Rousseau is considered to be at once the most modern political thinker of the 18th century and the most ancient in his allegiance to classical republicanism. These essays address the place of liberty in his moral and political philosophy, and the origins, meaning, strength, weakness and significance of his argument.

Causal pluralists hold that that there is not just one determinate kind of causation. Some causal pluralists hold that ‘cause’ is ambiguous among these different kinds. For example, Hall (2004) argues that ‘cause’ is ambiguous between two causal relations, which he labels dependence and production. The view that ‘cause’ is ambiguous, however, wrongly predicts zeugmatic conjunction reduction, and wrongly predicts the behaviour of ellipsis in causal discourse. So ‘cause’ is not ambiguous. If we are to disentangle (...) causal pluralism from the ambiguity claim, we need to consider what other linguistic approaches are available to the causal pluralist. I consider and reject proposals that ‘cause’ is a general term, that the term is an indexical, and that the term conveys different kinds of causation through implicature or presupposition. Finally, I argue that causal pluralism is better handled by treating ‘cause’ as a univocal term within a dynamic interpretation framework. (shrink)

In general, the false cause fallacy occurs when the “link between premises and conclusion depends on some imagined causal connection that probably does not exist”. There are three different ways an argument can commit the false cause fallacy: post hoc ergo propter hoc; cum hoc ergo propter hoc; and ignoring common cause. This chapter focuses on one of the common fallacies in Western philosophy, 'ignoring common cause'. The commercial exploited the false cause fallacy to get (...) consumers to buy its product. This fallacy is also present in many of the arguments concerning the alleged connection between vaccines and autism despite the plethora of evidence otherwise. The chapter consists of various examples showcasing the fallacy of ignoring common cause. It also compares the examples, showing that slight changes can lead the other types of false cause fallacies to occur. (shrink)

This chapter focuses on one of the common fallacies in Western philosophy called the false cause fallacy. This fallacy occurs when the “link between premises and conclusion depends on some imagined causal connection that probably does not exist”. There are three different ways an argument can commit the false cause fallacy: post hoc ergo propter hoc; cum hoc ergo propter hoc; and ignoring common cause. The chapter deals with post hoc ergo propter hoc. One example of the (...) post hoc ergo propter hoc fallacy that has had a great impact on public health has been the recent fear of childhood vaccines, particularly the measles, mumps, and rubella (MMR) vaccine and the concern it can cause autism. (shrink)

This chapter focuses on one of the common fallacies in Western philosophy: 'false cause'. In general, the false cause fallacy occurs when the “link between premises and conclusion depends on some imagined causal connection that probably does not exist”. There are three different ways an argument can commit the false cause fallacy: post hoc ergo propter hoc; cum hoc ergo propter hoc; and ignoring common cause. Like the post hoc ergo propter hoc fallacy, this fallacy is (...) guilty of trying to establish a causal connection between two events on dubious grounds. The way to avoid committing the cum hoc ergo propter hoc fallacy is to study correlative relationships more carefully in order to decipher if an actual causal relationship exists rather than assuming the latter follows from the former. (shrink)

It is now—at least loosely—acknowledged that most health and clinical outcomes are influenced by different interacting causes. Surprisingly, medical research studies are nearly universally designed to study—usually in a binary way—the effect of a single cause. Recent experiences during the coronavirus disease 2019 pandemic brought to the forefront that most of our challenges in medicine and healthcare deal with systemic, that is, interdependent and interconnected problems. Understanding these problems defy simplistic dichotomous research methodologies. These insights demand a shift in (...) our thinking from ‘cause and effect’ to ‘causes and effects’ since this transcends the classical way of Cartesian reductionist thinking. We require a shift to a ‘causes and effects’ frame so we can choose the research methodology that reflects the relationships between variables of interest—one-to-one, one-to-many, many-to-one or many-to-many. One-to-one (or cause and effect) relationships are amenable to the traditional randomized control trial design, while all others require systemic designs to understand ‘causes and effects’. Researchers urgently need to re-evaluate their science models and embrace research designs that allow an exploration of the clinically obvious multiple ‘causes and effects’ on health and disease. Clinical examples highlight the application of various systemic research methodologies and demonstrate how ‘causes and effects’ explain the heterogeneity of clinical outcomes. This shift in scientific thinking will allow us to find the necessary personalized or precise clinical interventions that address the underlying reasons for the variability of clinical outcomes and will contribute to greater health equity. (shrink)

