In this paper, we shall give another proof of the faithfulness of Blass translation of the propositional fragment \ of Leśniewski’s ontology in the modal logic \ by means of Hintikka formula. And we extend the result to von Wright-type deontic logics, i.e., ten Smiley-Hanson systems of monadic deontic logic. As a result of observing the proofs we shall give general theorems on the faithfulness of B-translation with respect to normal modal logics complete to certain sets of well-known accessibility relations (...) with a restriction that transitivity and symmetry are not set at the same time. As an application of the theorems, for example, B-translation is faithful for the provability logic \ ), that is, \ \ \ \supset \Box \phi \). The faithfulness also holds for normal modal logics, e.g., \, \, \, \. We shall conclude this paper with the section of some open problems and conjectures. (shrink)

In this paper, we shall show that the following translation \(I^M\) from the propositional fragment \(\bf L_1\) of Leśniewski's ontology to modal logic \(\bf KTB\) is sound: for any formula \(\phi\) and \(\psi\) of \(\bf L_1\), it is defined as (M1) \(I^M(\phi \vee \psi) = I^M(\phi) \vee I^M(\psi)\), (M2) \(I^M(\neg \phi) = \neg I^M(\phi)\), (M3) \(I^M(\epsilon ab) = \Diamond p_a \supset p_a. \wedge. \Box p_a \supset \Box p_b.\wedge. \Diamond p_b \supset p_a\), where \(p_a\) and \(p_b\) are propositional variables corresponding to (...) the name variables \(a\) and \(b\), respectively. In the last, we shall give some comments including some open problems and my conjectures. (shrink)

We prove that Flagg and Friedman's translation from epistemic to intuitionistic predicate logics is not faithful.

Mutations in the daf-2 and age-1 genes cause constitutive dauer larva formation and double adult life span in C. elegans. Their effect on life span has excited considerable interest and their effect on dauer formation has facilitated rapid progress in their genetic and molecular analysis. Two recent papers12,13 report that daf-2 encodes a member of the insulin-receptor family and that age-1 encodes a PI3 kinase subunit, a second-messenger producing enzyme known to act downstream of the mammalian insulin receptor. These findings (...) provide the first mechanistic insight into the well-established link between metabolism and aging. BioEssays 20:113–115, 1998. © 1998 John Wiley & Sons, Inc. (shrink)