The frame problem is the difficulty of explaining how non-magical systems think and act in ways that are adaptively sensitive to context-dependent relevance. Influenced centrally by Heideggerian phenomenology, Hubert Dreyfus has argued that the frame problem is, in part, a consequence of the assumption (made by mainstream cognitive science and artificial intelligence) that intelligent behaviour is representation-guided behaviour. Dreyfus' Heideggerian analysis suggests that the frame problem dissolves if we reject representationalism about intelligence and recognize that human agents realize the property (...) of thrownness (the property of being always already embedded in a context). I argue that this positive proposal is incomplete until we understand exactly how the properties in question may be instantiated in machines like us. So, working within a broadly Heideggerian conceptual framework, I pursue the character of a representation-shunning thrown machine. As part of this analysis, I suggest that the frame problem is, in truth, a two-headed beast. The intra-context frame problem challenges us to say how a purely mechanistic system may achieve appropriate, flexible and fluid action within a context. The inter-context frame problem challenges us to say how a purely mechanistic system may achieve appropriate, flexible and fluid action in worlds in which adaptation to new contexts is open-ended and in which the number of potential contexts is indeterminate. Drawing on the field of situated robotics, I suggest that the intra-context frame problem may be neutralized by systems of special-purpose adaptive couplings, while the inter-context frame problem may be neutralized by systems that exhibit the phenomenon of continuous reciprocal causation. I also defend the view that while continuous reciprocal causation is in conflict with representational explanation, special-purpose adaptive coupling, as well as its associated agential phenomenology, may feature representations. My proposal has been criticized recently by Dreyfus, who accuses me of propagating a cognitivist misreading of Heidegger, one that, because it maintains a role for representation, leads me seriously astray in my handling of the frame problem. I close by responding to Dreyfus' concerns. (shrink)

This paper examines the application of the mutual manipulability criterion as a way to demarcate constituents of cognitive systems from resources having a mere causal influence on cognitive systems. In particular, it is argued that on at least one interpretation of the mutual manipulability criterion, the criterion is inadequate because the criterion is conceptualized as identifying synchronic dependence between higher and lower ‘levels’ in mechanisms. It is argued that there is a second articulation of the mutual manipulability criterion available, and (...) that it should be preferred for at least two reasons. The first is that the criterion of mutual manipulability is an instance of continuous reciprocal causation. The second is that it has implications for how to understand this distinction between causation and constitution. It is shown that when considering dynamic systems, continuous reciprocal causation - ubiquitous in dynamical systems - is a form of constitutive causality, which entails that causal factors may, in the right circumstances, by genuine constitutive factors of cognition. This notion of constitutive causality lends support to conceiving of the mutual manipulability criterion as a genuine demarcation principle in the debate over the boundaries of mind. (shrink)

Laland and colleagues have sought to challenge the proximate–ultimate distinction claiming that it imposes a unidirectional model of causation, is limited in its capacity to account for complex biological phenomena, and hinders progress in biology. In this article the core of their argument is critically analyzed. It is claimed that contrary to their claims Laland et al. rely upon the proximate–ultimate distinction to make their points and that their alternative conception of reciprocal causation refers to phenomena that (...) were already accounted for by standard theory. (shrink)

Kevin Laland and colleagues have put forward a number of arguments motivating an extended evolutionary synthesis. Here I examine Laland et al.'s central concept of reciprocal causation. Reciprocal causation features in many arguments supporting an expanded evolutionary framework, yet few of these arguments are clearly delineated. Here I clarify the concept and make explicit three arguments in which it features. I identify where skeptics can—and are—pushing back against these arguments, and highlight what I see as the (...) empirical, explanatory, and methodological issues at stake. (shrink)

In recent years, biologists and philosophers of science have argued that evolutionary theory should incorporate more seriously the idea of ‘reciprocal causation.’ This notion refers to feedback loops whereby organisms change their experiences of the environment or alter the physical properties of their surroundings. In these loops, in particular niche constructing activities are central, since they may alter selection pressures acting on organisms, and thus affect their evolutionary trajectories. This paper discusses long-standing problems that emerge when studying such (...) reciprocal causal processes between organisms and environments. By comparing past approaches to reciprocal causation from the early twentieth century with contemporary ones in niche construction theory, we identify two central reoccurring problems: All of these approaches have not been able to provide a conceptual framework that allows maintaining meaningful boundaries between organisms and environments, instead of merging the two, and integrating experiential and physical kinds of reciprocal causation. By building on case studies of niche construction research, we provide a model that is able to solve these two problems. It allows distinguishing between mutually interacting organisms and environments in complex scenarios, as well as integrating various forms of experiential and physical niche construction. (shrink)

