopment, of all the disciplines which study man, philosophy is the one which until now said the least. At first sight, to mark off in the topic of underdevelopment an area of real philosophical concern does not seem feasible (...) indeed. Underdevelopment understood as a mere failure of development appears to be within the competence of the disciplines of the social sciences, especially of economics, rather than that of philosophy. Viewed as a technical problem, it could thereby be declared outside the sphere of direct philosophical inquiry.But this reason is no sooner accepted than it comes up against itself by the very nature of the conflicts in which the social sciences got bogged down in their attempt to understand underdevelopment. Although not directly spelt out, the conflicts are unmistakably pervaded throughout by philosophical questions. It suffices to point out the main cause of the theoretical split which divides scholars, in the general issue of tradition and modernity, to really bring out the eminently philosophical nature of the debate. We are obviously referring to the conflict between modernization theory and the neo-Marxist school. The purpose of this paper is precisely to show that the topic of underdevelopment, not only raises one of the basic and oldest problems of philosophy, namely the nature of the connection between the spiritual and the material, but also helps positively to reformulate it. (shrink)

Thomas Brown was one of the tail-enders of the Scottish Enlightenment. He shared with Dugald Stewart the chair of Moral Philosophy at the University of Edinburgh from 1810 until his premature death in 1820. He is sometimes classed with the Scottish common-sense philosophers and, to some extent at least, his basic philosophical principles were akin to those of the common-sense philosophy. He did, for instance, forfeit the issue of the justification of some of our most basic beliefs and rested them, (...) instead, on their being intuitively irresistible; in particular, he thought that some of our most basic beliefs could be seen as permanent principles of human nature—a claim made popular by Thomas Reid. Based on his theory of the workings of the human mind—which was developed in a course of lectures on the philosophy of mind presented at the University of Edinburgh and appeared posthumously as a book titled Lectures on the Philosophy of the Human Mind—some philosophers and psychologists have characterised him as an ‘associationist’.1 Brown ’s main contribution to the philosophy of causation was his book Inquiry into the Relation of Cause and Effect, published in 1818. This is, actually, the third edition of his little book titled Observations on. (shrink)

Discussions of John Locke’s theory of causation tend, understandably, to focus on the related notion of power and in particular the dialectic with David Hume. But Locke faces a very different threat, one that is internal to his view. For he argues both that causation is a relation and that relations are not real. The obvious conclusion is intolerable. And yet the premises, I argue, are unassailable. Building on an interpretation of Locke’s treatment of relations I have developed (...) elsewhere, I show how Locke can at once speak meaningfully of causation and deny its mind-independent existence. (shrink)

In biology and philosophy of biology, discussing the notion of interaction leads to an examination of interactionism, which is, broadly speaking, the view that rejects gene-centrism and gene determinism and instead emphasizes the fact that traits of organisms are always the result of genes and environments. It has long been asserted that the nature-nurture problem requires an interactionist solution of sorts, the so-called interactionist consensus. This consensus, however, has been deemed insufficient and challenged by several authors triggering an extension of (...) the debate among contestants and defenders. Unfortunately, part of the problem is that the views on causation that would ground claims about interactionism are not always made explicit in this debate, which renders those views somewhat complicated to assess. Moreover, it seems to be assumed that causal complexity excludes the possibility of characterizing, distinguishing, or comparing among causal contributions. By turning to a detailed survey of the origin of the debate and to some developments in the philosophy of causation, we will contend that this view is unwarranted, and that much of the debate around interactionism is based on the drawing of this conclusion. We also examine implications of this analysis for the project to develop a framework based on the notion of inter-identities. (shrink)

