This chapter focuses on the experimental practices and reasoning strategies employed in nineteenth century investigations on the causal origin of the phenomenon of Brownian movement. It argues that there was an extensive and sophisticated experimental work done on the phenomenon throughout the nineteenth century. Investigators followed as rigorously as possible the methodological standards of their time to make causal claims and advance causal explanations of Brownian movement. Two major methodological strategies were employed. The first was the experimental (...) strategy of varying the circumstances. Suspected causal factors were varied and the resulting effect on Brownian movement was studied. The main goal of this strategy was the identification of difference-making factors, i.e., factors having a causal influence on the phenomenon. The second was the so-called method of hypothesis. Rather than relying exclusively on the ability of experiments to identify difference-making factors, its proponents tried to show how the independently developed tenets of the new kinetic-molecular conception of matter provided a plausible causal explanation of Brownian movement. Each one of these methodological strategies had its distinctive practices and notions of control. None of these strategies could, on its own, establish molecular motion as the cause of Brownian movement. It was only the fruitful combination of the two strategies and of their accompanying practices and notions of control that led, at the end of the nineteenth century, to the recognition of molecular motion as the most probable cause of Brownian movement. (shrink)

The idea that intentions make the difference between voluntary and non-voluntary behaviors is simple and intuitive. At the same time, we lack an understanding of how voluntary actions actually come about, and the unquestioned appeal to intentions as discrete causes of actions offers little if anything in the way of an answer. We cite evidence suggesting that the origin of actions varies depending on context and effector, and argue that actions emerge from a causal web in the brain, rather (...) than a central origin of intentional action. We argue that this causal web need not be confined to the central nervous system, and that proprioceptive feedback might play a counterintuitive role in the decision process. Finally we argue that the complex and dynamic origins of voluntary action and their interplay with the brain’s propensity to predict the immediate future are better studied using a dynamical systems approach. (shrink)

Studies of primate cognition have conclusively shown that humans and apes share a range of basic cognitive abilities. As a corollary, these same studies have also focussed attention on what makes humans unique, and on when and how specifically human cognitive skills evolved. There is widespread agreement that a major distinguishing feature of the human mind is its capacity for causal reasoning. This paper argues that causal cognition originated with the use made of indirect natural signs by early (...) hominins forced to adapt to variable late Miocene and early Pliocene environments; that early hominins evolved an innate tendency to search for such signs and infer their causes; that causal inference required the existence of incipient working memory; and that causal relationships were stored through being integrated into spatial maps to create increasingly complex causal models of the world. (shrink)

There are several interpretations of the argument structure of Darwin's Origin of Species, representing Covering-Law, Inference-to-the-Best-Explanation, and (more recently) Semantic models. I argue that while all three types of interpretation enjoy some textual support, none succeeds in capturing the overall strategy of the Origin, consistent with Darwin's claim that it is 'one long argument'. I provide detailed criticisms of all three current models, and then offer an alternative interpretation based on the view that there are three main argument strategies in (...) the Origin, all supporting the 'causal efficacy' of Darwin's theory. This interpretation provides both a more unified treatment of the text, and some important implications concerning the relation between general philosophical models of scientific theory support and specific historical cases. (shrink)

In his well-known 1952 dialogue Max Black describes a counterexample to the Principle of the Identity of Indiscernibles (PII). The counterexample is a world containing nothing but two purportedly indiscernible iron spheres. Reflecting on Black's example, Robert Adams uses the possibility of a world containing two almost indiscernible spheres to argue for the possibility of the indiscernible spheres world. One of Adams's almost indiscernible spheres has a small impurity, and, Adams writes, "Surely... the absence of the impurity would not make (...) such a universe impossible." The appeal to "surely" constitutes a gap in Adams's argument. This paper bridges the gap with a premise that exploits the counterfactual conditional and the related notion of causal independence. The paper then argues that causal independence bears in a similar way on an issue Nathan Salmon raises concerning Kripke's argument for the Essentiality of Origins. (shrink)

Did affluence lead to psychological changes such as reduced discounting, and did these changes facilitate the innovation associated with the Industrial Revolution? I argue that claims of this sort are best made when they can be supported by causal evidence and good psychological measurement. When we have neither identifying variation nor adequate measures, the toolbox of psychologists is not useful.

