We present a probabilistic extension to active path analyses of token causation. The extension uses the generalized notion of intervention presented in : we allow an intervention to set any probability distribution over the intervention variables, not just a single value. The resulting account can handle a wide range of examples. We do not claim the account is complete --- only that it fills an obvious gap in previous active-path approaches. It still succumbs to recent counterexamples by Hiddleston, (...) because it does not explicitly consider causal processes. We claim three benefits: a detailed comparison of three active-path approaches, a probabilistic extension for each, and an algorithmic formulation. (shrink)

Judea Pearl (2000) has recently advanced a theory of token causation using his structural equations approach. This paper examines some counterexamples to Pearl's theory, and argues that the theory can be modified in a natural way to overcome them.

Recent literature on causation invokes a distinction between deviant and default behavior to account for token causation. Critical examination of two prominent attempts to employ a distinction between deviants and defaults reveals that the distinction is far from clear. I clarify and develop the distinction by appeal to the notion of a modally robust process, and show how the distinction can be employed by causal process theorists to respond to cases of causation by omission. This shows (...) that the default/deviant distinction is not so much a tool for counterfactual accounts of causation, but rather for causal process theory. (shrink)

Causal Bayes nets (CBNs) provide one of the most powerful tools for modelling coarse-grained type-level causal structure. As in other fields (e.g., thermodynamics) the question arises how such coarse-grained characterisations are related to the characterisation of their underlying structure (in this case: token-level causal relations). Answering this question meets what is called a “coherence-requirement” in the reduction debate: How are different accounts of one and the same system (or kind of system) related to each other. We argue that CBNs (...) as tools for type-level causal inference are abstract enough to roughly fit any current token-level theory of causation as long as certain modelling assumptions are satisfied, but accounts of actual causation, i.e. accounts that attempt to infer token-causation based on CBNs, for the very same reason, face certain limitations. (shrink)

Qiyās, or “correlational inference”, comprises a primary set of methodological tools recognized by a majority of premodern Sunnī jurists. Its elements, valid modes, and proper applications were the focus of continual argument and refinement. A particular area of debate was the methodology of determining or justifying the ʿilla: the legal cause giving rise to a ruling in God’s Law. This was most often discussed under the rubric of “the modes of causal justification”. Among these modes was the much debated test (...) of dawarān. In brief, proponents of dawarān employed it to justify claims that a property occasioned the ruling in an authoritative source-case. In concert with other considerations, the demonstrated co-presence and co-absence of property and ruling—that is, their concomitance “in existence” and “in nonexistence” — was taken as an indication that the property was the ruling’s ʿilla. Delving further into dawarān and causation, the current study interprets “in existence” and “in nonexistence” not as a kind of metaphor for true and false, but as an accurate terminology vis-à-vis the meaning of causality statements, fully compatible with dominant Islamicate views on causal agency. In brief, a deeper logical and linguistic analysis of the different existential modes of dawarān strongly suggests that we should distinguish property and ruling as types as opposed to tokens. Our reading of dawarān as shaped by a finer-grained structure not only allows us to identify the efficient occasioning process as a function which takes some particular token of the ʿilla and renders a token of the general ruling type, but it allows us to elucidate the role of taʿlīl in shaping an epistemological theory of argument to the best explanation: a sophisticated, premodern manifestation of abductive reasoning. (shrink)

In this paper, I argue that causal theories of memory are typically committed to two independent, non-mutually entailing theses. The first thesis pertains to the necessity of appropriate causation in memory, specifying a condition token memories need to satisfy. The second pertains to the explanation of memory reliability in causal terms and it concerns memory as a type of mental state. Post-causal theories of memory can reject only the first (weak post-causalism) or both (strong post-causalism) theses. Upon this (...) backdrop, I examine Werning’s (2020) causalist argument from probabilistic correlation. I argue that it doesn’t establish the necessity of appropriate causation and thus it can only target strong post-causalist theories. I end up by presenting some general considerations, suggesting that memories may not always be causally linked to past experiences. (shrink)

