Early modern philosophers rejected various important aspects of Aristotelianism. Current scholarship debates the question to what extent the early moderns rejected final causation. Leibniz explicitly endorsed it. I argue that his notion of final causation should be understood in connection with his resurrection of substantial forms and his seeing such forms on the model of the soul. I relate Leibniz’ conception of final causation to the Aristotelian background as well as Descartes’s treatment of teleology. (...) I argue that he agreed with a view found in Aristotelianism that genuine efficiently causal powers require immanent teleology and with Descartes that immanent teleology presupposes cognition in the agent. (shrink)

John Carriero has argued that for Spinoza there is no final causality in the Aristotelian sense and that the striving of things is merely to be understood in terms of metaphysical inertia. This paper makes a case against this claim. First it is argued that Spinoza’s notion of striving does in principle meet Thomas Aquinas’ criterion for final causation. Second it is shown that Carriero’s denial of final causation in Spinoza leads to a deflationary interpretation (...) of Spinoza’s notions of the good and striving of things, which is at odds with many passages of Spinoza’s Ethics. One can only do justice to these passages if one assumes that Spinoza did accept final causation in the traditional sense. John Carriero argumentierte, dass Spinoza keine finale Kausalität im aristotelischen Sinne vertrete, sondern das Streben von Dingen als Ausdruck einer Art metaphysischen Trägheit verstehe. Dagegen, so wird hier ausgeführt, sprechen zwei Gründe: Erstens genügt Spinozas Charakterisierung des Strebens von Dingen prinzipiell Thomas von Aquins Definition finaler Verursachung. Zweitens führt Carrieros Zurückweisung finaler Verursachung zu einer deflationären Lesart von Spinozas Begriffen des Guten und des Strebens von Dingen, die mit vielen Passagen aus Spinozas Ethik unvereinbar sind. Diesen Passagen kann nur Rechnung getragen werden, wenn man annimmt, dass Spinoza finale Verursachung im traditionellen Sinn akzeptierte. (shrink)

In semiotics, final causation can be related to the process of determination (PAPE, 1993). From Peirce’s point of view, determination is not a causal determinism, but a delimitation of a range of possibilities. One starts from objects towards interpretants, in a process mediated by the sign, in which the dynamic object works as a force that constrains interpretants to correspond to their objects. The correspondence between object and interpretant is important because it is through a generated interpretant that (...) the object of a sign can be known. Even though this process of determination coincides with the idea of final causation, there is a certain indeterminacy in it. For Peirce (EP 2:353, 1905), vagueness and generality are two types of indeterminacy. In the terms of the phenomenological categories, vagueness is an indeterminacy of the order of firstness, generality an indeterminacy of the order of thirdness, and both, to some extent, are opposed to that which is defined, which belongs to secondness. Each aspect of the sign may vary according to the three phenomenological categories. Consequently, degrees of imprecision are added to the semiotic process, which is a determination process. Peirce asserts that the perfect precision of thought is theoretically unattainable (SS 11, 1903). Every sign is vague or general at least to some degree. In this paper, we seek to perceive degrees of indetermination and causality from an analysis of the kinds of objects and interpretants proposed by Peirce in the system of 28 sign classes. (shrink)

For many areas of philosophy, we lack an understanding of their developments between the fourteenth and seventeenth centuries. One such area is the development of the notion of final causation. The rejection of final causation is often described as one of the distinguishing hallmarks of so called Early Modern philosophy in relation to the Scholastic philosophical tradition. Our lack of understanding of the development of this notion in philosophy therefore impedes our ability to write an adequate (...) history of philosophy spanning these centuries. In this article, the notion of final causation as treated in the works of Chrysostom Javellus (1472–1538) and Francis Silvestri (of Ferrara) (1474–1526) is presented. It is argued that the treatment of final causation in these thinkers is already shaped by concerns regarding finality that we find in Early Modern philosophy. (shrink)

ABSTRACT: With a view to suggesting the possible relevance of Aristotelian thought to current notions of complexity and self-organization, studies Aristotlenard cells, and the theories of Schneider, Kay, and D. Sagan.

According to a standard picture of Leibniz’s mature views on creaturely causation, Leibniz held what some interpreters have described as his ‘thesis of spontaneity’: “every non-initial, nonmiraculous state of every created substance has as a real cause some preceding state of that very substance.”2 Evidence for this thesis is abundantly available throughout Leibniz’s mature work and here are some prominent instances.

This paper revives the idea of final cause as a way of considering how the meaning of occurrences--what they have permitted in our lives, as opposed to what produced them or how they fit into a prior plan--is essential to our understanding them.

