Causal selection is the task of picking out, from a field of known causally relevant factors, some factors as elements of an explanation. The Causal Parity Thesis in the philosophy of biology challenges the usual ways of making such selections among different causes operating in a developing organism. The main target of this thesis is usually gene centrism, the doctrine that genes play some special role in ontogeny, which is often described in terms of information-bearing or programming. (...) This paper is concerned with the attempt of confronting the challenge coming from the Causal Parity Thesis by offering principles of causal selection that are spelled out in terms of an explicit philosophical account of causation, namely an interventionist account. I show that two such accounts that have been developed, although they contain important insights about causation in biology, nonetheless fail to provide an adequate reply to the Causal Parity challenge: Ken Waters's account of actual-difference making and Jim Woodward's account of causal specificity. A combination of the two also doesn't do the trick, nor does Laura Franklin-Hall's account of explanation (in this volume). We need additional conceptual resources. I argue that the resources we need consist in a special class of counterfactual conditionals, namely counterfactuals the antecedents of which describe biologically normal interventions. (shrink)

This paper questions the form and prospects of “extended theories” which have been simultaneously and independently advocated both in the philosophy of mind and in the philosophy of biology. It focuses on Extend Mind Theory (EMT) and Developmental Systems Theory (DST). It shows first that the two theories vindicate a parallel extension of received views, the former concerning extending cognition beyond the brain, the latter concerned with extending evolution and development beyond the genes. It also shows that both arguments rely (...) on the demonstration of causal parities, which have been undermined by the classical received view. Then I question whether the argument that there is an illegitimate inference from parities or coupling to constitution claims, which has been objected by Adams and Aizawa in The bounds of cognition, (2008) to EMT, also holds against DST. To this aim, I consider two defenses against DST that are parallel to two defenses against EMT, one about intrinsic content, the other about the difference between what’s in principle possible and what happens in practice. I conclude by claiming that the weaknesses and strengths of both theories are different regarding these two kinds of objections. (shrink)

Several authors have used the notion of causal specificity in order to defend non-parity about genetic causes (Waters 2007, Woodward 2010, Weber 2017, forthcoming). Non-parity in this context is the idea that DNA and some other biomolecules that are often described as information-bearers by biologists play a unique role in life processes, an idea that has been challenged by Developmental Systems Theory (e.g., Oyama 2000). Indeed, it has proven to be quite difficult to state clearly what the (...) alleged special role of genetic causes consists in. In this paper, I show that the set of biomolecules that are normally considered to be information-bearers (DNA, mRNA) can be shown to be the most specific causes of protein primary structure, provided that causal specificity is measured over a relevant space of biological possibilities, disregarding physical as well as logically possible states of the causal variables. (shrink)

The received view in philosophy of biology is that there is a well-understood, philosophically rigorous account of information—causal information. I argue that this view is mistaken. Causal information is fatally undermined by misinterpretations and conflations between distinct independent accounts of information. As a result, philosophical arguments based on causal information are deeply flawed. I end by briefly considering what a correct application of the relevant accounts of information would look like in the biological context.

I discuss the general form of arguments that profess to prove that the view that things endure in tensed time through causally produced change (the dynamic view) must be false because it involves contradictions. I argue that these arguments implicitly presuppose what has been called the temporal parity thesis, i.e. that all moments of time are equally existent and real, and that this thesis must be understood as the denial of the dynamic view. When this implicit premise is made (...) explicit, the arguments turn out to be either circular, they presuppose what they profess to prove, or mere demonstrations of the fact that the dynamic view is incompatible with its own negation. Furthermore, I discuss the metaphysical consequences of accepting the temporal parity thesis, arguing that it deprives us of the means to provide natural explanations to empirical phenomena. (shrink)

Dualism holds that some mental events are fundamental and non-physical. I develop a prima facie plausible causal argument for dualism. The argument has several significant implications. First, it constitutes a new way of arguing for dualism. Second, it provides dualists with a parity response to causal arguments for physicalism. Third, it transforms the dialectical role of epiphenomenalism. Fourth, it refutes the view that causal considerations prima facie support physicalism but not dualism. After developing the causal (...) argument for dualism and drawing out these implications, I subject the argument to a battery of objections. Some prompt revisions to the argument. Others reveal limitations in scope. It falls out of the discussion that the causal argument for dualism is best used against physicalism as a keystone in a divide and conquer strategy. (shrink)

Causal selection and priority are at the heart of discussions of the causal parity thesis, which says that all causes of a given effect are on a par, and that any justified priority assigned to a given cause results from causal explanatory interests. In theories of causation that provide necessary and sufficient conditions for the truth of causal claims, status as cause is an either/or issue: either a given cause satisfies the conditions or it does (...) not. Consequently, assessments of causal parity and priority require more resources, which can either be additional or part of the causal analysis itself. While adding resources in terms of a theory of causal explanation has been standard, here we develop a unified conceptual analysis that includes a range of different precise causal concepts that allows for assessments of causal priority in terms of different kinds of causal relevance. (shrink)

