Several authors have recently claimed that the notorious causal exclusion problem, according to which higher-level causes are threatened with causal pre-emption by lower-level causes, can be avoided if causal relevance is understood in terms of Woodward's interventionist account of causation. They argue that if causal relevance is defined in interventionist terms, there are cases where only higher-level properties, but not the lower-level properties underlying them, qualify as causes of a certain effect. In this (...) article, I show that the line of reasoning supposed to establish this claim does not succeed and that interventionism is not better capable of dealing with higher-level causal claims than other accounts of causation. According to Woodward, higher-level causal claims are nonetheless more adequate than lower-level ones if they describe a realization-independent dependency relationship and, hence, meet the requirement that causes should be proportional to their effects. I argue, however, that combining interventionism with proportionality considerations raises difficulties and that, therefore, Woodward's account does not vindicate higher-level causation. (shrink)

One of Stuart Glennan's most prominent contributions to the new mechanist debate consists in his reductive analysis of higher-level causation in terms of mechanisms (Glennan, 1996). In this paper I employ the causal Bayes net framework to reconstruct his analysis. This allows for specifying general assumptions which have to be satis ed to get Glennan's approach working. I show that once these assumptions are in place, they imply (against the background of the causal Bayes net machinery) that (...) higher-level causation indeed reduces to interactions between component parts of mechanisms. I also briefly discuss the plausibility of these assumptions and some consequences for the mechanism debate. (shrink)

Supporters of the autonomy of higher-level causation (or explanation) often appeal to proportionality, arguing that higher-level causes are more proportional than their lower-level realizers. Recently, measures based on information theory and causal modeling have been proposed that allow one to shed new light on proportionality and the related notion of specificity. In this paper we apply ideas from this literature to the issue of higher vs. lower-level causation (and explanation). Surprisingly, proportionality (...) turns out to be irrelevant for the question of whether higher-level causes (or explanations) can be autonomous; specificity is a much more informative notion for this purpose. (shrink)

We have a familiar idea of levels of description or levels of theory in science: microphysics, atomic physics, chemistry, biology, psychology, and the various social sciences. It is clear that philosophers - such as Terry Horgan - who want to be nonreductive materialists with regard to the mental must hold that this is not mere description; there must be genuine higher-level causes, and hence, genuine higher-level properties, in particular mental properties and causes. But there appears to (...) be a deep problem concerning mental causes. The (micro-) physical world is causally closed. Mental states are - or depend on or are realized by - physical states. It seems, then, that the physical state on which a mental state depends will be responsible for any alleged effects of the mental state. There will be no room for mental causation. And if properties exist insofar at they have a causal role, there will be no room for mental properties either. Many philosophers - Horgan included - have seen this problem of the "causalexclusion" ofthe mental as a specialcase of a general problem:the exclusion of higher-level causes by the causal closure of microphysics. Suppose one higher-level state, H1 leads to another higher-level state, H2. H1 is realized by some base level state, B1, which leads to a base-level state, B2, which in turn realizes H2. All of the casual work, so to speak, takes place at the base level. There is no room for any genuine causal connection between H1, as such, and H2, as such. I argue that there is no problem about higher-order causation in general. There are genuine, unsurprising higher-level causes and properties. A ball roles, for example, or breaks a window. If there is a problem of exclusion regarding putative mental causes, it is not an instance of a general exclusion problem, but is sui generis, and mental causation remains mysterious. (shrink)

A wide range of gene knockout experiments shows that functional stability is an important feature of biological systems. On this backdrop, we present an argument for higher‐level causation based on counterfactual dependence. Furthermore, we sketch a metaphysical picture providing resources to explain the metaphysical nature of functional stability, higher‐level causation, and the relevant notion of levels. Our account aims to clarify the role empirical results and philosophical assumptions should play in debates about reductionism and (...) higher‐level causation. It thereby contributes to the development of a philosophical foundation for systems biology. †To contact the authors, please write to: CSMN/IFIKK, University of Oslo, Box 1020 Blindern, N‐0315, Oslo, Norway; e‐mail: [email protected] , [email protected]. (shrink)

