Naïve realism (also called ‘relationalism’ or ‘object view’) is becoming increasingly popular, but the specific outline of its commitments remains often underspecified by proponents and misunderstood by critics. Naïve realism is associated with two claims, both concerning genuine, veridical perceptual experience (where this excludes hallucinations). Constitutive Claim (CC): The phenomenal character of perception is (partly) constituted by the mind-independent objects in one’s surrounding and their properties. Relational Claim (RC): Perception is a relation to mind-independent objects in the environment and their (...) properties. Some philosophers use the two claims interchangeably while talking about naïve realism, while others use only one or the other, although they do not explicitly discuss if the other claim is also a core commitment of naïve realism, or if naïve realism can be held without the other claim. This raises the question of how RC and CC relate to one another, together with the most pressing question of what each claim ultimately commits one to. After discussing the shortcomings of alternative interpretations, I argue that naïve realism should be understood as committed to, first and foremost, RC. This should be understood as a claim about the phenomenal character of perception, rather than about its nature, structure or essence (whatever that means). CC, on the other hand, should be understood as a corollary of RC. This doesn’t only offer a better characterisation of how naïve realists understand phenomenal character: it also helps us understand how we can simultaneously claim that the object of perception is a constitutive element of perception, while also allowing for it to play a causal role in determining perception. (shrink)

The phenomenological method, characterized by the suspension of judgment (epoché), has helped analyzing the subjective experience of patients affected by mental disorders. Psychiatry, dealing with the human being itself in its complexity and unicity, is placed between the biological positivistic attempt, for which the symptoms of mental illness are a mere consequence of brain dysfunctions and the phenomenological-existential approach, inclined to consider the symptoms as meaningful phenomena of the person’s subjective experience. Eugène Minkowski, Ludwig Binswanger, Arthur Tatossian, Kimura Bin, Henri (...) Maldiney and Hubertus Tellenbach are fundamental authors in the phenomenological psychopathology of depression; they described the alterations of the lived time, space, body and others experienced by the depressed. Starting from the main theoretical contributions of the authors, we will focus on the psychopathology and discuss the key themes of clinical depression: guilt, poverty and hypochondriasis. Finally we will focus on the typus melancholicus construct. (shrink)

Implanting deep brain stimulation (DBS) electrodes in patients with Parkinson’s disease often results in the appearance of a non-infectious, delayed-onset edema that disappears over time. However, the time window between the DBS electrode and DBS stimulating device implant is often used to record local field potentials (LFPs) which are used both to better understand basal ganglia pathophysiology and to improve DBS therapy. In this work, we investigated whether the presence of post-surgery edema correlates with the quality of LFP recordings in (...) eight patients with advanced Parkinson’s disease implanted with subthalamic DBS electrodes. The magnetic resonance scans of the brain after 8.5 ± 1.5 days from the implantation surgery were segmented and the peri-electrode edema volume was calculated for both brain hemispheres. We found a correlation (ρ = −0.81, p < 0.0218, Spearman’s correlation coefficient) between left side local field potentials of the low beta band (11–20 Hz) and the edema volume of the same side. No other significant differences between the hemispheres were found. Despite the limited sample size, our results suggest that the effect on LFPs may be related to the edema localization, thus indicating a mechanism involving brain networks instead of a simple change in the electrode-tissue interface. (shrink)