In this paper a theory relating to the causal factors operative in the origin of living systems is presented.Let us consider living forms as material systems exhibiting, in addition to those properties held in common with all matter, systemic properties of a specific nature. If, then, the matter of the earth is classified from this standpoint, it is found that these systems are distributed only over the surface of the earth or in a shallow upper layer. This fact indicates (...) that the special properties of these systems are related in some manner to factors introduced at the surface and not operative elsewhere. (shrink)

„Errors are the greatest obstacles to the progress of science; to correct such errors is of more practical value than to achieve new knowledge,“ asserted Eugen Bleuler. Basic error of several prevailing classification schemes of pathological conditions, as for example ICD-10, lies in confusing and mixing symptoms with diseases, what makes them unscientific. Considering the need to bring order into the chaos and light into terminological obscureness, I introduce the Causal classification of diseases originating from the notion of bodily (...) wholeness and causality of its loss. (shrink)

This paper poses an original puzzle about the relationship between causation and moral responsibility called The Moral Difference Puzzle. Using the puzzle, the paper argues for three related ideas: (1) the existence of a new sort of moral luck; (2) an intractable conflict between the causal concepts used in moral assessment; and (3) inability of leading theories of causation to capture the sorts of causal differences that matter for moral evaluation of agents’ causal contributions to outcomes.

Discovering high-level causal relations from low-level data is an important and challenging problem that comes up frequently in the natural and social sciences. In a series of papers, Chalupka etal. (2015, 2016a, 2016b, 2017) develop a procedure forcausal feature learning (CFL) in an effortto automate this task. We argue that CFL does not recommend coarsening in cases where pragmatic considerations rule in favor of it, and recommends coarsening in cases where pragmatic considerations rule against it. We propose a new (...) technique, pragmatic causal feature learning (PCFL), which extends the original CFL algorithm in useful and intuitive ways. We show that PCFL has the same attractive measure-theoretic properties as the original CFL algorithm. We compare the performance of both methods through theoretical analysis and experiments. (shrink)

It is well established that the temporal proximity of two events is a fundamental cue to causality. Recent research with adults has shown that this relation is bidirectional: events that are believed to be causally related are perceived as occurring closer together in time—the so‐called temporal binding effect. Here, we examined the developmental origins of temporal binding. Participants predicted when an event that was either caused by a button press, or preceded by a non‐causal signal, would occur. We (...) demonstrate for the first time that children as young as 4 years are susceptible to temporal binding. Binding occurred both when the button press was executed via intentional action, and when a machine caused it. These results suggest binding is a fundamental, early developing property of perception and grounded in causal knowledge. (shrink)

Possibly the most fundamental scientific problem is the origin of time and causality. The inherent difficulty is that all scientific theories of origins and evolution consider the existence of time and causality as given. We tackle this problem by starting from the concept of self-organization, which is seen as the spontaneous emergence of order out of primordial chaos. Self-organization can be explained by the selective retention of invariant or consistent variations, implying a breaking of the initial symmetry exhibited by (...) randomness. In the case of time, we start from a random graph connecting primitive “events”. Selection on the basis of consistency eliminates cyclic parts of the graph, so that transitive closure can transform it into a partial order relation of precedence. Causality is assumed to be carried by causal “agents” which undergo a more traditional variation and selection, giving rise to causal laws that are partly contingent, partly necessary. (shrink)

Causal Modeling Semantics (CMS, e.g., Galles and Pearl 1998; Pearl 2000; Halpern 2000) is a powerful framework for evaluating counterfactuals whose antecedent is a conjunction of atomic formulas. We extend CMS to an evaluation of the probability of counterfactuals with disjunctive antecedents, and more generally, to counterfactuals whose antecedent is an arbitrary Boolean combination of atomic formulas. Our main idea is to assign a probability to a counterfactual (A ∨ B) > C at a causal model M as (...) a weighted average of the probability of C in those submodels that truthmake A∨ B (Briggs 2012; Fine 2016, 2017). The weights of the submodels are given by the inverse distance to the original model M, based on a distance metric proposed by Eva, Stern, and Hartmann (2019). Apart from solving a major problem in the epistemology of counterfactuals, our paper shows how work in semantics, causal inference and formal epistemology can be fruitfully combined. (shrink)