In reworking a variety of biological concepts, Developmental Systems Theory (DST) has made frequent use of parity of reasoning. We have done this to show, for instance, that factors that have similar sorts of impact on a developing organism tend nevertheless to be invested with quite different causal importance. We have made similar arguments about evolutionary processes. Together, these analyses have allowed DST not only to cut through some age-old muddles about the nature of development, but also to (...) effect a long-delayed reintegration of development into evolutionary theory. Our penchant for causal symmetry, however (or 'causal democracy', as it has recently been termed), has sometimes been misunderstood. This paper shows that causal symmetry is neither a platitude about multiple influences nor a denial of useful distinctions, but a powerful way of exposing hidden assumptions and opening up traditional formulations to fruitful change. (shrink)

This thesis aims to clarify a number of conceptual aspects of the debate surrounding algorithmic fairness. The particular focus here is the role of causal modeling in defining criteria of algorithmic fairness. In Chapter 1, I argue that in the discussion of algorithmic fairness, two fundamentally distinct notions of fairness have been conflated. Subsequently, I propose that what is usually taken to be the problem of algorithmic fairness should be divided into two subproblems, the problem of predictive fairness, and (...) the problem of allocative fairness. At the core of Chapter 2 is the proof of a theorem that establishes that three of the most popular (predictive) fairness criteria are pairwise incompatible. In particular, I show that under certain conditions, a predictive algorithm that satisfies a criterion called counterfactual fairness will with logical necessity violate two other popular predictive fairness criteria called equalized odds and predictive parity. In Chapter 3, a new predictive fairness criterion is developed using a mathematical framework for causal modeling. This fairness criterion, which I call causal relevance fairness, is a relaxation of another popular fairness criterion, counterfactual fairness, but turns out to be more closely in line with philosophical theories of discrimination. In Chapter 4, another infamous impossibility result in algorithmic fairness is analyzed through the lens of causality. I argue that by using a causal inference method called matching, we can modify the two fairness criteria equalized odds and predictive parity in a way that resolves the impossibility. Lastly, Chapter 5 contains an empirical case study. In it, the fairness of a popular recidivism risk prediction tool is analyzed using the criteria of (predictive) fairness developed earlier. (shrink)

Griffiths et al. (2015) have proposed a quantitative measure of causal specificity and used it to assess various attempts to single out genetic causes as being causally more specific than other cellular mechanisms, for example, alternative splicing. Focusing in particular on developmental processes, they have identified a number of important challenges for this project. In this discussion note, I would like to show how these challenges can be met.

The concept of causal specificity is drawing considerable attention from philosophers of biology. It became the rationale for rejecting (and occasionally, accepting) a thesis of causal parity of developmental factors. This literature assumes that attributing specificity to causal relations is at least in principle a straightforward (if not systematic) task. However, the parity debate in philosophy of biology seems to be stuck at a point where it is not the biological details that will help move (...) forward. In this paper, I take a step back to reexamine the very idea of causal specificity and its intended role in the parity dispute in philosophy of biology. I contend that the idea of causal specificity across variations as currently discussed in the literature is irreducibly twofold in nature: it is about two independent components that are not mutually entailed. I show this to be the source of prior complications with the notion of specificity itself that ultimately affect the purposes for which it is often invoked, notably to settle the parity dispute. (shrink)

I present a reconstruction of F.H.C. Crick's two 1957 hypotheses "Sequence Hypothesis" and "Central Dogma" in terms of a contemporary philosophical theory of causation. Analyzing in particular the experimental evidence that Crick cited, I argue that these hypotheses can be understood as claims about the actual difference-making cause in protein synthesis. As these hypotheses are only true if restricted to certain nucleic acids in certain organisms, I then examine the concept of causal specificity and its potential to counter claims (...) about causal parity of DNA and other cellular components. I first show that causal specificity is a special kind of invariance under interventions, namely invariance of generalizations that range over finite sets of discrete variables. Then, I show that this notion allows the articulation of a middle ground in the debate over causal parity. (shrink)

According to Russo and Williamson (Int Stud Philos Sci 21(2):157–170, 2007, Hist Philos Life Sci 33:389–396, 2011a, Philos Sci 1(1):47–69, 2011b ), in order to establish a causal claim of the form, ‘_C_ is a cause of _E_’, one typically needs evidence that there is an underlying mechanism between _C_ and _E_ as well as evidence that _C_ makes a difference to _E_. This thesis has been used to argue that hierarchies of evidence, as championed by evidence-based movements, tend (...) to give primacy to evidence of difference making over evidence of mechanisms and are flawed because the two sorts of evidence are required and they should be treated on a par. An alternative approach gives primacy to evidence of mechanism over evidence of difference making. In this paper, we argue that this alternative approach is equally flawed, again because both sorts of evidence need to be treated on a par. As an illustration of this parity, we explain how scientists working in the ‘EnviroGenomarkers’ project constantly make use of the two evidential components in a dynamic and intertwined way. We argue that such an interplay is needed not only for causal assessment but also for policy purposes. (shrink)

