An examination of the statement, criticism, and reformulation of the Pirenne, Turner, and Weber theses as causal explanations makes possible a clarification of the nature and justification of causal theses in history. Criticisms of such theses typically attack either the description of the cause-phenomenon or the effect-phenomenon, or they attack the generalization or theory which justifies the claim of causal connection. Theses are defended by redescribing the phenomena so as to make the underlying theory a stronger justification. (...) The analysis clarifies the essential connection between description and explanation, and shows that "colligatory" descriptions are always theoretical relative to lower-level factual descriptions. (shrink)

The received view in philosophy of biology is that there is a well-understood, philosophically rigorous account of information—causal information. I argue that this view is mistaken. Causal information is fatally undermined by misinterpretations and conflations between distinct independent accounts of information. As a result, philosophical arguments based on causal information are deeply flawed. I end by briefly considering what a correct application of the relevant accounts of information would look like in the biological context.

I present a reconstruction of F.H.C. Crick's two 1957 hypotheses "Sequence Hypothesis" and "Central Dogma" in terms of a contemporary philosophical theory of causation. Analyzing in particular the experimental evidence that Crick cited, I argue that these hypotheses can be understood as claims about the actual difference-making cause in protein synthesis. As these hypotheses are only true if restricted to certain nucleic acids in certain organisms, I then examine the concept of causal specificity and its potential to counter claims (...) about causal parity of DNA and other cellular components. I first show that causal specificity is a special kind of invariance under interventions, namely invariance of generalizations that range over finite sets of discrete variables. Then, I show that this notion allows the articulation of a middle ground in the debate over causal parity. (shrink)

Causal selection is the task of picking out, from a field of known causally relevant factors, some factors as elements of an explanation. The Causal Parity Thesis in the philosophy of biology challenges the usual ways of making such selections among different causes operating in a developing organism. The main target of this thesis is usually gene centrism, the doctrine that genes play some special role in ontogeny, which is often described in terms of information-bearing or (...) programming. This paper is concerned with the attempt of confronting the challenge coming from the Causal Parity Thesis by offering principles of causal selection that are spelled out in terms of an explicit philosophical account of causation, namely an interventionist account. I show that two such accounts that have been developed, although they contain important insights about causation in biology, nonetheless fail to provide an adequate reply to the Causal Parity challenge: Ken Waters's account of actual-difference making and Jim Woodward's account of causal specificity. A combination of the two also doesn't do the trick, nor does Laura Franklin-Hall's account of explanation (in this volume). We need additional conceptual resources. I argue that the resources we need consist in a special class of counterfactual conditionals, namely counterfactuals the antecedents of which describe biologically normal interventions. (shrink)

Some have argued that no analysis of counterfactual conditionals can succeed without appealing to causal notions. Such authors claim that, in determining what would transpire had some events gone differently, we hold fixed everything that is causally independent from those events. Call this view Causal Independence. Some have argued that we need Causal Independence to accommodate intuitive judgments about certain kinds of counterfactuals in indeterministic worlds. The aim of this paper is to show that, contra these authors, (...) Causal Independence systematically delivers counterintuitive results for a certain subset of such counterfactuals-namely, those involving causation by double prevention. I conclude that intuitions about such counterfactuals do not motivate Causal Independence, at least in any form in which it has thus far been articulated. However, I suggest that a refined Causal Independence thesis that presupposes a kind of causal pluralism might be able to accommodate these intuitions, though such a refined version of Causal Independence may not conflict with reductive analyses of causal notions that appeal to counterfactuals after all. (shrink)

In this thesis, I give a metascientific account of causality in medicine. I begin with two historical cases of causal discovery. These are the discovery of the causation of Burkitt’s lymphoma by the Epstein-Barr virus, and of the various viral causes suggested for cervical cancer. These historical cases then support a philosophical discussion of causality in medicine. This begins with an introduction to the Russo- Williamson thesis (RWT), and discussion of a range of counter-arguments against it. Despite (...) these, I argue that the RWT is historically workable, given a small number of modifications. I then expand Russo and Williamson’s account. I first develop their suggestion that causal relationships in medicine require some kind of evidence of mechanism. I begin with a number of accounts of mechanisms and produce a range of consensus features of them. I then develop this consensus position by reference to the two historical case studies with an eye to their operational competence. In particular, I suggest that it is mechanistic models and their representations which we are concerned with in medicine, rather than the mechanism as it exists in the world. -/- I then employ these mechanistic models to give an account of the sorts of evidence used in formulating and evaluating causal claims. Again, I use the two human viral oncogenesis cases to give this account. I characterise and distinguish evidence of mechanism from evidence of difference-making, and relate this to mechanistic models. I then suggest the relationship between types of evidence presents us with a means of tackling the reference-class problem. This sets the scene for the final chapter. Here, I suggest the manner in which these two different classes of evidence become integrated is also reflected in the way that developing research programmes change as their associated causal claims develop. (shrink)

Several philosophers have recently defended Causal Essentialism—the view that every property confers causal powers, and whatever powers it confers, it confers essentially. I argue that on the face of it, Causal Essentialism implies a form of Monism, and in particular, the thesis I call ‘Mereological Monism’: that there is some concretum that is a part of every concretum. However, there are three escape routes, three views which are such that if one of them is true, (...) class='Hi'>Causal Essentialism does not imply any form of Monism at all. I survey the costs associated with taking these escape routes along with the costs associated with accepting Mereological Monism. (shrink)

