Non-reductive interventionist theories of causation and methodologies of causal reasoning embedded in that theoretical framework have become increasingly popular in recent years. This paper argues that one variant of an interventionist account of causation, viz. the one presented, for example, in Woodward (2003), is unsuited as a theoretical fundament of interventionist methodologies of causal reasoning, because it renders corresponding methodologies incapable of uncovering a causal structure in a finite number of steps. This finding runs counter to Woodward's own assessment and (...) to other recent studies which presume that Woodward's version of interventionism is effectively applicable to uncover causal structures, e.g. Campbell (2007) or Shapiro and Sober (2007). (shrink)

This paper defends an interventionist account of causation by construing this account as a contribution to methodology, rather than as a set of theses about the ontology or metaphysics of causation. It also uses the topic of causation to raise some more general issues about the relation between, on the one hand, methodology, and, on the other hand, ontology and metaphysics, as these are understood in contemporary philosophical discussion, particularly among so-called analytic metaphysicians. It concludes with the suggestion that issues (...) about the ontology of causation often can be fruitfully reconstrued as methodological proposals. (shrink)

This is one of a pair of discussion notes comparing some features of the account of causation in Wolfgang Spohn’s Laws of Belief with the “interventionist” account in James Woodward’s Making Things Happen. This note locates the core difference of the accounts in the fact that Woodward’s account follows an epistemological order, while Spohn’s follows a conceptual order. This unfolds in five further differences: type- versus token-level causation, reference to time, actual/counterfactual intervention versus epistemic/suppositional wiggling, a circular versus a circle-free (...) conception of the circumstances of a direct causal relation, and absolute versus model-relative causation. (shrink)

We start this paper by arguing that causality should, in analogy with force in Newtonian physics, be understood as a theoretical concept that is not explicated by a single definition, but by the axioms of a theory. Such an understanding of causality implicitly underlies the well-known theory of causal nets and has been explicitly promoted by Glymour. In this paper we investigate the explanatory warrant and empirical content of TCN. We sketch how the assumption of directed cause–effect relations can be (...) philosophically justified by an inference to the best explanation. We then ask whether the explanations provided by TCN are merely post-facto or have independently testable empirical content. To answer this question we develop a fine-grained axiomatization of TCN, including a distinction of different kinds of faithfulness. A number of theorems show that although the core axioms of TCN are empirically empty, extended versions of TCN have successively increasing empirical content. (shrink)

Causation is a macroscopic phenomenon. The temporal asymmetry displayed by causation must somehow emerge along with other asymmetric macroscopic phenomena like entropy increase and the arrow of radiation. I shall approach this issue by considering ‘causal inference’ techniques that allow causal relations to be inferred from sets of observed correlations. I shall show that these techniques are best explained by a reduction of causation to structures of equations with probabilistically independent exogenous terms. This exogenous probabilistic independence imposes a recursive order (...) on these equations and a consequent distinction between dependent and independent variables that lines up with the temporal asymmetry of causation. (shrink)

The most serious candidates for common causes that fail to screen off and thus violate the causal Markov condition refer to quantum phenomena. In her seminal debate with Hausman and Woodward, Cartwright early on focussed on unfortunate non-quantum examples. Especially, Hausman and Woodward’s redescriptions of quantum cases saving the CMC remain unchallenged. This paper takes up this lose end of the discussion and aims to resolve the debate in favour of Cartwright’s position. It systematically considers redescriptions of ICC structures, including (...) those by Hausman and Woodward, and explains why these are inappropriate, when quantum mechanics is true. It first shows that all cases of purported quantum ICCs are cases of entanglement and then, using the tools of causal modelling, it provides an analysis of the quantum mechanical formalism for the case that the collapse of entangled systems is best described as a causal model with an ICC. (shrink)

Some explanations appeal to facts about the causal structure of a system in order to shed light on a particular phenomenon; these are explanations which do more than cite the causes X and Y of some state-of-affairs Z, but rather appeal to “macro-level” causal features—for example the fact that A causes B as well as C, or perhaps that D is a strong inhibitor of E—in order to explain Z. Appeals to these kinds of “macro-level” causal features appear in a (...) wide variety of social scientific and biological research; statements about features such as “patriarchy,” “healthcare infrastructure,” and “functioning DNA repair mechanism,” for instance, can be understood as claims about what would be different in a system with a different causal structure. I suggest interpreting counterfactual questions involving structural features as questions about alternative parameter settings of causal models, and propose an extension of the usual interventionist framework for causal explanation which enables scientists to explore the consequences of interventions on “macro-level” structure. (shrink)

According to manipulationist accounts of causal explanation, to explain an event is to show how it could be changed by intervening on its cause. The relevant change must be a ‘serious possibility’ claims Woodward 2003, distinct from mere logical or physical possibility—approximating something I call ‘scientific possibility’. This idea creates significant difficulties: background knowledge is necessary for judgments of possibility. Yet the primary vehicles of explanation in manipulationism are ‘invariant’ generalisations, and these are not well adapted to encoding such knowledge, (...) especially in the social sciences, as some of it is non-causal. Ceteris paribus (CP) laws or generalisations labour under no such difficulty. A survey of research methods such as case and comparative studies, randomised control trials, ethnography, and structural equation modeling, suggests that it would be more difficult and in some instances impossible to try to represent the output of each method in invariant generalisations; and that this is because in each method causal and non-causal background knowledge mesh in a way that cannot easily be accounted for in manipulationist terms. Ceteris paribus-generalisations being superior in this regard, a theory of explanation based on the latter is a better fit for social science. (shrink)

