For the framework of event causation—i.e. the framework according to which causation is a relation between events—absences or omissions pose a problem. Absences, it is generally agreed, are not events; so, under the framework of event causation, they cannot be causally related. But, as a matter of fact, absences are often taken to be causes or effects. The problem of absence causation is thus how to make sense of causation that apparently involves absences as (...) causes or effects. In an influential paper, Helen Beebee offers a partial solution to the problem by giving an account of causation by absence. I argue that Beebee's account can be extended to cover causation of absence as well. More importantly, I argue that the extended Beebeeian account calls for a major modification to David Lewis's theory of causal explanation, usually taken as standard. Compared to the standard theory, the.. (shrink)

Several theories of causation reject causation of or by absences. They thereby clash with much of what we think and say about what causes what. This paper examines a way in which one kind of theory, causal dispositionalism, can be modified so as to accept absence causation, while still retaining a fundamental commitment of dispositionalism. The proposal adopts parts of a strategy described by David Lewis. The paper concludes with a brief discussion of the problem of (...) the proliferation of causes. (shrink)

If _presentism_ is true, then no wholly non-present events exist. If _absence orthodoxy_ is true, then no absences exist. I discuss a well-known causal argument against presentism, and develop a very similar argument against absence orthodoxy. I argue that solutions to the argument against absence orthodoxy can be adopted by the presentist as solutions to the argument against presentism. The upshot is that if the argument against absence orthodoxy fails, then so does the argument against presentism.

A major criticism of David Lewis’ counterfactual theory of causation is that it allows too many things to count as causes, especially since Lewis allows, in addition to events, absences to be causes as well. Peter Menzies has advanced this concern under the title “the problem of profligate causation.” In this paper, I argue that the problem of profligate causation provides resources for exposing a tension between Lewis’ acceptance of absence causation and his modal realism. (...) The result is a different problem of profligate causation—one that attacks the internal consistency of Lewisian metaphysics rather than employing common sense judgments or intuitions that conflict with Lewis’ extensive list of causes. (shrink)

I argue that so-called ‘absence causation’must be treated in terms of counterfactuals about causation such as ‘had a occurred, a would have caused b’. First, I argue that some theories of causation that accept absence causation are unattractive because they undermine the idea of possible causation. And second, I argue that accepting absence causation violates a principle commonly associated with relativity.

In this paper, I defend a counterfactual approach to causation by absences from some recent criticisms due to Sartorio.

In this paper, we argue that, omissions are not events or actions, but rather fact-like entities, and that, insofar as only events and actions can be causes, omissions cannot be causes. Nevertheless, since omissions can, and often do, play a role in the explanations of events, their place in such explanations must be found; and an attempt to find such a place is made.

This item has been retired at the request of its author.

Many photographs seem to be images of absences: for instance, a photograph of a shadow seems to be an image of an absence, as shadows are plausibly thought of as being absences of light. Absence photography is puzzling, however, as, first, it is a common idea that photographs can only be images of things that have caused them, and, second, it is unclear whether absences can cause anything. In this paper, I look at various ways to unravel the (...) puzzle. Along the way, I also hope to cast some light on the idea that photography is a causal medium. (shrink)

Many photographs seem to be images of absences: for instance, a photograph of a shadow seems to be an image of an absence, as shadows are plausibly thought of as being absences of light. Absence photography is puzzling, however, as, first, it is a common idea that photographs can only be images of things that have caused them, and, second, it is unclear whether absences can cause anything. In this paper, I look at various ways to unravel the (...) puzzle. Along the way, I also hope to cast some light on the idea that photography is a causal medium. (shrink)

This paper comments on Carolina Sartorio’s Causation and Free Will, challenging the non-modal conception of reasons-sensitivity that Sartorio advances.