A central argument for non-reductive accounts of group agency is that complex social entities are capable of exerting causal influence independently of and superseding the causal efficacy of the individuals constituting them. A prominent counter is that non-reductionists run into an insuperable dilemma between identity and redundancy – with identity undermining independent higher-level efficacy and redundancy leading to overdetermination or exclusion. This paper argues that critics of non-reductionism can manage with a simpler and more persuasive reductio strategy called (...) mapping: allow that group agents are causally efficacious in their own right and chart how their causal efficacy is carried out; how it relates to the causal efficacy of individual determiners; how it connects to the causal relevance of background structural factors. The focus exclusively on whether groups are or are not causally efficacious black-boxes implementation, while close attention to how causation is wired increases the visibility of individualist arguments and countenances structure-oriented explanations. (shrink)

Kim (1998) has argued that a genuine robust physicalism does not leave any room for real, causally efficacious mental properties. Despite all of his concerns about the reality and causal efficacy of mental phenomena Kim does not eliminate all higher-level macro causation. Kim’s problem with the mental is that most current cognitive theories imply that the mind is not higher-level but higher-order. In this paper I argue that connectionism makes meaning higher-level and (...) therefore by Kim’s own standards puts meaning on the same footing as other real causally efficacious higher-level properties. The upshot is that we can side-step the current debate about mental causation by moving the mind to the shared uncontroversially real and physical common-ground. (shrink)

In their Every Thing Must Go, Ladyman and Ross defend a novel version of Neo- Russellian metaphysics of causation, which falls into three claims: (1) there are no fundamental physical causal facts (orthodox Russellian claim), (2) there are higher-level causal facts of the special sciences, and (3) higher-level causal facts are explanatorily emergent. While accepting claims (1) and (2), I attack claim (3). Ladyman and Ross argue that higher-level causal facts are explanatorily emergent, (...) because (a) certain aspects of these higher-level facts (their universality) can be captured by renormalization group (RG) explanations, and (b) RG explanations are not reductive explanations. However, I argue that RG explanation should be understood as reductive explanations. This result undermines Ladyman and Ross’s RG-based argument for the explanatory emergence of higher-level causal facts. (shrink)

The idea of a higher level phenomenon having a downward causal influence on a lower level process or entity has taken a variety of forms. In order to discuss the relation between emergence and downward causation, the specific variety of the thesis of downward causation (DC) must be identified. Based on some ontological theses about inter-level relations, types of causation and the possibility of reduction, three versions of DC are distinguished. Of these, the (...) `Strong' form of DC is held to be in conflict with contemporary science; the `Medium' version of DC may for instance describe thoughts constraining neurophysiological states, while the `Weak' form of DC is physically acceptable but may not in practice be a feasible description of the mind/brain or the cell/molecule relation. All forms have their specific problems, but the Medium and the Weak version seems to be most promising. (shrink)

I have two main aims. The first is general, and more philosophical. The second is specific, and more closely related to physics. The first aim is to state my general views about laws and causation at different ”levels’. The main task is to understand how the higher levels sustain notions of law and causation that ”ride free’ of reductions to the lower level or levels. I endeavour to relate my views to those of other symposiasts. The (...) second aim is to give a framework for describing dynamics at different levels, emphasizing how the various levels’ dynamics can mesh or fail to mesh. This framework is essentially that of elementary dynamical systems theory. The main idea will be, for simplicity, to work with just two levels, dubbed ”micro’ and ”macro’, which are related by coarse-graining. I use this framework to describe, in part, the first four of Ellis’ five types of top-down causation. (shrink)

This paper tracks the commitments of mechanistic explanations focusing on the relation between activities at different levels. It is pointed out that the mechanistic approach is inherently committed to identifying causal connections at higher levels with causal connections at lower levels. For the mechanistic approach to succeed a mechanism as a whole must do the very same thing what its parts organised in a particular way do. The mechanistic approach must also utilise bridge principles connecting different causal terms of (...) different theoretical vocabularies in order to make the identities of causal connections transparent. These general commitments get confronted with two claims made by certain proponents of the mechanistic approach: William Bechtel often argues that within the mechanistic framework it is possible to balance between reducing higher levels and maintaining their autonomy at the same time, whereas, in a recent paper, Craver and Bechtel argue that the mechanistic approach is able to make downward causation intelligible. The paper concludes that the mechanistic approach imbued with identity statements is no better candidate for anchoring higher levels to lower ones while maintaining their autonomy at the same time than standard reductive accounts are, and that what mechanistic explanations are able to do at best is showing that downward causation does not exist. (shrink)

