If sociopathy is a biological adaptation, it probably evolved in small social groups in which individuals lacked the social mobility required for a con-man strategy to work. On the other hand, conflicts between groups may have provided a large niche for sociopathy throughout human history.

We propose that Mealey's model is limited in its description of sociopathy because it does not provide an adequate role for the main organ mediating genes and behavior, namely, the brain. Further, on the basis of our research, we question the view of sociopaths as a homogeneous group.

Mealey's evolutionary reasoning is logically flawed. Furthermore, the evidence presented in favor of a genetic contribution to the causation of sociopathy is overinterpreted. Given the potentially large societal impact of sociobiological speculation on the roots of criminality, more-than-usual caution in interpreting data is called for.

The validity of the classification of “primary sociopaths” as a qualitatively distinct group in the general population is questioned. Cenetic variation in the experience and expression of emotions may play a role in the development of antisocial behavior. However, research clearly documents that socialization environments powerfully modify the expression of genetic biases in a manner that increases or decreases the risk for “sociopathy.”.

Mealey's article on the sociobiology of sociopathy raises questions about the relationship between adaptiveness and abnormality. Although sociopathy may be adaptive, simply defining medical and psychiatric disorders in terms of evolutionary function is problematic. Rather, sociopathy may be characterized as a relatively atypical disorder, an entity to which it may be useful to extend the metaphors of the medical model.

Definitional slippage threatens to equate secondary sociopathy with mere criminality and leaves the status of noncriminal sociopaths ambiguous. Primary sociopathy appears to show more environmental contingency than would be implied by a strong genetic trait approach. A reinterpretation in terms of hypermasculinity and hypofemininity is compatible with the data.

Behavioral biologists have long sought to link behavioral units (e.g., aggression, depression, sociopathy) with biological units (e.g., genes, neurotransmitters, hormones, neuroanatomical loci). These units, originally contrived for descriptive purposes, often lead to misunderstandings when they are reified for purposes of causal analysis. This genetic and biochemical explanation for sociopathy reflects such problems.

Evolutionary analysis suggests that policies based on deterrence may cope effectively with primary sociopathy if the threat of punishment fits the crime in the cost/benefit calculus of the sociopath, not that of the public. On the other hand, policies designed to offset serious disadvantage in social competition may help inhibit the development of secondary sociopathy, rather than deter its expression.

Mealey's analysis of secondary sociopathy has much in common with Belsky, Steinberg, and Draper's (1991) evolutionary theory of socialization. Both draw attention to the potential influence of early rearing in the promotion of a cold, detached, manipulative, and opportunistic style of relating to others and, in so doing, raise the question of whether secondary sociopathy represents a facultative reproductive strategy.

Recent studies lend support to the two-pathway model of the evolution of sociopathy with evidence that: 1) psychopathy (primary sociopathy) is a discrete type and 2) in general, sociopaths have relatively high levels of reproductive success. Hare's Psychopathy Checklist may provide a start for the revision of terminology that will be necessary to distinguish between primary and secondary trajectories.

Sociopaths are “outstanding” members of society in two senses: politically, they draw our attention because of the inordinate amount of crime they commit, and psychologically, they hold our fascination because most ofus cannot fathom the cold, detached way they repeatedly harm and manipulate others. Proximate explanations from behavior genetics, child development, personality theory, learning theory, and social psychology describe a complex interaction of genetic and physiological risk factors with demographic and micro environmental variables that predispose a portion of the population (...) to chronic antisocial behavior. More recent, evolutionary and game theoretic models have tried to present an ultimate explanation of sociopathy as the expression of a frequency-dependent life strategy which is selected, in dynamic equilibrium, in response to certain varying environmental circumstances. This paper tries to integrate the proximate, developmental models with the ultimate, evolutionary ones, suggesting that two developmentally different etiologies of sociopathy emerge from two different evolutionary mechanisms. Social strategies for minimizing the incidence of sociopathic behavior in modern society should consider the two different etiologies and the factors that contribute to them. (shrink)

Mealey argued that sociopathy is an evolutionary stable strategy subject to frequency-dependent selection – high levels of sociopathy being advantageous to the individual if population-wide frequencies of it are low, and vice versa. I argue that at least one alternative hypothesis exists that explains her data equally well. Alternative hypotheses must be formulated and tested before any theory can be validated.