Davidson's account of mental causation initiated in “Mental Events” forms a backdrop to much subsequent discussion of the topic. Davidson is commonly taken to defend token identity – token mental events are identical with token physical events – but type diversity – mental types “supervene on,” but are not reducible to or identical with physical types, where types are understood as properties. Mental events are physical events, but one and the same event can have a physical (...) property and, by virtue of having this property, have a mental property as well. There are two salient difficulties with this account. (1) It gives rise to the problem of “causal relevance”: granted that a mental event can cause a physical event, could it do so by virtue of being mental, by virtue of its possession of a mental property? (2) The view thus described is not Davidson's. This chapter discusses Davidson actual account and shows that, whatever its flaws, they are not what they are commonly thought to be. (shrink)

We present a probabilistic extension to active path analyses of token causation (Halpern & Pearl 2001, forthcoming; Hitchcock 2001). The extension uses the generalized notion of intervention presented in (Korb et al. 2004): we allow an intervention to set any probability distribution over the intervention variables, not just a single value. The resulting account can handle a wide range of examples. We do not claim the account is complete --- only that it fills an obvious gap in previous (...) active-path approaches. It still succumbs to recent counterexamples by Hiddleston (2005), because it does not explicitly consider causal processes. We claim three benefits: a detailed comparison of three active-path approaches, a probabilistic extension for each, and an algorithmic formulation. (shrink)

In this paper, I provide a probabilistic account of factual knowledge, based on the notion of chance, which is a function of an event given a prior history. This account has some affinity with my chance account of token causation, but it neither relies on it nor presupposes it. Here, I concentrate on the core cases of perceptual knowledge and of knowledge by memory. The analysis of knowledge presented below is externalist. The underlying intuition guiding the treatment of (...) knowledge in this paper is that knowledge boils down to high token discriminative indicativeness. Type indicativeness or type discriminability are neither necessary nor sufficient for knowledge: the token aspect comes out in the strong dependence on the specific circumstances and chances of the case. The main condition of the first section, the indicativity condition, properly refined, yields pertinent indicativity as a main constituent. Very roughly, it involves the chance of the content clause p being higher given the subject's believing that p than otherwise. The discriminability condition in question captures the sense of discriminability appropriate for knowledge and yield the indicativity condition: it is an extension of the indicativity condition KI. Roughly, the subject’s ability to discriminate the object in front of her being red from its being green is captured by holding fixed, in the indicativity condition, the condition “the object in front of her is red or green.” A major element in the analysis is the so-called Contrast Class, which governs the scope of discriminability. This is the class of features that have to be taken into account in the discriminability condition, and it is characterized by two constraints. Very roughly, according to the first constraint, for a feature to be in the contrast class, it must not represent a sub-type of the predicate in the content clause. According to the second constraint, which is a central condition with many implications, the chance that the object specified in the content clause has a feature represented in the contrast class must not under the circumstances be too low. This constraint, within the framework of the discriminability condition, brings out a major constitutive aspect of knowledge: knowledge amounts to a limited vulnerability to mistakes of the belief in question under the circumstances at hand. The contrast class plays a major role in my treatment of skepticism. The second constraint on the Contrast Class together with the VHP condition below bring out precisely the way in which perceptual knowledge is fallible. (shrink)

I provide a theory of causation within the causal modeling framework. In contrast to most of its predecessors, this theory is model-invariant in the following sense: if the theory says that C caused (didn't cause) E in a causal model, M, then it will continue to say that C caused (didn't cause) E once we've removed an inessential variable from M. I suggest that, if this theory is true, then we should understand a cause as something which transmits deviant (...) or non-inertial behavior to its effect. (shrink)

Donald Davidson has two central aims in his celebrated paper ‘Mental Events.’ First, he argues for the impossibility of ‘strict … laws on the basis of which mental events can be predicted and explained’. I shall call the resulting view ‘mental anomalism.’ Second, he argues, based partially on this impossibility, for a version of monism which holds that every mental event is token-identical with some physical event. This second aim puts constraints on how the argument for mental anomalism can (...) plausibly proceed. That argument cannot assume anything that presupposes monism or adversely affects the argument for it. When elaborated, this constraint by itself rules out otherwise attractive conceptions of how the argument for mental anomalism works. More generally, in order to be compelling, the overall argument for anomalous monism ought not to incorporate excessively controversial presuppositions even if they do not themselves beg any questions about monism. The argument needs to be built upon relatively intuitive premises. (shrink)