In “The Fixation of Belief,” Peirce apparently accepts the causal claim that real physical objects cause us to reach an indefeasible “final opinion” concerning them. In “How to Make Our Ideas Clear,” he apparently accepts the constitutive claim that for physical objects to be real just is for them to be represented in that opinion. These claims initially seem inconsistent, since causal claims are explanatory and since equivalent claims cannot explain one another. Contrary to prominent suggestions that Peirce rejected (...) the constitutive claim, however, he actually accepted both, reconciling them via a Humean denial that causal claims are genuinely explanatory. (shrink)

Mental causation is notoriously threatened by the causal exclusion argument. A prominent strategy to save mental causation from causal exclusion consists in subscribing to an interventionist account of causation. This move has, however, recently been challenged by several authors. In this paper, we do two things: We (i) develop what we consider to be the strongest version of the interventionist causal exclusion argument currently on the market and (ii) propose a new way how it can in principle (...) be overcome. In particular, we propose to replace strict supervenience in the assumption that the mental supervenes on the physical by probabilistic supervenience and show how this move has the potential to license the inference to mental causation. Finally, we argue that probabilistic supervenience captures some of the most important intuitions that strict supervenience captures and discuss possible objections to weakening strict supervenience in the way we suggest. (shrink)

In The Biopsychosocial Model of Health and Disease, Derek Bolton and Grant Gillett argue that a defensible updated version of the biopsychosocial model requires a metaphysically adequate account of disease causation that can accommodate biological, psychological, and social factors. This present paper offers a philosophical critique of their account of biopsychosocial causation. I argue that their account relies on claims about the normativity and the semantic content of biological information that are metaphysically contentious. Moreover, I suggest that these (...) claims are unnecessary for a defence of biopsychosocial causation, as the roles of multiple and diverse factors in disease causation can be readily accommodated by a more widely accepted and less metaphysically contentious account of causation. I then raise the more general concern that they are misdiagnosing the problem with the traditional version of the biopsychosocial model. The challenge when developing an explanatorily valuable version of the biopsychosocial model, I argue, is not so much providing an adequate account of biopsychosocial causation, but providing an adequate account of causal selection. Finally, I consider how this problem may be solved to arrive at a more explanatorily valuable and clinically useful version of the biopsychosocial model. (shrink)

The world contains objective causal relations and universals, both of which are intimately connected. If these claims are true, they must have far-reaching consequences, breathing new life into the theory of empirical knowledge and reinforcing epistemological realism. Without causes and universals, Professor Fales argues, realism is defeated, and idealism or scepticism wins. Fales begins with a detailed analysis of David Hume's argument that we have no direct experience of necessary connections between events, concluding that Hume was mistaken on this fundamental (...) point. Then, adopting the view of Armstrong and others that causation is grounded in a second-order relation between universals, he explores a range of topics for which the resulting analysis of causation has systematic implications. In particular, causal identity conditions for physical universals are proposed, which generate a new argument for Platonism. The nature of space and time is discussed, with arguments against backward causation and for the view that space and time can exist independently of matter or causal process. Many of Professor Fales's conclusions seem to run counter to received opinion among contemporary empiricists. Yet his method is classically empiricist in spirit, and a chief motive for these metaphysical explorations is epistemological. The final chapters investigate the perennial question of whether an empiricist, internalist and foundational epistemology can support scientific realism. (shrink)

This dissertation begins by addressing the question of when a causal model is apt for deciding questions of actual causation with respect to some target situation. I first provide relevant background about causal models, explain what makes them promising as a tool for analyzing actual causation, and motivate the need for a theory of aptness as part of such an analysis (Chapter 1). I then define what it is for a model on a given interpretation to be accurate (...) of, that is, say only true things about, some target situation. This involves a systematization of various representational principles mentioned and/or discussed throughout the literature into a method of interpretation, which I propose be taken as standard (Chapter 2). Next, I explain and address two reasons for which accuracy as I’ve defined it is insufficient for aptness. The first reason – already discussed in the literature – is the problem of structural isomorphs. In response, I propose the aptness condition of Explicit Partial Mediation (Chapter 3). The second reason – which has yet to be noticed – is the problem of the indeterminacy of accuracy. As I demonstrate, a model is accurate of a target situation only relative to a set of background possibilities – what I call a modal profile. It follows that a model represents a situation only relative to some modal profile or other. I go on to discuss the ramifications of this observation for a theory of actual causation in terms of models. I argue that the relativity be taken at face value and built into our metaphysical account of causation, resulting in a view that I call causal relativism (Chapter 4). I explore one advantage of this view in detail: that the resulting account can defend the principle of strong proportionality against several objections (Chapter 5). Finally, I apply the earlier discussion of aptness to attempts to provide a semantics of counterfactuals in terms of causal models – an interventionist semantics. I show how just as a similarity semantics relies on an opaque notion of similarity, an interventionist semantics relies on an analogous notion of aptness. The challenge of articulating aptness thus undermines the claim that an interventionist semantics avoids representational problems inherent in a similarity semantics (Chapter 6). I close with a recap and suggestions for future research (Chapter 7). -/- . (shrink)