In lieu of an abstract, here is a brief excerpt of the content:51. HOW NOT TO REFUTE HUME'S THEORY OF CAUSALITY: A REPLY TO GRAY Mr. Robert Gray's alleged refutation of Hume's theory of causality does not strike me as being in reality conclusive. The essential element in his alleged refutation, if I have understood it correctly, is that when two billiard balls strike one another and stop - a paradigm of cause and effect - the striking and the stopping (...) constitute one event as opposed to two. They occupy, as a result, one and the same time, a state of affairs inconsistent with the Humean view that the cause immediately precedes the effect. But, surely, they would, if they are the same event, occupy by parity of reasoning one and the same position. For Hume's theory of space and time, as Gray describes it, implies that there is a one-to-one mapping between positions and times. If, therefore, their being the same event implies that they occupy one and the same time, it would also imply that they occupy one and the same position. And if they occupy the same position, they are not spatially contiguous, a state of affairs inconsistent with the Humean view that the cause is spatially contiguous to the effect. A Humean solution in both cases seems clear. It is to allow that the striking and stopping of the billiard balls are the same event and to seek its cause in their moving towards one another, i.e. to seek it in billiard ball A's being at A, and billiard ball B's being at B,, where both A, and B, are spatially contiguous with and immediately precede the striking and stopping of the respective billiard balls B and A. Gray resists this solution, but only because he is implicitly committed to the anti-Humean view that one and the same event can occupy two different positions at one and the same time. As a result, where A designates the 52. position where billiard ball A strikes billiard ball B and stops it and B designates the position where billiard ball B strikes billiard ball A and stops it - this being simply a re-descriptiön in Gray's view of what happens in the single event of two billiard balls striking one another and stopping - he holds that A is different from B. If A is different from B, then by relocating the cause of billiard ball A's stopping at B, we have sacrificed the spatial contiguity of cause and effect. For B will intervene between position B,, that of the cause, and A, that of the effect. And the same will hold with obvious adjustments when we relocate the cause of billiard ball B's stopping at A,. Fortunately, however, Hume need only reply that A is one and the same as B. Indeed, if he is to be consistent with his theory of space and time, this must be his reply. This reply, moreover, is necessary to Hume's explanation of how the reciprocal striking of billiard ball A and billiard ball B can exist in the first place. Such a phenomenon requires a point of contact, a point common to a part of both billiard balls. This point of contact, it should be clear, is provided by the point designated by both A and B. Robert A. Imlay University of Toronto "A Refutation of Hume's Theory of Causality," Hume Studies, Vol. II (1976), pp. 76-85.... (shrink)

In lieu of an abstract, here is a brief excerpt of the content:51. HOW NOT TO REFUTE HUME'S THEORY OF CAUSALITY: A REPLY TO GRAY Mr. Robert Gray's alleged refutation of Hume's theory of causality does not strike me as being in reality conclusive. The essential element in his alleged refutation, if I have understood it correctly, is that when two billiard balls strike one another and stop - a paradigm of cause and effect - the striking and the stopping (...) constitute one event as opposed to two. They occupy, as a result, one and the same time, a state of affairs inconsistent with the Humean view that the cause immediately precedes the effect. But, surely, they would, if they are the same event, occupy by parity of reasoning one and the same position. For Hume's theory of space and time, as Gray describes it, implies that there is a one-to-one mapping between positions and times. If, therefore, their being the same event implies that they occupy one and the same time, it would also imply that they occupy one and the same position. And if they occupy the same position, they are not spatially contiguous, a state of affairs inconsistent with the Humean view that the cause is spatially contiguous to the effect. A Humean solution in both cases seems clear. It is to allow that the striking and stopping of the billiard balls are the same event and to seek its cause in their moving towards one another, i.e. to seek it in billiard ball A's being at A, and billiard ball B's being at B,, where both A, and B, are spatially contiguous with and immediately precede the striking and stopping of the respective billiard balls B and A. Gray resists this solution, but only because he is implicitly committed to the anti-Humean view that one and the same event can occupy two different positions at one and the same time. As a result, where A designates the 52. position where billiard ball A strikes billiard ball B and stops it and B designates the position where billiard ball B strikes billiard ball A and stops it - this being simply a re-descriptiön in Gray's view of what happens in the single event of two billiard balls striking one another and stopping - he holds that A is different from B. If A is different from B, then by relocating the cause of billiard ball A's stopping at B, we have sacrificed the spatial contiguity of cause and effect. For B will intervene between position B,, that of the cause, and A, that of the effect. And the same will hold with obvious adjustments when we relocate the cause of billiard ball B's stopping at A,. Fortunately, however, Hume need only reply that A is one and the same as B. Indeed, if he is to be consistent with his theory of space and time, this must be his reply. This reply, moreover, is necessary to Hume's explanation of how the reciprocal striking of billiard ball A and billiard ball B can exist in the first place. Such a phenomenon requires a point of contact, a point common to a part of both billiard balls. This point of contact, it should be clear, is provided by the point designated by both A and B. Robert A. Imlay University of Toronto "A Refutation of Hume's Theory of Causality," Hume Studies, Vol. II (1976), pp. 76-85.... (shrink)

According to the proponents of Developmental Systems Theory and the Causal Parity Thesis, the privileging of the genome as “first among equals” with respect to the development of phenotypic traits is more a reflection of our own heuristic prejudice than of ontology - the underlying causal structures responsible for that specified development no more single out the genome as primary than they do other broadly “environmental” factors. Parting with the methodology of the popular responses to the Thesis, (...) this paper offers a novel criterion for ‘causal primacy’, one that is grounded in the ontology of the unique causal role of dispositional properties. This paper argues that, if the genome is conceptualised as realising dispositional properties that are “directed toward” phenotypic traits, the parity of ‘causal roles’ between genetic and extra-genetic factors is no longer apparent, and further, that the causal primacy of the genome is both plausible and defensible. (shrink)