In this doctoral thesis, I will argue that in his De motu (1721, ‘On motion’), Bishop George Berkeley (c.1684–1753) develops a pragmatist theory of causation regarding mechanical theories outlined previously with Newtonianism. I place chief emphasis on the importance of logic and mathematics in Berkeley’s scientific approach, on which the other levels of semantics, epistemology, and mechanics build up. On my rendering, Berkeley’s pragmatic method to conceive or mathematically imagine causation makes sense in terms of mechanical causes or (...) ‘mathematical hypotheses’. For the mechanist maintains the usefulness and truthfulness of causation by the following definition. Definition. A pragmatist theory of causation is one which holds that: 1 Causal terms are indispensable in scientific deliberation for their usefulness; they cannot be eliminated [contra reductionism]. 2 What a cause is is defined by one’s temporal deliberative practices, independent of a temporal structure that theories hold [contra structuralism]. 3 Causal laws (theories formulated in causal terms) are genuinely true, not fictitious, when one confirms and deduces them [contra instrumentalism]. By justifying this definition, I will object to three rival readings—reductionism, structuralism, and instrumentalism—to my reading of Berkeley as a pragmatist about causation. In particular, my pragmatist reading criticises the most popular instrumentalist reading because, according to the latter, talk of causal terms like forces can be false or merely fictitious inasmuch as one can hold the utility of theories in mechanical practices. The instrumentalist reading is then compatible with mathematical formalism, according to which it is not truth or meaning that counts as formal manipulation or game of meaningless symbols, thereby eschewing a platonist attitude towards mathematical objects. However, I rebut the formalistic instrumentalism from Berkeley’s logical and realist standpoint, maintaining the irreducibility of occult qualities that mathematical objects have in their formulation from hypotheses to propositions. Light shall be shed on the tenet I propose that law-propositions formulated in hypothetical, causal terms must be true, neither false nor fictitious, when we (1) frame, (2) confirm, and (3) express them to the extent of our discursive thinking (in three steps). [339 words]. (shrink)

This dissertation presents a new interpretation of Rene Descartes' views on body/body causation by examining them within their historical context. Although Descartes gives the impression that his views constitute a complete break with those of his predecessors, he draws on both Scholastic Aristotelian concepts of the efficient cause and existing anti-Aristotelian views. ;The combination of Aristotelian and anti-Aristotelian elements in Descartes' theory of causation creates a tension in his claims about the relationship between the first cause, God, and (...) the secondary causes, created things. To make sense of Descartes' position on secondary causation I study the 16th and 17th century debates on secondary causation, God's concurrence, and the action of efficient causes. I examine the Aristotelian commentaries of the Jesuit philosophers Francisco Suarez, Francisco Toledo, Antonio Rubio and the Coimbran Commentators. I also analyze the anti-Aristotelian views on nature and causation found in Sebastian Basson's textbook on natural philosophy. ;The Jesuit philosophers, like Descartes, deny that the extendedness and impenetrability of matter requires the existence of occult forces to penetrate the matter and exercise causality. Rather they argue that causality is exercised by a thing's form and that causal interaction requires only contiguity not penetration. They also downplay the importance of the final cause, making the efficient cause prior in the study of nature. While Descartes rejects their hylomorphism, he agrees with the Jesuits that there are secondary efficient causes which determine God's action, causing a specific kind of effect. However, for Descartes the secondary causes are the laws of nature, not individual substances. ;Descartes' reduction of all natural change to the local motion of material particles is reminiscent of Basson's anti-Aristotelian natural philosophy. Basson reacts against the Jesuit view of secondary causation, arguing that it commits them to attributing cognition to physical causes. His own account of natural causation contains both mechanistic elements, which we find in a more developed form in Descartes' philosophy, and Neoplatonic and Stoic elements which Descartes rejects. (shrink)

The origins and development of the problem of mental causation are outlined. The underlying presuppositions which give rise to the problem are identified. Possible strategies for solving, or dissolving the problem are examined.

Summary A sufficient condition for a revolution in physics is a change in the concept of cause. To demonstrate this, we examine three developments in physical theory. After informally characterizing a theory in terms of an heuristic and a set of equations, we show how tensions between these two dimensions lead to the development of alternative theoretical accounts. In each case the crucial move results in a refinement of our account of cause. All these refinements taken together result in (...) the emergence of a new conceptual framework in which ‘causation’ is evolving in a manner unrelated to the common sense understanding of the concept. (shrink)

Professor Chisholm’s lively “Reflections on Human Agency” develop themes which have appeared in at least nine earlier papers of his on action and the kindred topic of events. His latest variations on the Incompatibility thesis will be my sole concern here. This is the doctrine that fully voluntary deeds of a free agent, for which we may justifiably hold him accountable, cannot result from earlier or contemporaneous events. Chisholm’s general Incompatibility formula reads.