Arguments for an extended evolutionary synthesis often center on the concept of “reciprocal causation.” Proponents argue that reciprocal causation is superior to standard models of evolutionary causation for at least two reasons. First, it leads to better scientific models with more predictive power. Second, it more accurately represents the causal structure of the biological world. Simply put, proponents of an extended evolutionary synthesis argue that reciprocal causation is empirically and explanatorily apt relative to (...) competing causal frameworks. In this paper, I present quantitative survey data from faculty members in biology departments at universities across the United States to evaluate this claim. The survey data indicate that a majority of the participants do not agree (i.e., most either disagree or neither agree nor disagree) that the concept of reciprocal causation confers a larger advantage on research practices. However, a majority of the participants agree that the causal framework of the extended evolutionary synthesis more accurately represents the structure of the biological world. These results demonstrate that the explanatory merits of a conceptual framework and its practical utility can come apart in interesting and informative ways. (shrink)

Philosophers of dispositionalism deny the Humean account of causality in terms of constant conjunction, contiguity, temporal priority and contingency. And some of them go further to explain the causal relation not between events or objects, but between properties, in terms of reciprocity, simultaneity, ubiquity, intentionality and holism. But their exposition seems to remain fragmented even though they try to make use of the notions of intentionality and holim. I would inquire reasons why it is piecemeal, by analysing that they employ (...) these notions of intentionality and holism which has been constructed within the dualistic tradition. If one wants to make the dispositional project to be powerful, she should look for the notions of intentionality and holism which are free of the dualistic traits and which have been constructed in the tradition of naturalism. The concept of integrationality may be a candidate for such a task. This conception was coined in Zhongyong under the name of cheng in that there is nothing without cheng, and was developed in one of Sok-hon Ham's thesis that any entity in the world contains "seed" within it to realize objectives shared by all involved. Humans are privileged and distinct from other natures, but there is continuity between them. An example of the integrationality applied to the case of humans would be that realization of myself and realizations of all others are one and not two. I would summarize notions of "cheng" and "seed" by offering the integrationality thesis, that is, that there is integrationality in any entity like dispositions such that the integrationality is a power to realize the embedded objective of it in the context where it interacts with all others. I would claim that integrationality is a metaphysical basis for a way of human thinking, by arguing that it is a necessary part of presupposition for our understanding of the world without which the world is not intelligible. The notion of integrationality is involved empirically as well as conceptually. Empirically, there are numerous reports from physics, biology and information science, which provide with a clue which may well be taken to show the ubiquitous integrationality. But some conceptual considerations would also indicate necessarily toward the same direction. These would include various theses of ontology, epistemology, disposition, modality, naturalized intentionality, fitting and topics like these. I will investigate some of them in order to justify my claim of this paper. (shrink)

Among advocates and critics of the “extended evolutionary synthesis” (EES), “reciprocal causation” refers to the view that adaptive evolution is a bidirectional phenomenon, whereby organisms and environments impinge on each other through processes of niche construction and natural selection. I argue that reciprocal causation is incompatible with the view that natural selection is a metaphysically emergent causal process. The emergent character of selection places reciprocal causation on the horns of dilemma, and neither horn can (...) rescue it. I conclude that proponents of the EES must abandon the claim that the process of natural selection features in cycles of reciprocal causation. (shrink)