The Lotka–Volterra predator-prey-model is a widely known example of model-based science. Here we reexamine Vito Volterra’s and Umberto D’Ancona’s original publications on the model, and in particular their methodological reflections. On this basis we develop several ideas pertaining to the philosophical debate on the scientific practice of modeling. First, we show that Volterra and D’Ancona chose modeling because the problem in hand could not be approached by more direct methods such as causal inference. This suggests a philosophically insightful motivation (...) for choosing the strategy of modeling. Second, we show that the development of the model follows a trajectory from a “how possibly” to a “how actually” model. We discuss how and to what extent Volterra and D’Ancona were able to advance their model along that trajectory. It turns out they were unable to establish that their model was fully applicable to any system. Third, we consider another instance of model-based science: Darwin’s model of the origin and distribution of coral atolls in the Pacific Ocean. Darwin argued more successfully that his model faithfully represents the causal structure of the target system, and hence that it is a “how actually” model. (shrink)

The evolution of the human mind through natural selection mandates that our conscious experiences are causally potent in order to leave a tangible impact upon the surrounding physical world. Any attempt to construct a functional theory of the conscious mind within the framework of classical physics, however, inevitably leads to causally impotent conscious experiences in direct contradiction to evolution theory. Here, we derive several rigorous theorems that identify the origin of the latter impasse in the mathematical properties of ordinary differential (...) equations employed in combination with the alleged functional production of the mind by the brain. Then, we demonstrate that a mind--brain theory consistent with causally potent conscious experiences is provided by modern quantum physics, in which the unobservable conscious mind is reductively identified with the quantum state of the brain and the observable brain is constructed by the physical measurement of quantum brain observables. The resulting quantum stochastic dynamics obtained from sequential quantum measurements of the brain is governed by stochastic differential equations, which permit genuine free will exercised through sequential conscious choices of future courses of action. Thus, quantum reductionism provides a solid theoretical foundation for the causal potency of consciousness, free will and cultural transmission. (shrink)

Norton has recently argued that causation is merely a useful folk concept and that it fails to hold for some simple systems even in the supposed paradigm case of a causal physical theory – namely Newtonian mechanics. The purpose of this article is to argue against this devaluation of causality in physics. My main argument is that Norton’s alleged counterexample to causality within standard Newtonian physics fails to obey what I shall call the causal core of Newtonian mechanics. (...) In particular, I argue, Norton’s example is not in conformity with Newton’s first law. Moreover, Norton’s reformulation of this first law seems insufficient as a replacement for the original version since the notion of inertial frames in the resulting reformulated theory lacks a physical justification, and since an intelligible notion of time in Newtonian mechanics appears to be closely tied to Newton’s first law in its standard form. I will finally suggest how, given a plausible relationist account of time, the causal core of Newtonian mechanics may play a central role also in relativity and quantum theory. (shrink)

The paper argues that on three out of eight possible hypotheses about the EPR experiment we can construct novel and realistic decision problems on which (a) Causal Decision Theory and Evidential Decision Theory conflict (b) Causal Decision Theory and the EPR statistics conflict. We infer that anyone who fully accepts any of these three hypotheses has strong reasons to reject Causal Decision Theory. Finally, we extend the original construction to show that anyone who gives any of the (...) three hypotheses any non-zero credence has strong reasons to reject Causal Decision Theory. However, we concede that no version of the Many Worlds Interpretation (Vaidman, in Zalta, E.N. (ed.), Stanford Encyclopaedia of Philosophy 2014) gives rise to the conflicts that we point out. (shrink)

The idea that causation can be reduced to transmission of an amount of some conserved quantity between events is spelled out and defended against important objections. Transmission is understood as a symmetrical relation of copresence in two distinct events. The actual asymmetry of causality has its origin in the asymmetrical character of certain irreversible physical processes and then spreads through the causal net. This conception is compatible with the possibility of backwards causation and with a causal theory of (...) time. Genidentity, the persistence of concrete objects, can be given an explanation in causal terms. The transmission theory is shown to escape difficulties faced by two important alternative theories of causation: Salmon's (1984) Mark Transmission Theory and Dowe's (1992a) Conserved Quantities Theory. (shrink)