The standard objection to dualist theories of mind is that they seemingly cannot account for the obvious fact that mental phenomena cause our behaviour. On the plausible assumption that all our behaviour is physically necessitated by entirely physical phenomena, there appears to be no room for dualist mental causation. Some argue that dualists can address this problem by making minimal adjustments in their ontology. I argue that no such adjustments are required. Given recent developments in philosophy of causation, (...) it is plausible that mental phenomena cause behaviour in standard dualist ontologies. (shrink)

In this paper, we analyse the fruitfulness of Ronald Giere’s comparative model for causation in populations. While the original model was primarily developed to capture the meaning of causal claims in the biomedical and health sciences, we want to show that the model is not only useful in these domains, but can also fruitfully be applied to other scientific domains. Specifically, we demonstrate that the model is fruitful for characterizing the meaning of causal claims found in classical genetics, epidemiology (...) and electoral sociology. Additionally, we propose an adapted comparative model which is needed to get a grip on higher level causal claims. We show that such claims are present in population genetics, epidemiology and electoral systems research. We conclude with some reflections on the implications of our findings for the issue of causal pluralism. (shrink)

This article considers interventionist arguments for downward causation and non-fundamental level causal explanation from the point of view of inferring causation from experiments. Several authors have utilised the interventionist theory of causal explanation to argue that the causal exclusion argument is moot and that higher-level as well as downward causation is real. I show that this argument can be made when levels are understood as levels of grain, leaving us with a choice between causal (...) explanations pitched at different levels. Causal proportionality has been suggested as a principle for choosing the correct level of description for causal explanations, but this suggestion has serious problems. I offer an alternative principle, based on the idea that explanations should track actual difference-makers. I then consider claims about systems causally influencing their own parts. Such claims risk conceptual confusion for the same reason as the exclusion argument, by conflating causal and constitutive dependencies. The distinction between causation and constitution is used to give criteria for the correct use of the idea of downward causation. (shrink)

The issue of downward causation (and mental causation in particular), and the exclusion problem is discussed by taking into account some recent advances in the philosophy of science. The problem is viewed from the perspective of the new interventionist theory of causation developed by Woodward. It is argued that from this viewpoint, a higher-level (e.g., mental) state can sometimes truly be causally relevant, and moreover, that the underlying physical state which realizes it may fail to (...) be such. (shrink)

In many biological experiments, due to gene-redundancy or distributed backup mechanisms, there are no visible effects on the functionality of the organism when a gene is knocked out or down. In such cases there is apparently no counterfactual dependence between the gene and the phenotype in question, although intuitively the gene is causally relevant. Due to relativity of causal relations to causal models, we suggest that such cases can be handled by changing the resolution of the causal model that represents (...) the system. By decreasing the resolution of our causal model, counterfactual dependencies can be established at a higher level of abstraction. By increasing the resolution, stepwise causal dependencies of the right kind can serve as a sufficient condition for causal relevance. Finally, we discuss how introducing a temporal dimension in causal models can account for causation in cases of non-modular systems dynamics. (shrink)

Emergence is interpreted in a non-dualist framework of thought. No metaphysical distinction between the higher and basic levels of organization is supposed, but only a duality of modes of access. Moreover, these modes of access are not construed as mere ways of revealing intrinsic patterns of organization: They are supposed to be constitutive of them, in Kant’s sense. The emergent levels of organization, and the inter-level causations as well, are therefore neither illusory nor ontologically real: They are objective (...) in the sense of transcendental epistemology. This neo-Kantian approach defuses several paradoxes associated with the concept of downward causation, and enables one to make good sense of it independently of any prejudice about the existence (or inexistence) of a hierarchy of levels of being. (shrink)