What are the possible varieties of cognition-artifact relations, and which dimensions are relevant for exploring these varieties? This question is answered in two steps. First, three levels of functional and informational integration between human agent and cognitive artifact are distinguished. These levels are based on the degree of interactivity and direction of information flow, and range from monocausal and bicausal relations to continuous reciprocal causation. In these levels there is a hierarchy of integrative processes in which there (...) is an increasing degree of hybridization. Therefore, from a functional and informational level of abstraction, it becomes increasingly difficult to distinguish between agent and artifact in this hierarchy. Second, a multidimensional matrix for exploring cognition-artifact relations is sketched. The dimensions in the matrix include reliability, durability, trust, procedural and representational transparency, individualization, bandwidth, speed of information flow, distribution of computation, and cognitive and artifactual transformation. Together, these dimensions constitute a multidimensional space in which particular cognition-artifact relations can be located. The higher a particular cognition-artifact relation scores on these dimensions, the more integration and hybridization occurs, and the more tightly coupled the overall system is. It is then better, for explanatory reasons, to see agent and artifact as one cognitive system with a distributed informational architecture. (shrink)

Recent work in cognitive and computational neuroscience depicts the human cortex as a multi-level prediction engine. This ‘predictive processing’ framework shows great promise as a means of both understanding and integrating the core information processing strategies underlying perception, reasoning, and action. But how, if at all, do emotions and sub-cortical contributions fit into this emerging picture? The fit, we shall argue, is both profound and potentially transformative. In the picture we develop, online cognitive function cannot be assigned to either the (...) cortical or the sub-cortical component, but instead emerges from their tight co-ordination. This tight co-ordination involves processes of continuous reciprocal causation that weave together bodily information and ‘top-down’ predictions, generating a unified sense of what’s out there and why it matters. The upshot is a more truly ‘embodied’ vision of the predictive brain in action. (shrink)

I examine the following question: Do actions require representations that are intrinsic to the action itself? Recent work by Mark Rowlands, Michael Wheeler, and Andy Clark suggests that actions may require a minimal form of representation. I argue that the various concepts of minimal representation on offer do not apply to action per se and that a non‐representationalist account that focuses on dynamic systems of self‐organizing continuous reciprocal causation at the sub‐personal level is superior. I further recommend (...) a scientific pragmatism regarding the concept of representation. (shrink)

Advancing the reductionist conviction that biology must be in agreement with the assumptions of reductive physicalism (the upward hierarchy of causal powers, the upward fixing of facts concerning biological levels) A. Rosenberg argues that downward causation is ontologically incoherent and that it comes into play only when we are ignorant of the details of biological phenomena. Moreover, in his view, a careful look at relevant details of biological explanations will reveal the basic molecular level that characterizes biological systems, defined (...) by wholly physical properties, e.g., geometrical structures of molecular aggregates (cells). In response, we argue that contrary to his expectations one cannot infer reductionist assumptions even from detailed biological explanations that invoke the molecular level, as interlevel causal reciprocity is essential to these explanations. Recent very detailed explanations that concern the structure and function of chromatin—the intricacies of supposedly basic molecular level—demonstrate this. They show that what seem to be basic physical parameters extend into a more general biological context, thus rendering elusive the concepts of the basic level and causal hierarchy postulated by the reductionists. In fact, relevant phenomena are defined across levels by entangled, extended parameters. Nor can the biological context be explained away by basic physical parameters defining molecular level shaped by evolution as a physical process. Reductionists claim otherwise only because they overlook the evolutionary significance of initial conditions best defined in terms of extended biological parameters. Perhaps the reductionist assumptions (as well as assumptions that postulate any particular levels as causally fundamental) cannot be inferred from biological explanations because biology aims at manipulating organisms rather than producing explanations that meet the coherence requirements of general ontological models. Or possibly the assumptions of an ontology not based on the concept of causal powers stratified across levels can be inferred from biological explanations. The incoherence of downward causation is inevitable, given reductionist assumptions, but an ontological alternative might avoid this. We outline desiderata for the treatment of levels and properties that realize interlevel causation in such an ontology. (shrink)

It is often argued that constitution and causation are different kinds of metaphysical relations. Constitution, like other grounding relations, is assumed to be synchronic, while causation is diachronic. It is this synchronic-diachronic division that, more than other difference-makers, is argued to distinguish grounding relations such as constitution from causation. This paper develops an account of a species of constitution that happens over time. We call this type of constitution, diachronic constitution. We show how diachronic constitution is a (...) consequence of a common type of causation, often ignored in metaphysical discussions of causation: continuous reciprocal causation. Hence, constitution is not only sometimes like causation; it can also be a form of causation. We argue that temporalising the constitution relation is neither as remarkable nor as problematic as it might initially seem. It is inevitable, given local interactions between microscale and macroscale states in dynamical systems. (shrink)