The philosophical problem of mental causation concerns a clash between commonsense and scientific views about the causation of human behaviour. On the one hand, commonsense suggests that our actions are caused by our mental states—our thoughts, intentions, beliefs and so on. On the other hand, neuroscience assumes that all bodily movements are caused by neurochemical events. It is implausible to suppose that our actions are causally overdetermined in the same way that the ringing of a bell may be (...) overdetermined by two hammers striking it at the same time. So how are we to reconcile these two views about the causal origins of human behaviour? One philosophical doctrine effects a nice reconciliation. Neuralism, or the token-identity theory, states that every particular mental event is a neurophysiological event and that every action is a physically specifiable bodily movement. If these identities hold, there is no problem of causal overdetermination: the apparently different causal pathways to the behaviour are actually one and the same pathway viewed from different perspectives. This attractively simple view is enjoying a recent revival in fortunes. (shrink)

The basic idea of counterfactual theories of causation is that the meaning of causal claims can be explained in terms of counterfactual conditionals of the form “If A had not occurred, C would not have occurred”. While counterfactual analyses have been given of type-causal concepts, most counterfactual analyses have focused on singular causal or token-causal claims of the form “event c caused event e”. Analyses of token-causation have become popular in the last thirty years, especially since (...) the development in the 1970's of possible world semantics for counterfactuals. The best known counterfactual analysis of causation is David Lewis's (1973b) theory. However, intense discussion over thirty years has cast doubt on the adequacy of any simple analysis of singular causation in terms of counterfactuals. Recent years have seen a proliferation of different refinements of the basic idea to achieve a closer match with commonsense judgements about causation. (shrink)

The so-called problem of mental causation as discussed in the recent literature raises three central challenges for an adequate solution from a physicalist perspective: the threat of epiphenomenalism, the problem of externalism (or the difficulty in accounting for the causal efficacy of extrinsic mental properties) and the problem of causal exclusion (or the threat of over determination). We wish to account for mental causationas a real phenomenon within a physicalistic framework without accepting epiphenomenalism or overdetermination. The key ideas of (...) our proposal are an internal realism of causation combined with a relative notion of individuating events. We are arguing?contra Davidson?tha there is no absolute notion of events (neither as types nor as tokens) but rather one which is relative to explanatory interests and our intuitions concerning a relevant spatial and temporal overlap. Furthermore, we are presupposing a metaphysics of internal realism: We can only characterize entities by means of concepts produced within our epistemological framework. Physical concepts and mental concepts crossclassify the world as it is. Relying on this framework we try to explain how mental causation can be adequately described: Although mental concepts are not reducible to physical concepts and mental event-tokens may be different from "underlying" physical event-tokens, mental events are real phenomena that are realized by physical phenomena in special context conditions. (shrink)

The starting point in the development of probabilistic analyses of token causation has usually been the naïve intuition that, in some relevant sense, a cause raises the probability of its effect. But there are well-known examples both of non-probability-raising causation and of probability-raising non-causation. Sophisticated extant probabilistic analyses treat many such cases correctly, but only at the cost of excluding the possibilities of direct non-probability-raising causation, failures of causal transitivity, action-at-a-distance, prevention, and causation by (...) absence and omission. I show that an examination of the structure of these problem cases suggests a different treatment, one which avoids the costs of extant probabilistic analyses. (shrink)

The literature on causation distinguishes between causal claims relating properties or types and causal claims relating individuals or tokens. Many authors maintain that corresponding to these two kinds of causal claims are two different kinds of causal relations. Whether to regard causal relations among variables as yet another variety of causation is also controversial. This essay maintains that causal relations obtain among tokens and that type causal claims are generalizations concerning causal relations among these tokens.

In this paper I consider whether a powers ontology facilitates a reduction of causal relations to intrinsic powers of the causal relata. I first argue that there is a tension in the view that powerful causation is an internal relation in this sense. Powers are ontologically dependent on other powers for their individuation, but in that case—given an Aristotelian conception of properties as immanent universals—powers will not be intrinsic on several extant analyses of ‘intrinsic’, since to possess a given (...) power P requires the existence of other concrete particulars as bearers of the powers that individuate P. I suggest several ways for Aristotelians to resolve this tension, but all tenable options involve individuative type-level causal relations between powers. While these individuative relations between powers are internal in the sense that the powers are essentially related, this is a different sense of ‘ internal ’ to the one that entails reducibility. The proposed reduction of token-causal relations to powers succeeds only at the cost of irreducible type-level causal relations between the powers themselves. (shrink)