Butterfield's (1992a,b,c) claim of the equivalence of absence of Lewisian probabilistic counterfactual causality (LC) to Hellman's stochastic Einstein locality (SEL) is questioned. Butterfield's assumption on which the proof of his claim is based would suffice to prove that SEL implies absence of LC also for appropriately given versions of these notions in algebraic quantum field theory, but the assumption is not an admissible one. The conclusion must be that the relation of SEL and absence of LC is (...) open, and that they may be independent. (shrink)

Causation is at once familiar and mysterious. Neither common sense nor extensive philosophical debate has led us to anything like agreement on the correct analysis of the concept of causation, or an account of the metaphysical nature of the causal relation. Causation: A User's Guide cuts a clear path through this confusing but vital landscape. L. A. Paul and Ned Hall guide the reader through the most important philosophical treatments of causation, negotiating the terrain by taking (...) a set of examples as landmarks. They clarify the central themes of the debate about causation, and cover questions about causation involving omissions or absences, preemption and other species of redundant causation, and the possibility that causation is not transitive. Along the way, Paul and Hall examine several contemporary proposals for analyzing the nature of causation and assess their merits and overall methodological cogency.The book is designed to be of value both to trained specialists and those coming to the problem of causation for the first time. It provides the reader with a broad and sophisticated view of the metaphysics of the causal relation. (shrink)

Folk theories—untutored people’s (often implicit) theories about various features of the world—have been fashionable objects of inquiry in psychology for almost two decades now (e.g., Hirschfeld and Gelman 1994), and more recently they have been of interest in experimental philosophy (Nichols 2004). Folk theories of psy- chology, physics, biology, and ethics have all come under investigation. Folk meta- physics, however, has not been as extensively studied. That so little is known about folk metaphysics is unfortunate for (at least) two reasons. (...) First, folk metaphysics is almost certainly implicit, and it is likely to be our default way of thinking about metaphysical problems. Moreover, one’s metaphysical commitments can have pro- found consequences—in scientific, religious, and ethical contexts, for example. Thus, folk metaphysics ought to be dragged out into the open and exposed to criticism. As Peirce eloquently remarked (1994, 1.129; see also 1994, 7.579). (shrink)

Carolina Sartorio argues that only the actual causes of our behaviour matter to our freedom. The key, she claims, lies in a correct understanding of the role played by causation in a view of that kind. Causation has some important features that make it a responsibility-grounding relation, and this contributes to the success of the view. Also, when agents act freely, the actual causes are richer than they appear to be at first sight; in particular, they reflect the (...) agents' sensitivity to reasons, where this includes both the existence of actual reasons and the absence of other reasons. So acting freely requires more causes and quite complex causes, as opposed to fewer causes and simpler causes, and is compatible with those causes being deterministic. The book connects two different debates, the one on causation and the one on the problem of free will, in new and illuminating ways. (shrink)

In “A Subjectivist’s Guide to Objective Chance,” David Lewis says that he is “led to wonder whether anyone but a subjectivist is in a position to understand objective chance.” The present essay aims to motivate this same Lewisean attitude, and a similar degree of modest subjectivism, with respect to objective causation. The essay begins with Newcomb problems, which turn on an apparent tension between two principles of choice: roughly, a principle sensitive to the causal features of the relevant situation, (...) and a principle sensitive only to evidential factors. Two-boxers give priority to causal beliefs, and one-boxers to evidential beliefs. The essay notes that a similar issue can arise when the modality in question is chance, rather than causation. In this case, the conflict is between decision rules based on credences guided solely by chances, and rules based on credences guided by other sorts of probabilistic evidence. Far from excluding cases of the latter kind, Lewis’s Principal Principle explicitly allows for them, in the form of the caveat that credences should follow beliefs about chances only in the absence of “inadmissible evidence.” The essay then exhibits a tension in Lewis’s views on these two matters, by presenting a class of decision problems—some of them themselves Newcomb problems—in which Lewis’s view of the relevance of inadmissible evidence seems in tension with his causal decision theory. It offers a diagnosis for this dilemma and proposes a remedy, based on an extension of a proposal due to Ned Hall and others from the case of chance to that of causation. The remedy suggests a new view of the relation between causal decision theory and evidential decision theory, namely, that they stand to each other much as chance stands to credence, being objective and subjective faces of the same practical coin. This has much the same metaphysical benefits as Lewis’s own view of chance and also throws interesting new light on Newcomb problems, providing an irenic resolution of the apparent disagreement between causal and evidential decision rules. (shrink)