A case study in historical cognitive science, this paper addresses two claims made by radical proponents of new dynamical approaches. It queries their historical narrative, which sees embodied, situated cognition as correcting an individualist, atemporal framework originating in Descartes. In fact, new Descartes scholarship shows that 17th-century animal spirits neurophysiology realized a recognizably distributed model of memory; explicit representations are patterns of spirit flow, and memory traces are changes left by experience in connections between brain pores. This historical sketch supports (...) the second dynamicist claim, that connectionists' stress on the cognitive importance of pattern-recreation needs supplementing by dynamicists' real-time focus and attention to the active roles of body and environment. Animal spirits theory exhibits just the 'continuous reciprocal causation' between brain, body, and environment which Andy Clark sees as dynamicism's central contribution, and allows for the embedding of brains in culture as well as the physical world. (shrink)

In this article, I set forth my understanding of reciprocal causality between God and finite entities in three stages, beginning with Aristotle’s analysis of change in this world. Afterwards, I examine the way in which Aquinas used the causal scheme of Aristotle in his Christian understanding of the God-world relationship. Finally, I indicate how both Aristotle’s philosophy and Aquinas’s approach to the God-world relationship should be rethought so as to be more in line with contemporary scientific understanding of the (...) evolution of life from non-life and the emergence of progressively higher-order life-systems out of lower-order life-systems within creation. (shrink)

Neurophenomenology (Varela 1996) is not only philosophical but also empirical and experimental. Our purpose in this article is to illustrate concretely the efficiency of this approach in the field of neuroscience and, more precisely here, in epileptology. A number of recent observations have indicated that epileptic seizures do not arise suddenly simply as the effect of random fluctuations of brain activity, but require a process of pre-seizure changes that start long before. This has been reported at two different levels of (...) description: on the one hand, the epileptic patient often experiences some warning symptoms that precede seizures from several minutes to hours in the form of very specific lived events. On the other hand, the analyses of brain electrical activities have provided strong evidence that it is possible to detect a pre-seizure state in the neuronal dynamics several minutes before the electro-clinical onset of a seizure. We review here some of the ongoing work of our research group concerning seizure anticipation. In particular, we discuss experimental evidence of upward (local-to-global) formation of conscious experience and its neural substrate, but also of the downward (global-to-local) determination of local neuronal activity by situated conscious activity and its substrate large-scale neural assemblies. This causal role of conscious experience may lead to new kinds of therapy for epileptic patients. (shrink)

We propose that all actual causes are simultaneous with their direct effects, as illustrated by both everyday examples and the laws of physics. We contrast this view with the sequential conception of causation, according to which causes must occur prior to their effects. The key difference between the two views of causation lies in differing assumptions about the mathematical structure of time.

Analyses of singular causation often make use of the idea that a cause increases the probability of its effect. Of particular salience in such accounts are the values of the probability function of the effect, conditional on the presence and absence of the putative cause, analysed around the times of the events in question: causes are characterized by the effect’s probability function being greater when conditionalized upon them. Put this way, it becomes clearer that the ‘behaviour’ of probability functions (...) in small intervals about the times in question ought to be of concern. In this article, I make an extended case that causal theorists employing the ‘probability raising’ idea should pay attention to the continuity question. Specifically, if the probability functions are ‘jumping about’ in ways typical of discontinuous functions, then the stability of the relevant probability increase is called into question. The rub, however, is that sweeping requirements for either continuity or discontinuity are problematic and, as I argue, this constitutes a ‘continuity bind’. Hence more subtle considerations and constraints are needed, two of which I consider: utilizing discontinuous first derivatives of continuous probability functions, and abandoning point probability for imprecise probability. _1_ Introduction _2_ Probability Trajectories and Continuity _2.1_ Probability trajectories _2.2_ Causation as discontinuous jumps _2.3_ Against systematic discontinuity _3_ Broader Discontinuity Concerns _4_ The Continuity Bind _4.1_ Retaining continuity with discontinuous first derivatives _4.2_ Imprecise probability trajectories _5_ Concluding Remarks Appendix. (shrink)