Mealey distinguishes two types of sociopathy: (1) or obligate, and (2) or facultative. Either sociopathy evolved twice, or one form is derived from the other, e.g., through: (1) genetic assimilation generating polymorphism in the relative strength of biases favoring the development of otherwise facultative strategies, or (2) independently heritable but strategically relevant characteristics biasing the optimal selection of facultative strategies.

Sociobiological explanation requires both a reliable and a valid definition of the sociopathy phenotype. Mealey assumes that such reliable and valid definition of sociopathy exists in her A review of psychiatric literature on the diagnosis of antisocial personality disorder clearly demonstrates that this assumption is faulty. There is substantial disagreement among diagnostic systems (e.g., RDC, DSM-III) over what constitutes the antisocial phenotype, different systems identify different individuals as sociopathic. Without a valid definition of sociopathy, sociobiological theories like (...) Mealey's should be viewed as entirely speculative. (shrink)

Questions are raised about several issues discussed by Mealey: (1) the nature of the distinction between primary and secondary sociopaths, (2) some difficulties with a general arousal theory of criminality, and (3) the possible role of countervailing forces in the development of sociopathy. An important area that calls for attention is the patterning of different specific emotions in the lives of sociopaths as compared to other groups.

Understanding the bases of complex behavioral phenotypes, such as sociopathy, is assisted by an evolutionary approach, in addition to other theoretical perspectives. Unraveling genetic and environmental factors underlying variant forms of sociopathy remains a key challenge for behavioral science investigators. Twin research methods (e.g., longitudinal analyses; twins reared apart) offer informative means of assessing novel hypotheses relevant to sociopathic behaviors.

We doubt that primary sociopathy is adaptive, for three reasons: First, its prevalence is too low to require an adaptive explanation. Second, a common sequela of damage to the orbito-frontal lobes is Any pattern of behavior that can be produced by brain damage is unlikely to be adaptive. Third, we argue that most human social behavior is not under tight genetic control, but is produced by open-ended calculation of fitness-contingencies.

We make three suggestions with regard to Mealey's work. First, her lack of a cognitive analysis of the sociopath results in underspecified mappings between sociobiology and behavior. Second, the developmental literature indicates that Mealey's implicit assumption, that moral socialisation is achieved through punishment, is invalid. Third, we advance the use of causal modelling to map the developmental relationships between biology, cognition, and behaviour.

Endorsing Mealey's analysis, it is pointed out that increasing rates of crime and violence are due to increasing proportions of children being reared in circumstances radically different from the extendedfamily environment to which we are evolntionarily adapted, that is, they are reared without fathers.

Metaethical questions are typically held to be a priori , and therefore impervious to empirical evidence. Here I examine the metaethical claim that motive-internalism about belief , the position that moral beliefs are intrinsically motivating, is true. I argue that belief-internalists are faced with a dilemma. Either their formulation of internalism is so weak that it fails to be philosophically interesting, or it is a substantive claim but can be shown to be empirically false. I then provide evidence for the (...) falsity of substantive belief-internalism. I describe a group of brain-damaged patients who sustain impairment in their moral sensibility: although they have normal moral beliefs and make moral judgments, they are not inclined to act in accordance with those beliefs and judgments. Thus, I argue that they are walking counterexamples to the substantive internalist claim. In addition to constraining our conception of moral reasoning, this argument stands as an example of how empirical evidence can be relevantly brought to bear on a philosophical question typically viewed to be a priori. (shrink)

Emotion is critical in Mealey's conceptual arguments. However, several of her assertions about the role of emotion in sociopathy are problematic. Questions are raised regarding the link between lack of anxiety and low levels of secondary emotions such as love and sympathy, the argument that sociopaths are low in anxiety but high in neuroticism, and the designation of anxiety as a secondary emotion.

Although serotonin, testosterone, and genes contribute to sociopathy, the relationships are probably indirect and subject to modifiers (e.g., present only under certain conditions of rearing and temperament). Age at menarche may be a marker variable as well as a causal factor. Since the genders differ in all four areas, sex differences in sociopathy represent a very complex interaction of these factors.

Mealey's sociobiological model of sociopathy could profit from attachment theory, in particular, the theory and research on the basis of the Adult Attachment Interview (Main & Goldwyn 1985–1993). Findings of an adult attachment study in a forensic psychiatric setting are summarized. Three attachment-oriented strategies for families, schools, and forensic settings are proposed to help reduce or prevent secondary sociopathy and criminal recidivism.

Mealey's (1995a) psychological explanation of the sociopath's antisocial activity appeals to an incomplete or nonstandard theory of mind. This is not the only possible mechanism of mental state attribution. The simulation theory of mental state ascription offers a better hope of explaining the diverse elements of sociopathy reported by Mealey.