A critical analysis of recent interventionist responses to the causal exclusion problem is presented. It is argued that the response can indeed offer a solution to the problem, but one that is based on renouncing the multiple realizability thesis. The account amounts to the rejection of nonreductive physicalism and would thus be unacceptable to many. It is further shown that if the multiple realizability thesis is brought back in and conjoined with the interventionist notion of causation, inter-level (...) causation is ruled out altogether. (shrink)

Causal essentialists hold that a property essentially bears its causal and nomic relations. Further, as many causal essentialists have noted, the main motivations for causal essentialism also motivate holding that properties are individuated in terms of their causal and nomic relations. This amounts to a kind of identity of indiscernibles thesis; properties that are indiscernible with respect to their causal and nomic relations are identical. This can be compared with the more well-known identity (...) of indiscernibles thesis, according to which particulars that are qualitatively indiscernible are identical. Robert Adams has developed a well-known objection to this thesis by considering a series of possibilities involving nearly qualitatively indiscernible particulars that naturally leads to a possibility involving qualitatively indiscernible particulars. I argue that we can construct parallel cases involving a series of possibilities involving properties that are nearly indiscernible with respect to their causal and nomic relations that naturally lead to possibilities involving properties that are indiscernible with respect to their causal and nomic relations. The same features that make Adams’ argument forceful also carry over to my cases, giving us a powerful objection to the causal essentialist identity of indiscernibles thesis. (shrink)

The thesis explores and suggests a solution to a problem that I identify in John McDowell’s and Lynne Rudder Baker’s approaches to mental and intention-dependent (ID) causation in the physical world. I begin (chapter 1) with a brief discussion of McDowell’s non-reductive and anti-scientistic account of mind and world, which I believe offers, through its vision of the unbounded conceptual and the world as within the space of reasons, to liberate and renew philosophy. However, I find an inconsistency in (...) McDowell’s criticism of Davidson’s anomalous monism (chapter 2), stemming from a tension between McDowell’s naïve common sense view of mental causation and an understanding of ordinary physical causation which I think McDowell ought to embrace, which portrays it as both objective, in the sense of being recognition- independent, and as belonging within the space of reasons. The question of the relation between these two concepts of causation is an aspect of the more general question of the relation between the space of reasons and the realm of law. In chapter 3 I begin examining the possibility that Baker’s account of material and property constitution could form the basis of a bridge between the two spaces, and find it generally promising. However, I find that her defence of her version of non-reductive monism against Kim’s causal arguments also runs into problems, which I attribute to the fact that she holds a view of causation as secondary to causal explanation. 2 3 In chapter 4 I develop an account of what I call manifest physical causation – of objective causal relations in the world of Sellars’s manifest image. Based upon McDowell’s transcendental empiricism, which takes the existence of the ordinary perceived world as a condition of the possibility of our possession of conceptual capacities, I contend that it is this picture of reality, rather than that of fundamental physics, that should be the starting point of our theorizing. Causation in the manifest image, I argue, covers the behaviour of the familiar physical world as well as that of its well-understood extensions into the special sciences and engineering. Manifest physical causation, on my account, is productive, acts through mechanisms which are almost entirely mechanical, electromagnetic, and/ or chemical, and is causally closed. In my view, normative, semantic, contentful property-instances are not part of the manifest physical causal nexus. In my final chapter I suggest a modification of Baker’s constitution account, which I call Constituted Causation, whereby higher-level – mental and other ID – causal relations are constituted, in favourable circumstances, by lower-level ones. ID causal relations belong in their own causal nexus but are connected to the manifest physical world through constitution, a relation of unity without identity. Causation and constitution are, respectively, intra- and inter-level relations, and they are non-overlapping. The constituted network of rational and normative relations bears, I believe, striking parallels with McDowell’s view of reality. According to Baker’s view of constitution, the essential properties of constituted entities subsume those of their constituters; extending this to my account enables us to say that the real cause and explanation of someone’s action is that they consciously performed it 4 rather than that certain manifest causal processes occurred at the lower level. (shrink)

In this dissertation, I develop and defend a model of causation that allows for dualist mental causation in worlds where the physical domain is physically complete. In Part I, I present the dualist ontology that will be assumed throughout the thesis and identify two challenges for models of mental causation within such an ontology: the exclusion worry and the common cause worry. I also argue that a proper response to these challenges requires a thoroughly lightweight account of causation, i.e. (...) an account that allows for causes to be metaphysically distinct from the phenomena that produce or physically necessitate their effects. In Part II, I critically evaluate contemporary responses to these challenges from the philosophical literature. In particular, I discuss List and Stoljar’s criticism of exclusion worries, Kroedel’s alternative dualist ontology, concerns about the notion of causal sufficiency, and Lowe’s models of dualist mental causation. I argue that none of these proposals provide independent motivation for a thoroughly lightweight account of causation and therefore leave room for improvement. In the first four chapters of Part III, I develop a thoroughly lightweight model of causation, which builds on interventionist approaches to causation. First, I explain how so-called ‘holding fixed’-requirements in standard interventionist accounts stand in the way of dualist mental causation. I then argue that interventionist accounts should impose a robustness condition on causal correlations and that, with this condition in place, the ‘holding fixed’-requirements can be weakened such that they do allow for dualist mental causation. I dub the interventionist model with such weakened ‘holding fixed’-requirements ‘insensitive interventionism’, argue that it can counter the exclusion worry as well as the common cause worry, and explain under which circumstances it would predict there to be dualist mental causation. Importantly, these circumstances might, for all we know, hold in the actual world. In the final three chapters of Part III, I defend insensitive interventionism against some objections. I consider the objection that causation must be productive, the objection that causes must physically necessitate their effects, and the objection that insensitive interventionism is too permissive. I respond by drawing from the literature on causation by absences and on the relation between causation and fundamental physics. Overall, insensitive interventionism performs as well as standard interventionist accounts. I conclude that insensitive interventionism is a credible model of causation. (shrink)