People generally accept that there is causation by omission—that the omission of some events cause some related events. But this acceptance elicits the selection problem, or the difficulty of explaining the selection of a particular omissive cause or class of causes from the causal conditions. Some theorists contend that dependence theories of causation cannot resolve this problem. In this paper, we argue that the appeal to norms adequately resolves the selection problem for dependence theories, and we provide novel (...) experimental evidence for it. (shrink)

Hunting Causes and Using Them argues that causation is not one thing, as commonly assumed, but many. There is a huge variety of causal relations, each with different characterizing features, different methods for discovery and different uses to which it can be put. In this collection of new and previously published essays, Nancy Cartwright provides a critical survey of philosophical and economic literature on causality, with a special focus on the currently fashionable Bayes-nets and invariance methods - and it exposes (...) a huge gap in that literature. Almost every account treats either exclusively how to hunt causes or how to use them. But where is the bridge between? It's no good knowing how to warrant a causal claim if we don't know what we can do with that claim once we have it. This book will interest philosophers, economists and social scientists. (shrink)

In Causes, Laws, and Free Will, Kadri Vihvelin argues that we can have free will even if everything we do is predictable given the laws of nature and the past. The belief that determinism robs us of free will springs from mistaken beliefs about the metaphysics of causation, the nature of laws, and the logic of counterfactuals.

Most people are skeptical of the claim that the expectation that a person would have a life that would be well worth living provides a reason to cause that person to exist. In this essay I argue that to cause such a person to exist would be to confer a benefit of a noncomparative kind and that there is a moral reason to bestow benefits of this kind. But this conclusion raises many problems, among which is that it (...) must be determined how the benefits conferred on people by causing them to exist weigh against comparable benefits conferred on existing people. In particular, might the reason to cause people to exist ever outweigh the reason to save the lives of existing people? (shrink)

This volume offers an updated analysis of the use, meaning, and scope of the classical notion of aitía. It clarifies philosophical and philological questions about aitia and offers bold and innovative interpretations of this key concept of ancient philosophy. The numerous meanings and nuances of aitia remain difficult to grasp. Ancient philosophers use aitia to explain the existence and activity of substances, bodies, souls, or gods, Paradoxically, its own definition remains difficult to establish. This book reconstructs some of the most (...) important uses, variants, and scopes of the term aitia within different philosophical perspectives in antiquity, including early Greek philosophy, Plato, Aristotle, Stoicism, and Islamic philosophy. The chapters analyze metaphysical aspects, epistemological issues, and logical implications of aitia. They engage with the most relevant critical literature generated in several modern languages. In doing so, they offer an inclusive and overarching re-evaluation of our assumptions about causation and explanation in ancient philosophy. Cause and Explanation in Ancient Philosophy will be of interest to scholars and advanced students working on pre-Socratic philosophy, Plato, Aristotle, Hellenistic philosophy, late antiquity, and medieval philosophy. (shrink)

A just cause for war is a type of wrong that may make those responsible for it morally liable to military attack as a means of preventing or rectifying it. This claim has implications that conflict with assumptions of the current theory of just war.

Michael T. Ferejohn presents a new analysis of Aristotle's theory of explanation and scientific knowledge, in the context of its Socratic roots. Ferejohn shows how Aristotle resolves the tension between his commitment to the formal-case model of explanation and his recognition of the role of efficient causes in explaining natural phenomena.

Paul Pietroski presents an original philosophical theory of actions and their mental causes. We often act for reasons: we deliberate and choose among options, based on our beliefs and desires. However, bodily motions always have biochemical causes, so it can seem that thinking and acting are biochemical processes. Pietroski argues that thoughts and deeds are in fact distinct from, though dependent on, underlying biochemical processes within persons.

Biologists studying complex causal systems typically identify some factors as causes and treat other factors as background conditions. For example, when geneticists explain biological phenomena, they often foreground genes and relegate the cellular milieu to the background. But factors in the milieu are as causally necessary as genes for the production of phenotypic traits, even traits at the molecular level such as amino acid sequences. Gene-centered biology has been criticized on the grounds that because there is parity among causes, the (...) “privileging” of genes reflects a reductionist bias, not an ontological difference. The idea that there is an ontological parity among causes is related to a philosophical puzzle identified by John Stuart Mill: what, other than our interests or biases, could possibly justify identifying some causes as the actual or operative ones, and other causes as mere background? The aim of this paper is to solve this conceptual puzzle and to explain why there is not an ontological parity among genes and the other factors. It turns out that solving this puzzle helps answer a seemingly unrelated philosophical question: what kind of causal generality matters in biology? (shrink)