Developmental Systems Theory is a theoretical reinterpretation of biological phenomena that challenges the conventional gene-centered account of development and evolution. In this article, I focus on Griffiths and Gray’s version of DST and particularly analyze their reconceptualization of inheritance. First, I present their concept of expanded and diffused inheritance; then, I examine and criticize their rejection of the multiple inheritance system model; finally, I present and oppose Griffiths and Gray’s extension of what they call the “causal parity thesis” (...) from development to evolution. I argue that their proposal is an interesting and programmatic philosophical perspective on biological phenomena but fails to provide either additional heuristic tools for empirical investigation in biology or a more realistic representation of the biological world. (shrink)

David Bohm's interpretation of quantum mechanics yields a quantum potential, Q. In his early work, the effects of Q are understood in causal terms as acting through a real (quantum) field which pushes particles around. In his later work (with Basil Hiley), the causal understanding of Q appears to have been abandoned. The purpose of this paper is to understand how the use of certain metaphors leads Bohm away from a causal treatment of Q, and to evaluate (...) the use of those metaphors. (shrink)

This article offers a reading of Eriugena’s thought that is inspired by Heidegger’s claim according to which being is constituted in a dialectical interplay of revelation and concealment. Beginning with an analysis of how “causality as concealing revelation” works on the level of God’s inner-Trinitarian life, the piece moves on to a consideration of the way in which the human soul reveals itself in successive stages of exteriorization that culminate in the creation of the body, its “image.” The body, however, (...) conceals as much as it reveals true human nature. Moreover, it is shown that for Eriugena all of reality, as theophany, possesses this character of concealing its fundamental truth. These insights lead Eriugena to a recognition of radical human finitude, as genuine wisdom requires an acknowledgement of our fundamental ignorance. (shrink)

Special Issue: The Material Bases of Cognition Guest Editors: Fred Adams · Kenneth Aizawa -/- Compositional Explanatory Relations and Mechanistic Reduction K.L. Theurer 287 -/- Constitution, and Multiple Constitution, in the Sciences: Using the Neuron to Construct a Starting Framework C. Gillett 309 -/- The Mark of the Cognitive F. Adams · R. Garrison 339 -/- Dynamics and Cognition L.A. Shapiro 353 -/- Causal Parity and Externalisms: Extensions in Life and Mind P. Huneman 377 -/- Did I Do (...) That? Brain–Computer Interfacing and the Sense of Agency P. Haselager 405 . (shrink)

This paper responds to Kenneth Waters’s account of actual difference making. Among other things, I argue that although Waters is right that researchers may sometimes be justified in focusing on genes rather than other causes of phenotypic traits, he is wrong that the apparatus of actual difference makers overcomes the traditional causal parity thesis.

The purpose of this article is to update and defend syntax-based gene concepts. I show how syntax-based concepts can and have been extended to accommodate complex cases of processing and gene expression regulation. In response to difficult cases and causal parity objections, I argue that a syntax-based approach fleshes out a deflationary concept defining genes as genomic sequences and organizational features of the genome contributing to a phenotype. These organizational features are an important part of accepted molecular explanations, (...) provide the theoretical basis for a large number of experimental techniques and practical applications, and play a crucial role in in annotating the genome, deriving predictions and constructing bioinformatics models. (shrink)

Developmental System Theory is a theoretical reinterpretation of biological phenomena challenging the conventional gene-centered account of development and evolution. In this paper, I focus on Griffiths and Gray’s version of Developmental Systems Theory and I particularly analyze their reconceptualization of inheritance. First, I present their concept of expanded and diffused inheritance; then, I examine and criticize their refusal of the multiple inheritance system model; finally, I present and contrast Griffiths and Gray’s extension of what they call the “causal (...) class='Hi'>parity thesis” from development to evolution. I argue that their proposal is an interesting and programmatic philosophical perspective on biological phenomena but, because of their commitment to holism, fails to provide both more heuristic tools for empirical investigation in biology and a more realistic representation of the biological world. (shrink)

I want to exhibit the deeper metaphysical reasons why some common ways of describing the causal role of genes in development and evolution are problematic. Specifically, I show why using the concept of information in an intentional sense in genetics is inappropriate, even given a naturalistic account of intentionality. Furthermore, I argue that descriptions that use notions such as programming, directing or orchestrating are problematic not for empirical reasons, but because they are not strictly causal. They are intentional. (...) By contrast, other notions that are part of the received view in genetics and evolutionary theory are defensible if understood correctly, in particular the idea that genes are the main replicators in evolution. The paper concludes that dropping all intentional or intentionally laden concepts does not force us to accept the so-called causal parity thesis, at least not in its stronger form. (shrink)

Minimal genetic pre-formationism is defended, in that primacy is ascribed to DNA in the structuring of molecules through molecular codes. This together with the importance of such codes for stability and variation in living systems makes DNA categorically different from other causal factors. It is argued that post-transcriptional and post-translational processing in protein synthesis does not rob DNA of this structuring role. Notions of structuring causal powers that may vary in degree, of arbitrary molecular codes that are more (...) or less realized, of partial templating and of genetic information as a subspecies of mechanistic information are brought in to support this and to rival causal and semantic notions of information. It is concluded that the primacy of genes in their structuring of molecules goes together with parity between genes and non-genetic causal factors in regulation of living systems. This is seen to hold independently of the radical reconceptualization of organism cum environment that has been suggested in developmental systems theory. (shrink)