This work presents a causal conception of metaphysical modality in which a state of affairs is metaphysically possible if and only if it can be caused (in the past, the present or the future) by current entities. The conception is contrasted with what is called the "combinatorial" conception of modality, in which everything can coexist with anything else. This work explains how the notion of 'causality' should be construed in the causal theory, what difference exists between modalities thus (...) defined from nomological modality, how accessibility relations between possible worlds should be interpreted, and what is the relation between the causal conception and the necessity of origin. (shrink)

The causal characters (spacelike, lightlike, timelike) of the coordinate lines, coordinate surfaces and coordinate hypersurfaces of a coordinate system in Relativity define what is called its causal class. It is known that, in any relativistic space-time, there exist one hundred and ninety nine such causal classes. But in Newtonian physics (where only spacelike and timelike characters exist) the corresponding causal classes have not been discussed until recently. Here it is shown that, in sharp contrast with the (...) relativistic case, in Newtonian space-time the different causal classes of coordinate systems are drastically reduced to four. These causal classes admit simple geometric descriptions and physical interpretations. For example, it is shown that one can generate coordinate systems of the four causal classes by means of the sole linear synchronization group, i.e. by coordinate transformations that only change the origin of time linearly. The relativistic analogs of these examples are also considered. (shrink)

Perfect adaptation in a dynamical system is the phenomenon that one or more variables have an initial transient response to a persistent change in an external stimulus but revert to their original value as the system converges to equilibrium. With the help of the causal ordering algorithm, one can construct graphical representations of dynamical systems that represent the causal relations between the variables and the conditional independences in the equilibrium distribution. We apply these tools to formulate sufficient graphical (...) conditions for identifying perfect adaptation from a set of first-order differential equations. Furthermore, we give sufficient conditions to test for the presence of perfect adaptation in experimental equilibrium data. We apply this method to a simple model for a protein signalling pathway and test its predictions in both simulations and using real-world protein expression data. We demonstrate that perfect adaptation can lead to misleading orientation of edges in the output of causal discovery algorithms. (shrink)

The finding of fractal scaling (FS) in behavioral sequences has raised a debate on whether FS is a pervasive property of the cognitive system or is the result of specific processes. Inferences about the origins of properties in time sequences are causal. That is, as opposed to correlational inferences reflecting instantaneous symmetrical relations, causal inferences concern asymmetric relations lagged in time. Here, I integrate Granger‐causality with inferences about FS. Four simulations illustrate that causal analyses can isolate (...) distinct FS sources, whereas correlational techniques cannot. I then analyze three simultaneous sequences of responses from a database of word‐naming trials. I find that two, or perhaps three, distinct sources account for the presence of FS in these sequences, but FS is not a general property of the system. This suggests that FS arises due to the properties of a limited number of identifiable psychological and/or neural processes. Finally, I reanalyze a previously published dataset of acoustic frequency spectra using the new tools. The causality/criticality combination introduced here offers a new important perspective in the study of cognition. (shrink)

It is natural to think of remembering in terms of causation: I can recall a recent dinner with a friend because I experienced that dinner. Some fifty years ago, Martin and Deutscher (1966) turned this basic thought into a full-fledged theory of memory, a theory that came to dominate the landscape in the philosophy of memory. Remembering, Martin and Deutscher argue, requires the existence of a specific sort of causal connection between the rememberer's original experience of an event and (...) his later representation of that event: a causal connection sustained by a memory trace. In recent years, it has become apparent that this reference to memory traces may be out of step with memory science. Contemporary proponents of the causal theory are thus confronted with the question: is it possible to develop an empirically adequate version of the theory, or is it time to move beyond it? This chapter traces the recent history of the causal theory, showing how increased awareness of the theory’s problems has led to the development of modified version of the causal theory and ultimately to the emergence of postcausal theories. (shrink)

This book, first published in 1936, divides into roughly two parts: a re-examination of historical material; and a positive theory of causation suggested by the results of this re-examination. The historical study discloses an ambiguity in the meanings of causation and determinism; it discloses also that this ambiguity is transferred to the meaning of freedom.