Absences pose a dilemma for theories of causation. Allowing them to be causes seems to make theories too permissive (Lewis, 2000). Banning them from being causes seems to make theories too restrictive (Schaffer, 2000, 2004). An increasingly popular approach to this dilemma is to acknowledge that norms can affect which absences count as causes (e.g., Thomson, 2003; McGrath, 2005; Henne et al., 2017; Willemsen, 2018). In this article, I distinguish between two influential implementations of such ‘abnormality’ approaches and argue (...) that so-called ‘double-prevention mechanisms’ provide counterexamples against both. (shrink)

The physical and/or intrinsic connection approach to causation has become prominent in the recent literature, with Salmon, Dowe, Menzies, and Armstrong among its leading proponents. I show that there is a type of causation, causation by disconnection, with no physical or intrinsic connection between cause and effect. Only Hume-style conditions approaches and hybrid conditions-connections approaches appear to be able to handle causation by disconnection. Some Hume-style, extrinsic, absence-relating, necessary and/or sufficient condition component of the causal (...) relation proves to be needed. (shrink)

I focus on token, deterministic causal claims as they feature in causal explanations. Adequately handling absences is difficult for most causal theories, including theories of causal explanation. Yet so is adequately handling cases of late preemption. The best account of absence-causal claims as they appear in causal explanations is Jonathan Schaffer's quaternary, contrastive account. Yet Schaffer's account cannot handle preemption. The account that best handles late preemption is James Woodward's interventionist account. Yet Woodward's account is inadequate when it comes (...) to absences. I propose an account that handles both absences and preemption by transposing Schaffer's account into an interventionist framework. (shrink)

The explanatory role of natural selection is one of the long-term debates in evolutionary biology. Nevertheless, the consensus has been slippery because conceptual confusions and the absence of a unified, formal causal model that integrates different explanatory scopes of natural selection. In this study we attempt to examine two questions: (i) What can the theory of natural selection explain? and (ii) Is there a causal or explanatory model that integrates all natural selection explananda? For the first question, we argue (...) that five explananda have been assigned to the theory of natural selection and that four of them may be actually considered explananda of natural selection. For the second question, we claim that a probabilistic conception of causality and the statistical relevance concept of explanation are both good models for understanding the explanatory role of natural selection. We review the biological and philosophical disputes about the explanatory role of natural selection and formalize some explananda in probabilistic terms using classical results from population genetics. Most of these explananda have been discussed in philosophical terms but some of them have been mixed up and confused. We analyze and set the limits of these problems. (shrink)

In this article, I defend a biconditional counterfactual account of causation, which places equal emphasis on what I call “the presence condition” and “the absence condition,” whereas Lewis's classical counterfactual theory focuses only on the absence condition. I attempt to show that biconditionalism provides a promising treatment of supervenient causation, namely, causal cases involving the supervenience relationship. Although some philosophers confuse this account with the proportionality constraint on causation, I argue that biconditionalism is distinct from (...) and superior to proportionalism in accommodating our reliable causal intuitions. (shrink)

Hume thought that if you believed in powers, you believed in necessary connections in nature. He was then able to argue that there were none such because anything could follow anything else. But Hume wrong-footed his opponents. A power does not necessitate its manifestations: rather, it disposes towards them in a way that is less than necessary but more than purely contingent. -/- In this paper a dispositional theory of causation is offered. Causes dispose towards their effects and often (...) produce them. But a set of causes, even though they may succeed in producing an effect, cannot necessitate it since the effect could have been counteracted by some additional power. This would require a separation of our concepts of causal production and causal necessitation. The most conspicuous cases of causation are those where powers accumulate and pass a requisite threshold for an effect to occur. -/- We develop a model for representing powers as constituent vectors within an n-dimensional quality space, where composition of causes appears as vector addition. Even our resultant vector, however, has to be understood as having dispositional force only. This model throws new light on causal modality and cases of prevention, causation by absence and probabilistic causation. (shrink)