The paper builds on the basically Humean idea that A is a cause of B iff A and B both occur, A precedes B, and A raises the metaphysical or epistemic status of B given the obtaining circumstances. It argues that in pursuit of a theory of deterministic causation this ‘status raising’ is best explicated not in regularity or counterfactual terms, but in terms of ranking functions. On this basis, it constructs a rigorous theory of deterministic causation that (...) successfully deals with cases of overdetermination and pre-emption. It finally indicates how the account's profound epistemic relativization induced by ranking theory can be undone. Introduction Variables, propositions, time Induction first Causation Redundant causation Objectivization. (shrink)

In this article I examine the relation between causation and moral responsibility. I distinguish four possible views about that relation. One is the standard view: the view that an agent's moral responsibility for an outcome requires, and is grounded in, the agent's causal responsibility for it. I discuss several challenges to the standard view, which motivate the three remaining views. The final view – the view I argue for – is that causation is the vehicle of transmission (...) of moral responsibility. According to this view, although moral responsibility does not require causation, causation still grounds moral responsibility. (shrink)

I defend the thesis that, if there are substances, substance causation (i.e., causation by substances) is the only sort of causation in the universe – or the only fundamental sort. Subsequently, I develop an account of substance causation that is partly grounded on a peculiar interpretation of absolute change (i.e., of entities' coming and ceasing to be) and qualitative change, on some ontological assumptions about modes (i.e., individual properties that ontologically depend on their bearers) and powers. (...) Finally, I reply to some objections against substance causation in general (e.g., C. D. Broad's objection against causes' being non-essentially-dated entities) and to some possible objections against my own account and I briefly explore three advantages given by the acceptance of the latter. (shrink)

<b> </b>Abstract: It is generally accepted that the most serious threat to the possibility of mental causation is posed by the causal self-sufficiency of physical causal processes. I argue, however, that this feature of the world, which I articulate in principle I call Completeness, in fact poses no genuine threat to mental causation. Some find Completeness threatening to mental causation because they confuse it with a stronger principle, which I call Closure. Others do not simply conflate Completeness (...) and Closure, but hold that Completeness, together with certain plausible assumptions, _entails_ Closure. I refute the most fully worked-out version of such an argument. Finally, some find Completeness all by itself threatening to mental causation. I argue that one will only find Completeness threatening if one operates with a philosophically distorted conception of mental causation. I thereby defend what I call naïve realism about mental causation. (shrink)

Reasoning about causation in fact is an essential element of attributing legal responsibility. Therefore, the automation of the attribution of legal responsibility requires a modelling effort aimed at the following: a thorough understanding of the relation between the legal concepts of responsibility and of causation in fact; a thorough understanding of the relation between causation in fact and the common sense concept of causation; and, finally, the specification of an ontology of the concepts that are minimally (...) required for (automatic) common sense reasoning about causation. This article offers a worked-out example of the indicated analysis. Such example consists of: a definition of the legal concept of responsibility (in terms of liability and accountability); a definition of the legal concept of causation in fact (in terms of the initiation of physical processes by an agent and of the provision of reasons and/or opportunities to other agents); CausatiOnt, an AI-like ontology of the common sense (causal) concepts that are minimally needed for reasoning about the legal concept of causation in fact (in particular, the concepts of category, dimension, object, agent, process, event and act). (shrink)

In what follows, I will contend that the commonsense view of ourselves as fundamental causal agents - for which some have used the term “unmoved movers" but which I think might more accurately be expressed as “not wholly moved movers” - is theoretically understandable, internally consistent, and consistent with what we have thus far come to know about the nature and workings of the natural world. In the section that follows, I try to show how the concept of ‘agent’ (...) class='Hi'>causation can be understood as a distinct species (from ‘event’ causation) of the primitive idea, which I’ll term “causal production”, underlying realist or non-Humean conceptions of event causation. In section III, I respond to a number of contemporary objections to the theory of agent causation. Sections IV-V are devoted to showing that the theory is compatible with ordinary reasons explanations of action, which then places me in a position to respond, in the final section, to the contention that we could never know, in principle, whether the agency theory actually describes a significant portion of human activity. (shrink)