One standard criticism of the doctrine of continuous creation is that it entails the occasionalist position that God alone is a true cause and that the events we commonly identify as causes are merely the occasions upon which God brings about effects. I begin by clearly stating Malebranche's argument from continuous creation to occasionalism. Next, I examine two strategies for resisting Malebranche's argument – strong and weak concurrentism – and argue that weak concurrentism is the more promising strategy. (...) Finally, I argue that weak concurrentism requires a necessitarian approach to secondary causation. (shrink)

: Descartes provides an original and puzzling argument for the traditional theological doctrine that the world is continuously created by God. His key premise is that the parts of the duration of anything are "completely independent" of one another. I argue that Descartes derives this temporal independence thesis simply from the principle that causes are necessarily simultaneous with their effects. I argue further that it follows from Descartes's version of the continuous creation doctrine that God is the instantaneous and (...) total cause of everything that happens, and that this is just what his physics demands. But although God is the total cause of everything, he is not the only cause, since Descartes considers it obvious that finite minds have the power to move bodies. In the face of this apparent paradox, several recent commentators have suggested that Descartes accepted the late scholastic view that God and finite causes somehow collaborate or concur in the production of numerically identical effects. But close examination reveals that the case for Cartesian concurrentism is weak. Fortunately, there is a simpler and more fruitful solution to the problem of reconciling divine and human action. I argue that that in Descartes's world certain motions, such as voluntary movements of our bodies, are causally overdetermined This account allows Descartes to avoid occasionalism without embracing an elaborate metaphysics of secondary causality. Finally, I argue that the overdeterminist interpretation sheds new light on two longstanding problems of Cartesian metaphysics. First, it resolves a serious difficulty with a standard reading of Descartes's conception of human freedom. Second, it offers a novel approach to the old problem of reconciling genuine human action with the principle of the conservation of total quantity of motion. (shrink)

One standard criticism of the doctrine of continuous creation is that it entails the occasionalist position that God alone is a true cause and that the events we commonly identify as causes are merely the occasions upon which God brings about effects. I begin by clearly stating Malebranche's argument from continuous creation to occasionalism. Next, I examine two strategies for resisting Malebranche's argument ??? strong and weak concurrentism ??? and argue that weak concurrentism is the more promising strategy. (...) Finally, I argue that weak concurrentism requires a necessitarian approach to secondary causation. (shrink)

This chapter discusses Mina Bissell's pathbreaking research on cancer. Along with her colleagues and students, Bissell focused her attention on how the causal pathways regulating cell behavior were a two way street. Healthy cells’ and cancer cells’ behavior are both highly context-dependent. The pathway to this insight was not direct. Bissell’s work began with research into cellular metabolism. As a result of this early research, she found that cells can “change their fate” – revert to, or activate, functions not typical (...) of cells in the differentiated state. This had important implications for our understanding of cancer's etiology and treatment. Bissell - among others - emphasized in her research that it was not simply cell intrinsic properties – such as mutations to “oncogenes” and “tumor suppressor” genes – that determine the typical behaviors of cancer cells, but also a variety of extrinsic properties in the surrounding environment: metabolic and other signaling molecules, the extracellular matrix, and tissue architecture. (shrink)

Causal connectionists need to provide an account of causal linkage and of causal direction. This chapter distinguishes between two kinds of causal connection, namely, discrete and continuous. Causal connectionists have a number of options for explaining the linkage between causes and effects in the case of discrete causation. The chapter provides some popular options. If some causation is discrete, and the exercise of causal powers provides a direction to discrete causation, then the causal direction of processes (...) can be derived from the causal direction of discrete interaction. David Hume famously made the claim about the knowledge of causation in his Enquiry Concerning Human Understanding. Hume offers, in defense of his principle, a set of thought‐experiments that seem to show that we can always imagine any event's having a different result from the one we in fact observe. Causal processes like locomotion may provide counterexamples to Hume's Epistemic Principle. (shrink)

From the perspective of ecological psychology, perception and action are not separate, linear, and mechanistic processes that refer to the immediate present. Rather, they are reciprocal and continuous and refer to the impending future. Therefore, from the perspective of ecological psychology, delays in perception and action are impossible, and delay compensation mechanisms are unnecessary.