Peter Alexander: It is presumably admitted that laws, whether causal or not, are universal in form; they are appropriately stated in universal categoricals or unrestricted hypotheticals. I assume that this is not at issue in the question set. I take our question to be this: given that causal laws are universal statements, can they be said to be about, to apply to, to hold for, individual things? -/- Peter Downing: Mr. Alexander maintains that there are 'irreducibly singular' (...) class='Hi'>causal statements, and that from this it follows that causal laws need not be purely general. The view that there are irreducibly singular causal statements appears to commit Alexander to rejecting two theses: (i) that singular causal statements are analysable into statements containing specific generalizations, [...] (ii) That singular causal statements are analysable into statements containing incompletely specified generalizations [...]. Thesis (i) is, I think, clearly false. [...] Thesis (ii), however, is not easily refuted. (shrink)

Russo and Williamson put forward the following thesis: in order to establish a causal claim in medicine, one normally needs to establish both that the putative cause and putative effect are appropriately correlated and that there is some underlying mechanism that can account for this correlation. I argue that, although the Russo-Williamson thesis conflicts with the tenets of present-day evidence-based medicine, it offers a better causal epistemology than that provided by present-day EBM because it better explains (...) two key aspects of causal discovery. First, the thesis better explains the role of clinical studies in establishing causal claims. Second, it yields a better account of extrapolation. (shrink)

In psychiatry, pharmacological drugs play an important experimental role in attempts to identify the neurobiological causes of mental disorders. Besides being developed in applied contexts as potential treatments for patients with mental disorders, pharmacological drugs play a crucial role in research contexts as experimental instruments that facilitate the formulation and revision of neurobiological theories of psychopathology. This paper examines the various epistemic functions that pharmacological drugs serve in the discovery, refinement, testing, and elaboration of neurobiological theories of mental disorders. I (...) articulate this thesis with reference to the history of antipsychotic drugs and the evolution of the dopamine hypothesis of schizophrenia in the second half of the twentieth century. I argue that interventions with psychiatric patients through the medium of antipsychotic drugs provide researchers with information and evidence about the neurobiological causes of schizophrenia. This analysis highlights the importance of pharmacological drugs as research tools in the generation of psychiatric knowledge and the dynamic relationship between practical and theoretical contexts in psychiatry. (shrink)

Causal selection and priority are at the heart of discussions of the causal parity thesis, which says that all causes of a given effect are on a par, and that any justified priority assigned to a given cause results from causal explanatory interests. In theories of causation that provide necessary and sufficient conditions for the truth of causal claims, status as cause is an either/or issue: either a given cause satisfies the conditions or it does (...) not. Consequently, assessments of causal parity and priority require more resources, which can either be additional or part of the causal analysis itself. While adding resources in terms of a theory of causal explanation has been standard, here we develop a unified conceptual analysis that includes a range of different precise causal concepts that allows for assessments of causal priority in terms of different kinds of causal relevance. (shrink)

This thesis addresses the problem of causal asymmetry. This problem may be characterized as follows: what is the relation R such that if an event c causes an event e c bears relation R to e but e does not bear relation R to e. The traditional Humean account of causal asymmetry is that "R" may be replaced by "temporally prior." Difficulties with this account based on consideration of cases of simultaneous causation and backward causation have given (...) rise to non-Humean accounts of causal asymmetry. This dissertation analyzes some of these difficulties along with various non-Humean approaches. I offer a new theory of causal asymmetry which does not depend upon the temporal priority of causes to their effects and which differs from any of the other theories available. ;In chapter one the problem of causal asymmetry is clarified. This problem is distinguished from related issues. At the same time, I articulate the various philosophical considerations which bear on the scope and importance of this problem. I also state what I take to be the conditions an adequate account of causal asymmetry must satisfy. In the second chapter I discuss the classical Humean analysis of causal priority in terms of temporal priority. It is argued that arguments which have been put forward to show that backwards causation is logically impossible are unconvincing and that there are cases of simultaneous causation. In the third chapter the manipulability theory of causation is treated. Various formulations of this theory, from the most anthropomorphic to the least anthropomorphic are treated as they bear on the problem of causal asymmetry. Manipulability theory is found to be inadequate. ;In chapter four counterfactual theories of causation are considered. A number of counterexamples to these theories are presented and developed. In chapter five, two views which are grouped under "regularity theory" are analyzed and criticized: those of Bromberger and Berofsky. Mackie's earlier and later accounts of causal asymmetry along with Sanford's view are discussed in chapter six. In chapter seven, Erik Brown's recent view is considered alongside "transference theory." In the final chapter I develop a new theory of causal priority which utilizes the "circumstantial" character of the causal relation in accounting for causal asymmetry. (shrink)