In this article, I begin by giving a brief history of melanoma causation. I then discuss the current manner in which malignant melanoma is classified. In general, these systems of classification do not take account of the manner of tumour causation. Instead, they are based on phenomenological features of the tumour, such as size, spread, and morphology. I go on to suggest that misclassification of melanoma is a major problem in clinical practice. I therefore outline an alternative means (...) of classifying these tumours based on causal factors. By analogy with similar systems that have recently emerged for other cancers, I suggest that this causal classification is likely to be both workable and helpful, even in the absence of a full causal-mechanistic understanding of the aetiology of the tumour. (shrink)

Analyses of singular causation often make use of the idea that a cause increases the probability of its effect. Of particular salience in such accounts are the values of the probability function of the effect, conditional on the presence and absence of the putative cause, analysed around the times of the events in question: causes are characterized by the effect’s probability function being greater when conditionalized upon them. Put this way, it becomes clearer that the ‘behaviour’ of probability (...) functions in small intervals about the times in question ought to be of concern. In this article, I make an extended case that causal theorists employing the ‘probability raising’ idea should pay attention to the continuity question. Specifically, if the probability functions are ‘jumping about’ in ways typical of discontinuous functions, then the stability of the relevant probability increase is called into question. The rub, however, is that sweeping requirements for either continuity or discontinuity are problematic and, as I argue, this constitutes a ‘continuity bind’. Hence more subtle considerations and constraints are needed, two of which I consider: utilizing discontinuous first derivatives of continuous probability functions, and abandoning point probability for imprecise probability. _1_ Introduction _2_ Probability Trajectories and Continuity _2.1_ Probability trajectories _2.2_ Causation as discontinuous jumps _2.3_ Against systematic discontinuity _3_ Broader Discontinuity Concerns _4_ The Continuity Bind _4.1_ Retaining continuity with discontinuous first derivatives _4.2_ Imprecise probability trajectories _5_ Concluding Remarks Appendix. (shrink)

Moral and political philosophers no longer condemn harm inflicted on nonhuman animals as self-evidently as they did when animal welfare and animal rights advocacy was at the forefront in the 1980s, and sentience, suffering, species-typical behavior, and personhood were the basic concepts of the discussion. The article shows this by comparing the determination with which societies seek responsibility for human harm to the relative indifference with which law and morality react to nonhuman harm. When harm is inflicted on humans, policies (...) concerning negligence and duty of care and principles such as the ‘but for’ rule and the doctrine of double effect are easily introduced. When harm is inflicted on nonhumans, this does not happen, at least not any more. As an explanation for the changed situation, the article offers a shift in discussion and its basic terminology. Simple ethical considerations supported the case for nonhuman animals, but many philosophers moved on to debate different views on political justice instead. This allowed the creation of many conflicting views that are justifiable on their own presuppositions. In the absence of a shared foundation, this fragments the discussion, focuses it on humans, and ignores or marginalizes nonhuman animals. (shrink)

But if there aren’t, then ‘they’ are not caused by anything and do not cause anything. That certainly appears to be false, however. John’s absence from our party might have been caused by his having fallen ill, and might cause a commotion. Dick’s not eating his soup might have been caused by his having fallen ill, and might cause a commotion.

But if there aren’t, then ‘they’ are not caused by anything and do not cause anything. That certainly appears to be false, however. John’s absence from our party might have been caused by his having fallen ill, and might cause a commotion. Dick’s not eating his soup might have been caused by his having fallen ill, and might cause a commotion.

In this article I review the core elements of Carolina Sartorio’s actual causal sequence account of free will and moral responsibility, and propose two revisions. First, I suggest replacing the contested notion of absence causation by the relatively uncontroversial notion of causal explanation by absences. Second, I propose retaining explanation by unreduced dispositions, of which Sartorio appears to be wary. I then set out a response to her critical treatment of manipulation arguments against compatibilism. Lastly, I point out (...) that Sartorio’s reasons-sensitivity condition on moral responsibility is amenable to a conception of moral responsibility that, unlike the one she endorses, dispenses with basic desert. (shrink)