There are striking parallels, largely unexplored in the literature, between skeptical arguments against theism and against moral realism. After sketching four arguments meant to do this double duty, I restrict my attention to an explanatory argument that claims that we have most reason to deny the existence of moral facts (and so, by extrapolation, theistic ones), because such putative facts have no causal-explanatory power. I reject the proposed parity, and offer reasons to think that the potential vulnerabilities of (...) moral realism on this front are quite different from those of the theist. Key Words: causal power • explanatory power • Gilbert Harman • moral facts • moral realism • theism. (shrink)

Biologists studying complex causal systems typically identify some factors as causes and treat other factors as background conditions. For example, when geneticists explain biological phenomena, they often foreground genes and relegate the cellular milieu to the background. But factors in the milieu are as causally necessary as genes for the production of phenotypic traits, even traits at the molecular level such as amino acid sequences. Gene-centered biology has been criticized on the grounds that because there is parity among (...) causes, the “privileging” of genes reflects a reductionist bias, not an ontological difference. The idea that there is an ontological parity among causes is related to a philosophical puzzle identified by John Stuart Mill: what, other than our interests or biases, could possibly justify identifying some causes as the actual or operative ones, and other causes as mere background? The aim of this paper is to solve this conceptual puzzle and to explain why there is not an ontological parity among genes and the other factors. It turns out that solving this puzzle helps answer a seemingly unrelated philosophical question: what kind of causal generality matters in biology? (shrink)

Recently internalists have mounted a counter-attack on the attempt to redefine the bounds of cognition. The counter-attack is aimed at a radical project which I call "cognitive integration," which is the view that internal and external vehicles and processes are integrated into a whole. Cognitive integration can be defended against the internalist counter arguments of Adams and Aizawa (A&A) and Rupert. The disagreement between internalists and integrationists is whether the manipulation of external vehicles constitutes a cognitive process. Integrationists think that (...) they do, typically for reasons to do with the close coordination and causal interplay between internal and external processes. The internalist criticisms of the manipulation thesis fail because they misconstrue the nature of manipulation, ignore the hybrid nature of cognition, and take the manipulation thesis to be dependent upon a weak parity principle. (shrink)

This chapter delves deeper into the two “waves” of arguments for EM as discussed in the last chapter. The first wave focuses on questions of functional parity between internal and external processes and focuses mainly on the functional role of causal coupling between internal and external vehicles. The second wave, on the other hand, focuses on questions regarding the complementarity of internal and external vehicles and their consequent integration into a cognitive whole. In contrast to the first, it (...) approaches cognition as being constituted by man’s bodily activities in the world in conjunction with neural processes and vehicles. In this chapter, the first-wave arguments are referred to as “extended-mind-style” arguments; the second-wave arguments are referred to as “cognitive integration-style” arguments. (shrink)

Small numbers are spontaneously associated with left space and larger numbers with right space (the SNARC effect), for example when classifying numbers by parity. This effect is often attributed to reading habits but a causal link has so far never been documented. We report that bilingual Russian-Hebrew readers show a SNARC effect after reading Cyrillic script (from left-to-right) that is significantly reduced after reading Hebrew script (from right-to-left). In contrast, they have similar SNARC effects after listening to texts (...) in either language. These results support the view that spatially directional scanning habits contribute to the spatial association of numbers but also emphasize its flexibility. (shrink)

What exactly is it that makes someone a parent? Many people hold that parenthood is grounded, in the first instance, in the natural derivation of one person's genetic constitution from the genetic constitutions of others. We refer to this view as "Geneticism". In Part I we distinguish three forms of geneticism on the basis of whether they hold that direct genetic derivation is sufficient, necessary, or both sufficient and necessary, for parenthood. Parts two through four examine three arguments for geneticism: (...) the property argument, the causal argument, and the parity argument. We conclude that none of these arguments succeeds. (shrink)

This paper offers a non-traditional account of what was really at stake in debates over the legitimacy of teleology and teleological explanations in the later medieval and early modern periods. It is divided into four main sections. The first section highlights two defining features of ancient and early medieval views on teleology, namely, that teleological explanations are on a par (or better) with efficient causal explanations, and that the objective goodness of outcomes may explain their coming about. The second (...) section argues that it was the first thesis – the thesis of explanatory parity –that came under attack by mainstream philosophers in the later medieval and early modern traditions, rather than teleology per se. The third section argues similarly that it was the second thesis – the thesis of explanatory goodness – that was the object of Spinoza’s critique of final causes, rather than teleology itself. Finally, the fourth section argues that Leibniz’s claim to be a champion of traditional teleology rests on his willingness to defend both the theses of explanatory parity and goodness against the very charges that led to their demise. The essay concludes by contrasting the narrative offered here with Don Garrett’s recent account according to which “it is not Leibniz but Spinoza who holds the position on teleology and teleological explanation nearest to that of Aristotle” (312). (shrink)