This paper will be a limited attempt to make sense of divine causation in Leibniz. I will not be able to discuss whether divine causation is immanent causation, nor explore the metaphysics of creation, emanation, and miracles in detail. Rather, I will focus on adjudicating the degree to which Leibniz’s famous denial of the interaction of substances bears on his views of divine causation. (edited).

As is well known, Aristotelian natural philosophy, and many other systems of natural philosophy since, have relied heavily on teleology and teleological causation. Somehow, the purpose or end of an obj ect can be used to predict and explain what that object does: once you know that the end of an acorn is to become an oak, and a few things about what sorts of circumstances are conducive to the attainment of this end, you can predict a lot about (...) the sprouting of the acorn and the subsequent behaviour of the piece of vegetation that results. Once you know that a rock seeks to move towards the centre of the Earth, you gain some insight into why it falls when released, and why it deforms the carpet or foot that it lands on. Once you know that the rabbit seeks to preserve itself, you can predict it will run from the fox. And so on. There are at least three features of Aristotle’s teleology, and more generally of an Aristotelian frame of mind about teleology, that may induce suspicion. One is that an end can serve as a "cause": as well as the sort of causation we all recognize, efficient causation, there are other forms, one of which is teleological causation. However, this can look less odd if we think of causes as things that figure in "because" answers to "why" questions. Whether or not self-preservation, or the rabbit’s continued existence, or something similar, causes the rabbit to run, the reply "because it seeks to continue in existence" certainly makes sense as an answer, or part of an answer, to a question about why it ran from the fox. (At present we are only. (shrink)

Consensus in the contemporary philosophical literature has it that conserved quantity theories of causation such as that of Dowe [2000]—according to which causation is to be analysed in terms of the exchange of conserved quantities (e.g., energy)—face damning problems when confronted with contemporary physics, where the notion of conservation becomes delicate. In particular, in general relativity it is often claimed that there simply are no conservation laws for (say) total-stress energy. If this claim is correct, it is difficult (...) to see how conserved quantity theories of causation could survive. In this article, we resist the above consensus and defend conserved quantity theories from this conclusion, at least when focusing on the apparent problems posed by general relativity. We argue that this approach to causation can continue to be defended in general relativity, once one appreciates (a) the availability of approximate symmetries in generic general relativistic spacetimes, and (b) the role of modelling and idealisation in that theory. Given these points, conserved quantity theories of causation must stand or fall on other grounds. (shrink)

Causation is one of the most controversial topics in philosophy. There is a wide range of philosophical accounts of causation, for example, the regularity account, the probabilistic account, the counterfactual account, the interventionist account, which can be all classified as ‘difference-making’ accounts; and the mechanistic account. Many argue that only one of these accounts is correct as there is only one type of causal relation (causal monism), while others maintain that there are multiple types of causation (causal (...) pluralism). In addition, there are eliminativists argue that science has no need of causation at all, while primitivists maintain that causation is unanalysable. Recently, the difference-making and mechanistic approaches have dominated recent philosophical discussion of causation. Other approaches and positions have been insufficiently discussed and assessed, especially in the context of philosophy of science. This volume explores and examines alternative approaches to causation. It revisits causal primitivism and causal eliminativism in the context of recent literature. It further explores the pluralistic approach, the fictionalist approach, the inferentialist approach, and the informational approach. It also examines the application of the dispositional approach, the epistemic approach, and the powerful particulars approach to the natural and social sciences. Overall, the volume is complementary to the recent discussion on the difference-making and mechanistic approaches and sheds new light on the metaphysical, epistemological, conceptual and methodological issues on causation. As such, it provides foundations for further research and teaching of this hotly debated topic. (shrink)

Reciprocal altruism was originally formulated in terms of individual selection and most theorists continue to view it in this way. However, this interpretation of reciprocal altruism has been challenged by Sober and Wilson (1998). They argue that reciprocal altruism (as well as all other forms of altruism) evolves by the process of group selection. In this paper, we argue that the original interpretation of reciprocal altruism is the correct one. We accomplish this by arguing that if (...) fitness attaches to (at minimum) entire life cycles, then the kind of fitness exchanges needed to form the group-level in such situations is not available. Reciprocal altruism is thus a result of individual selection and when it evolves, it does so because it is individually advantageous. (shrink)

I argue that, in his effort to overcome causation as an obstacle to agency or free will, Raymond Tallis’ self-described “Humean” re-working of David Hume’s analysis of causation falters on historicotextual and conceptual grounds.