The finding of fractal scaling (FS) in behavioral sequences has raised a debate on whether FS is a pervasive property of the cognitive system or is the result of specific processes. Inferences about the origins of properties in time sequences are causal. That is, as opposed to correlational inferences reflecting instantaneous symmetrical relations, causal inferences concern asymmetric relations lagged in time. Here, I integrate Granger-causality with inferences about FS. Four simulations illustrate that causal analyses can isolate (...) distinct FS sources, whereas correlational techniques cannot. I then analyze three simultaneous sequences of responses from a database of word-naming trials. I find that two, or perhaps three, distinct sources account for the presence of FS in these sequences, but FS is not a general property of the system. This suggests that FS arises due to the properties of a limited number of identifiable psychological and/or neural processes. Finally, I reanalyze a previously published dataset of acoustic frequency spectra using the new tools. The causality/criticality combination introduced here offers a new important perspective in the study of cognition. (shrink)

There has been no shortage of such conceptual analyses and no shortage of counterexamples to all of them. The counterexamples exploit, at least partly, situations in which we are presumed to have clear intuitions about what causes what, but which intuitions are not being respected by the suggested philosophical analysis. The counterexamples typically lead to a battery of sophisticated attempts to revise or amend the philosophical analysis so that it is saved from refutation. These attempts, typically, either deny the intuitions (...) on which the counterexamples are based or accommodate the problematic cases within the theory by adding further clauses to the original philosophical analysis. The result of all this is that where the original philosophical theory rested on a simple, forceful and intuitively plausible idea (e.g., that causation consists in a relation of counterfactual dependence between discrete events), the modified philosophical theory becomes very convoluted, somewhat ad hoc and implausible. (shrink)

The first part of this paper shows that Qualitative Comparative Analysis (QCA)--also in its most recent forms as presented in Ragin (2000, 2008)--, does not correctly analyze data generated by causal chains, which, after all, are very common among causal processes in the social sciences. The incorrect modeling of data originating from chains essentially stems from QCA’s reliance on Quine-McCluskey optimization to eliminate redundancies from sufficient and necessary conditions. Baumgartner (2009a,b) has introduced a Boolean methodology, termed Coincidence Analysis (...) (CNA), that is related to QCA, yet, contrary to the latter, does not eliminate redundancies by means of Quine-McCluskey optimization. The second part of the paper applies CNA to chain-generated data. It will turn out that CNA successfully detects causal chains in small-N data. (shrink)

The doctrine of the immaculate conception, which is a dogma binding on all Roman Catholics and also held by members of some other Christian denominations, holds that Mary the mother of Jesus Christ was conceived without the stain of original sin as a result of the redeeming effects of Christ’s later life, passion, death, and resurrection. In this paper I argue first that, even on an orthodox reading of this doctrine, the immaculate conception seems to result in a kind of (...) causal time loop. I then consider several common philosophical objections to causal time loops, showing how each is either not a serious problem for causal time loops in general or is not a serious problem for the immaculate conception time loop in particular because of some particular features of that particular loop. The upshot of this discussion is that it shows that anyone who is committed to the dogma of the immaculate conception is also committed to the possibility, and, indeed, the actuality, of at least one causal time loop, but also that this is no reason to reject the dogma, since all of the major worries for causal time loops can be resolved in one way or another. (shrink)

Not only has the philosophical debate on causation been gaining ground in the last few decades, but it has also increasingly addressed the sciences. The biomedical sciences are among the most prominent fields that have been considered, with a number of works tackling the understanding of the notion of cause, the assessment of genuinely causal relations and the use of causal knowledge in applied contexts. Far from denying the merits of the debate on causation and the major theories (...) it comprises, this paper is meant as a stimulus for theorists of causation in the philosophy of biomedicine, with a focus on clinical matters. Without aiming at putting forward an original theory of causation and starting from the narration of two actual but paradigmatic cases at the joints between biomedical research and clinical practice, we want to point out that some pathological situations addressed by molecular medicine actually prove resistant to some of our major epistemological accounts of causal explanation. Given this scenario, which is very frequent in our hospitals, our analysis aims to provide a stimulus for the debate among theorists of causation in biomedicine interested in real cases in science in practice. We believe that this might in turn encourage some more general rethinking of the complex intertwinement of science, philosophy of science and ethics, as well as of the role of philosophy of science for clinical medicine itself. (shrink)

The essence of temporal universe creation is that any analytical solution has to comply with the boundary condition of our universe; dimensionality and causality constraints. The essence of this book is to show that everything has a price within our temporal (t > 0) universe; energy and time.