This paper explores several paths by which the extended cognition thesis may overcome the coupling-constitution fallacy. In so doing, I address a couple of shortcomings in the contemporary literature. First, on the dimension of first-wave EC, I argue that constitutive arguments based on functional parity suffer from either a threat of cognitive bloat or an impasse with respect to determining the correct level of grain in the attribution of causal-functional roles. Second, on the dimension of second-wave EC, I (...) argue that especially the complementarity approach suffers from a similar sort of dilemma as first-wave EC: an inability to justify just what entails the ontological claim of EC over the scaffolding claim of weaker approaches in cognitive science. In this paper I show that two much more promising explanations by which to ground the ontological claim of EC are available, both starting from an exploration of the coordination dynamics between environmental resources and neural resources. On the one hand, I argue that second-wave EC based on cognitive integration, with its focus on bodily manipulations constrained by cognitive norms, is capable of resolving the coupling-constitution fallacy. On the other hand, I argue that the framework of cognitive integration can be supplemented by philosophical accounts of mechanistic explanation, because such accounts enable us to explain the emergence of higher-level cognitive properties due to a system's organization-dependent structure. (shrink)

In a recent paper, Alvin Plantinga defends occasionalism against an important moral objection: if God is the sole direct cause of all the suffering that results from immoral human choices, this causal role is difficult to reconcile with God’s perfect goodness. Plantinga argues that this problem is no worse for occasionalism than for any of the competing views of divine causality; in particular, there is no morally relevant difference between God directly causing suffering and God indirectly causing it. First, (...) we examine Plantinga’s moral parity argument in detail and offer a critical evaluation of it. Then we provide a positive argument, based on the doctrine of doing and allowing, to show why there is a morally relevant difference between God’s direct and indirect causation of suffering. (shrink)

The CPT theorem states that any causal, Lorentz-invariant, thermodynamically well-behaved quantum field theory must also be invariant under a reflection symmetry that reverses the direction of time, flips spatial parity, and conjugates charge. Although its physical basis remains obscure, CPT symmetry appears to be necessary in order to unify quantum mechanics with relativity. This paper attempts to decipher the physical reasoning behind proofs of the CPT theorem in algebraic quantum field theory. Ultimately, CPT symmetry is linked to a (...) systematic reversal of the C*-algebraic Lie product that encodes the generating relationship between observables and symmetries. In any physically reasonable relativistic quantum field theory it is always possible to systematically reverse this generating relationship while preserving the dynamics, spectra, and localization properties of physical systems. Rather than the product of three separate reflections, CPT symmetry is revealed to be a single global reflection of the theory’s state space. (shrink)

The notion of innateness is widely used, particularly in philosophy of mind, cognitive science and linguistics. Despite this popularity, it remains a controversial idea. This is partly because of the variety of ways in which it can be explicated and partly because it appears to embody the suggestion that we can determine the relative causal contributions of genes and environment in the development of biological individuals. As these causes are not independent, the claim is metaphysically suspect. This paper argues (...) that there is a plausible reconstruction of the notion of innateness. This involves defining it sufficiently broadly to cover most of the current usages as well as making it an informational rather than a causal property. This has two consequences. Firstly, innateness becomes a matter of degree. Secondly, we have to abandon the idea, originally proposed by ethologists, that innate traits are necessarily the products of genetic information. (shrink)

The author of “Parsimony and inference to the best mathematical explanation” argues for platonism by way of an enhanced indispensability argument based on an inference to yet better mathematical optimization explanations in the natural sciences. Since such explanations yield beneficial trade-offs between stronger mathematical existential claims and fewer concrete ontological commitments than those involved in merely good mathematical explanations, one must countenance the mathematical objects that play a theoretical role in them via an application of the relevant mathematical results. The (...) nominalist’s challenge is thus to undermine the platonistic force of such explanations by way of alternative nominalistic ones. The author’s contention is that such nominalistic explanations should provide a paraphrase of the proofs of the mathematical results being applied. There are reasons to doubt that proofs, construed here as formal derivations, actually contribute to the platonistc force to be undermined and, by parity, that nominalized proofs should bear responsability for the corresponding undermining. A discussion of two examples and of associated arguments by Lange, Pincock, Steiner and Tallant, point to a a wealth of worries concerning the construal of this explanatory role. Among those figure the distinction between the weak and strong role of proofs, the distinction between causal or “ordinary” explanations and genuine mathematical ones, and the unifying role of optimization explanations. More generally, the very idea that the explanatory advantages yielded by applied mathematical claims may be construed as gradual or progressive and the associated notion that the feasibility of their nominalistic paraphrases decreases as the generality and force of these claim increases, deserves a closer attention. (shrink)

I introduce "nomic-role nonreductionism" as an alternative to traditional causal-role functionalism in the philosophy of mind. Rather than identify mental properties by a theory that describes their intra-level causal roles via types of inputs, internal states, and outputs, I suggest that one identify mental properties by a more comprehensive theory that also describes inter-level realization roles via types of lower-level engineering, internal mental states, and still higher-level states generated by them. I defend this position on grounds that mental (...) properties should be understood by our best scientific theories, which at present include informatioin about mental engineering. I further defend this claim by a "parity of reasons" argument. Causal-role functionalists are justified to include sensory stimuli in their theory of mind as opposed to, say, the remote causes of sensory stimuli because the former but not the latter are items of direct mental production. But ditto for the system's physical realizations. They too directly produce mental states, only not by "causing" them but by "realizing" them. Engineering realizations and their input triggering conditions work in tandem. In addition, I tell a related but more general metaphysical story about property identity, namely, that the traditional causal theory should be replaced by a more comprehensive nomic theory that individuates properties by their intra-level causal powers as well as their inter-level realization capacities. (shrink)