This paper defends the notion of downward causation. I will seek to elucidate this notion, explain why it is a useful way of thinking, and respond to criticisms attacking its intelligibility. My account of downward causation will be in many respects similar to the account recently advanced by Ellis. The overall framework I will adopt is the interventionist treatment of causation I have defended elsewhere: X causes Y when Y changes under a suitable manipulation of X. When (...) X is at a higher “level” than Y this allows for the possibility of downward causation from X to Y. True claims of downward causation must meet certain additional conditions, some of which have already been discussed by Ellis. These include the condition that X must have a homogenous effect on Y in the sense that the effect of X on Y must be the same regardless of how X is “realized” at lower levels. In addition, the most plausible examples of downward causation will involve causes X, that in a sense that I will try to specify, are capable of being manipulated by macro-level interventions that have a coordinated or organized impact on them, as when one manipulates the temperature of a gas by placing it in a heat bath.Three common criticisms of the notion of downward causation that I will consider are: the claim that this involves a whole acting downward on its parts which is an objectionable idea because wholes and parts are not sufficiently distinct to stand in causal relationships, that downward causation commits us to the existence of causal cycles in which X causes Y which in turn causes X and that the asymmetric nature of the causal relation rules out such cycles, and causal exclusion type worries, according to which all of the causal action occurs among “low level” variables, so that upper level variables are deprived of causal efficacy. In response I will argue that plausible examples of downward causation in the scientific literature do not involve whole to part causation, there is nothing wrong with causal cycles, which are common in, for example, biological contexts, exclusion type worries do not arise within the interventionist framework that I favor. (shrink)

Frank Jackson, Philip Pettit, and Jaegwon Kim put forward two models of higher-level causal explanation. Advocates of both versions are inclined to draw the conclusion that the models don't differ substantially. I argue, on the contrary, that there are relevant metaphysical differences between Jackson and Pettit's notion of programme explanation on the one hand, and Kim's idea of supervenient causation on the other. These can be traced back to underlying differences between the contents of their physicalisms.

In this paper I distinguish two types of mental causation, called 'higher-level causation' and 'exploitation'. These notions superficially resemble the traditional problematic notions of supervenient causation and downward causation, but they are different in crucial respects. My new distinction is supported by a radically externalist competitor of the so-called Standard View of mental states, i.e. the view that mental states are brain states. I argue that on the Alternative View, the notions of 'higher- (...) class='Hi'>level causation' and 'exploitation' can in combination dissolve the problem of mental causation as standardly discussed. (shrink)

The methodological nonreductionism of contemporary biology opens an interesting discussion on the level of ontology and the philosophy of nature. The theory of emergence (EM), and downward causation (DC) in particular, bring a new set of arguments challenging not only methodological, but also ontological and causal reductionism. This argumentation provides a crucial philosophical foundation for the science/theology dialogue. However, a closer examination shows that proponents of EM do not present a unified and consistent definition of DC. Moreover, they (...) find it difficult to prove that higher-order properties can be causally significant without violating the causal laws that operate at lower physical levels. They also face the problem of circularity and incoherence in their explanation. In our article we show that these problems can be overcome only if DC is understood in terms of formal rather than physical (efficient) causality. This breakdown of causal monism in science opens a way to the retrieval of the fourfold Aristotelian notion of causality. (shrink)

The Exclusion Argument, which aims to deny the causal efficacy of irreducible mental properties, is probably the most serious challenge to non-reductive physicalism. Many proposed solutions to the exclusion problem can only reject simplified exclusion arguments, but fail to block a sophisticated version I introduce. In this paper, I attempt to show that we can refute the sophisticated exclusion argument by appeal to a sophisticated understanding of causation, what I call the 'Dual-condition Conception of Causation'. Specifically, I argue (...) that the dual-condition account of causation gives strong support to the so-called 'Autonomy Solution', which contends that even if mental properties are unable to cause (fundamental) physical properties, they can still cause higher-level properties (such as mental, behavioral, and social properties)—if so, human agency would be preserved in the physical world. (shrink)