An influential tradition in the philosophy of causation has it that all token causal facts are, or are reducible to, facts about difference-making. Challenges to this tradition have typically focused on pre-emption cases, in which a cause apparently fails to make a difference to its effect. However, a novel challenge to the difference-making approach has recently been issued by Alyssa Ney. Ney defends causal foundationalism, which she characterizes as the thesis that facts about difference-making depend upon facts (...) about physical causation. She takes this to imply that causation is not fundamentally a matter of difference-making. In this paper, I defend the difference-making approach against Ney’s argument. I also offer some positive reasons for thinking, pace Ney, that causation is fundamentally a matter of difference-making. (shrink)

Whether or not an intentional explanation of action necessarily involves law-like statements is related to another question, namely, is it a causal explanation? The Popper-Hempel Thesis , which answers both questions affirmatively, inevitably faces a dilemma between realistic and universalistic requirements. However, in terms of W.C. Salmon’s concept of causal explanation, intentional explanation can be a causal one even if it does not rely on any laws. Based on this, we are able to refute three characteristic (...) arguments for the claim “reason is not a cause of action,” namely, the “proper logical” argument, the “logical relation” argument, and the “rule-following” argument. This rebuttal suggests that the causal relationship between reason and action can provide a justification for intentional explanations. (shrink)

I propose an argument for the thesis that laws of nature are necessary in the sense of holding in all worlds sharing the properties of the actual world, on the basis of a principle I propose to call the Causal Criterion of Reality . The CCR says: for an entity to be real it is necessary and sufficient that it is capable to make a difference to causal interactions. The crucial idea here is that the capacity to (...) interact causally - or to contribute to determining causal interactions - is not only the ultimate metaphysical ground for the existence of an entity, but it also provides a criterion for determining the nature of that entity, i.e. its properties. The alternative is to conceive of laws of nature as contingent: they could be different from what they are like in the actual world, where that possibility is understood to be metaphysical, not only epistemic. For the sake of this paper, I shall accept Armstrong's thesis that laws of nature are relations between universals. I also follow Armstrong in the view that both the existence and the properties of particulars are metaphysically independent of the existence and identity of other particular. However, what is controversial and what I shall challenge is his thesis that universals are like particulars in the following respect: according to Armstrong, each universal is a logically distinct entity whose existence and identity is independent of the existence and identity of other universals. My aim in this paper is to show that the identity of a universal is entirely determined by its lawful relations to other universals. The crucial premise I use is the thesis that the CCR is a universal criterion, which applies both to particulars and universals. From the thesis that the identity of a universal is exclusively determined by laws, it follows that laws are necessary in the sense that they cannot differ without the universals they link also being different. This creates a difficulty for those authors who, as Armstrong, accept the CCR but nevertheless defend the view that laws are contingent. (shrink)

The thesis addresses the nature of causation. It is argued that causation exists and is as local as its causes and effects. As a consequence, the position advocated is contrary to the as yet prevailing view that no 'causal tie' between cause and effect exists. Moreover, it is suggested that this tie can be perceived. The essay attempts to elucidate the nature of causes, effects, and causal mechanisms. It is argued that they are facts rather than particulars (...) or universals. Furthermore it is suggested that these facts can be conceived of as tropes. Finally, in the light of this thesis a few traditional issues are discussed: what is the relation between causation and explanation? How should we conceive of causal laws? Does the idea of mechanisms with different degrees of reliability further our understanding of the determinism/indeterminism issue? (shrink)

Martin and Deutscher’s causal theory of remembering holds that a memory trace serves as a necessary causal link between any genuine episode of remembering and the event it enables one to recall. In recent years, the causal theory has come under fire from researchers across philosophy and cognitive science, who argue that results from the scientific study of memory are incompatible with the kinds of memory traces that Martin and Deutscher hold essential to remembering. Of special note, (...) these critics observe, is that a single memory trace can be shaped by multiple past experiences. This appears to prevent traces from underwriting Martin and Deutscher’s distinction between remembering an event and merely forming an accurate representation of it. This paper accepts such criticisms of the standard causal theory and, through considering the phenomenon forgetting through repetition, raises several others. A substantially revised causal theory is then developed, compatible with the thesis that individual memory traces are shaped by multiple past experiences. The key strategy is to conceive of episodic remembering not as the simple retrieval and projection of a static memory trace, but as a complex quasi-inferential process that makes use of multiple forms of information and cues—“prop-like” memory traces included—in generating the experience known as episodic remembering. When remembering is understood as a multi-componential process, there are a variety of ways in which a representation of the past may be appropriately causally dependent upon a prior experience of the event remembered. (shrink)