The counterfactual tradition to defining actual causation has come a long way since Lewis started it off. However there are still important open problems that need to be solved. One of them is the (in)transitivity of causation. Endorsing transitivity was a major source of trouble for the approach taken by Lewis, which is why currently most approaches reject it. But transitivity has never lost its appeal, and there is a large literature devoted to understanding why this is so. (...) Starting from a survey of this work, we will develop a formal analysis of transitivity and the problems it poses for causation. This analysis provides us with a sufficient condition for causation to be transitive, a sufficient condition for dependence to be necessary for causation, and several characterisations of the transitivity of dependence. Finally, we show how this analysis leads naturally to several conditions a definition of causation should satisfy, and use those to suggest a new definition of causation. (shrink)

Professor Chisholm’s lively “Reflections on Human Agency” develop themes which have appeared in at least nine earlier papers of his on action and the kindred topic of events. His latest variations on the Incompatibility thesis will be my sole concern here. This is the doctrine that fully voluntary deeds of a free agent, for which we may justifiably hold him accountable, cannot result from earlier or contemporaneous events. Chisholm’s general Incompatibility formula reads.

In The Biopsychosocial Model of Health and Disease, Derek Bolton and Grant Gillett argue that a defensible updated version of the biopsychosocial model requires a metaphysically adequate account of disease causation that can accommodate biological, psychological, and social factors. This present paper offers a philosophical critique of their account of biopsychosocial causation. I argue that their account relies on claims about the normativity and the semantic content of biological information that are metaphysically contentious. Moreover, I suggest that these (...) claims are unnecessary for a defence of biopsychosocial causation, as the roles of multiple and diverse factors in disease causation can be readily accommodated by a more widely accepted and less metaphysically contentious account of causation. I then raise the more general concern that they are misdiagnosing the problem with the traditional version of the biopsychosocial model. The challenge when developing an explanatorily valuable version of the biopsychosocial model, I argue, is not so much providing an adequate account of biopsychosocial causation, but providing an adequate account of causal selection. Finally, I consider how this problem may be solved to arrive at a more explanatorily valuable and clinically useful version of the biopsychosocial model. (shrink)

While it is generally agreed that the concept of homology refers to individuated traits that have been inherited from common ancestry, we still lack an adequate account of trait individuation or inheritance. Here I propose that we utilize a counterfactual criterion of causation to link each trait with a developmental-causal (DC) gene. A DC gene is made up of the genetic information (which might or might not be physically contiguous in the genome) that is needed for the production of (...) the organismic attributes that comprise the trait. I argue that individuated traits—phenes—correspond to organismic features that are caused by DC genes. Using such an approach, we can define a DC map, which shows the relations between each pair of phenes and provides a succinct summary of genotype-phenotype relationships and phenotypic complexity. Phenes in parents and offspring are judged to be homologous if their DC genes are composed of orthologous genetic factors. When comparing more distantly related organisms, traits are homologous when linked by a chain of parent-offspring homologs along the path of ancestry that links the two organisms. There are three possible ways to deal with the potential for multiple equivalent DC genes: maximal, minimal, and consensus homology. Whereas maximal homology has limited utility, the other two approaches have value and can help to guide research at the intersection of evolution and development. (shrink)

This paper examines a promising probabilistic theory of singular causation developed by David Lewis. I argue that Lewis' theory must be made more sophisticated to deal with certain counterexamples involving pre-emption. These counterexamples appear to show that in the usual case singular causation requires an unbroken causal process to link cause with effect. I propose a new probabilistic account of singular causation, within the framework developed by Lewis, which captures this intuition.