In this paper I consider possible causation, specifically, would-cause counterfactuals of the form ‘had an event of kind A occurred, it would have caused an event of kind B’. I outline some difficulties for the Lewis program for understanding would-cause counterfactuals, and canvass an alternative. I then spell out a view on their significance, in relation to (i) absence causation, where claims such as ‘A’s not occurring caused B’s not occurring’ seem to make sense when understood in (...) terms of the would-cause counterfactual ‘had an event of kind A occurred, it would have caused an event of kind B’; (ii) contrastive causal explanation, where to explain why E rather than E* occurred we might appeal to the causal history of E and the counterfactual causal history of E*, an approach which appeals directly to would-cause counterfactuals ‘had an event of kind C* occurred, it would have caused an event of kind E*’; and (iii) dispositions, where the claim ‘the glass is fragile’ clearly has some connection or other with would-cause counterfactuals such as ‘were the glass to be struck, the striking would cause the glass to break’. (shrink)

Much contemporary debate on the nature of mechanisms centers on the issue of modulating negative causes. One type of negative causability, which I refer to as “causation by absence,” appears difficult to incorporate into modern accounts of mechanistic explanation. This paper argues that a recent attempt to resolve this problem, proposed by Benjamin Barros, requires improvement as it overlooks the fact that not all absences qualify as sources of mechanism failure. I suggest that there are a number of (...) additional types of effects caused by absences that need to be incorporated to account for the diversity of causal connections in the biological sciences. Furthermore, it is argued that recognizing natural variability in mechanisms, such as attenuation, leads to some interesting line-drawing issues for contemporary philosophy of mechanisms. (shrink)

Much contemporary debate on the nature of mechanisms centers on the issue of modulating negative causes. One type of negative causability, which I refer to as "causation by absence," appears difficult to incorporate into modern accounts of mechanistic explanation. This paper argues that a recent attempt to resolve this problem, proposed by Benjamin Barros, requires improvement as it overlooks the fact that not all absences qualify as sources of mechanism failure. I suggest that there are a number of (...) additional types of effects caused by absences that need to be incorporated to account for the diversity of causal connections in the biological sciences. Furthermore, it is argued that recognizing natural variability in mechanisms, such as attenuation, leads to some interesting line-drawing issues for contemporary philosophy of mechanisms. (shrink)

Salmon has argued that science provides explanations by describing a causal nexus: For Salmon, this nexus is a network of processes and interactions. I argue that this picture of the causal nexus is insufficient for an account of scientific explanation: a taxonomy of causal relevance is also needed. ;Probabilistic theories of causation seem to provide such a taxonomy in their dichotomy between promoting and inhibiting causes. However, standard probabilistic theories are beset by a difficulty called the problem of disjunctive (...) factors. According to such theories, an effect is more probable in the presence of a cause than in its absence . But there are many ways in which a particular cause may come about, and many in which it may be absent; these ways need not confer the same probabilities upon the effect. Thus, there is no direct comparison of two probabilities to be made. My solution is to abandon the oversimplified dichotomy between promoting and inhibiting causes. Causal claims convey qualitative information about more complex probabilistic relationships. Interestingly, this picture of causation meshes naturally with extant theories of explanation. ;I show how this framework may be employed in resolving several outstanding problems in the theories of causation and explanation, such as the relationship between singular and general causal claims, and the role of contrastive stress in causal and explanatory claims. ;Finally, I show how my account of causal explanation can be connected with the semantic conception of scientific theories. According to this conception, a theory is characterized in terms of a class of mathematical models. I describe the relationship between these mathematical models and the probabilistic relationships that are described by causal explanations. These ideas are illustrated in an extended discussion of causation and explanation in evolutionary theory. (shrink)

More than a century ago, Russell launched a forceful attack on causation, arguing not only that modern physics has no need for causal notions but also that our belief in causation is a relic of a pre-scientific view of the world. He thereby initiated a debate about the relations between physics and causation that remains very much alive today. While virtually everybody nowadays rejects Russell's causal eliminativism, many philosophers have been convinced by Russell that the fundamental physical (...) structure of our world doesn't contain causal relations. This raises the question of how to reconcile the central role of causal concepts in the special sciences and in common sense with the putative absence of causation in fundamental physics. (shrink)