This paper attempts to elucidate three characteristics of causal relationships that are important in biological contexts. Stability has to do with whether a causal relationship continues to hold under changes in background conditions. Proportionality has to do with whether changes in the state of the cause “line up” in the right way with changes in the state of the effect and with whether the cause and effect are characterized in a way that contains irrelevant detail. Specificity is connected both to (...) David Lewis’ notion of “influence” and also with the extent to which a causal relation approximates to the ideal of one cause–one effect. Interrelations among these notions and their possible biological significance are also discussed. (shrink)

Causation The question, “What is causation?” may sound like a trivial question—it is as sure as common knowledge can ever be that some things cause another; that there are causes and they necessitate certain effects. We say that we know that what caused the president’s death was an assassin’s shot. But when asked why, we … Continue reading Causation →.

Introduction , Sophie Gibb 1. Mental Causation , John Heil 2. Physical Realization without Preemption , Sydney Shoemaker 3. Mental Causation in the Physical World , Peter Menzies 4. Mental Causation: Ontology and Patterns of Variation , Paul Noordhof 5. Causation is Macroscopic but not Irreducible , David Papineau 6. Substance Causation, Powers, and Human Agency , E. J. Lowe 7. Agent Causation in a Neo-Aristotelian Metaphysics , Jonathan D. Jacobs and Timothy O’Connor 8. (...) Mental Causation and Double Prevention , Sophie Gibb 9. The Identity Theory as a Solution to the Exclusion Problem , David Robb 10. Continuant Causation, Fundamentality, and Freedom , Peter Simons 11. There is no Exclusion Problem , Steinvor Tholl Arnadottir and Tim Crane. (shrink)

The widely accepted “continuing bonds” model of grief tells us that rather than bereavement necessitating the cessation of one’s relationship with the deceased, very often the relationship continues instead in an adapted form. However, this framework appears to conflict with philosophical approaches that treat reciprocity or mutuality of some form as central to loving relationships. Seemingly the dead cannot be active participants, rendering it puzzling how we should understand claims about continued relationships with them. In this article, we resolve this (...) tension by highlighting two fundamental aspects of paradigmatic loving relationships that can, and often do, continue in an adapted form following bereavement: love and mutual shaping of interests, choices, and self-concepts. Attention to these continuing features of relationships helps to capture and clarify the phenomenological and behavioral features of continuing bonds. However, love and mutual shaping must also change in important ways following bereavement. Love becomes unreciprocated, and although the dead continue to shape our interests, choices, and self-concepts, we predominantly shape their legacies and memories in return. These changes place important constraints upon the nature of our interpersonal connections with the dead. (shrink)

The paper concerns the metaphysical possibility of life after death. It argues that the existence of a psychological duplicate is insufficient for resurrection, even if psychological continuity suffices for personal identity. That is because our persistence requires immanent causation. There are at most three ways of having life after death: if we are immaterial souls; if we are snatched bodily from our deathbeds; or if there is immanent causation ‘at a distance’ as Zimmerman proposes--but this requires an ontology (...) of temporal parts. (shrink)

In this paper I discuss a recent exchange of articles between Hugh McLachlan and John Coggon on the relationship between omissions, causation, and moral responsibility. My aim is to contribute to their debate by isolating a presupposition I believe they both share and by questioning that presupposition. The presupposition is that, at any given moment, there are countless things that I am omitting to do. This leads both McLachlan and Coggon to give a distorted account of the relationship between (...) causation and moral or (as the case may be) legal responsibility and, in the case of Coggon, to claim that the law’s conception of causation is a fiction based on policy. Once it is seen that this presupposition is faulty, we can attain a more accurate view of the logical relationship between causation and moral responsibility in the case of omissions. This is important because it will enable us, in turn, to understand why the law continues to regard omissions as different, both logically and morally, from acts, and why the law seeks to track that logical and moral difference in the legal distinction it draws between withholding life-sustaining measures and euthanasia. (shrink)