It is widely thought that causal cognition underpins technical reasoning. Here we suggest that understanding causal cognition as a thinking system that includes theory of mind (i.e., social cognition) can be a productive theoretical tool for the field of evolutionary cognitive archaeology. With this contribution, we expand on an earlier model that distinguishes seven grades of causal cognition, explicitly presenting it together with a new analysis of the theory of mind involved in the different grades. We then (...) suggest how such thinking may manifest in the archaeological or stone tool record and techno-behaviors of the last three million years or so. Our thesis is threefold: (a) theory of mind is an integral element of causal cognition; (b) generally speaking, the more advanced causal cognition is, the more it is dependent on theory of mind; and (c) the evolution of causal cognition depends more and more on mental representations of hidden variables. Ultimately, the final or seventh grade of causal cognition allows us to reason from a network of hidden variables that, amongst other things, enables the learning, manufacture, and use of complex technological systems. It also facilitates the seamless mapping of knowledge between personal (egocentric), physical, and social networks that allows for newly devised and innovative technical and social outcomes. (shrink)

After precisely specifying the thesis of the causal theory of time, Grünbaum's program developed to support this thesis is examined. Four objections to his definition of temporal order in terms of a more primitive causal relation are put and held to be conclusive. Finally, the philosophical arguments that Grünbaum has proposed supporting the desirability of establishing a causal theory of time are shown to be either invalid or inconclusive.

I present an argument for causal fundamentality, understood as the thesis that the causal history of every being, whose existence has a causal explanation, includes some uncaused beings. I argue that this thesis is a consequence of an actualist account of metaphysical modality whose novelty lies in its hybrid dispositional-essentialist foundation. I argue that my modal theory is extensionally correct and minimalistic. Its range of metaphysical necessities and possibilities is just as wide as needed to (...) capture the pre-theoretical notion of modality. Moreover, my theory is immune from the necessitism of standard essentialist accounts of modality and addresses the challenge of global possibilities facing dispositionalist modal theories thanks to its essentialist component. (shrink)

The aim of this paper is to argue that there are categorical properties as well as causal powers, and that the world would not exist as we know it without them. For categorical properties are needed to define the powers—to locate them, and to specify their laws of action. These categorical properties, I shall argue, are not dispositional. For their identities do not depend on what they dispose their bearers to do. They are, as Alexander Bird would say, ’quiddities’. (...) But there is nothing wrong with quiddities. And, in the second half of this paper, I shall defend the thesis that all categorical properties are quiddities. (shrink)

The Church-Turing thesis makes a bold claim about the theoretical limits to computation. It is based upon independent analyses of the general notion of an effective procedure proposed by Alan Turing and Alonzo Church in the 1930''s. As originally construed, the thesis applied only to the number theoretic functions; it amounted to the claim that there were no number theoretic functions which couldn''t be computed by a Turing machine but could be computed by means of some other kind (...) of effective procedure. Since that time, however, other interpretations of the thesis have appeared in the literature. In this paper I identify three domains of application which have been claimed for the thesis: (1) the number theoretic functions; (2) all functions; (3) mental and/or physical phenomena. Subsequently, I provide an analysis of our intuitive concept of a procedure which, unlike Turing''s, is based upon ordinary, everyday procedures such as recipes, directions and methods; I call them mundane procedures. I argue that mundane procedures can be said to be effective in the same sense in which Turing machine procedures can be said to be effective. I also argue that mundane procedures differ from Turing machine procedures in a fundamental way, viz., the former, but not the latter, generate causal processes. I apply my analysis to all three of the above mentioned interpretations of the Church-Turing thesis, arguing that the thesis is (i) clearly false under interpretation (3), (ii) false in at least some possible worlds (perhaps even in the actual world) under interpretation (2), and (iii) very much open to question under interpretation (1). (shrink)

The past century has witnessed numerous methodological innovations in probabilistic and statistical methods of causal inference (e.g., the graphical modelling and the potential outcomes frameworks, as introduced in Chapter 1). These innovations have not only enhanced the methodologies by which scientists across diverse domains make causal inference, but they have also made a profound impact on the way philosophers think about causation. The philosophical issues discussed in this thesis are stimulated and inspired by these methodological innovations. Chapter (...) 2 addresses the question of how the holding of screening-off conditions for a causal model depends on the choice of variables. As bridge principles between probability and causation, screening-off conditions (especially the Causal Markov Condition) play a key role in causal inference. However, it has been known that these conditions may fail due to poor variable choice. My aim in this chapter is to further examine those constraints on variable choice that are deemed necessary for the satisfaction of screening-off conditions. The idea of a well-defined (hypothetical) intervention is also crucial for reliable causal inference. Chapter 3 explores the question of when interventions invoked in causal inference are “well-defined” or unambiguous, and how this requirement constrains the choice of cause-variables. I propose that an intervention is well-defined just in case the effect of interest is well-defined (under ideal interventions), and that the intervention can serve as a suitable means to identify that effect. Based on this proposal, several distinct types of ambiguous interventions are identified. Methodological progress in causal inference also poses the following question: Can such progress shed light on the ontology of causation? My answer is yes. In Chapter 4, I develop a Carnapian-pragmatist approach to the ontology of causation as an alternative to existing metaphysical approaches. I argue that, compared to traditional metaphysics, the pragmatist approach provides a superior picture of how the ontology and methodology of causation interact with each other in scientific practice. I conclude in Chapter 5 that the thing we call “causation” consists in both the right worldly infrastructure (e.g., screening-off patterns and possibilities for interventions) and appropriate ways of framing this infrastructure. (shrink)