The starting point in the development of probabilistic analyses of token causation has usually been the naïve intuition that, in some relevant sense, a cause raises the probability of its effect. But there are well-known examples both of non-probability-raising causation and of probability-raising non-causation. Sophisticated extant probabilistic analyses treat many such cases correctly, but only at the cost of excluding the possibilities of direct non-probability-raising causation, failures of causal transitivity, action-at-a-distance, prevention, and causation by (...) class='Hi'>absence and omission. I show that an examination of the structure of these problem cases suggests a different treatment, one which avoids the costs of extant probabilistic analyses. (shrink)

The counterfactual analysis of causation has focused on one particular counterfactual conditional, taking as its starting-point the suggestion that C causes E iff (C E). In this paper, some consequences are explored of reversing this counterfactual, and developing an account starting with the idea that C causes E iff (E C). This suggestion is discussed in relation to the problem of pre-emption. It is found that the 'reversed' counterfactual analysis can handle even the most difficult cases of pre-emption with (...) only minimal complications. The paper closes with a discussion of the wider philosophical implications of developing a reversed counterfactual analysis, especially concerning the differentiation of causes from causal conditions, causation by absences, and the extent to which causes suffice for their effects. (shrink)

Functionalist theories have been proposed for just about everything: mental states, dispositions, moral properties, truth, causation, and much else. The time has come for a functionalist theory of nothing. Or, more accurately, a role functionalist theory of those absences that are causes and effects.

Philosophers of mind tend to take it for granted that causal relations are part of the mind-independent, objective fabric of the physical world. In fact, their status has been hotly contested since Russell famously observed that the closest thing to causal relations in physics are timesymmetric dynamical laws relating global time slices of world-history. 1 These bear a distant relationship to the local, asymmetric relations that form the core of the folk notion of cause. Nancy Cartwright, in an influential response, (...) agreed about the absence of causal relations from physics, but argued that Russell’s position was not viable because agents choosing among potential actions need specifically causal information to distinguish effective from ineffective strategies for bringing about ends. 2 Causal beliefs play an ineliminable role in practical deliberation In recent years, there has been a great deal of progress in understanding the relationship between causal concepts and the dynamical laws that appear in advanced physics, together with a proliferation of new tools for representing and discovering causal structure. (shrink)

Christopher Franklin argues that the hard luck view, which I have recently defended, is misnamed: the arguments turn on absence of control and not on luck. He also argues that my objections to agent-causal libertarianism depend on a demand, for a contrastive explanation that guarantees the choice the agent makes, which would be question-begging in the dialectical context. In response to the first objection, I argue that though Franklin may be right that it is absence of control that (...) matters to freedom and not luck, we often are unable to discern whether an agent lacks sufficient control without asking whether the event is lucky for the agent; hence we cannot dispense with worries about luck in favor of concern about control. In response to the second worry, I argue that though Franklin is right in claiming that there is a perfectly good explanation regarding how the agent-causal power is exercised, it is not an adequate explanation given the context, because the same kind of explanation is available on event-causal theories. Agent-causal libertarians aim to avoid the luck objection which they acknowledge is powerful against event-causal libertarians; if it is true that they can offer nothing more than the latter kinds of theories in the way of control over actions, they fail in their aim. (shrink)

This chapter discusses the causal requirements on perceptual success in putative cases of the perception of absence – in particular, in cases of hearing silence and seeing darkness. It is argued that the key to providing the right account of the respect in which we can perceive silence and darkness lies in providing the right account of the respect in which we can have conscious perceptual contact with intervals of time and regions of space within which objects can potentially (...) be perceived. In this account, a significant explanatory role is assigned to comparatively invariant structural features of our conscious experience of regions of space and intervals of time. The chapter discusses how the explanatory role assigned to these structural features affects our view of the causal requirements on perceptual success. (shrink)

Negative causation occurs when an absence serves as cause, effect, or causal intermediary. Negative causation is genuine causation, or so I shall argue. It involves no physical connection between cause and effect. Thus causes need not be physically connected to their effects.