The evolutionary emergence of biological processes in organisms with inner, qualitative aspects has not been explained in any sufficient way by neurobiology, nor by the traditional neo-Darwinian paradigm — natural selection would appear to work just as well on insentient zombies (with the right behavioral input-output relations) as on real sentient animals. In consciousness studies one talks about the ‘hard problem’ of qualia. In this paper I sketch a set of principles about sign action, causality and emergent evolution. On the (...) basis of these principles, I characterize a concept of cause that would allow for a naturalistic explanation of the origin of consciousness. The suggested account of causation also turns the ‘hard problem’ of qualia into the easier problem of relating experimental biology to experiential biology. (shrink)

The famous Cartesian Nicolas Malebranche (1638-1715) espoused the occasionalist doctrine that ‘there is only one true cause because there is only one true God; that the nature or power of each thing is nothing but the will of God; that all natural causes are not true causes but only occasional causes’ (LO, 448, original italics). One of Malebranche’s well-known arguments for occasionalism, known as, the ‘no necessary connection’ argument (or, NNC ) stems from the principle that ‘a true cause… is (...) one such that the mind perceives a necessary connection between it and its effect’ (LO, 450). The outline of this paper is as follows. I explicitly layout NNC and articulate some of its prima facie strengths (§1). I then critically discuss, what I take to be, the two main arguments against NNC of the Lee-Pyle interpretation (§2). The main conclusion from (§2) is that Malebranche did not abandon NNC in his later works given textual evidence from the Dialogues, contrary to the Lee-Pyle interpretation. In (§3) I discuss in what ways Suárez, Leibniz, Régis and Spinoza all accepted the main premise of NNC. Then, I rebut Steven Nadler’s influential and unchallenged criticism that Malebranche conflated causal and logical necessity, and provide a more accurate interpretation of Malebranche that only commits him to a partial reduction of causal to logical necessity (§4). (shrink)

The paper discusses the regularity account of causation but finds it insufficient as a complete account of our notion of causality. The attractiveness of the regularity account is its attempt to understand causation in terms of empirically accessible features of the world. However, this account does not match our intuition that singular causality is prior in normal epistemic situations and that there is more to causation than mere succession. Apart from succession and regularity, the concept of causality also contains a (...) modal feature which allows us to engage in counterfactual discourses about singular causal events and to claim that a particular cause is both sufficient and necessary for its effect in the circumstances. However, we may directly observe singular causes, but the modal element is not something we can possibly observe. Rather, this element is something we add to our perception of succession. Thus, the paper suggests that the modal feature of causality is a mental construction which was originally formed by our knowledge of certain structural features of similar events in other situations. It stems not from what we actually observe but from what we have observed or may observe under different but relevant circumstances. So the concept of causation has partly an empirical content and partly a constructed one. (shrink)

Donald J. Zeyl begins the historical section of the book by tracing divine causation throughout classical Greek philosophy. Some of the Pre-Socratics held to a single god as the source of rational order or change. These views suggested that the cosmos may be explained teleologically. Plato takes up that suggested promise in his Phaedo and finds it wanting. Instead, he looks to Forms as (formal) causes of natural processes. This direction of inquiry leads him to postulate, in the Republic, the (...) Form of the Good as the “unhypothetical (causal) first principle.” In Plato’s later writings, most notably in the Timaeus, Plato presents a full-blown teleological account of both the universe at large and the varieties of living things that inhabit the earth. The agent that brings about the conformity of the world to the Forms is the divine Demiurge. -/- Aristotle inherits from Plato the belief that processes in the natural world are to be understood teleologically. These processes are explained by the nature (or form) of the organism itself. Aristotle’s world has no beginning and thus has no place for an original efficient cause to get the world started. Aristotle’s “Unmoved Mover” plays no causal role in the processes of the natural world, all of which are exhaustively explained in terms of Aristotle’s well-known theory of the “four causes.” Nevertheless, the Unmoved Mover is a final cause. -/- After Aristotle, only the Stoics offered an account of divine causal agency. As pantheists, the Stoics identified the world with a divine rational principle they named the Logos. All events, natural and social, are completely determined by Reason, so the Stoics were strict determinists, both in their natural philosophy and in their social and ethical thought. (shrink)