Do causes necessitate their effects? Causal necessitarianism is the view that they do. One major objection—the “monotonicity objection”—runs roughly as follows. For many particular causal relations, we can easily find a possible “blocker”—an additional causal factor that, had it also been there, would have prevented the cause from producing its effect. However—the objection goes on—, if the cause really necessitated its effect in the first place, it would have produced it anyway—despite the blocker. Thus, CN must be (...) false. Though different from Hume’s famous attacks against CN, the monotonicity objection is no less important. In one form or another, it has actually been invoked by various opponents to CN, past and present. And indeed, its intuitive appeal is quite powerful. Yet, this paper argues that, once carefully analysed, the objection can be resisted—and should be. First, I show how its success depends on three implicit assumptions concerning, respectively, the notion of cause, the composition of causal factors, and the relation of necessitation. Second, I present general motivations for rejecting at least one of those assumptions: appropriate variants of them threaten views that even opponents to CN would want to preserve—in particular, the popular thesis of grounding necessitarianism. Finally, I argue that the assumption we should reject is the one concerning how causes should be understood: causes, I suggest, include an element of completeness that excludes blockers. In particular, I propose a way of understanding causal completeness that avoids common difficulties. (shrink)

This thesis presents an alternative method for the detection of MIMIC models. Previous methods (such as factor analysis) suffer from a number of significant aws and limitations, which the new method (a causal search algorithm) doesn't suffer. A new algorithm is introduced, followed by a worked-through example of its application. Discussion focuses on some of the limiting assumptions the algorithm currently requires. Finally, recommendations for future work address improvements of the algorithm, as well as its applicability.

In her ‘Causality and Determination’, Anscombe argues for the strong thesis that despite centuries of philosophical assumption to the contrary, the supposition that causality and necessity have something essential to do with one another is baseless. In this paper, I assess Anscombe’s arguments and endorse her conclusion. I then attempt to argue that her arguments remain highly relevant today, despite the fact that most popular general views of causation today are firmly probabilistic in orientation and thus show no trace (...) of the assumptions Anscombe hoped to undermine. My suggestion is that Anscombe's interests in causality are distinct from those which mostly animate the modern debate about the general nature of causality and that in those specialized areas of philosophy in which those concerns still dominate, one can still see the effects of the fallacies and confusions to which she alerts us. I conclude by offering two possible complementary explanations of the tendency to suppose that causation is a variety of necessitation. (shrink)

In an earlier paper (Dupré 1984), I criticized a thesis sometimes defended by theorists of probabilistic causality, namely, that a probabilistic cause must raise the probability of its effect in every possible set of causally relevant background conditions (the "contextual unanimity thesis"). I also suggested that a more promising analysis of probabilistic causality might be sought in terms of statistical relevance in a fair sample. Ellery Eells (1987) has defended the contextual unanimity thesis against my objections, and (...) also raised objections of his own to my positive claims. In this paper I defend and amplify both my objections to the contextual unanimity thesis and my constructive suggestion. (shrink)

It is widely thought that causal cognition underpins technical reasoning. Here we suggest that understanding causal cognition as a thinking system that includes theory of mind (i.e., social cognition) can be a productive theoretical tool for the field of evolutionary cognitive archaeology. With this contribution, we expand on an earlier model that distinguishes seven grades of causal cognition, explicitly presenting it together with a new analysis of the theory of mind involved in the different grades. We then (...) suggest how such thinking may manifest in the archaeological or stone tool record and techno-behaviors of the last three million years or so. Our thesis is threefold: (a) theory of mind is an integral element of causal cognition; (b) generally speaking, the more advanced causal cognition is, the more it is dependent on theory of mind; and (c) the evolution of causal cognition depends more and more on mental representations of hidden variables. Ultimately, the final or seventh grade of causal cognition allows us to reason from a network of hidden variables that, amongst other things, enables the learning, manufacture, and use of complex technological systems. It also facilitates the seamless mapping of knowledge between personal (egocentric), physical, and social networks that allows for newly devised and innovative technical and social outcomes. (shrink)

This thesis aims to clarify a number of conceptual aspects of the debate surrounding algorithmic fairness. The particular focus here is the role of causal modeling in defining criteria of algorithmic fairness. In Chapter 1, I argue that in the discussion of algorithmic fairness, two fundamentally distinct notions of fairness have been conflated. Subsequently, I propose that what is usually taken to be the problem of algorithmic fairness should be divided into two subproblems, the problem of predictive fairness, (...) and the problem of allocative fairness. At the core of Chapter 2 is the proof of a theorem that establishes that three of the most popular (predictive) fairness criteria are pairwise incompatible. In particular, I show that under certain conditions, a predictive algorithm that satisfies a criterion called counterfactual fairness will with logical necessity violate two other popular predictive fairness criteria called equalized odds and predictive parity. In Chapter 3, a new predictive fairness criterion is developed using a mathematical framework for causal modeling. This fairness criterion, which I call causal relevance fairness, is a relaxation of another popular fairness criterion, counterfactual fairness, but turns out to be more closely in line with philosophical theories of discrimination. In Chapter 4, another infamous impossibility result in algorithmic fairness is analyzed through the lens of causality. I argue that by using a causal inference method called matching, we can modify the two fairness criteria equalized odds and predictive parity in a way that resolves the impossibility. Lastly, Chapter 5 contains an empirical case study. In it, the fairness of a popular recidivism risk prediction tool is analyzed using the criteria of (predictive) fairness developed earlier. (shrink)