I present an alternative account of causation in the biomedical and social sciences according to which the meaning of causal claims is given by their inferential relations to other claims. Specifically, I will argue that causal claims are inferentially related to certain evidential claims as well as claims about explanation, prediction, intervention and responsibility. I explain in some detail what it means for a claim to be inferentially related to another and finally derive some implication of the proposed account (...) for the epistemology, semantics and metaphysics of causation. (shrink)

A phenome occurs through the many pathways of the complex net of interaction between the phenome and its environment; therefore researching and understanding how it arises requires investigation into many possible causes that are in constant interaction with each other. The most comprehensive investigations in biology are the ones in which many biologists from different sub-areas—evolutionary biology, developmental biology, molecular biology, physiology, genetics, epigenetics, ecology—have collaborated. Still, biologists do not always need to collaborate or look for the most comprehensive explanations. (...) A more standard investigation in biology occurs within a single subarea, and uses well-defined experiments with very specific conditions. This paper is about causation and related explanation in plant phenome research and its relevance to Aristotle’s Theory of Four Causes. I argue that there are causes which resemble Aristotle’s formal, material, and efficient causes in phenotype explanation and occurrence; but causes which resemble Aristotle’s final causes occur in phenotype explanation only, not in the occurrence. (shrink)

The current consensus view of causation in physics, as commonly held by scientists and philosophers, has several serious problems. It fails to provide an epistemology for the causal knowledge that it claims physics to possess; it is inapplicable in a prominent area of physics (classical thermodynamics); and it is difficult to reconcile with our everyday use of causal concepts and claims. In this dissertation, I use historical examples and philosophical arguments to show that the interventionist account of causation (...) constitutes a promising alternative for a “physically respectable” account of causation. The interventionist account explicates important parts of the experimental practice of physics and important aspects of the ways in which physical theory is used and applied. Moreover, the interventionist account succeeds where the consensus view of causation in physics fails. I argue that the interventionist account provides an epistemology of causal knowledge in physics that is rooted in experiment. On the interventionist view, there is a close link between experiment and the testing of causal claims. I give several examples of experiments from the early history of thermodynamics that scientists used in interventionist-type arguments. I also argue that interventionist claims made in the context of a physical theory can be epistemically justified by reference to the experimental interventions and observations that serve as evidence for the theory. I then show that the interventionist account of causation is well-suited to the patterns of reasoning that are intrinsic to thermodynamic theory. I argue that interventionist reasoning constitutes the structural foundation of thermodynamic theory, and that thermodynamic theory can provide clear answers to meaningful questions about whether or not a certain variable is a cause of another in a given context. Finally, I argue that the interventionist account offers the prospect of a unification of “physically respectable” causation and our everyday notion of causation. I conclude the dissertation by sketching an anti-foundationalist unification of causation, according to which causal reasoning occurs in the same manner in physics as it does in other branches of life and scientific research. (shrink)

In many biological experiments, due to gene-redundancy or distributed backup mechanisms, there are no visible effects on the functionality of the organism when a gene is knocked out or down. In such cases there is apparently no counterfactual dependence between the gene and the phenotype in question, although intuitively the gene is causally relevant. Due to relativity of causal relations to causal models, we suggest that such cases can be handled by changing the resolution of the causal model that represents (...) the system. By decreasing the resolution of our causal model, counterfactual dependencies can be established at a higher level of abstraction. By increasing the resolution, stepwise causal dependencies of the right kind can serve as a sufficient condition for causal relevance. Finally, we discuss how introducing a temporal dimension in causal models can account for causation in cases of non-modular systems dynamics. (shrink)

Downward causation exercised by emergent properties of wholes upon their lower-level constituents’ properties has been accused of conceptual and metaphysical incoherence. Only upward causation is usually peacefully accepted. The aim of this paper is to criticize and refuse the traditional hierarchical-vertical way of conceiving both types of causation, although preserving their deepest ontological significance, as well as the widespread acceptance of the traditional atomistic-combinatorial view of the entities and the relations that constitute the so-called ‘emergence base’. Assuming (...) those two perspectives with no reserves, we are condemned to confine our debate to the question of whether reified wholes can have the power to downwardly change or influence their lower-level parts, a question which seems profoundly misleading to me. I therefore propose an alternative relational ontological view, assuming a straightforward horizontal and intra-level way of representing those putative cases of cross-level causation. I finally confront two recent replies to Kim’s well-known objections to DC—Craver and Bechtel and Kistler :595–609, 2009)—, emphasizing their global positive approaches, as well as the reasons why their accounts still seem insufficient to me. I conclude arguing that both Kim’s principle of the causal closure of the physical domain and its allegation of an overdetermination in cases of DC can be surpassed by the new relational ontological perspective presented here. (shrink)