The aim of this paper is to defend the causal homogeneity of functional, mental properties against Kim’s attack. It is argued that (a) token identity is sufficient for mental causation, that (b) token identity implies a sort of functional reduction, but that (c) nonetheless functional, mental properties can be causally homogeneous despite being multiply realizable: multiple composition is sufficient for multiple realizability, but multiple composition does not prevent the realizers from having their pertinent effects in common. Thus, (...) the causal exclusion problem provides no argument for abandoning the position that there are functional, mental properties that are natural kind properties. (shrink)

The paper investigates whether causation is extrinsic in Humean Supervenience in the sense that "being caused by" is an intrinsic relation between token causes and effects. The underlying goal is to test whether causality is extrinsic for Humeans and intrinsic for anti-Humeans in this sense. I argue that causation is typically extrinsic in HS, but it is intrinsic to event pairs that collectively most of the universe's history.

A popular solution to the causal exclusion problem in the non-reductive physicalist camp is the trope identity solution. But this solution is haunted by the “quausation problem” which charges that the trope only confers causal powers qua physical, not qua mental. Although proponents of the trope solution have responded to the problem by denying the existence of properties of tropes, I do not find their reply satisfactory. Rather, I believe they have missed the core presupposition behind the quausation problem. I (...) will argue that the presupposition is the generalist notion of causation. Then, for the trope theorists to solve the quausation problem, they need to abandon the generalist notion and adopt the singularist notion of causation. However, making that move will lead them to a new quausation problem, rendering irreducible mental types causally irrelevant and mental causal explanations reducible. Either adopting a generalist notion or a singularist notion of causation, a quausation problem awaits the trope solution. Given this dilemma, my conclusion is that the trope identity solution cannot solve the exclusion problem in a non-reductive way. Moreover, the dilemma can be generalized, showing that token physicalism is a shaky position. (shrink)

A key test of any philosophical account of the mind is its treatment of mental causation. Proponents of the token-identity theory point to its strengths in both “demystifying” mental causation — by identifying mental causes with the physical causal mechanisms underlying bodily movements — and in avoiding commitment to dubious forms of causal overdetermination. I argue against this account of mental causation, pointing out that it mistakenly identifies actions with bodily movements. I suggest instead treating action (...) explanations as explanations of redundant causalities in behavior, and the mental causes cited in such explanations as structural causes. (shrink)

Donald Davidson has two central aims in his celebrated paper ‘Mental Events.’ First, he argues for the impossibility of ‘strict … laws on the basis of which mental events can be predicted and explained’. I shall call the resulting view ‘mental anomalism.’ Second, he argues, based partially on this impossibility, for a version of monism which holds that every mental event is token-identical with some physical event. This second aim puts constraints on how the argument for mental anomalism can (...) plausibly proceed. That argument cannot assume anything that presupposes monism or adversely affects the argument for it. When elaborated, this constraint by itself rules out otherwise attractive conceptions of how the argument for mental anomalism works. More generally, in order to be compelling, the overall argument for anomalous monism ought not to incorporate excessively controversial presuppositions even if they do not themselves beg any questions about monism. The argument needs to be built upon relatively intuitive premises. (shrink)

Argument for Epiphenomenalism [I]: (A) Mental event-tokens are identical with physical event-tokens. (B) The causal powers of a physical event are determined only by its physical properties; and (C) mental properties are not reducible to physical properties.

An influential tradition in the philosophy of causation has it that all token causal facts are, or are reducible to, facts about difference-making. Challenges to this tradition have typically focused on pre-emption cases, in which a cause apparently fails to make a difference to its effect. However, a novel challenge to the difference-making approach has recently been issued by Alyssa Ney. Ney defends causal foundationalism, which she characterizes as the thesis that facts about difference-making depend upon facts about (...) physical causation. She takes this to imply that causation is not fundamentally a matter of difference-making. In this paper, I defend the difference-making approach against Ney’s argument. I also offer some positive reasons for thinking, pace Ney, that causation is fundamentally a matter of difference-making. (shrink)

Yablo suggests that we can understand the possibility of mental causation by supposing that mental properties determine physical properties, in the classic sense of determination according to which red determines scarlet. Determinates and their determinables do not compete for causal relevance, so if mental and physical properties are related as determinable and determinates, they should not compete for causal relevance either. I argue that this solution won''t work. I first construct a more adequate account of determination than that provided (...) by Yablo. I then consider two common accounts of the mental, token identity theories and dispositional theories, and argue that on neither do mental and physical properties satisfy the requirements for determination. (shrink)

One part of the true theory of actual causation is a set of conditions responsible for eliminating all of the non-causes of an effect that can be discerned at the level of counterfactual structure. I defend a proposal for this part of the theory.

