The dilemma of free will is that if actions are caused deterministically, then they are not free, and if they are not caused deterministically then they are not free either because then they happen by chance and are not up to the agent. I propose a conception of free will that solves this dilemma. It can be called agent causation but it differs from what Chisholm and others have called so.

The goal of this paper is two-fold: first, to emphasize causality in disease ontology and knowledge representation, presenting a general and cursory discussion of causality and causal chains; and second, to clarify and develop the River Flow Model of Diseases (RFM). The RFM is an ontological account of disease, representing the causal structure of pathology. It applies general knowledge of causality using the concept of causal chains. The river analogy of disease is explained, formal descriptions are offered, and the RFM (...) disease definition is refined by describing causal chains in terms of causal relations. The definition is updated to coincide with the actual RFM classes found in its upper-level ontology, YAMATO, which brings to light some challenges in developing both YAMATO and the RFM. The RFM is also discussed in relation to another ontological account of disease. Strategies are offered toward interoperability between these theories. (shrink)

Critics often charge that agent-causal libertarianism is unintelligible due to the uniqueness of agent-causation—the sui generis causal relationship said to be involved when agents make free choices. This paper presents five objections, which are taken to be the only good objections, to agent-causal libertarianism and argues they all fail to show agent-causal libertarianism is unintelligible. The first four objections fail outright. The fifth objection fails in a special way. Naturalistic agent-causal libertarian theories succumb to this fifth objection; theistic agent-causal libertarian (...) theories do not. This entails that if agent-causal libertarianism is intelligible, then it is only so within theism. (shrink)

One standard criticism of the doctrine of continuous creation is that it entails the occasionalist position that God alone is a true cause and that the events we commonly identify as causes are merely the occasions upon which God brings about effects. I begin by clearly stating Malebranche's argument from continuous creation to occasionalism. Next, I examine two strategies for resisting Malebranche's argument – strong and weak concurrentism – and argue that weak concurrentism is the more promising strategy. Finally, I (...) argue that weak concurrentism requires a necessitarian approach to secondary causation. (shrink)

Richard Swinburne is one of the best-known names in the international philosophical scene. His apologetic project is considered one of the largest and most impactful and profound of the last century. The interview conducted here explores many biographical and theoretical issues (Omniscience, Eternity, God’s existence, Free will, Analogy, Relational ontology and Powers ontology, Soul-Body relation, Trinity, Evil) and it aims to trace a broad (albeit necessarily partial) path through his numerous works. The interview took place in 2016, in Oxford, at (...) Swinburne’s home. Today, Swinburne’s thought is discussed in monographs and doctoral theses, witnessing to an influence that will undoubtedly be lasting. (shrink)

I argue that the debate between proponents of substance causation and proponents of causation by powers, as to whether substances or their powers are causes, hinges on whether or not powers are self-exemplifying or non-self-exemplifying properties. Substance causation is committed to powers being non-self-exemplifying properties while causation by powers is committed to powers being self-exemplifying properties. I then argue that powers are non-self-exemplifying properties, in support of substance causation.

A growing number of philosophers now hold that agent causation is required for agency, or free will, or moral responsibility. To clarify what is at issue, this paper begins with a distinction between agent causation that is ontologically fundamental and agent causation that is reducible to or realized in causation by events or states. It is widely accepted that agency presents us with the latter; the view in question claims a need for the former. The paper then examines a “disappearing (...) agent” argument from Derk Pereboom that is aimed at showing that free will requires agent causation that is ontologically fundamental. It is argued that the argument fails. Further, it is argued that, contrary to Pereboom’s claim, the issue raised by his disappearing agent argument is distinct from the problem of present luck that libertarian theories of free will face. The paper concludes with an assessment of the prospects for success of a disappearing agent argument showing that agent causation that is ontologically fundamental is required for agency tout court. (shrink)

Several philosophers claim that the phenomenology of one’s own agency conflicts with standard causal theories of action, couched in terms of causation by mental events or states. Others say that the phenomenology is prima facie incompatible with such a theory, even if in the end a reconciliation can be worked out. Here it is argued that the type of action theory in question is consistent with what can plausibly be said to be presented to us in our experience of our (...) agency. Several routes to a claim that there is nevertheless a prima facie incompatibility are examined, and all are found wanting. The phenomenology of agency, it is argued, is no threat to a standard causal theory of action. (shrink)

A theory of causation with ‘tendencies’ as causal con- nections is proposed. Not, however, as ‘necessary connec- tions’: causes are not sufficient, they do not necessitate their effects. The theory is not an analysis of the concept of causation, but a description of what is the case in typical cases of causation. Therefore it does not strictly contradict any analysis of the concept of causation, not even reduct- ive ones. It would even be supported by a counterfactual or a probabilistic (...) analysis. (shrink)

The tendency theory of causation and its use in ontological engineering is described.