How do people understand questions about cause and prevent? Some theories propose that people affirm that A causes B if A's occurrence makes a difference to B's occurrence in one way or another. Other theories propose that A causes B if some quantity or symbol gets passed in some way from A to B. The aim of our studies is to compare these theories' ability to explain judgements of causation and prevention. We describe six experiments that compare judgements for (...) causal paths that involve a mechanism, i.e. a continuous process of transmission or exchange from cause to effect, against paths that involve no mechanism yet a change in the cause nevertheless brings about a change in the effect. Our results show that people prefer to attribute cause when a mechanism links cause to effect. In contrast, prevention is sensitive both to the presence of an interruption to a causal mechanism and to a change in the outcome in the absence of a mechanism. In this sense, ‘prevent’ means something different than ‘cause not'. We discuss the implications of our results for existing theories of causation. (shrink)

Huw Price has argued that on an interventionist account of cause the distinction is perspectival, and the claim prompted some interesting responses from interventionists and in particular an exchange with Woodward that raises questions about what it means to say that one or another structure is perspectival. I’ll introduce his reasons for claiming that the distinction between cause and effect on an interventionist account is perspectival. Then I’ll introduce a distinction between different ways in which a class of (...) concepts can be said to depend on facts about their users. Three importantly different forms of dependence will emerge from the discussion: Pragmatic dependence on us: truth conditions for x-beliefs can be given by a function f \ of more fundamental physical structures making no explicit reference to human agents. But there are any other number of functions ) ontologically on a par with x and what explains the distinguished role f plays in our practical and epistemic lives are facts about us. Implicit relativization: truth conditions for x-beliefs are relative to agent or context; the context supplies the value of a hidden parameter that determines the truth of x-beliefs. Indexicals: like implicit relativization except that the surface syntax contains a term whose semantic value is context-dependent. I suggest that Price’s insights are best understood in the first way. This will draw a crucial disanalogy with his central examples of perspectival concepts, but it will refine the thesis in a way that is more faithful to what his arguments show. The refined thesis will also support generalization to other concepts, and clarify the foundations of the quite distinctive research program that Price has been developing for a number of years. (shrink)

We propose a new definition of actual causes, using structural equations to model counterfactuals. We show that the definition yields a plausible and elegant account of causation that handles well examples which have caused problems for other definitions and resolves major difficulties in the traditional account.

This paper considers and criticises what appears to be a suggestion by Hart and Honore in 'Causation in the Law' that there is a category of basic doings, which ought not themselves to be regarded as causings. It argues instead that all actions are causings by the agent.

The present volume discuss aspects of "Cause, Knowledge and Responsibility" from various theoretical points of view. The contributions provide input to intense discussions on these contested topics. The volume is based on results of the conference of the Institut International de Philosophie (IIP) in 9-13 September 2009, at the University of Coimbra.

'A truly revolutionary idea.... Clear, tight, disciplined, beautifully structured, and brilliantly reasoned.'--Ayn Rand. Self-sacrifice, oriental mysticism, racial 'truth,' the public good, doing one's duty -- these are among the seductive catchphrases that circulated in pre-Nazi Germany. Objectivist author and philosopher Leonard Peikoff was Ayn Rand's long-time associate. In The Cause of Hitler's Germany -- previously published in The Ominous Parallels -- Peikoff demonstrates how unreason and collectivism led the seemingly civilized German society to become a Nazi regime.

Within evolutionary biology a distinction is frequently made between proximate and ultimate causes. One apparently plausible interpretation of this dichotomy is that proximate causes concern processes occurring during the life of an organism while ultimate causes refer to those processes (particularly natural selection) that shaped its genome. But ultimate causes are not sought through historical investigations of an organisms lineage. Rather, explanations referring to ultimate causes typically emerge from functional analyses. But these functional analyses do not identify causes of any (...) kind, much less ultimate ones. So-called ultimate explanations are not about causes in any sense resembling those of proximate explanations. The attitude, implicit in the term ultimate cause, that these functional analyses are somehow superordinate to those involving proximate causes is unfounded. Ultimate causes are neither ultimate nor causes. (shrink)

Causation is everywhere in the world: it features in every science and technology. But how much do we understand it? Mumford and Anjum develop a new theory of causation based on an ontology of real powers or dispositions. They provide the first detailed outline of a thoroughly dispositional approach, and explore its surprising features.

Race and nationality.--Ancients and moderns.--Liberty.--Ultimate facts in economics.--Auto-suggestion.--Nervous tri-unity.--The laws of the mind.--The brain as a laboratory.--Time and space.--Vocabulary and grammar.--Logic.--Motives and feelings.--Free will, trial, and choice.--The foundations of morality.--The development of art.--Amusement.