Quentin Smith’s idea is that God being the originating cause of the universe is logically inconsistent with all extant definitions of causation, and thus logically impossible. Thus, for example the God of the Philosophers couldn’t have created the Universe, not even in both its senses, in both literal and analogical senses. The thesis is advanced by accounts of the usual views of “cause”. It is maintained these is successful. Such I shall then offer an account of divine causation (...) of my own, and thus attempt to argue that Smith has not shown that the relation that God has to the universe is not a causal relation. Such as a Humean or that of David Lewis sense and of the “singularist” view of C. J. Ducasse would fail the analogical. And Malebranche’s “occasionalism” is surely an exception. If we turn to the other kind then it seems to be a case of “if the data are analogical-in, then the data will be that too”. Finally, it is argued that it is more productive to consider particular individual theistic powers and perfections, for these are mongrels which literality and of analogy are compounded. (shrink)

Downward causation is a widespread and problematic phenomenon. It is typically defined as the causation of lower-level effects by higher-level entities. Downward causation is widespread, as there are many examples of it across different sciences: a cell constraints what happens to its own constituents; a body regulates its own processes; two atoms, when they are appropriately related, make it the case that their own electrons are distributed in certain ways. However, downward causation is also problematic. Roughly, (...) it seems to be at odds with specific scientific and/or epistemological desiderata: first and foremost, that everything can be reduced (one day or another) to the fundamental, micro-physical constituents and goings-on of the universe, so as to provide a unified explanation of everything and a unification of all the sciences " from the bottom ". Indeed, downward causation (if it is an irreducible phenomenon) introduces special causings not only at the higher levels, but also at the lower ones: if, in principle, we cannot fully understand what happens to the electrons without paying attention to the atoms (at the higher level) and we cannot fully understand what happens to the atoms by only paying attention to the electrons (at the lower level), there is no fully lower-level explanation for both higher-level and lower-level goings-on. In this introduction, we shall try to describe the prospects for downward causation in metaphysics and the philosophy of science. After having delved into the connections between downward causation, emergence and levels (§1), we shall discuss the irreducibility of downward causation (§2). We shall then briefly consider how specific metaphysical and epistemological assumptions bear on our understanding of downward causation and of its possibility (§3) and describe some views according to which downward causation is actually non-causal (or it is a special causal relation) (§4). We shall also mention some problems for the connection between downward causation and mental causation (§5) and some scientific examples of downward causation (§6). Finally, we shall summarize the contents of the contributions in this book (§7). (shrink)

Agent causation presupposes that actions are behaviors under the causal control of the agent’s mental states, its beliefs and desires. Here the idea of conscious causation in causal explanations of actions is examined, specifically, actions said to be the result of conscious efforts. Causal–functionalist theories of consciousness purport to be naturalistic accounts of the causal efficacy of consciousness. Flanagan argues that his causal–functionalist theory of consciousness satisfies naturalistic constraints on causation and that his causal efficacy thesis is (...) compatible with results of Libet’s experiments on conscious causation. First, the notions of conscious effort and conscious causation are analyzed with respect to the project of naturalizing the mind, that is, the attempt to assimilate folk-psychological explanation to the causal model of explanation in the natural sciences. It is argued that a serious obstacle for any naturalist program is that mental states are individuated by their semantic content, not the mechanistic, physical properties of their neural state instantiations. In particular, it is argued that explanation by reference to mental state content yields not a causal but an interpretive or rationalizing account of action in which the question of causal efficacy is irrelevant. Then, Flanagan’s causal–functionalist theory of consciousness is critically assessed; specifically his interpretations of Libet’s negative experimental results on the causal efficacy of consciousness are diagnosed and disputed. It is contended that Flanagan misinterprets the results of Libet’s consciousness experiments and that his functionalist concept of consciousness fails to yield an adequate explanation of the alleged causal efficacy of consciousness. Finally, his thesis is countered with other experimental results that appear to favor an epiphenomenalist view over the causal efficacy account of consciousness. (shrink)

In recent years, a number of philosophers have attempted to fix paradoxes of the counterfactual account of causation by making causation contrastive. In this framework, causation is understood to be not a two-place relationship between a cause and an effect but a three or four-place relationship between a cause, an effect and a contrast on the side of the cause, the effect or both. I argue that contrasting helps resolving certain paradoxes only if an account of admissibility (...) of the chosen set of contrasts is given. I show by means of numerous examples that it is contextual features that determine admissibility. This way, context becomes part of the semantics of causation. I finally argue that once contextualised, explicit contrasting is redundant: causation is therefore a three-place relationship between a cause, an effect and a context. (shrink)