The aim of this paper is to defend the causal homogeneity of functional, mental properties against Kim's attack. It is argued that token identity is sufficient for mental causation, that token identity implies a sort of functional reduction, but that nonetheless functional, mental properties can be causally homogeneous despite being multiply realizable: multiple composition is sufficient for multiple realizability, but multiple composition does not prevent the realizers from having their pertinent effects in common. Thus, the causal exclusion (...) problem provides no argument for abandoning the position that there are functional, mental properties that are natural kind properties. (shrink)

My dissertation concerns the long-standing mind-body problem in a contemporary context. I investigate whether a content property of a mental state can be causally efficacious in bringing about behavior. I argue that general objections against the causal efficacy of content are not warranted. I then propose my own account of the causal efficacy of content. ;In Chapter 1, I examine the claim that the supervenience thesis renders mental causation incompatible with underlying physical causation. I argue that if we (...) understand the supervenience thesis and the concept of causation probabilistically, it turns out that the causal efficacy of the mental is compatible with that of the physical. ;In Chapter 2, I claim that given the compatibility claim established in the previous chapter, the computational theory of the mind can be understood as a causal hypothesis. I examine this hypothesis in details and Fodor's "narrow content" proposal. I argue that Fodor's proposal has a serious problem. ;In Chapter 3, I discuss individualism which implies that content is not a psychologically relevant property. I argue that individualism is in conflict with legitimate scientific practices and that there is no reason not to individuate psychological properties in terms of diachronic, causal relations. ;In Chapter 4, I investigate the screening-off argument that since content is unnecessary and redundant for the genesis of behavior, content cannot be causally efficacious. I argue that the argument is not valid. I also critically examine three accounts of the causal efficacy of content and claim that they fall short of conferring causal efficacy to content. ;In Chapter 5, I propose an account of causal efficacy which is consistent with the requirement of causal transaction via local, physical mechanism. My account is based on the notion of causal connection which is, in turn, understood as a category of token probabilistic causal significances. According to my account, content can be causally efficacious in bringing about behavior. I conclude that reasons qua reasons have causal have causal efficacy. (shrink)

Sungho Choi has criticised Michael Strevens's counterexample to DavidLewis's final theory of "token" causation, causation as "influence." Iargue that, even if Choi's points are correct, Strevens's counterexampleremains useful in revealing a shortcoming of Lewis's theory. Thisshortcoming is that Lewis's theory does not properly account for*degrees* of causation. That is, even if Choi's points are correct,Lewis's theory does not capture an intuition we have about the*comparative* causal statuses of those events involved in Strevens'scounterexample (we might, for example, (...) intuit that Sylvie's ball-firingis *as much*/*more*/*less* a cause of the jar's shattering as/than isBruno's ball-firing). (shrink)

This paper criticizes a recent account of token causation that states that negative causation involving absences of events is of a fundamentally different kind from positive causation involving events. The paper employs the structural equations framework to advance a theory of token causation that applies uniformly to positive and negative causation alike.

This paper makes the case for a wider acceptance of a probabilistic approach to causation in negligence. This acceptance would help to remove much of the incoherence which has come to afflict the English law of personal injury law. This incoherence can also be found in other common law jurisdictions (notably those of the United States, Canada and Australia). Concentrating upon recent UK case law, the argument opposes the contention that ‘naked statistics’ can play no role in establishing (...) class='Hi'>causation. The argument is controversial but it can be reduced to three unremarkable grounds: (1) With its acceptance (albeit in certain carefully prescribed circumstances) of liability for a negligently increased risk which has eventuated, the common law has already embraced a probabilistic conception of causation; (2) The English common law already employs a probabilistic (frequentist) approach to identifying coincidences; and (3) With the ‘balance of probabilities’ as the standard of proof in civil cases, the common law has long had a probabilistic (epistemic) concept at its core. Probabilistic approaches (at both the type and token level) are shown to be consistent with laypersons’ understanding of the concepts such as risk, chance, odds and likelihood. Moreover, a wider acceptance of a probabilistic perspective on causation would entail no major challenge to the fundamental aims of tort, viz. deterrence and corrective justice. (shrink)