When two rocks shatter the window at once, what causes the window to shatter? Is the throwing of each individual rock a cause of the window shattering, or are the throwings only causes collectively? This question bears on the analysis of causation, and the metaphysics of macro-causation. I argue that the throwing of each individual rock is a cause of the window shattering, and generally that individual overdeterminers are causes.

A discussion of Aristotle’s thought on determinism and culpability, Necessity, Cause, and Blame also reveals Richard Sorabji’s own philosophical commitments. He makes the original argument here that Aristotle separates the notions of necessity and cause, rejecting both the idea that all events are necessarily determined as well as the idea that a non-necessitated event must also be non-caused. In support of this argument, Sorabji engages in a wide-ranging discussion of explanation, time, free will, essence, and purpose in nature. (...) He also provides historical perspective, arguing that these problems remain intimately bound up with modern controversies. “ Necessity, Cause and Blame would be counted by all as one of Sorabji’s finest. The book is essential for philosophers—both specialists on the Greeks and modern thinkers about free will—and also compelling for non-specialists.”—Martha Nussbaum “Original and important . . . The book relates Aristotle’s discussions to both the contemporary debates on determinism and causation and the ancient ones. It is especially detailed on Stoic arguments about necessity . . . and on the social and legal background to Aristotle’s thought.”— Choice “It is difficult to convey the extraordinary richness of this book. . . . A Greekless philosopher could read it with pleasure . . . At the same time, its learning and scholarship are enormous.”—G. E. M. Anscombe, Times Literary Supplement. (shrink)

A condition is formulated in terms of the probabilities of two pairs of correlated events in a classical probability space which is necessary for the two correlations to have a single (Reichenbachian) common-cause and it is shown that there exists pairs of correlated events probabilities of which violate the necessary condition. It is concluded that different correlations do not in general have a common common-cause. It is also shown that this conclusion remains valid even if one weakens slightly (...) Reichenbach's definition of common-cause. The significance of the difference between common-causes and common common-causes is emphasized from the perspective of Reichenbach's Common Cause Principle. (shrink)

I argue that the criterion of just cause is not independent of proportionality and other valid jus ad bellum criteria. One cannot know whether there is a just cause without knowing whether the other (valid) criteria (apart from ‘right intention’) are satisfied. The advantage of this account is that it is applicable to all wars, even to wars where nobody will be killed or where the enemy has not committed a rights violation but can be justifiably warred against (...) anyway. This account also avoids the inefficiency of having proportionality considerations come up at two different points: in a separate criterion of just cause and in the criterion of proportionality proper. ‘Right intention’, the subjective element of the justification of a war, on the other hand, is not to be subsumed under the criterion of just cause: there can be a just cause without anybody knowing it. Conversely, however, the subjective element requires that those responsible for waging the war do know that the justifying objective conditions are fulfilled. This is in one sense more demanding than traditional just war theory; in another sense, however, it is less demanding: nobody needs to intend to fight for a ‘just aim’. (shrink)

Negative causation occurs when an absence serves as cause, effect, or causal intermediary. Negative causation is genuine causation, or so I shall argue. It involves no physical connection between cause and effect. Thus causes need not be physically connected to their effects.

We propose a new definition of actual causes, using structural equations to model counterfactuals. We show that the definition yields a plausible and elegant account of causation that handles well examples which have caused problems for other definitions and resolves major difficultiesn in the traditional account.

We propose a new definition of actual causes, using structural equations to model counterfactuals. We show that the definition yields a plausible and elegant account of causation that handles well examples which have caused problems for other definitions and resolves major difficulties in the traditional account. 1. Introduction2. Causal models: a review2.1Causal models2.2Syntax and semantics3. The definition of cause4. Examples5. A more refined definition6. DiscussionAAppendix: Some Technical IssuesA.1The active causal processA.2A closer look at AC2(b)A.3Causality with infinitely many variablesA.4Causality in nonrecursive (...) models. (shrink)

Kyle Stanford has recently given substance to the problem of unconceived alternatives, which challenges the reliability of inference to the best explanation (IBE) in remote domains of nature. Conjoined with the view that IBE is the central inferential tool at our disposal in investigating these domains, the problem of unconceived alternatives leads to scientific anti-realism. We argue that, at least within the biological community, scientists are now and have long been aware of the dangers of IBE. We re-analyse the nineteenth-century (...) study of inheritance and development (Stanford’s case study) and extend it into the twentieth century, focusing in particular on both classical Mendelian genetics and the studies by Avery et al. on the chemical nature of the hereditary substance. Our extended case studies show the preference of the biological community for a different methodological standard: the vera causa ideal, which requires that purported causes be shown on non-explanatory grounds to exist and be competent to produce their effects. On this basis, we defend a prospective realism about the biological sciences. 1 Introduction2 Stanford on Unconceived Alternatives3 Pangenesis: A Re-analysis4 The Vera Causa Ideal I: Negative Aspect5 Rendering Heredity Accessible6 The Vera Causa Ideal II: Positive Aspect7 Implications for Scientific Realism8 Conclusion. (shrink)