Downward causation is a widespread and problematic phenomenon. It is typically defined as the causation of lower-level effects by higher-level entities. Downward causation is widespread, as there are many examples of it across different sciences: a cell constraints what happens to its own constituents; a body regulates its own processes; two atoms, when they are appropriately related, make it the case that their own electrons are distributed in certain ways. However, downward causation is (...) also problematic. Roughly, it seems to be at odds with specific scientific and/or epistemological desiderata: first and foremost, that everything can be reduced (one day or another) to the fundamental, micro-physical constituents and goings-on of the universe, so as to provide a unified explanation of everything and a unification of all the sciences " from the bottom ". Indeed, downward causation (if it is an irreducible phenomenon) introduces special causings not only at the higher levels, but also at the lower ones: if, in principle, we cannot fully understand what happens to the electrons without paying attention to the atoms (at the higher level) and we cannot fully understand what happens to the atoms by only paying attention to the electrons (at the lower level), there is no fully lower-level explanation for both higher-level and lower-level goings-on. In this introduction, we shall try to describe the prospects for downward causation in metaphysics and the philosophy of science. After having delved into the connections between downward causation, emergence and levels (§1), we shall discuss the irreducibility of downward causation (§2). We shall then briefly consider how specific metaphysical and epistemological assumptions bear on our understanding of downward causation and of its possibility (§3) and describe some views according to which downward causation is actually non-causal (or it is a special causal relation) (§4). We shall also mention some problems for the connection between downward causation and mental causation (§5) and some scientific examples of downward causation (§6). Finally, we shall summarize the contents of the contributions in this book (§7). (shrink)

What exactly do social scientists and biologists say when they make causal claims? This question is one of the central puzzles in philosophy of science. Alexander Reutlinger sets out to answer this question. He aims to provide a theory of causation in the special sciences (that is, a theory causation in the social sciences, the biological sciences and other higher-level sciences). According one recent prominent view, causation is that causation is intimately tied to manipulability (...) and the possibility of intervene. Reutlinger's main negative target is to argue interventionist account of causation is not adequate. Where do interventionist accounts go wrong? Reutlinger argues that the central concept of the interventionist theories – that is, the very concept of an intervention – is tremendously problematic. Reutlinger's main positive claim consists in replacing the interventionist approach by an alternative explication of causation in the special sciences, the comparative variability theory of causation. This alternative preserves many insights of the interventionist account without a commitment to the claim that causation and interventions are intimately tied together. (shrink)

In many fields in the life sciences investigators refer to downward or top-down causal effects. Craver and Bechtel defended the view that such cases should be understood in terms of a constitution relation between levels in a mechanism and causation as solely an intra-level relation. Craver and Bechtel, however, provided insufficient specification as to when entities constitute a higher-level mechanism. In this paper I appeal to graph-theoretic representations of networks that are now widely employed in systems (...) biology and neuroscience to identify mechanisms with the modules that exhibit high clustering. As a result of the interconnections of nodes in these modules/mechanisms, they often exhibit complex dynamic behaviors that constrain how the individual components respond to external inputs, an important feature of cases viewed as involving top-down causation. (shrink)

Enabling constraints are bottom up causes which create the possibility of the existence of a system. Disabling constraints reduce the degrees of freedom and narrow the choices of the system which are structural, functional, meaningful relations that assign executive roles to the component parts. In this paper, we discuss causality as enabling and disabling constraints in order to critique the absurdity of transitivity in causal relations. If downward causation is viewed as causation by constraints, we argue that it (...) will not face the absurdity of self-causation which is incurred because of the transitivity principle. In the process of self-organization, organizationally new structures are created from the lower level subcomponents of the system. Such self-organizing systems can be broadly considered as sui-generis and transcend the transitivity principle by causally influencing its subcomponents. These higher level systemic constraints that causally influence the components in a self-organizing system without violating any physical laws via causal constraints are termed as Downward Causation. (shrink)

This paper argues that scale-dependence of physical and biological processes offers resistance to reductionism and has implications that support a specific kind of downward causation. I demonstrate how insights from multiscale modeling can provide a concrete mathematical interpretation of downward causation as boundary conditions for models used to represent processes at lower scales. The autonomy and role of macroscale parameters and higher-level constraints are illustrated through examples of multiscale modeling in physics, developmental biology, and systems biology. (...) Drawing on these examples, I defend the explanatory importance of constraining relations for understanding the behavior of biological systems. (shrink)