Allow me to recapitulate some territory that will be familiar to most readers. Here is how the problem of mental causation has typically been set up since shortly after the onset of non-reductive physicalism. It is now widely assumed that the realm of the physical is causally closed: every physical event has a complete physical cause, a cause that is sufficient for the event’s occurrence. This apparently leaves us with a limited number of options concerning psychological causation, none (...) of which appear hugely attractive. Either: (a) the psychological is epiphenomenal and can have no causal impact on the physical, or (b) the psychological is identical with the physical, or (c) thoughts and actions are all over-determined, each one having two distinct sufficient causes. Option (b) subdivides into two further options. Either (b1) the psychological reduces to the physical and every psychological property is identical with some physical property, or (b2) token psychological events are identical with or constituted from token physical events but psychological properties are not identical with physical properties. (b1) is widely held to be inconsistent with the multiple realisation of the psychological by the physical. And (b2) appears to bring us back to the original problematic, with the properties as the locus of tension. If one event causes another it does so in virtue some of its properties and not others. If I throw a stone at a window and the window breaks, it is because the stone was hard and heavy that it broke the window and not, say, because it was grey and millions of years old. The properties in virtue of which an event has a particular effect are typically called the ‘causally efficacious properties of the cause with respect to the effect.’ Suppose, then that token neural event causes an action. We can ask ‘Does it do so in virtue of its physical properties or its psychological properties?’ and we are back to choosing between options (a) and (c) or returning to (b1).. (shrink)

An actual cause of some token effect is itself a token event that helped to bring about that effect. The notion of an actual cause is different from that of a potential cause – for example a pre-empted backup – which had the capacity to bring about the effect, but which wasn't in fact operative on the occasion in question. Sometimes actual causes are also distinguished from mere background conditions: as when we judge that the struck match was (...) a cause of the fire, while the presence of oxygen was merely part of the relevant background against which the struck match operated. Actual causation is also to be distinguished from type causation: actual causation holds between token events in a particular, concrete scenario; type causation, by contrast, holds between event kinds in scenario kinds. (shrink)

The paper outlines an analysis of token causation . It contains reasons for the choice of event tokens and a list of properties of causal relations. After defining the notion event it is shown, in which form classical philosophical problems arise and how they can be solved: Causality is interpreted as an second order property of first order relations. Temporal order can be induced from causal order. Token causation does not involve neither any kind of necessity, (...) nor probability. Causal laws are generalizations on sentences about token causation by quantifying over event types. Two explications of Causal law are suggested, differing in the ability of allowing for exceptions. (shrink)

The paper outlines an analysis of token causation. It containsreasons for the choice of event tokens and a list of properties of causal relations. After defining the notion event it is shown, in which form classicalphilosophical problems arise and how they can be solved: Causality is interpreted as an second order property of first order relations. Temporal ordercan be induced from causal order. Token causation does not involve neither any kind of necessity, nor probability. Causal laws (...) are generalizationson sentences about token causation by quantifying over event types. Twoexplications of Causal law are suggested, differing in the ability of allowingfor exceptions. (shrink)

In this paper I argue that the problem of mental causation can be solved by distinguishing between classificatory mental properties, like being a pain, and instances of those properties.Antireductive physicalism allows only that the former be irreducibly mental. Consequently, properties like being a pain cannot have causal commerce with the physical without violating causal closure. But instances of painfulness, according to the token identity thesis, are identical with various physical tokens and can therefore have causal efficacy in the (...) physical world. Since we expect particular mental phenomena, not types or classes of mental phenomena to be involved in causal interactions, it is argued that antireductive physicalism can explain satisfactorily mental causation, despite the protests of Kim, Sosa, Honderich, and others. Being a mental state of a certain sort may have no causal efficacy, but the intentional and phenomenal properties of such states should, if my argument is correct. (shrink)