Systems involving many interacting variables are at the heart of the natural and social sciences. Causal language is pervasive in the analysis of such systems, especially when insight into their behavior is translated into policy decisions. This is exemplified by economics, but to an increasing extent also by biology, due to the advent of sophisticated tools to identify the genetic basis of many diseases. It is argued here that a regularity notion of causality can only be meaningfully defined for (...) systems with linear interactions among their variables. For the vastly more important class of nonlinear systems, no such notion is likely to exist. This thesis is developed with examples of dynamical systems taken mostly from mathematical biology. It is discussed with particular reference to the problem of causal inference in complex genetic systems, systems for which often only statistical characterizations exist. (shrink)

This work consists of two parts. Part I will be a contribution to a philo- sophical discussion of the nature of causal explanation. It will present my contrastive counterfactual theory of causal explanation and show how it can be used to deal with a number of problems facing theories of causal explanation. Part II is a contribution to a discussion of the na- ture of interest explanation in social studies of science. The aim is to help to (...) resolve some controversies concerning interest explanation by explicating the concept of interest and its explanatory uses by using the account of explanation developed in Part I. (shrink)

This paper defends the thesis of learning from non-causal models: viz. that the study of some model can prompt justified changes in one’s confidence in empirical hypotheses about a real-world target in the absence of any known or predicted similarity between model and target with regards to their causal features. Recognizing that we can learn from non-causal models matters not only to our understanding of past scientific achievements, but also to contemporary debates in the philosophy of (...) science. At one end of the philosophical spectrum, my thesis undermines the views of those who, like Cartwright (Erkenntnis 70:45–58, 2009), follow Hesse (Models and Analogies in Science, Notre Dame, University of Notre Dame Press, 1963) in restricting the possibility of learning from models to only those situations where a model identifies some causal factors present in the target. At the other end of the spectrum, my thesis also helps undermine some extremely permissive positions, e.g., Grüne-Yanoff’s (Erkenntnis 70(1):81–99, 2009, Philos Sci 80(5): 850–861, 2013) claim that learning from a model is possible even in the absence of any similarity at all between model and target. The thesis that we can learn from non-causal models offers a cautious middle ground between these two extremes. (shrink)

Recent research on “causal cognition” in adults and infants shows that we can perceive singular causal relations not previously experienced. In particular, infants that are able to perceive causality seem to rely on innate beliefs and principles that allow a priori inference of a connection between cause and effect. Can causal cognition in infants justify the thesis of causal realism? On the one hand, it weakens the central pillar of the Humean arguments: the impossibility of (...) a synthetic a priori causal inference. On the other hand, if perception is the privileged way of justifying the reality of objects of the external world, that is valid in the case of causal relations as well. Moreover, the perception of causal relations, based on innate principles and beliefs, reflects the selective results of the interaction between the real constraints of the physical structure of the world and the evolution of the human mind. (shrink)

Scientific conflicts often stem from differences in the conceptual framework through which scientists view and understand their own field. In this chapter, I analyze the ontological and methodological assumptions of three traditions in evolutionary biology, namely, Ernst Mayr’s population thinking, the gene-centered view of the Modern Syn thesis, and the Extended Evolutionary Synthesis. Each of these frameworks presupposes a different account of "evolutionary causes," and this discrepancy prevents mutual understanding and objective evaluation in the recent contention surrounding the EES. (...) From this perspective, the chapter characterizes the EES research program as an attempt to introduce causal structures beyond genes as additional units of evolution, and compares its research methodology and objectives with those of the traditional MS framework. (shrink)

Economists reason causally. Like many other scientists, they aim at formulating justified causal claims about their object of study. This thesis contributes to our understanding of how causal reasoning proceeds in economics. By using the research on the causes of unemployment as a case study, three questions are adressed. What are the meanings of causal claims? How can a causal claim be adequately supported by evidence? How are causal beliefs affected by incoming facts? In (...) the process of answering these semantic, epistemic and dynamical questions, the richness of causal reasoning in economics is brought to the foreground. In the part on semantics, it is argued that the meaning of causal generalizations in policy-oriented economics is best captured by using the concept of ‘inferential relation’ rather than the one of ‘referential relation’. This part contributes to develop an inferentialist semantics by distinguishing among types of inferential relations. In the part on epistemology, it is argued that justification in sciences like economics often relies, and ought to rely, on evidential variety―i.e. the combination of evidence from multiple sources. An interpretation of independence among evidential sources is offered: two evidential sources are fully independent if and only if the reasons why they would be unreliable do not overlap. It is then shown that the variety-of-evidence thesis does not hold under extreme circumstances. The part on dynamics focuses on deviant-case study. A storyline is provided for what deviant-case study aims at and how it proceeds in what is characterized as an ‘eclectic science’. (shrink)