The absence of a necessary connection in singular causation is a key step in the Humean argument against any form of necessity in causation. I argue that Hume's defence of this step is unsuccessful, and that the step could be skipped, accepting the possibility of necessary a posteriori truths. Still this does not suffice to guarantee a necessary connection in singular causation. Necessary a posteriori truths should be backed by necessary a priori truths. Thus, a main (...) object of this paper is to argue that an a priori philosophical concept of causality involves a necessary connection between its terms. (shrink)

This is a brief philosophical introduction to, and an annotated translation of, the section on absence from Śālikanātha’s Pramāṇapārāyaṇa (Study of the Instruments of Knowledge), a foundational work of Prābhākara epistemology. In this section, which focuses on the epistemology of absence, Śālikanātha argues against the Bhāṭṭa view that there is a sui generis instrument of knowledge (pramāṇa) by which we learn of absence (abhāva). He does so by arguing for a subjective reductionist thesis about absence, according (...) to which the absence of a perceivable (dṛśya) object at a locus is identical with a positive state of awareness (buddhi) whose content includes the locus but not the perceivable object. If correct, Śālikanātha argues, we should therefore learn of absence in the same ways we acquire self-knowledge more generally. While developing his reductionism about absence, Śālikanātha responds to a range of concerns, including the objection that his view cannot explain causation by absence. (shrink)

A number of philosophers have argued that causation is not an objective feature of the microphysical world but rather is a perspectival phenomenon that holds only between “coarse-grained” entities such as those that figure in the special sciences. This view seems to pose a problem for arguments for physicalism that rely on the alleged causal completeness of physics. In this paper, I address this problem by arguing that the completeness of physics has two components, only one of which is (...) causal. These two components of completeness can be used in an argument for physicalism that is supported by strong inductive evidence even in the absence of microphysical causation. (shrink)

Whether there exist causal relations between guns firing and people dying, between pedals pressed and cars accelerating, or between carbon dioxide emissions and global warming, is typically taken to be a mind-independent, objective, matter of fact. However, recent contributions to the literature on causation, in particular theories of contrastive causation and causal modelling, have undermined this central causal platitude by relativising causal facts to models or to interests. This thesis flies against the prevailing wind by arguing that we (...) must pay greater attention to which elements of our causal talk vary with context and which elements track genuine features of the world around us. I will argue that once these elements are teased apart we will be in a position to better understand some of the most persistent problems in the philosophy of causation: pre-emption cases, absence causation, failures of transitivity and overdetermination. The result is a naturalist account of causation, concordant with the contextual variability we find in our ordinary causal talk, and parsimonious with respect to the theoretical entities posited. (shrink)

In this dissertation, I develop and defend a model of causation that allows for dualist mental causation in worlds where the physical domain is physically complete. In Part I, I present the dualist ontology that will be assumed throughout the thesis and identify two challenges for models of mental causation within such an ontology: the exclusion worry and the common cause worry. I also argue that a proper response to these challenges requires a thoroughly lightweight account of (...) causation, i.e. an account that allows for causes to be metaphysically distinct from the phenomena that produce or physically necessitate their effects. In Part II, I critically evaluate contemporary responses to these challenges from the philosophical literature. In particular, I discuss List and Stoljar’s criticism of exclusion worries, Kroedel’s alternative dualist ontology, concerns about the notion of causal sufficiency, and Lowe’s models of dualist mental causation. I argue that none of these proposals provide independent motivation for a thoroughly lightweight account of causation and therefore leave room for improvement. In the first four chapters of Part III, I develop a thoroughly lightweight model of causation, which builds on interventionist approaches to causation. First, I explain how so-called ‘holding fixed’-requirements in standard interventionist accounts stand in the way of dualist mental causation. I then argue that interventionist accounts should impose a robustness condition on causal correlations and that, with this condition in place, the ‘holding fixed’-requirements can be weakened such that they do allow for dualist mental causation. I dub the interventionist model with such weakened ‘holding fixed’-requirements ‘insensitive interventionism’, argue that it can counter the exclusion worry as well as the common cause worry, and explain under which circumstances it would predict there to be dualist mental causation. Importantly, these circumstances might, for all we know, hold in the actual world. In the final three chapters of Part III, I defend insensitive interventionism against some objections. I consider the objection that causation must be productive, the objection that causes must physically necessitate their effects, and the objection that insensitive interventionism is too permissive. I respond by drawing from the literature on causation by absences and on the relation between causation and fundamental physics. Overall, insensitive interventionism performs as well as standard interventionist accounts. I conclude that insensitive interventionism is a credible model of causation. (shrink)

