And this paper is an attempt to say precisely how, thus addressing a philosophical problem which is commonly taken to be a serious one. It does so, however, in quite an idiosyncratic way. It is based on the account of inductive schemes I have given in (1988) and (1990a) and on the conception of causation I have presented in (1980), (1983), and (1990b), and it intends to fill one of many gaps which have been left by these papers. Still, I (...) have tried to make this paper self-contained. Section 1 explains the philosophical question this paper is about; in more general terms it asks what might be meant by objectifying epistemic states or features of them and to which extent epistemic states can be objectified. The next sections introduce the basis I rely on with formal precision and some explanation; section 2 deals with induction and section 3 with causation. Within these confines, section 4 attempts to give an explication of the relevant sense of objectification and section 5 investigates the extent to which various features of epistemic states are objectifiable. The two most salient results are roughly that the relation "A is a reason for B" cannot be objectified at all and that the relation "A is a cause of B" can be objectified only under substantial, though reasonable restrictions. What has all of this to do with probability? A lot. The paper trades on a pervasive duality between probabilistic and deterministic epistemology, between a probabilistic representation of epistemic states together with a theory of probabilistic causation and another representation of epistemic states which I call deterministic because it lends itself, in a perfectly parallel fashion, to a theory of deterministic causation. Here I explicitly deal only with the deterministic side, but the duality should pave the way for further conclusions concerning objective probabilities and statistical laws. This outlook is briefly expanded in the final section 6. (shrink)

Many candidates have been tried out as proximate causes of actions: belief-desire pairs, volitions, motives, intentions, and other kinds of pro-attitudes. None of these mental states or events, however, seems to be able to do the trick, that is, to get things going. Each of them may occur without an appropriate action ensuing. After reviewing several attempts at closing the alleged “causal gap”, it is argued that on a correct analysis, there is no missing link waiting to be discovered. On (...) the counterfactual account of singular causation, the onset of belief or desire may perfectly well cause an action, although no kind of mental antecedent is ever a causally sufficient condition for a specific kind of action to occur. (shrink)

The paper builds on the basically Humean idea that A is a cause of B iff A and B both occur, A precedes B, and A raises the metaphysical or epistemic status of B given the obtaining circumstances. It argues that in pursuit of a theory of deterministic causation this ‘status raising’ is best explicated not in regularity or counterfactual terms, but in terms of ranking functions. On this basis, it constructs a rigorous theory of deterministic causation that successfully deals (...) with cases of overdetermination and pre-emption. It finally indicates how the account's profound epistemic relativization induced by ranking theory can be undone. Introduction Variables, propositions, time Induction first Causation Redundant causation Objectivization. (shrink)

The paper extends the framework of outcomes in branching space-time (Kowalski and Placek [1999]) by assigning probabilities to outcomes of events, where these probabilities are interpreted either epistemically or as weighted possibilities. In resulting models I define the notion of common cause of correlated outcomes of a single event, and investigate which setups allow for the introduction of common causes. It turns out that a deterministic common cause can always be introduced, but (surprisingly) only special setups permit the introduction of (...) truly stochastic common causes. I analyse next the Bell-Aspect experiment and derive the Bell-CH inequalities. I observe that we postulate there not a common cause for outcomes of a single event but rather a common common cause that accounts for outcomes of many events, where 'events' mean 'measurements with (different) directions of polarization'. Since the inequalities are violated, I claim that no causal story can be told about the Bell correlations, where causality is subliminal and restricted by screening-off condition. Similarly, given certain intuitive principles, no deterministic story can be told about these correlations. (shrink)

Wolfgang Spohn’s Frege prize lecture, like the work on which it is based, is a tour de force of rich, elegant, coherent argument about how the projected world that we experience is constructed. But we do not live in this projected world nor reason about it. The things Spohn constructs are there from the start—or so my Stanford School pragmatism teaches. This paper explores a deep difference in philosophical approaches—Spohn’s elegant proofs versus the stocky, tangled arguments I advocate—and illustrates how (...) these play out in far more detailed disputes about the nature of causality and causal inference. (shrink)

The paper displays the similarity between the theory of probabilistic causation developed by Glymour et al. since 1983 and mine developed since 1976: the core of both is that causal graphs are Bayesian nets. The similarity extends to the treatment of actions or interventions in the two theories. But there is also a crucial difference. Glymour et al. take causal dependencies as primitive and argue them to behave like Bayesian nets under wide circumstances. By contrast, I argue the behavior of (...) Bayesian nets to be ultimately the defining characteristic of causal dependence. (shrink)

As the paper explains, it is crucial to epistemology in general and to the theory of causation in particular to investigate the properties of conditional independence as completely as possible. The paper summarizes the most important results concerning conditional independence with respect to two important representations of epistemic states, namely (strictly positive) probability measures and natural conditional (or disbelief or ranking) functions. It finally adds some new observations.

This paper is the most complete presentation of my views on deterministic causation. It develops the deterministic theory in perfect parallel to my theory of probabilistic causation and thus unites the two aspects. It also argues that the theory presented is superior to all regularity and all counterfactual theories of causation.

The paper pleads for compatibilism by distinguishing the first-person’s normative and the observer’s empirical perspective. In the normative perspective one’s own actions are uncaused and free, in the empirical perspective they are caused and may be predetermined. Still, there is only one notion of causation that is able to account for the relation between the causal conceptions within the two perspectives. The other main idea for explicating free will by explaining free actions or intentions as appropriately caused in a specified (...) way is acknowledged, but not discussed. The paper finally argues that the normative and the empirical perspective are on a par; none is prior; even from within the empirical perspective the normative perspective is ineliminable. (shrink)

The paper attempts to reconcile two very different approaches to the concept of causation. In the original form, it is the opposition found in Laplace between his doctrine of constant and variable causes on the one hand and his mechanistic determinism on the other. This tension was described clearly only by Maxwell who stressed the contrast between the statistical and the dynamical method (calling the latter also the historical or strictly kinetic method). A similar dichotomy surfaces in the work of (...) Wesley Salmon who distinguished statistical from aleatory causation. Acknowledging that we seem to have conflicting intuitions about causality and explanation, I argue that assigning probabilistic laws a fundamental role in scientific explanation does not conflict with the assumption of Laplacian determinism. This is due to the fact that in most systems of interest, we confront a complex interaction of multiple causal factors and an extreme sensitivity to small variations of initial conditions. (shrink)

This paper deals with Hans Reichenbach's common cause principle. It was propounded by him in, and has been developed and widely applied by Wesley Salmon, e.g. in and. Thus, it has become one of the focal points of the continuing discussion of causation. The paper addresses five questions. Section 1 asks: What does the principle say? And section 2 asks: What is its philosophical significance? The most important question, of course, is this: Is the principle true? To answer that question, (...) however, one must first consider how one might one argue about it at all. One can do so by way of examples, the subject of section 3, or more theoretically, which is the goal of section 4. Based on an explication of probabilistic causation proposed by me in,, and, section 4 shows that a variant of the principle is provable within a classical framework. The question naturally arises whether the proved variant is adequate, or too weak. This is pursued in section 5. My main conclusion will be that some version of Reichenbach's principle is provably true, and others may be. This may seem overly ambitious, but it is not. The paper does not make any progress on essential worries about the common cause principle arising in the quantum domain; it only establishes more rigorously what has been thought to be plausible at least within a classical framework. (shrink)