It may seem obvious that causing disability in another person is morally problematic in a way that removing or preventing a disability is not. This suggests that there is a moral asymmetry between causing disability and causing non-disability. This chapter investigates whether there are any differences between these two types of actions that might explain the existence of a general moral asymmetry. After setting aside the possibility that having a disability is almost always bad or harmful for a person (a (...) view that we have critiqued at length elsewhere), seven putative differences are considered. Ultimately, it is concluded that none of these seven factors can ground a general moral asymmetry between causing disability and causing non-disability, though each factor can provide some moral reason to avoid causing disability in certain particular cases. (shrink)

We investigate a hitherto under-considered avenue of response for the logical pluralist to collapse worries. In particular, we note that standard forms of the collapse arguments seem to require significant order-theoretic assumptions, namely that the collection of admissible logics for the pluralist should be closed under meets and joins. We consider some reasons for rejecting this assumption, noting some prima facie plausible constraints on the class of admissible logics which would lead a pluralist admitting those logics to resist such closure (...) conditions. (shrink)

This book explores and defends George Saunders’ causal thesis that successful stories are those that establish causation well. The book includes an in-depth discussion of causation’s role in several different key craft elements of fiction writing and examines different theories of causation and their implications for causation in fiction. Other discussions include the role of causation in building suspense, character and causation, causation in dialogue and connections between fiction and counterfactuals (or hypotheticals). The book also considers a number of objections (...) to the causal thesis and offers a reply. (shrink)

The interventionist account of causation offers a criterion to distinguish causes from non-causes. It also aims at defining various desirable properties of causal relationships, such as specificity, proportionality and stability. Here we apply an information-theoretic approach to these properties. We show that the interventionist criterion of causation is formally equivalent to non-zero specificity, and that there are natural, information-theoretic ways to explicate the distinction between potential and actual causal influence. We explicate the idea that the description of causes should be (...) proportional to that of their effects. Then we draw a distinction between two ideas in the existing literature, the range of invariance of a causal relationship and its stability. The range of invariance is related to specificity and range of causal values. Stability concerns the effect of additional variables on the relationship between some focal pair of cause and effect variables. We show how to distinguish and measure the direct influence of background variables on the effect variable, and their influence on the relationship between the focal cause and the effect variable. Finally, we discuss the limitations of the information-theoretic approach, and offer prospects for complementary approaches. (shrink)

Much of the philosophical literature on causation has focused on the concept of actual causation, sometimes called token causation. In particular, it is this notion of actual causation that many philosophical theories of causation have attempted to capture.2 In this paper, we address the question: what purpose does this concept serve? As we shall see in the next section, one does not need this concept for purposes of prediction or rational deliberation. What then could the purpose be? We will argue (...) that one can gain an important clue here by looking at the ways in which causal judgments are shaped by people‘s understanding of norms. (shrink)

Is addiction a behavioral pattern, or the underlying cause of a behavioral pattern? Both views are found in prominent accounts of addiction, but theorists generally do not notice that they are taking a controversial position, let alone justify it. A third possibility is that addiction consists in both addictive behavior and its causes, though this view is less obviously present in the literature. I argue that two important considerations favor the "cause view" over the "behavior" and "hybrid" views. (...) The first is that we want to be able to explain addictive behavior with reference to addiction. The second is that addiction seems to persist long after addictive behavior has ceased. Only the cause view can easily accommodate both data points. (shrink)

This paper is concerned with the connection between the causal exclusion argument and the supervenience argument and, in particular, with two exclusion principles that figure prominently in these arguments. Our aim is, first, to reconstruct the dialectics of the two arguments by formalizing them and by relating them to an anti-physicalist argument by Scott Sturgeon. In a second step, we assess the conclusiveness of the two arguments. We demonstrate that the conclusion of both the causal exclusion argument and the supervenience (...) argument (a negation of the so called ?non-identity premise?) is not tantamount to the claim that all mental events are identical to physical events and that a cause-supervenience exclusion principle is actually required for the validity of the first stage of the supervenience argument. Moreover, we show that this new exclusion principle makes an epiphenomenalism with respect to mental events an impossible position, and that there are good reasons to believe in its falsity, rendering the supervenience argument inconclusive. (shrink)