This dissertation defends the view that concepts encode causal information and, for the first time, applies this view to a range of topics in the philosophy of language and social philosophy. In my first chapter (“Cognitive Essentialism and the Structure of Concepts”), I survey the current empirical and theoretical literature on causal-essentialist theories of concepts. In my second chapter (“Meaning Externalism and Causal Model Theory”), I propose an account of natural kind concepts according to which they encode (...) statistical information of features of a natural kind, and represents these features as causally related to each other. I show that this internalist model of concepts correctly predicts intuitions about Putnam’s twin earth scenario and Kripke’s conceivability cases that historically motivated philosophers of language to accept externalist accounts of meaning. The defended theory of concepts also informs topics that go beyond traditional issues in philosophy of language. In my third chapter (“An Essentialist Theory of the Meaning of Slurs”), I defend the view that slurs are, too, a species of kind terms: slur concepts encode mini-theories which represent an essence-like element that is causally connected to a set of negatively-valenced stereotypical features of a social group. This explains both the peculiar linguistic behavior of slurs and slurs’ dehumanizing effects. In my fourth chapter, I build on this insight, showing that the explicit language in and around pornography depicts women as ‘kinds’ or ‘breeds’ that are naturally made to enjoy certain sexual acts, and argue that this deterministic picture of women dehumanizes them. (shrink)

The identity of a natural kind can be construed in terms of its causal profile. This conception is more appropriate to science than two alternatives. The identity of a natural kind is not determined by one causal role because one natural kind can have many causal roles and several functions and because some functions are shared by different kinds. Furthermore, the microstructuralist thesis is wrong: The identity of certain natural kinds is not determined by their microstructure. (...) It is true that if A and B have the same microstructural composition then a sample of a chemical substance A is of the same chemical substance as a sample of B. However, the reverse does not hold. It is not the case that if a sample of a chemical substance A is of the same chemical substance as a sample of B then A and B have the same microstructural composition. This is because a macroscopic NK can be “multiconstituted” by different microstructures. (shrink)

The author of the paper critically analyzes a quasi-theory of future contingents (PFC) given by Marcin Tkaczyk and proposes his own explication of its theses and terms. The author makes it by introducing operational definitions of temporal and modal concepts, distinguishing between the causal and functional determination, discussing the status of the principle of bivalence, and replacing Tkaczyk’s theses by their new formulations. As a result, the author states, among other things, that (contrary to Tkaczyk) there is no contradiction (...) between the thesis about the opened future and the thesis about divine omniscience, because it requires the causal (but not functional as it is a case) determination between a true proposition and its determined fact. The author also shows that Tkaczyk’s examples of the retroactive causes are not an accurate solution of the antinomy because they are, at most, the examples of the factors which change the picture of the past but not the past itself. (shrink)

The term “historical epistemology” can be read in two different ways: (1) as referring to a program of ‘historicizing’ epistemology, in the sense of a critique of traditional epistemology’s tendency to gloss over historical context, or (2) as a manifesto of ‘epistemologizing’ history, i.e. as a critique of radical historicist and relativist approaches. In this paper I will defend a position in this second sense. I show that one can account for the historical development and diversity of science without disavowing (...) the relevance of a (normatively understood) epistemology and without denying the existence of human cognitive universals across historical and cultural differences. In support of my thesis, I draw on cognitive scientific research on causal and symbolic cognition, arguing that causal understanding constitutes a basic part of science, which, in the course of its development, becomes more and more superimposed by a culturally and historically variable symbolic superstructure. (shrink)

Frankfurt‐style cases suggest that an agent's moral responsibility for an action supervenes on the causal history of that action—at least when epistemic considerations are held constant. However, PAP‐style cases suggest that moral responsibility does not supervene on causal history, for judgments concerning an agent's responsibility for an action are also sensitive to the presence of alternative—and causally idle—possibilities. I appeal to the causal modeling tradition and the definitions of actual causation that derive therefrom in an attempt to (...) resolve this contradiction. I show that even the weakest definitions of actual causation proposed in the literature establish that some PAP‐style cases constitute genuine counterexamples to the supervenience thesis. I consider several responses to these counterexamples on behalf of the defenders of supervenience and show that they fail. Our best current thinking on causation thus appears to be inconsistent with an intuitive and widely held claim concerning the nature of moral responsibility. (shrink)

My aim in this thesis is to explain how a non-reductionist metaphysics can accommodate the causal relevance of the psychological and of the special sciences generally. According to physicalism, all behavior is caused by brain-states; given "folk-psychology", behavior is caused by some psychological state. If psychological states are distinct from brain states, then our behavior is overdetermined and this, it is claimed, is unacceptable. I argue that this consequence is not unacceptable. I claim that our explanatory practice should (...) guide our ontological commitment. If we can offer true explanations that appeal to more than one event, then we are committed to overdetermination for the event explained. I argue that accepting overdetermination is not absurd and that we can give an adequate account of causal relevance for psychological and other supervenient properties. The result is a partial defense of both property and event pluralism. Recent work by Davidson, Fodor, Jackson, Kim, Pettit and Yablo receives explicit and critical discussion. (shrink)

This essay defends two theses that jointly establish a link between causal and explanatory asymmetry. The first thesis is that statements specifying facts about effects, unlike statements specifying facts about causes, are not "independently variable". The second thesis is that independent variability among purportedly explanatory factors is a necessary condition on scientific explanations.