Benjamin Callard has recently suggested that causation between Platonic objects—standardly understood as atemporal and non-spatial—and spatio-temporal objects is not ‘a priori’ unintelligible. He considers the reasons some have given for its purported unintelligibility: apparent impossibility of energy transference, absence of physical contact, etc. He suggests that these considerations fail to rule out a priori Platonic-object causation. However, he has overlooked one important issue. Platonic objects must causally affect different objects differently, and different Platonic objects must causally affect (...) the same objects differently. How are Platonic objects—ones outside space and time—supposed to do that? CiteULike Connotea Del.icio.us What's this? (shrink)

_What Thomas Hobbes has to say of the nature of causation itself in_ _Entire Causes_ _and Their Only Possible Effects_ _is carried further in the first of the two excerpts here_ _-- although not at its start. His second subject in this imperfectly sequential piece of_ _writing is determinism itself -- a deterministic philosophy of mind. In the mind, as_ _elsewhere, each event has a 'necessary cause' -- a cause that necessitates the event._ _His third subject in the first (...) excerpt is freedom, this being voluntariness, and its_ _relation to the determinism. He gives a statement of what is now known as_ _Compatibilism -- roughly the doctrine that determinism and freedom properly_ _understood do not conflict with but are consistent with one another. We can be_ _entirely subject to determinism or 'necessity' and also be perfectly free. Certainly a_ _distinction between freedom as 'the absence of opposition', which can co-exist with_ _determinism, and some other kind of freedom, had been made before Hobbes. But it_ _will take a better historian than me to say if he was anticipated by someone else who_ _said that the particular freedom consistent with determinism is all that we can_ _properly mean by the term 'freedom'. Certainly he got in ahead of lovely_. (shrink)

Loewer (in: Physicalism and its discontents, 2001; Philos Phenomenol Res 65:655–663, 2002; in: Contemporary debates in philosophy of mind, 2007) has argued that the nonreductive physicalist should respond to the exclusion problem by endorsing the overdetermination entailed by their view. Kim’s (Physicalism, or something near enough, 2005; in: Contemporary debates in the philosophy of mind, 2007) argument against this reply is based on the premise that mental causation must be a productive relation in order to sustain human agency. In (...) this paper, I challenge the premise that mental causation is a productive relation by appealing to the underlying double prevention structure of the physiological mechanisms of human action. Since the causal pathways from an agent’s mental events to bodily movement involves an absence, mental causation is not productive. This places Kim in a troublesome dilemma in his debate with Loewer: either surrender mental causation or deny that causation is a productive relation. With the support offered for productive mental causation undermined, responses to the exclusion problem based on accepting overdetermination remain viable options for the nonreductive physicalist. (shrink)

We often explain by citing an absence or an omission. Apart from the problem of assigning a causal role to such apparently negative factors as absences and omissions, there is a puzzle as to why only some absences and omissions, out of indefinitely many, should figure in explanations. In this paper we solve this ’many absences problem’ by using the contrastive model of explanation. The contrastive model of explanation is developed by adapting Peter Lipton’s account. What initially appears to (...) be only a trivial amendment to Lipton’s Difference Condition enables us both to offer a much more satisfactory solution to the ’many absences problem’ than David Lewis did, and also to explain why explanation in terms of absences and omissions should be so common. (shrink)

This paper criticizes a recent account of token causation that states that negative causation involving absences of events is of a fundamentally different kind from positive causation involving events. The paper employs the structural equations framework to advance a theory of token causation that applies uniformly to positive and negative causation alike.