Transcranial direct current stimulation has been previously used to investigate the causal relationships between the dorsolateral prefrontal cortex and task switching but has delivered inconclusive results that may be due to different switching tasks involving different cognitive control processes. In the current study, we manipulated task types and task predictability to investigate the role of DLPFC in task-switching performances. Notably, we distinguished the specific effects of anodal-tDCS on two types of tasks. Forty-eight participants were randomly assigned to four task (...) groups as follows; Group I who was assigned right anode parity/magnitude tasks, Group II who were assigned sham parity/magnitude tasks, Group III who were assigned RA parity/vowel-consonant tasks, and Group IV who were assigned sham parity/vowel-consonant tasks. Participants were asked to complete both predictable and unpredictable tasks. In the parity/magnitude task, we demonstrated a lower switch cost for the RA group compared to the sham group for unpredictable tasks. In contrast, in the parity/vowel-consonant task, the switch cost was higher for the RA group compared to the sham group for unpredictable and predictable tasks. These findings confirmed an anodal-tDCS-induced effect over the right DLPFC both in the parity/magnitude and parity/vowel-consonant tasks. Our data indicated that anodal tDCS may have a stronger influence on task-switching performance over the right DLPFC by changing the irrelevant task-set inhibition process. Also, the right DLPFC is unlikely to act by performing exogenous adjustment of predictable task switching. (shrink)

Much of the literature directed at the Extended Mind Thesis has revolved around parity issues, focussing on the problem of how to individuate the functional roles and on the relevance of these roles for the production of human intelligent behaviour. Proponents of EMT have famously claimed that we shouldn’t take the location of a process as a reliable indicator of the mechanisms that support our cognitive behaviour. This functionalist understanding of cognition has however been challenged by opponents of EMT (...) [such as Rupert ; Adams & Aizawa ], who have claimed that differences between internal, biological processes and putatively extended ones not only exist but are actually crucial to undermine the idea that inner and outer are functionally equivalent. This debate about how to individuate the functional roles has led to a treacherous stand-off, in which proponents of EMT have been trapped under the persistent accusation of causal/constitution conflation. My strategy for responding to this charge is to look precisely at those functional differences highlighted by critics of EMT. I reckon that extended cognitive systems are endowed with quite different properties from systems that are “brain bound” and argue that it is precisely these differences that allow human minds to transcend their biological limitations. I thus defend a complementarity version of the extended mind, according to which externally located resources and internal biological elements make a different but complementary contribution to bringing about intelligent behaviour [Sutton ]. My defence of complementarity is based on both the phylogeny and the ontogeny of cognitive systems. I initially explore the interrelation between brain and cognitive development from a neuroconstructivist perspective [Quartz & Sejnowski ; Mareshal et al. ] and then argue that our brains do not have fixed functional architectures but are sculpted and given form by the activities we repeatedly engage in. As a result of repeated engagements in socio-cultural tasks, relevant brain pathways undergo substantial rewiring. Development thus scaffolds our brains, which become geared into working in symbiotic partnership with external resources. [Kiverstein & Farina ]. On these grounds, I call into question any tendency to interpret the human biological nature as fixed and endogenously pre-determined and side with proponents of DST [Oyama ; Griffiths & Gray ] and ontogenetic niche construction [Stotz ] in arguing that we should think of natural selection as operating on whole developmental systems composed of living organisms in culturally enriched niches. [Wheeler & Clark]. Complementarity defences of EMT argue that many of the kinds of cognition humans excel at can only be accomplished by brains working together with a body that directly manipulates and acts on the world [Rowlands ; Menary ]. I take Sensory Substitution Devices as my empirical case study. SSDs exploit the remarkable plasticity of our brains and with training supply a novel perceptual modality that compensates for loss or impaired sensory channel. I argue that the coupling with these devices triggers a new mode of phenomenal access to the world, something I propose to label as a kind of “artificial synaesthesia [Ward & Meijer ].This new mode of access to the world transforms our cognitive skills and gives rise to augmented processes of deep bio-technological symbiosis. SSDs therefore become mind enhancing tools [Clark ] and a perfect case study for Complementarity. Having shown the relevance of SSDs for EMT, I then take up the possibility that these devices don’t just relocate the boundaries of cognition but may also stretch the bounds of perceptual awareness. I explore the possibility that perceivers using SSDs count as extended cognitive systems and therefore argue that the experiences they enjoy should be counted as extended conscious experiences.[Kiverstein & Farina, ]. SSDs are quite often said to involve some form of incorporation.[Clark ]. Rupert has challenged this idea and its relevance for EMT on the grounds of his embedded approach. Particularly, he has explained tool-use in terms of the causal interaction between the subject and its detached tool. In the final chapter of my dissertation I critically look at his objections and argue that all his arguments fail to apply to SSDs. In SSD perception in fact the tool becomes geared to work in symbiotic partnership with the active subject and then get factored into its’ body schema so that both of them come to form a single system of cognitive analysis. (shrink)