Counterfactual theories of causation have had difficulty in delivering the intuitively correct verdicts for cases of causation involving preemption, without generating further counterexamples. Hall has offered a pluralistic theory of causation, according to which there are two concepts of causation: counterfactual dependence and production. Hall’s theory does deliver the correct verdicts for many of the problematic kinds of preemption. It also deals successfully with cases of causation by omission, which have proved stubborn counterexamples to physical (...) process theories of causation. Hall’s theory therefore appears to be a significant improvement on extant univocal theories of causation, both physical and counterfactual. In this paper I present a series of counterexamples to Hall’s theory. I also describe cases in which our causal judgments appear to be sensitive to moral considerations. It does not seem likely that conventional theories of causation, which attempt to situate causation in an objective metaphysical picture of the world, will ever accord our intuitions in such cases. Finally, the notion of responsibility is considered, but rejected as an illuminating primitive for analyzing causation. (shrink)

I argue (1) that it is not philosophically significant whether causation is linguistically represented by a predicate or by a sentence connective; (2) that there is no philosophically significant distinction between event- and states-of-affairs-causation; (3) that there is indeed a philosophically significant distinction between agent- and event-causation, and that event-causation must be regarded as an analog of agent-causation. Developing this point, I argue that event-causation's being in the image of agent-causation requires, mainly, (a) (...) that the cause is temporally prior to the effect, (b) that the cause necessitates (is sufficient with necessity) for the effect. Causal necessity is explained as a derivative of nomological necessity, and finally, via a definition of the causal sentence connective, the logic of event-causation is shown to be a part of temporal modal logic. (shrink)

Aristotle thinks that we understand something when we know its causes. According to Aristotle but contrary to most recent approaches, causation and explanation cannot be understood separately. Aristotle complicates matters by claiming that there are four causes, which have come to be known as the formal, material, final, and efficient causes. To understand Aristotelian causation and its relationship to explanation, then, we must come to a precise understanding of the four causes, and how they are supposed to (...) be explanatory. Aristotle’s discussion of the causes, however, is compact, and he typically presents them without arguing for them. He thus leaves us with a number of questions, ranging from the highly specific to the highly general. One question in particular has captured the attention of scholars and philosophers over the last century, and it has had a strong influence on recent treatments of the four causes – namely, whether we are right to understand Aristotle as committed to a plurality of kinds of causation, or rather a plurality of kinds of explanation. Sometimes the question is raised as one of whether it would be more accurate to speak of the four ‘becauses’ rather than the four ‘causes’. This worry is highly general, and there are in fact several ways in which it might be formulated; nonetheless, it is important to clarify the precise nature of the problem, and the possible ways of responding to it. At issue is not just whether Aristotle’s notions are sufficiently like the modern notion of causation to be relevant to our concerns, but, more importantly, whether the distinctions he draws are ultimately metaphysical or epistemological in character. (shrink)

The 'completeness of physics' is the key premise in the causal argument for physicalism. Standard formulations of it fail to rule out emergent downwards causation. I argue that it must do this if it is tare in a valid causal argument for physicalism. Drawing on the notion of conferring causal power, I formulate a suitable principle, 'strong completeness'. I investigate the metaphysical implications of distinguishing this principle from emergent downwards causation, and I argue that categoricalist accounts of properties (...) are better equipped to sustain the distinction than dispositional essentialist accounts. Finally, I argue that the additional evidence needed for strong completeness renders the causal argument otiose for any properties amenable to scientific reduction. (shrink)

In this paper I suggest a new interpretation of the relations of inherence, causation and conception in Spinoza. I discuss the views of Don Garrett on this issue and argue against Della Rocca's recent suggestion that a strict endorsement of the PSR leads necessarily to the identification of the relations of inherence, causation and conception. I argue that Spinoza never endorsed this identity, and that Della Rocca's suggestion could not be considered as a legitimate reconstruction or friendly amendment (...) to Spinoza 's system because it creates several severe and irresolvable problems in the system. -/- In the first part of the paper, I present the considerations and arguments that motivated Don Garrett's and Della Rocca's interpretations. In the second part, I present and examine several problems that result from Della Rocca's reading. In the third and final part, I present my own view on the relation among inherence, causation, and conception; offer a new interpretation of the conceived through relation in Spinoza ; and finally, defend and justify the presence of bifurcations at the very center of Spinoza 's system. (shrink)