This chapter contains section titled: Communities and Persons A Phenomenological Distinction between Community and Society Community Society The Self and its Social Roles Dispositional Vectors and the Shaping of Personality The Personality The Sociopathic Personality Type Conclusion.

A consideration of the genetics of sociopathy suggests the following. The author's Evolutionary Stable Strategy (ESS) types 2 to 4 are more likely than types 1 and 5 in crimes of violence, and there may not be an ESS for crimes of property or for sociopathy. Correlations between sociopathy and crimes of property are also more likely due to environmental than to genetic variants, and correlations between sociopathy and crimes of property are due more to environmental (...) than genetic variants. (shrink)

Mealey's sociopathy model is an exemplar of popular diathesis-stress models. Although such models, when presented in descriptive language, offer the illusion of integrative explanation, their actual scientific value is very limited because they fail to make specific, quantitative, falsifiable predictions. Conceptual and quantitative weaknesses of such diathesis-stress models are discussed and the requirements for useful models are outlined.

New discoveries in neuroscience challenge our understanding of human responsibility and justice. Recent studies suggest that psychopaths not only exhibit specific behavioral patterns but may also have a distinct neuroanatomical blueprint. Scientists have shown that a significant number of individuals who have demonstrated psychopathic behaviors have reduced volume and other anatomical changes in various regions of the cerebral cortex as well as decreased functional connectivity between different brain areas (i.e., smaller dysfunctional amygdalae). These findings raise ethical questions about how our (...) criminal system deals with these individuals. Should dysfunctional anatomical or diseased features exonerate affected individuals from any responsibility for their criminal activity or the suffering they have caused to their victims? It is estimated that individuals with the signs and symptoms of antisocial personality disorder make up 25% of inmates. Should individuals classified with this disorder be incarcerated with other criminals? In this paper, we summarize the results of the latest brain imaging studies performed on individuals with antisocial personality traits, referred to as psychopaths or sociopaths, and we explore some of the ethical issues concerning psychopathy and the law. (shrink)

I argue that the use of heritable modifications for psychology, personality, and behavior should be limited to the reversal or prevention of relatively unambiguous instances of pathology or likely harm (e.g. sociopathy). Most of the likely modifications of psychological personality would not be of this nature, however, and parents therefore should not have the freedom to make such modifications to future children. I argue by examining the viewpoints of both the individual and society. For individuals, modifications would interfere with (...) their capacity for self-determination in a way that undermines the very concept of self-determination. I argue that modification of psychology and personality is unlike present parenting in morally significant ways. For society, modification offers a medium for power to manipulate the makeup of persons and populations, possibly causing biological harm to the species and altering our conceptions of social responsibility. (shrink)

In this paper, I present a general profile of individuals with psychopathy, autism, and acquired sociopathy as well as look specifically at the abilities of these individuals with respect to the moral domain. These individuals are individually and collectively interesting because of their significant affective and social impairments. I argue that none of these individuals should be considered full moral agents based on a proposed account of moral agency consisting of the following two necessary conditions: the capacity for moral (...) judgment and the capacity for moral motivation. Since each of these groups of individuals fails to satisfy the criteria in a different way, these arguments help to distinguish three primary ways in which emotion contributes to moral motivation. I conclude with a suggested general implication of this discussion for related debates in moral theory. (shrink)

In the Frank (1988) model, a small increase in the number of cheaters will soon be reversed. It is not clear that this prediction holds for sociopathy. There are also many attractive evolutionary models that do not admit a small, stable proportion of cheaters. Hence, without definitive evidence about the character of early human society, we cannot conclude that sociopathy has an evolutionary origin.

Mealey's interesting interpretation of sociopathy is based on an inappropriate two-person game model. A multiperson, compound game version of Chicken would be more suitable, because a population engaging in random pairwise interactions with that structure would evolve to an equilibrium in which a fixed proportion of strategic choices was exploitative, antisocial, and risky, as required by Mealey's interpretation.

As more criminals are imprisoned, other individuals change their behavior to replace them, as predicted by the theory of strategic behavior. The physiological correlates of sociopathy suggest that research in cognitive neuroscience can lead toward a solution. Promising pathways include building upon current knowledge of self-deceit, the independence of positive and negative emotions, the lateralization of risk and caution, and the conditions promoting prosocial behavior.