Myles Brand and Marshall Swain advocate the principle that if A is the set of conditions individually necessary and jointly sufficient for the occurrence of B, then if C is a set of conditions individually necessary for the occurrence of B, every member of C is a member of A. I agree with John Barker and Risto Hilpinen who each argue that this principle is not true for causal necessity and sufficiency, but I disagree with their claim that it (...) is true for logical necessity and sufficiency. The original appeal of the principle may be due to confusing two kinds of totality: to say that when every member of set A obtains, then every condition necessary for E obtains is not to say that every condition necessary for E is a member of A. All the authors mentioned believe that causal necessity precludes logical necessity. I deny this on the basis of an example from kinematics. Hume has not refuted definitions of causation in terms of logically necessary and sufficient conditions, nor have Brand and Swain. (shrink)

The adaptation of the Kramers-Kronig dispersion relations to the causal localization structure of QFT led to an important project in particle physics, the only one with a successful closure. The same cannot be said about the subsequent attempts to formulate particle physics as a pure S-matrix project.The feasibility of a pure S-matrix approach are critically analyzed and their serious shortcomings are highlighted. Whereas the conceptual/mathematical demands of renormalized perturbation theory are modest and misunderstandings could easily be corrected, the correct (...) understanding about the origin of the crossing property requires the use of the mathematical theory of modular localization and its relation to the thermal KMS condition. These new concepts, which combine localization, vacuum polarization and thermal properties under the roof of modular theory, will be explained and their potential use in a new constructive (nonperturbative) approach to QFT will be indicated. The S-matrix still plays a predominant role but, different from Heisenberg’s and Mandelstam’s proposals, the new project is not a pure S-matrix approach. The S-matrix plays a new role as a “relative modular invariant”. (shrink)

The online version of the original article can be found under doi:10.1007/s11948-007-9045-2.

All causal Lie products of solutions of the Klein-Gordon equation and the wave equation in Minkowski space are determined. The results shed light on the origin of the algebraic structures underlying quantum field theory.

How are causal judgements such as 'The ice on the road caused the traffic accident' connected with counterfactual judgements such as 'If there had not been any ice on the road, the traffic accident would not have happened'? This volume throws new light on this question by uniting, for the first time, psychological and philosophical approaches to causation and counterfactuals. Traditionally, philosophers have primarily been interested in connections between causal and counterfactual claims on the level of meaning or (...) truth-conditions. More recently, however, they have also increasingly turned their attention to psychological connections between causal and counterfactual understanding or reasoning. At the same time, there has been a surge in interest in empirical work on causal and counterfactual cognition amongst developmental, cognitive, and social psychologists--much of it inspired by work in philosophy. In this volume, twelve original contributions from leading philosophers and psychologists explore in detail what bearing empirical findings might have on philosophical concerns about counterfactuals and causation, and how, in turn, work in philosophy might help clarify the issues at stake in empirical work on the cognitive underpinnings of, and relationships between, causal and counterfactual thought. (shrink)

Ekstein has shown that causal independence neither implies nor is implied by commutativity in an infinite-dimensional, reducible construction. DeFacio and Taylor have presented a finite-dimensional irreducible example of Ekstein's proposition. Avishai and Ekstein have shown that the original question regarding locality for algebraic quantum field theories remainsopen. We concur with that claim and offer additional arguments. A new denumerably infinite-dimensional, irreducible example is presented here which shows that a sort of “orthogonality” among operators is involved. Some observations on localC*-andW*-algebras (...) are given. (shrink)

We suggest a rigorous theory of how objective single-case transition probabilities fit into our world. The theory combines indeterminism and relativity in the “branching space–times” pattern, and relies on the existing theory of causae causantes (originating causes). Its fundamental suggestion is that (at least in simple cases) the probabilities of all transitions can be computed from the basic probabilities attributed individually to their originating causes. The theory explains when and how one can reasonably infer from the probabilities of one “chance (...) set-up” to the probabilities of another such set-up that is located far away. (shrink)