In a series of recent papers, Cynthia MacDonald and Graham MacDonald offer a resolution to the twin problems of mental causation and mental causal relevance. They argue that the problem of mental causation is soluble via token monism – mental events are causally efficacious physical events. At the same time, the problem of mental causal relevance is solved by combining this causally efficacious mental property instance with the systematic co-variation between distinct mental properties of the cause and (...) the action-theoretic properties of the effect in question. In this paper we argue that the solution offered by Mac- Donald and MacDonald faces significant difficulties in resolving both of the twin problems of mental causation and mental causal relevance. (shrink)

In this paper I argue that the problem of mental causation can be solved by distinguishing between classificatory mental properties, like being a pain, and instances of those properties.Antireductive physicalism allows only that the former be irreducibly mental. Consequently, properties like being a pain cannot have causal commerce with the physical without violating causal closure. But instances of painfulness, according to the token identity thesis, are identical with various physical tokens and can therefore have causal efficacy in the (...) physical world. Since we expect particular mental phenomena, not types or classes of mental phenomena to be involved in causal interactions, it is argued that antireductive physicalism can explain satisfactorily mental causation, despite the protests of Kim, Sosa, Honderich, and others. Being a mental state of a certain sort may have no causal efficacy, but the intentional and phenomenal properties of such states should, if my argument is correct. (shrink)

Glennan (2002) argues for the mechanism theory of causation that it explicates both type-level and token-level causation in terms of mechanism. I argue against the mechanism theory that it is not sufficient for explicating cause-effect relations at the token-level. I put forth two counterexamples (first, absence of causes and second, a cause preempting another cause) to the theory, and show that descriptions of a mechanism are inert in explicating cause-effect relations at the token level. I (...) point out that the problems with the mechanism theory are due to explicating cause-effect relation in monolithic ways. (shrink)

Much of the philosophical literature on causation has focused on the concept of actual causation, sometimes called token causation. In particular, it is this notion of actual causation that many philosophical theories of causation have attempted to capture.2 In this paper, we address the question: what purpose does this concept serve? As we shall see in the next section, one does not need this concept for purposes of prediction or rational deliberation. What then could (...) the purpose be? We will argue that one can gain an important clue here by looking at the ways in which causal judgments are shaped by people‘s understanding of norms. (shrink)

A distinction is drawn between property causation and token causation. According to the former, a positive causal factor in a population raises the probability of its effects within "background contexts". The latter, which concerns "actual physical connections" between token events, is not explicated here although its distinctness from the first concept and its importance are discussed. The applicability of both is illustrated by two currently controversial issues in evolutionary theory -- the units of selection controversy and (...) the use of parsimony as an inference rule in phylogenetic reconstruction. (shrink)

In this paper I will argue that what makes our ordinary judgements about token causation true can be explicated in terms of interferences into quasi-inertial processes. These interferences and quasi-inertial processes can in turn be fully explicated in scientific terms. In this sense the account presented here is reductive. I will furthermore argue that this version of a process-theory of causation can deal with the traditional problems that process theories have to face, such as the problem of (...) misconnection and the problem of disconnection as well as with a problem concerning the mis-classification of pre-emption cases. (shrink)

A cause may influence its effect via multiple paths. Paradigmatically (Hesslow [1974]), taking birth control pills both decreases one’s risk of thrombosis by preventing pregnancy and increases it by producing a blood chemical. Building on Pearl ([2001]), I explicate the notion of a path-specific effect. Roughly, a path-specific effect of C on E via path P is the degree to which a change in C would change E were they to be transmitted only via P. Facts about such effects may (...) be gleaned from the structural equations commonly used to represent the causal relationships among variables. I contrast my analysis of the Hesslow case with those given by theorists of probabilistic causality, who mistakenly link it to issues of causal heterogeneity, token-causation and indeterminism. The reason probabilistic theories misdiagnose this case is that they pay inadequate attention to the structural relationships among variables. (shrink)

There are two concepts of causes, property causation and token causation. The principle I want to discuss describes an epistemological connection between the two concepts, which I call the Connecting Principle. The rough idea is that if a token event of type Cis followed by a token event of type E, then the support of the hypothesis that the first event token caused the second increases as the strength of the property causal relation of (...) C to E does. I demonstrate the principle, illustrate its application to phylogenies, infections, and rumours, and discuss its consequence for the conceptual distinctness of causal processes from the events they connect. Although I am by no means confident that the Connecting Principle is ultimately correct, it seems to be a useful point of departure into an important aspect of the epistemology of causality. (shrink)