The drift toward behavioral accounts of the cluster of psychological and behavioral traits that were interchangeably referred to as psychopathy, sociopathy and anti-social personality is interesting and well worth exploring. Justman's correct that before the work of the Feighner group and the adoption of Antisocial Personality Disorder in the Diagnostic and Statistical Manual of Mental Disorders -III, the choice of concept did not seem to be vital and in the Mask of Sanity, Cleckley mentions all three terms and does (...) not seem to think much hangs on which term we choose.It seems correct that many held the absence of guilt to be the psychological marker of psychopathy... (shrink)

Problems of definition and identification in the integrated evolutionary model of sociopathy are suggested by Schoenfeld's (1974) criticism of the field of race differences in intelligence. Moral judgments by those labeled primary and secondary sociopaths may offer a way to validate the assumptions of the model.

Psychopathy has as its central traits socialization, sensation seeking, and impulsivity. These are combined in a supertrait: Impulsive Unsocialized Sensation Seeking (ImpUSS). Secondary types are defined by combinations of ImpUSS and neuroticism or sociability. All broad personality traits have both genetic and environmental determination, and therefore different etiologies (primary as genetic, secondary as environmental) for primary and secondary sociopathy are unlikely.

Mealey's excellent target article rests on several assumptions that may be questioned, including the overarching assumption that sociopathy reflects the failure of a small minority of males to cooperate with the larger group. I suggest that violent competition in ancestral bands cheatinggame was the primary evolutionary precursor of sociopathy. Today's violent sociopath is far more a than a failed cooperator.

Currently, psychopathy and related terms such as antisocial personality disorder are popular yet problematic constructs within forensic psychology and other disciplines. Psychopathy is traced typically to the works of Pinel and Prichard in the early 19th century, and it has even been linked to biblical passages, although there appears to be little or no support for the latter claim. The first use of the term psychopathy in German psychiatry of the mid-19th century referred only to psychological disturbance in general, or (...) ‘personality diseases’, although German psychiatrists such as Kraepelin did propose more specific definitions of the term related to social deviance. Our modern understanding of psychopathy as a psychiatric disorder of an undetermined genetic origin involving antisocial elements and a lack of feeling or concern for others owes much to the clinical work and writings of Cleckley. Antisocial personality disorder appears similarly to derive from the work of psychiatric nosologists like Kraepelin and Schneider, while sociopathy is due more to the work of Partridge in the 1920s and 1930s. (shrink)

When Genotype × Environment (G × E) interactions are present, heritability estimates are not interpretable. Mealey cites abundant evidence for G × E interactions in the etiology of sociopathy, thereby completely undermining estimates of the heritability of sociopathy which form the foundation of her model. Without proper evidence for a genetic basis of sociopathy, Mealey's sociobiological model collapses under its own great weight.

Reports of increasing family violence abound in Nigeria. Although studies have implicated cultural constructs as the basis for this trend, this study specifically contemplated the nexus between the increased violence and Anti-Social Personality Disorder (ASPD). The purpose of this study was to connect these abusive and violent behaviours in their specific contexts as evidence of ASPD. The study was hinged on autoethnographic research which I systematically analysed as lived experiences with a sociopath. Data for the analyses was drawn from purposively (...) sampled participants (n-70) of victims of family violent abuse. Thematic, interpretative analytical approach and the theories of sociopathy, particularly Hare, Psychopathy Checklist-revised (PCL-R) were used to analyse the data from my personal experiences, and the interviews. The results indicated that only five participants representing 7.14% of the purposively selected population, admitted knowing sociopaths as conscienceless murderers and serial killers. The majority of participants (90%) contemplated sociopaths in religious terms (as incarnated evil beings). Only 2.86% rightly expressed their understanding of sociopathy and related personality disorders in a much-informed manner. Although the results of this study contrast with conventional propositions on ASPD, they revealed how religious belief and superstition among Nigerians frame the conversation on sociopathy and other variants of ASPD. Findings suggested that the growing superstitious beliefs on the causes, nature and remedy to ASPD particularly those associated with sociopathy provide the space for people living with these behavioural conditions (particularly violent sociopaths) to continue abusing unsuspecting individuals in the society. Contribution: The finding of this study specifically challenged the faith-based misconceptions associated with ASPD. Therefore, it is expected that the new insight on ASPD, particularly sociopathy ushers in proper understanding and ways of dealing with the morally insane. (shrink)