Physics underlies all complexity, including our own existence: how is this possible? How can our own lives emerge from interactions of electrons, protons, and neutrons? This book considers the interaction of physical and non-physical causation in complex systems such as living beings, and in particular in the human brain, relating this to the emergence of higher levels of complexity with real causal powers. In particular it explores the idea of top-down causation, which is the key effect allowing (...) the emergence of true complexity and also enables the causal efficacy of non-physical entities, including the value of money, social conventions, and ethical choices. (shrink)

Michael Polanyi argues that in the case of both organisms and machines the functionality of the higher level imposes boundary conditions that harness the operations of lower level components in the service of the higher level, systemic whole. Given the science of his day, however, Polanyi understands this shaping of boundary conditions in terms of the operation of an external agency. The essay argues that the science of nonlinear, far from equilibrium thermodynamics in general, and (...) the phenomenon of autocatalysis in particular, explains how the endogenous closure of context-sensitive dynamic constraints shapes their boundary conditions such that self-organized, causally effective properties emerge. (shrink)

Digital computers carry out algorithms coded in high level programs. These abstract entities determine what happens at the physical level: they control whether electrons flow through specific transistors at specific times or not, entailing downward causation in both the logical and implementation hierarchies. This paper explores how this is possible in the light of the alleged causal completeness of physics at the bottom level, and highlights the mechanism that enables strong emergence to occur. Although synchronic emergence (...) of higher levels from lower levels is manifestly true, diachronic emergence is generically not the case; indeed we give specific examples where it cannot occur because of the causal effectiveness of higher level variables. (shrink)

Digital computers carry out algorithms coded in high level programs. These abstract entities determine what happens at the physical level: they control whether electrons flow through specific transistors at specific times or not, entailing downward causation in both the logical and implementation hierarchies. This paper explores how this is possible in the light of the alleged causal completeness of physics at the bottom level, and highlights the mechanism that enables strong emergence (the manifest causal effectiveness of (...) application programs) to occur. Although synchronic emergence of higher levels from lower levels is manifestly true, diachronic emergence is generically not the case; indeed we give specific examples where it cannot occur because of the causal effectiveness of higher level variables. (shrink)

The philosopher Roberto Unger and the physicist Lee Smolin have recently argued that the current explanatory framework of cosmology, which presumes a timeless background of unchanging physical laws, should be replaced by a thoroughly relational framework in which time is fundamental and all laws are subject to change. Within this alternative framework, however, Unger and Smolin find themselves confronted by a dilemma: either the laws of nature evolve according to some higher set of “meta-laws,” which reinstates a timeless background (...) at a higher level, or the laws of nature evolve randomly and the path of inquiry is blocked. In response to this dilemma, the authors propose a theory of causal events as singular, freely chosen, and self-determined optimalities constrained only by their intrinsic relations to all other events. (shrink)

According to the so-called 'proportionality principle', causes should be proportional to their effects: they should be both enough and not too much for the occurrence of their effects. This principle is the subject of an ongoing debate. On the one hand, many maintain that it is required to address the problem of causal exclusion and take it to capture a crucial aspect of causation. On the other hand, many object that it renders accounts of causation implausibly restrictive and (...) often reject the principle wholesale. I argue that there is exaggeration on both sides. While one half of the principle is overly demanding, the other half is unobjectionable. And while the unobjectionable half does not block exclusion arguments on its own, it provides a nuanced picture of higher-level causation, fits with recent developments in philosophy of causation, and motivates adjustments to standard difference-making accounts of causation. I conclude that at least half of the proportionality principle is worth taking seriously. (shrink)

It seems that higher-level, nonbasic properties can only manifest their causal powers by exerting causal influence on lower-level, physically basic phenomena in the first place. A very influential line of reasoning conceives of this form of downward causation as either reducible to causation by physical properties or as ultimately untenable, because incompatible with the causal closure of physical reality. The paper argues that this is not so. It examines, first, why it is that a recent (...) attempt by Noordhof to substantiate the notion of supervenient causation in a nonreductive framework fails. The upshot of this examination is the claim that any attempted specification of the most basic causal factors which supposedly underlie a causal transaction cannot account for the counterfactually necessary connections with the effect in question. By contrast, the specification of these factors at a higher level would allow establishing such connections. The paper closes with a discussion of how this view of autonomous causation at the higher-level can coexist with the notion of a complete specification of the causes of any physical effect exclusively in physical terms. (shrink)

We argue that embedded cognition provides an argument against Jaegwon Kim’s neural reduction of mental causation. Because some mental, or at least psychological processes have to be cast in an externalist way, Kim’s argument can be said to lead to the conclusion that mental causation is as safe as any other form of higher-level of causation.