This review article takes up certain key issues that are at stake in the valuable collection of essays edited by Lovell. It considers critically the argument that the adoption of Fraser's perspectival dualism implies regression to a base—superstructure theory of the social. It assesses the advantages of extending the dualism of redistribution and recognition to include also the need for participatory parity in the post-Westphalian political order. It raises again the question of whether Honneth is sociologically more forceful than (...) Fraser in arguing that material inequality is merely a facet of a more profound disrespect (lack of recognition). Lovell's assessment of Honneth's `recognition monism' is commended in that it resurrects an earlier critique of structural-functionalism to make a helpful distinction between social integration and (non-normative) system integration. She addresses these issues in terms of a wider critical realism concerned with disaggregating those social relations which are more causally generative from others: an approach which permits illuminating debates over the degree to which patriarchy and heteronormativity are contingent or necessary to late capitalism. Lovell also introduces Bourdieu into the debate, and especially his conceptions of misrecognition and symbolic violence. Finally, Sayer's vigorous arguments about the moral significance of distribution and class are considered as contributing to the current ethical turn. The article ends with an attempt to defend Bourdieu, whom Sayer sees both as a profound sociologist of class but also a theorist oriented too exclusively to agents' egoistic search for strategic advantage. The author concludes that although Fraser's perspectival trinity (or dualism) approach seems preferable to Honneth's monism, this should not obscure the empirical interweaving of deficiencies of distribution and of recognition in social reality. Indeed, new studies show the far-reaching consequences of this. In those societies where maldistribution is most marked, exposure to certain psychosocial risks such as low status (recognition) and low social capital literally incorporate stress, via biological pathways. Hence unequal health outcomes of a surprising range can also be detected. (shrink)

This article explores the main similarities and differences between Derek Parfit’s notion of imprecise comparability and a related notion I have proposed of parity. I argue that the main difference between imprecise comparability and parity can be understood by reference to ‘the standard view’. The standard view claims that 1) differences between cardinally ranked items can always be measured by a scale of units of the relevant value, and 2) all rankings proceed in terms of the trichotomy of (...) ‘better than’, ‘worse than’, and ‘equally good’. Imprecise comparability, which can be understood in terms of the more familiar notions of cardinality and incommensurability, rejects only the first claim while parity rejects both claims of the standard view. -/- I then argue that insofar as those attracted to imprecise comparability assume that all rankings are trichotomous, as Parfit appears to, the view should be rejected. This is because imprecise equality is not a form of equality but is a sui generis ‘fourth’ basic way in which items can be ranked. We should, I argue, understand imprecise equality as parity, and imprecise comparability as entailing ‘tetrachotomy’ – that if two items are comparable, one must better than, worse than, equal to, or on a par with the other. Thus those attracted to the idea that cardinality can be imprecise should abandon trichotomy and accept parity and tetrachotomy instead. -/- Finally, I illustrate the difference between Parfit’s trichotomous notion of imprecise comparability and parity by examining how each notion might be employed in different solutions to the problem posed by the Repugnant Conclusion in population ethics. I suggest that parity provides the arguably more ecumenical solution to the problem. (shrink)

Andy Clark and David Chalmers (1998) argue that certain mental states and processes can be partially constituted by objects located beyond one’s brain and body: this is their extended mind thesis (EM). But they maintain that consciousness relies on processing that is too high in speed and bandwidth to be realized outside the body (see Chalmers, 2008, and Clark, 2009). I evaluate Clark’s and Chalmers’ reason for denying that consciousness extends while still supporting unconscious state extension. I argue that their (...) reason is not well grounded and does not hold up against foreseeable advances in technology. I conclude that their current position needs re-evaluation. If their original parity argument works as a defence of EM, they have yet to identify a good reason why it does not also work as a defence of extended consciousness. I end by advancing a parity argument for extended consciousness and consider some possible replies. (shrink)

This paper begins with a response to Josh Gert’s challenge that ‘on a par with’ is not a sui generis fourth value relation beyond ‘better than’, ‘worse than’, and ‘equally good’. It then explores two further questions: can parity be modeled by an interval representation of value? And what should one rationally do when faced with items on a par? I argue that an interval representation of value is incompatible with the possibility that items are on a par (a (...) mathematical proof is given in the appendix). I also suggest that there are three senses of ‘rationally permissible’ which, once distinguished, show that parity does distinctive practical work that cannot be done by the usual trichotomy of relations or by incomparability. In this way, we have an additional argument for parity from the workings of practical reason. (shrink)

Sylviane Agacinski has never shied away from controversy. Vilified by some--including many feminists--and celebrated by others as a pioneer of gender equality, she has galvanized the French political scene. Her articulation of the theory of "parity" helped inspire a law that went into effect in May 2000 requiring the country's political parties to fill 50 percent of the candidacies in every race with women. Sylviane Agacinski, according to _The New Yorker,_ "is sometimes credited with making _parité_ respectable." Agacinski begins (...) with the notion that sexual difference should be affirmed rather than denied. Sex, Agacinski points out, is not a social, cultural, or ethnic characteristic--it is a universal human trait. In her argument for the necessary recognition of sexual difference, she enters into today's most controversial social territory. Agacinski's model of parity does not strive for the nebulous ideal of "equality" between the sexes; instead, it demands a concrete formula for political contests: an equal number of female and male candidates in every election. It is a theory that has sparked impassioned debate across France: Are female politicians necessarily different from male politicians? Is parity democratic? Is it truly feminist? Agacinski's sophisticated polemic will stimulate debate on American shores as it has in France. _Parity of the Sexes_ sheds light on one of the crucial spheres of public life in which earlier French feminists left their work unfinished--the realm of political power. (shrink)