Reports of increasing family violence abound in Nigeria. Although studies have implicated cultural constructs as the basis for this trend, this study specifically contemplated the nexus between the increased violence and Anti-Social Personality Disorder (ASPD). The purpose of this study was to connect these abusive and violent behaviours in their specific contexts as evidence of ASPD. The study was hinged on autoethnographic research which I systematically analysed as lived experiences with a sociopath. Data for the analyses was drawn from purposively (...) sampled participants (n-70) of victims of family violent abuse. Thematic, interpretative analytical approach and the theories of sociopathy, particularly Hare, Psychopathy Checklist-revised (PCL-R) were used to analyse the data from my personal experiences, and the interviews. The results indicated that only five participants representing 7.14% of the purposively selected population, admitted knowing sociopaths as conscienceless murderers and serial killers. The majority of participants (90%) contemplated sociopaths in religious terms (as incarnated evil beings). Only 2.86% rightly expressed their understanding of sociopathy and related personality disorders in a much-informed manner. Although the results of this study contrast with conventional propositions on ASPD, they revealed how religious belief and superstition among Nigerians frame the conversation on sociopathy and other variants of ASPD. Findings suggested that the growing superstitious beliefs on the causes, nature and remedy to ASPD particularly those associated with sociopathy provide the space for people living with these behavioural conditions (particularly violent sociopaths) to continue abusing unsuspecting individuals in the society. Contribution: The finding of this study specifically challenged the faith-based misconceptions associated with ASPD. Therefore, it is expected that the new insight on ASPD, particularly sociopathy ushers in proper understanding and ways of dealing with the morally insane. (shrink)

The following topics are discussed, from psychoanalytic, philosophical, and empirical perspectives: -/- *Sociopathy *Pedantry *The nature of bureaucrats *The nature of bureaucratic institutions *Rationalization and Repression *The relationship between ignorance and mental health *The relationship sapience and mental illness *The relationship between ignorance and the ability to act *The relationship between hyper-sapience and the inability to act. *The psychological underpinnings of addiction; and finally *The differences between men and women.

Pt. 1. The Practice Of Prudence. 1.The Moral in the Theater of the World -- pt. 2. The Labor Of The Negative. 2.Terrorism: The Ideology of Identity -- 3. Psycho-Sociopathy: The Counterfeit of Conscience -- 4. The Technological Person: The Dominance of Desire.

In this article the causes and features of a shift from neurosis to narcissism and then autism were investigated. Moreover, special attention is paid not so much to the psychological or even psychiatric aspects of the problem, but to changes in the general features of a human being. Thus, when speaking of the transition from narcissism to autism, one is focusing on true autism, which is characterized by veracity, sincerity and authencity, which reveal themselves in the form of a special (...) kind of infantility, a kind of mature infantility. A suitable example of such infantility is found in the animated film Shrek, a certain barbarism of which is not so ironic or even knowing ironic, but to a greater extent it is warm, earnest and serious although childish. Among other infantile practices, it is worth noting the situationalist-lettrist practice of organizing a text in the form of an ABC alphabet book, a hornbook, which is actualized in a digimodern context. True autism and to some extent pseudoautism in a certain sense become a way to cope with the trance and amnesia of pseudomodernism and leave a hope for the future. Thus, paradoxically, wholeness that was lost is restored through desocialization and sociopathy. This article also draws parallels between digimodernism and speculative realism, conceptualization of interpassivity as well as automodernity. It is emphasized that personal autonomy and automation can be achieved through the use of devices as a means of achieving productive loneliness, which should be distinguished from alienation. In this case, the widespread use of sensory experience as a way of communication is especially important, as an example of which Hope Sandoval’s mode of being and work are considered. It suggests that the high-tech / high-touch approach is best released in DIY activities. The above considerations allow to conclude that true autism is one of the most actual post-postmodern modes of subjectivation. (shrink)

Mealey's data do not support her dichotomous model of primary and secondary sociopathy; this data supports the view that there is a continuum of degrees of sociopathy, from zero to the maximal manifestation. There are multitudes of factors that can contribute to sociopathy and the countless different mixes of them can produce multiple degrees and variations of sociopathic behavior.

Mealey's etiological distinction between primary and secondary sociopathy blurs the delineation between individual and group differences. She uses physiological evidence to support her claim of genetic influences, neglecting variability within social classes, frequency of delinquent behavior in upper and middle classes (measured by self-report), and discontinuity of criminal behavior across the life span. Finally, Mealey's proposals for differential intervention fall short of a future agenda, which should tailor to individual needs, not social classes.