This commentary explores biological models of analogical and associative learning in support of Illusion 1 and Illusion 4 in D. Noble’s target article. The intent is to support Noble’s theses of emergent higher level functionality from lower level stochastic dynamics and his etiological claim that “there is no privileged level of causation” through a biosemiotic lens. Upon these arguments, a case for creative agency via higher-dimensional constraints will also be made in support of Noble’s (...) claim that organismic behavior is actively directed by an agent. (shrink)

Two conceptual frameworks – in terms of phase space and in terms of structural equations – are sketched, in which downward causal influence of higher-level features on lower-level features is possible. The “Exclusion” principle, which is a crucial premise of the argument against the possibility of downward causation, is false in models constructed within both frameworks. Both frameworks can be supplemented with conceptual tools that make it possible to explain why downward causal influence is not only (...) conceivable and compatible with the “Closure” principle, but also why it is often relevant to causally explain facts in terms of downward causal influence. It is briefly shown that the analysis of downward causation in the two frameworks complements Bennett’s analysis of overdetermination, the analysis does not entail the failure of the “Closure” principle and it does not require the postulate of synchronic downward causation. (shrink)

I develop and defend a model of downward causation denoted as downward, structural, substance causation. This model is based on the idea that higher-level, strongly emergent substances can cause something at lower levels by imposing certain structures on lower-level goings-on. After providing a sketch of the model and of its ontological assumptions, I apply it to the analysis of how free mental causes can structure neural goings-on. I defend three theses following from such an analysis, (...) and I test the empirical plausibility of the model. Finally, I anticipate some objections and replies. (shrink)

This article reviews the causal exclusion argument and confronts it with some recently proposed refutations based on the interventionist account of causation. I first show that there are several technical and interpretative difficulties in applying the interventionist account to the exclusion issue. Different ways of accommodating the two to one another are considered and all are shown to leave the issue without a fully satisfactory resolution. Lastly, I argue that, on the most consistent construal, the interventionist approach can provide (...) grounds for thinking that higher-level causal notions are as legitimate as lower-level causal notions, but it does not provide grounds for postulating inter-level causal interactions. (shrink)

I discuss whether Michael Tye, in Ten Problems of Consciousness. Cambridge, Massachusetts: MIT Press, 1966, holds that phenomenal properties are neurological properties, but that what gives them their phenomenal property names are their highly complex interconnections with other neurological properties and, most especially, subjects' surroundings. Or, alternatively, whether he holds that they are higher-level, wide functional properties in the sense of being properties of having properties that fill some specified wide or distal roles.

Our aim in this paper is to evaluate Frank Jackson and Philip Pettit’s ‘program explanation’ framework as an account of the autonomy of the special sciences. We argue that this framework can only explain the autonomy of a limited range of special science explanations. The reason for this limitation is that the framework overlooks a distinction between two kinds of properties, which we refer to as ‘higher-level’ and ‘higher-order’ properties. The program explanation framework can account for the autonomy (...) of special science explanations that appeal to higher-level properties but it does not account for the autonomy of most of those explanations that appeal to higher-order properties. (shrink)

There is a difference between someone breaking a glass by accidentally brushing up against it and smashing a glass in a fit of anger. In the first case, the person's cognitive state has little to do with the event, but in the second, the mental state qua anger is quite relevant. How are we to understand this difference? What is the proper way to understand the relation between the mind, the brain, and the resultant behavior? This paper explores the popular (...) "middle ground" reply in which mental phenomena are claimed to be "as real as" other higher level properties. It argues that this solution fails to answer epistemological difficulties surrounding how to chose the appropriate factors in an explanation. A more sophisticated understanding of scientific theorizing and of the relation between ontology and explanation give us a framework in which we can determine when we should refer to mental states as being the causally efficacious agents for some behavior. (shrink)