Sometimes you make a choice. Whether or not you made it was up to you. The choice was free. But how can this be? A scientific view of the world may leave no room for free choice. Free will literature continually explodes. Yet experts still focus on control or on a power to do otherwise. Sadly, they neglect another intuitive feature of free will: being an underived source or ultimate originator. When acting freely, one is a self-determined, self-directed, sole author (...) of change. Surprisingly, though the literature is replete with this pre-theoretic feature of free will, virtually no one makes rigorous use of it when constructing arguments. Equipped with fresh research, this book contends that agent-causation—currently an unpopular theory—deserves serious reconsideration. Only agent-causation accounts for ultimate origination, and popular objections fail under rigorous scrutiny. This book is tightly argued. It challenges and deepens recent advances in free will scholarship made by Peter van Inwagen, Robert Kane, Carl Ginet, Randy Clarke, Stewart Goetz, Tim O’Connor, and others. (shrink)

This paper analyzes the metaphysical system developed in Cheyne’s Philosophical Principles of Religion. Cheyne was an early proponent of Newtonianism and tackled several philosophical questions raised by Newton’s work. The most pressing of these concerned the causal origin of gravitational attraction. Cheyne rejected the occasionalist explanations offered by several of his contemporaries in favor of a model on which God delegated special causal powers to bodies. Additionally, he developed an innovative approach to divine conservation. This allowed him to argue that (...) Newton’s findings provided evidence for God’s existence and providence without the need for continuous divine intervention in the universe. (shrink)

In this paper I take a look at what I take to be the best argument for dispositions. According to this argument we need dispositions in order to understand certain features of scientific practice. I point out that these dispositions have to be continuously manifestable. Furthermore I will argue that dispositions are not the causes of their manifestations. However, dispositions and causation are closely connected. What it is to be a cause can best be understood in terms of counterfactuals (...) that are based on dispositions. (shrink)

The observation that agents and structures are co-constituted is now commonplace, yet scholars continue to struggle to incorporate this insight. Rationalists tend to overemphasize actors’ agency in the constitution of social order while constructivists tend to overstate the degree to which structures determine action. This article uses The Gift to rethink the agent–structure debate, arguing that the model of social relations Mauss outlines in this work sheds new light on basic concepts in international relations theory such as reciprocity, hierarchy, and (...) obligation. Mauss’ social theory locates the generative structure of social order in diffuse exchange relations, what he terms gift exchange, and assumes that actors are both socially positioned within hierarchical relations of exchange and reflexive agents who are able to understand and strive to change those relations. In so doing, he avoids reducing social order to either deeply internalized social norms or instrumental interests, navigating between ag... (shrink)

Biomedical research and its translation continue to pose normative questions about the nature of relations between researcher and participant and the role of research involving human subjects in so...

The cause of an event must continue over a period at which the effect is not occurring and the whole period at which it is occurring. It follows that simultaneous causation and backward causation are metaphysically impossible. I distinguish among events said to occur at a time, ‘hard’ events which really occur solely at that time and ‘soft’ events which occur partly at another time. God’s beliefs at a time are hard events at that time. It follows that (...) if God is a temporal being, he cannot know infallibly what either we or he will do freely at a future time; and if God is timeless, he cannot know what happens in time. Hence we must define God’s ‘omniscience’ in such a way as to exclude any knowledge of future free actions. I discuss in an Appendix how far this view is compatible with Scripture and Church tradition. (shrink)

For well over a century the dominant narrative covering the major thinkers and themes of early modern British philosophy has been that of “British Empiricism”, within which the great triumvirate of Locke-Berkeley-Hume are taken to be the dominant figures. Although it is now common to question this schema as a way of analyzing and understanding the period in question, it continues to command considerable authority and acceptance. (One likely reason for this is that no credible or plausible alternatives structures or (...) schemas of analysis have suggested themselves.) Be this as it may, the analysis of the rival systems of Clarke, Berkeley and Hume makes clear that this narrative, however deeply entrenched it may be, is wholly misleading and both distorts and obscures key themes and figures in the period in question. (shrink)