This book provides the reader with an analysis of backward causation. The notion of backward causation faces many different paradoxes that threaten to make the notion inconsistent or incoherent. The book denies that these pose a real threat. It developed a theory of causation according to which the orientation of causation is not dependent on the direction of time. In this process it takes issues with David Lewis' contrafactual analysis of causation, and denies that the (...) direction of time is determined by irreversible processes . Likewise the book argues that the conceptual possibility of backward causation requires that the future is just as real as the past and present. Finally the book considers what the physical processes may look like if backward causation not only a a conceptual possibility but also a physical possibility. The conclusion is that no normal matter can possibly participate in backward causal processes. Instead objects moving backwards in time seem to posses negative mass and energy, a kind of objects that would be different from both ordinary objects and hypothetical tachyons. (shrink)

I apply some of the lessons from quantum theory, in particular from Bell’s theorem, to a debate on the foundations of decision theory and causation. By tracing a formal analogy between the basic assumptions of causal decision theory (CDT)—which was developed partly in response to Newcomb’s problem— and those of a local hidden variable theory in the context of quantum mechanics, I show that an agent who acts according to CDT and gives any nonzero credence to some possible causal (...) interpretations underlying quantum phenomena should bet against quantum mechanics in some feasible game scenarios involving entangled systems, no matter what evidence they acquire. As a consequence, either the most accepted version of decision theory is wrong, or it provides a practical distinction, in terms of the prescribed behaviour of rational agents, between some metaphysical hypotheses regarding the causal structure underlying quantum mechanics. (shrink)

Holtby, Allen and Fairchild are both recent and revolutionary decisions that address an important aspect of the indeterminate causation problem that frequently arises in tort litigation. In Holtby and Allen, the Court of Appeal departed from the traditional binary approach, under which a tort claimant either recovers compensation for his or her entire injury or is altogether denied recovery—depending on whether his or her case against the defendant is more probable than not. Holtby and Allen substituted this approach by (...) the proportionate recovery principle, under which the defendant compensates the claimant for a fraction of his or her injury that represents the defendant's statistical share in that injury. This article analyses this development within the particular domain of indeterminate causation, over which the proportionate recovery principle has been licensed to exercise control. The article claims that this development would constitute an improvement of the law from the perspectives of both optimal deterrence and corrective justice, if the courts properly formulate its scope. First, the proportionate recovery principle needs to be explicitly confined to cases that deal with recurrent wrongs. Second, determination of the defendant's share in the claimant's injury ought to be grounded on the probability of causation, rather than the risk of inflicting that injury. Third, judges must not apply unarticulated intuitions in determining the magnitude of the relevant risk or probability: their decisions in that area would be better informed by the statistical principle of ‘insufficient reason’, also known as the ‘indifference principle’. Fourth, courts are yet to relieve the doctrinal tension between the proportionate recovery principle, as recognized in Holtby and Allen, and the previous rejection of that principle by the House of Lords. Subsequently, the article analyses the House of Lords' decision in Fairchild—yet another instance of indeterminate causation in which proportionate recovery is preferable to the ‘all or nothing’ approach. In this case, the House of Lords allowed the claimants full recovery. This holding was grounded in the Law Lords' innovative approach to indeterminate causation, and the article unfolds the problematics of this approach. Finally, the article offers an adoption of yet another legal mechanism—the ‘evidential damage doctrine’—that replaces liability under uncertainty with liability for uncertainty. (shrink)

The aim of this paper is to assess the relation between causation and the notion of emptiness described in Buddhist philosophy. While the Madhyamaka school argues that some entity’s being caused implies its being empty, some contemporary authors have argued that there is a ‘Humean’ regularity account of causation that can both be understood as a plausible model of the earlier Buddhist Abhidharma account of causation and also block the Madhyamaka inference from causation to emptiness. After (...) describing the Abhidharma account of causation, the ‘Humean’ regularity account and the Madhyamaka argument from causation to emptiness, we assess some ways in which this argument may be developed, with particular focus on the ‘ladder of causation’ and on the Madhyamaka account of time. The debate about the relation between causation and emptiness, it appears, is a facet of a more comprehensive metaphysical debate between a (moderate) foundationalism and a thoroughgoing anti-foundationalism. (shrink)