Does inferential justification require the subject to be aware that her premises support her conclusion? Externalists tend to answer “no” and internalists tend to answer “yes”. In fact, internalists often hold the strong higher-level requirement that an argument justifies its conclusion only if the subject justifiably believes that her premises support her conclusion. I argue for a middle ground. Against most externalists, I argue that inferential justification requires that one be aware that her premises support her conclusion. Against (...) many internalists, I argue that this higher-level awareness needn’t be doxastic or justified. I also argue that the required higher-level awareness needn’t be caused in some appropriate way, e.g. by a reliable or properly functioning faculty. I suspect that this weaker higher-level requirement is overlooked because, at first glance, it seems absurd to allow nondoxastic, unjustified, and unreliably-caused higher-level awareness to contribute to inferential justification. One of the central goals of this paper is to explain how such weak awareness can make an essential contribution to inferential justification. (shrink)

Top-down causation is often taken to be a metaphysically suspicious type of causation that is found in a few complex systems, such as in human mind-body relations. However, as Ellis and others have shown, top-down causation is ubiquitous in physics as well as in biology. Top-down causation occurs whenever specific dynamic behaviors are realized or selected among a broader set of possible lower-level states. Thus understood, the occurrence of dynamic and structural patterns in physical and (...) biological systems presents a problem for reductionist positions. We illustrate with examples of universality and functional equivalence classes how higher-level behaviors can be multiple realized by distinct lower-level systems or states. Multiple realizability in both contexts entails what Ellis calls “causal slack” between levels, or what others understand as relative explanatory autonomy. To clarify these notions further, we examine procedures for upscaling in multi-scale modeling. We argue that simple averaging strategies for upscaling only work for simplistic homogenous systems, because of the scale-dependency of characteristic behaviors in multi-scale systems. We suggest that this interpretation has implications for what Ellis calls mechanical top-down causation, as it presents a stronger challenge to reductionism than typically assumed. (shrink)

This article examines Nietzsche’s analysis of the phenomenology of agent causation. Sense of agent causation, our sense of self-efficacy, is tenacious because it originates, according to Nietzsche’s hypothesis, in the embodied and situated experience of effort in overcoming resistances. It arises at the level of the organism and is sustained by higher-order cognitive functions. Based on this hypothesis, Nietzsche regards the sense of self as emerging from a homeostatic system of drives and affects that unify such (...) as to maintain self-efficacy levels. He relies on the same hypothesis to explain the emergence of an ascetic moral system and its specific, interpretive-affective ‘mechanism of willing’. The article aligns Nietzsche’s account of agent causation with Albert Bandura’s self-efficacy studies and Antonio Damasio’s recent account of self-systems as homeostatic systems. (shrink)

Multidisciplinary models aggregating ‘lower-level’ biological and ‘higher-level’ psychological and social determinants of a phenomenon raise a puzzle. How is the interaction between the physical, the psychological and the social conceptualized and explained? Using biopsychosocial models of pain as an illustration, I argue that these models are in fact level-neutral compilations of empirical findings about correlated and causally relevant factors, and as such they neither assume, nor entail a conceptual or ontological stratification into levels of description, explanation (...) or reality. If inter-level causation is deemed problematic or if debates about the superiority of a particular level of description or explanation arise, these issues are fueled by considerations other than empirical findings. (shrink)

Multidisciplinary models aggregating ‘lower-level’ biological and ‘higher-level’ psychological and social determinants of a phenomenon raise a puzzle. How is the interaction between the physical, the psychological and the social conceptualized and explained? Using biopsychosocial models of pain as an illustration, I argue that these models are in fact level-neutral compilations of empirical findings about correlated and causally relevant factors, and as such they neither assume, nor entail a conceptual or ontological stratification into levels of description, explanation (...) or reality. If inter-level causation is deemed problematic or if debates about the superiority of a particular level of description or explanation arise, these issues are fueled by considerations other than empirical findings. (shrink)