The thesis of this paper is that causation, when described and treated as a metaphor, increases in explanatory power, while diminishing the problems associated with standard analysis of it. I first present a description of the uses of metaphor in scientific and literary language. This is drawn primarily from Max Black's interaction view of metaphor, as well as the view forwarded by Donald Davidson in his What Metaphors Mean. I then outline some of the standard analyses in the field (...) of causation, followed by some of the standard replies to those analyses. Finally, I show how describing causation in terms of a metaphor will bypass many of these objections, while maintaining or increasing its explanatory power. (shrink)

The counterfactual and regularity theories are universal accounts of causation. I argue that these should be generalized to produce local accounts of causation. A hallmark of universal accounts of causation is the assumption that apparent variation in causation between locations must be explained by differences in background causal conditions, by features of the causal-nexus or causing-complex. The local account of causation presented here rejects this assumption, allowing for genuine variation in causation to be explained (...) by differences in location. I argue that local accounts of causation are plausible, and have pragmatic, empirical and theoretical advantages over universal accounts. I then report on the use of presheaves as models of local causation. The use of presheaves as models of local variation has precedents in algebraic geometry, category theory and physics; they are here used as models of local causal variation. The paper presents this idea as stemming from an approach using presheaves as models of local truth. Finally, I argue that a proper balance between universal and local causation can be assuaged by moving from presheaves to fully-fledged sheaf models. (shrink)

Between 1653 and 1655 Margaret Cavendish makes a radical transition in her theory of matter, rejecting her earlier atomism in favour of an infinitely-extended and infinitely-divisible material plenum, with matter being ubiquitously self-moving, sensing, and rational. It is unclear, however, if Cavendish can actually dispense of atomism. One of her arguments against atomism, for example, depends upon the created world being harmonious and orderly, a premise Cavendish herself repeatedly undermines by noting nature’s many disorders. I argue that her supposed difficulties (...) with atomism expose a deeper tension in her work between two fundamental metaphysical commitments each of which has substantial philosophical support: her monist theory of the material world (which maintains that there exists just one natural substance which is the single principal cause) and her occasional theory of causation (which requires multiple finite principal causes in nature -- causes that might be considered individual substances). Her monism undermines atomism while her theory of occasional cause seems to rest on a conception of nature that would be especially friendly to atomism. I argue further that we can solve this tension within a Cavendishian framework in such a way as to preserve her theory of causation and her monism, but that this solution depends upon our taking her monism in a particular (and weak) form. I finally note that we can best make sense of her unique and interesting form of monism by acknowledging her social-political motivations in addition to her motivations in natural philosophy. (shrink)

This paper is about what I call the Question of Iterated Causation (QIC): for any instance of causation in which c1…ck cause effect e, what are the causes of c1…ck’s causing of e? In short: what causes instances of causation or, as I will refer to these instances, the “causal goings‐on”? A natural response (which I call “dismissivism”) is that this is a bad question because causal goings‐on aren’t apt to be caused. After rebutting several versions of (...) dismissivism, I consider the view that QIC, though not illegitimate, is easy to answer: the causal goings‐on are apt to be caused but are plainly uncaused (“brutism”). However, I will argue that brutism too has a serious problem: namely, it leads to a highly implausible kind of armchair indeterminism. Next I consider some substantive candidate answers to QIC, none of which, I argue, is particularly promising. The paper’s final conclusion is twofold: QIC is at least as difficult as the more well‐known Question of Iterated Grounding; moreover, the largely overlooked regress problem that it raises gives us at least some defeasible reason to avoid causation in theory‐building. (shrink)

In Part I of ‘Causality and Determination” (CD), Anscombe writes that (1) we understand causality through understanding specific causal expressions, (2) efficient causation can be perceived, (3) “causality consists in the derivativeness of an effect from its causes”, and 4) no “analysis in terms of necessity or universality” has a place for this. Theses (1) and (2) represent fundamental and important insights. (3) is unsatisfactory; for, taken in a sense that does not already build on the general notion of (...) causation, deriving from does little to elucidate this notion. CD is however right to urge the need to identify a “core, the common feature, of causality in its various kinds”—a kind of criterion, such as the one suggested by Makin (2000), that takes account of (4) in that it is more substantial than, but does not entail, “necessity or universality”. What CD seems to underestimate is the important role of regularities, possibly neither necessary nor exceptionless, for our understanding of the causal relation. Finally, such understanding also requires us, not only to rely on a common criterion of causation, but also on a subjective component of this idea: the consciousness of our own causal agency. Anscombe’s own investigations into non-observational practical knowledge open the door to the study of this aspect of causality. (shrink)

Leibniz holds that created substances do not causally interact with each other but that there is causal activity within each such creature. Every created substance constantly changes internally, and each of these changes is caused by the substance itself or by its prior states. Leibniz describes this kind of intra-substance causation both in terms of final causation and in terms of efficient causation. How exactly this works, however, is highly controversial. I will identify what I take (...) to be the major interpretive issues surrounding Leibniz's views on causation and examine several influential interpretations of these views. In ‘Leibniz on Causation – Part 2’ I will then take a closer look at final causation. (shrink)

Holtby, Allen and Fairchild are both recent and revolutionary decisions that address an important aspect of the indeterminate causation problem that frequently arises in tort litigation. In Holtby and Allen, the Court of Appeal departed from the traditional binary approach, under which a tort claimant either recovers compensation for his or her entire injury or is altogether denied recovery—depending on whether his or her case against the defendant is more probable than not. Holtby and Allen substituted this approach by (...) the proportionate recovery principle, under which the defendant compensates the claimant for a fraction of his or her injury that represents the defendant's statistical share in that injury. This article analyses this development within the particular domain of indeterminate causation, over which the proportionate recovery principle has been licensed to exercise control. The article claims that this development would constitute an improvement of the law from the perspectives of both optimal deterrence and corrective justice, if the courts properly formulate its scope. First, the proportionate recovery principle needs to be explicitly confined to cases that deal with recurrent wrongs. Second, determination of the defendant's share in the claimant's injury ought to be grounded on the probability of causation, rather than the risk of inflicting that injury. Third, judges must not apply unarticulated intuitions in determining the magnitude of the relevant risk or probability: their decisions in that area would be better informed by the statistical principle of ‘insufficient reason’, also known as the ‘indifference principle’. Fourth, courts are yet to relieve the doctrinal tension between the proportionate recovery principle, as recognized in Holtby and Allen, and the previous rejection of that principle by the House of Lords. Subsequently, the article analyses the House of Lords' decision in Fairchild—yet another instance of indeterminate causation in which proportionate recovery is preferable to the ‘all or nothing’ approach. In this case, the House of Lords allowed the claimants full recovery. This holding was grounded in the Law Lords' innovative approach to indeterminate causation, and the article unfolds the problematics of this approach. Finally, the article offers an adoption of yet another legal mechanism—the ‘evidential damage doctrine’—that replaces liability under uncertainty with liability for uncertainty. (shrink)

Clarifying the causes of subway construction accidents has an important impact on reducing the probability of accidents and protecting workers’ lives and public property to a greater extent. A total of 138 investigation records of subway construction accidents from 2000 to 2020 were collected in this study. Based on a systemic analysis of 29 well-known accident causation models and the formative process of the subway construction accidents, we extracted the causative factors of subway construction accidents from the collected records. (...) Furthermore, a causation analysis index system of subway accidents was proposed based on fault tree analysis, where we considered subway construction accidents as the top event and the five dimensions, i.e., human, equipment, environment, management, and safety culture, as first-level intermediate events. Moreover, 17 causative factors were considered to be related to the severity of subway construction accidents. It is found that human factors are prone to be critical to high-risk accidents. Finally, a Bayesian network was formed to explore the causative factors of high-risk subway construction accidents. Based on the combined application of FTA and BN, this study discusses the complex influence factors and their action routes to unsafe accidents in subway construction sites, and makes efforts to correspond safety decision basis for the management of China subway construction. (shrink)

The aim of this dissertation is to provide an analysis of central concepts in philosophy of science from the perspective of current molecular and developmental research. Each chapter explores the ways in which particular phenomena or discoveries in molecular biology influences our philosophical understanding of the nature of scientific knowledge. The introductory prologue draws some general connections between the various threads, which revolve around two central themes: causation and explanation. Chapter Two identifies a particular type of causal relation which (...) is widespread across the sciences, but cannot be straightforwardly accommodated by extant accounts of causation and causal explanation. Chapter Three explores how the form of redundant causality identified in the previous chapter plays an important role in causal explanation, by making the effect stable and robust. Chapter Four offers a novel perspective on the debate over biological reductionism by distinguishing between different paradigms of molecular explanation. Chapter Five provides a philosophical analysis of the so-called "Developmental Synthesis" of evolutionary and developmental biology, and suggests a general account of scientific unification grounded in the notion of explanatory relevance. Chapter Six offers an account of dispositional properties inspired by mechanisms of gene regulation, according to which dispositions are not properties of entities, but properties that describe the behavior of abstract idealized models. Finally, Chapter Seven scrutinizes the concept of the molecular ecosystem, a metaphor frequently employed by biologists to describe cellular interactions, but seldom articulated in detail. (shrink)