Proponents of various causal exclusion arguments claim that for any given event, there is often a unique level of granularity at which that event is caused. Against these causal exclusion arguments, causal ecumenists argue that the same event or phenomenon can be caused at multiple levels of granularity. This paper argues that the Bayesian network approach to representing the causal structure of target systems is consistent with causal ecumenism. Given the ubiquity of Bayesian networks as a tool for representing causal (...) structure in both philosophy of science and science itself, this result speaks in favor of the ecumenical view, and against rival exclusionary accounts. Gebharter’s (Philos Phenomenol Res 95(2):353–375, 2017) argument that the Bayes nets formalism is consistent with causal exclusion is considered and rebutted. (shrink)

Whether mental properties are identical with neural properties is one of the central questions of contemporary philosophy of mind. Many philosophers agree that even if mental properties are identical with neural properties, the mind-brain identity thesis cannot be established on empirical grounds, but only be vindicated by theoretical philosophical considerations. In his paper ‘When Is a Brain Like the Planet?’, Clark Glymour proposes a causal criterion for local property identifications and claims that this criterion can be used to empirically establish (...) local identities between mental and neural properties. If successful, such an account would settle the debate on the mind-body problem. In this paper, I argue that Glymour’s approach falls short of its aims. The causal criterion which he proposes does not provide a sufficient condition for the local identification of properties. Moreover, his account does not succeed in rendering local mind-brain identities empirically testable. Therefore, the mind-body problem cannot be solved as easily as Glymour assumes. (shrink)

Woodward’s interventionist theory of causation is beset by a problem of circularity: the analysis of causes is in terms of interventions, and the analysis of interventions is in terms of causes. This is not in itself an argument against the correctness of the analysis. But by requiring us to have causal knowledge prior to making any judgements about causation, Woodward’s theory does make it mysterious how we can ever start acquiring causal knowledge. We present a solution to this problem by (...) showing how the interventionist notion of causation can be rationally generated from a more primitive agency notion of causation. The agency notion is easily and non-circularly applicable, but fails when we attempt to capture causal relations between non-actions. We show that the interventionist notion of causation serves as an appropriate generalisation of the agency notion. Furthermore, the causal judgements based on the latter generally remain true when rephrased in terms of the former, which allows one to use the causal knowledge gained by applying the agency notion as a basis for applying Woodward’s interventionist theory. We then present an overview of relevant empirical evidence from developmental psychology which shows that our proposed rational reconstruction lines up neatly with the actual development of causal reasoning in children. This gives additional plausibility to our proposal. The article thus provides a solution to one of the main problems of interventionism while keeping Woodward’s analysis intact. (shrink)

Philosophical theories of causation are commonly judged by their ability to correctly determine whether there is a causal relation present in intuitively clear example scenarios. If the theories survive this test, they are then used to answer big philosophical questions about causation. This Method of Examples is attractive because it seems to allow us to determine the quality of a theory of causation independently of answering the big philosophical questions; which is good, since it means that we can then non-circularly (...) use the theories judged to be best to answer those questions. However, the current article argues that this virtue of the Method of Examples is an illusion. In particular, I argue that the necessary step of judging whether a proposed analysis of causation is reductive can only be taken after many of the most vexing philosophical questions about causation have already been answered. It is then shown that a rejection of the methodological supremacy of the Method of Examples leads to a more pluralistic method of judging theories of causation, a pluralism that benefits non-standard approaches like interventionism and agency theories. (shrink)

In this paper I argue that constitutive relevance relations in mechanisms behave like a special kind of causal relation in at least one important respect: Under suitable circumstances constitutive relevance relations produce the Markov factorization. Based on this observation one may wonder whether standard methods for causal discovery could be fruitfully applied to uncover constitutive relevance relations. This paper is intended as a first step into this new area of philosophical research. I investigate to what extent the PC algorithm, originally (...) developed for causal search, can be used for constitutive relevance discovery. I also discuss possible objections and certain limitations of a constitutive relevance discovery procedure based on PC. (shrink)

Strong notions of free will are closely connected to the possibility to do otherwise as well as to an agent’s ability to causally influence her environment via her decisions controlling her actions. In this paper we employ techniques from the causal modeling literature to investigate whether a notion of free will subscribing to one or both of these requirements is compatible with naturalistic views of the world such as non-reductive physicalism to the background of determinism and indeterminism. We argue that (...) from a causal modeler’s perspective the only possibility to get both requirements consists in subscribing to reductive physicalism and indeterminism. (shrink)

In this paper we develop an analysis of dispositions by means of causal Bayes nets. In particular, we analyze dispositions as cause-effect structures that increase the probability of the manifestation when the stimulus is brought about by intervention in certain circumstances. We then highlight several advantages of our analysis and how it can handle problems arising for classical analyses of dispositions such as masks, mimickers, and finks.

The causal Bayes net framework specifies a set of axioms for causal discovery. This article explores the set of causal variables that function as relata in these axioms. Spirtes showed how a causal system can be equivalently described by two different sets of variables that stand in a non-trivial translation-relation to each other, suggesting that there is no “correct” set of causal variables. I extend Spirtes’ result to the general framework of linear structural equation models and then explore to what (...) extent the possibility to intervene or a preference for simpler causal systems may help in selecting among sets of causal variables. (shrink)

An interventionist account of causation characterizes causal relations in terms of changes resulting from particular interventions. I provide a new example of a causal relation for which there does not exist an intervention satisfying the common interventionist standard. I consider adaptations that would save this standard and describe their implications for an interventionist account of causation. No adaptation preserves all the aspects that make the interventionist account appealing. Part of the fallout is a clearer account of the difficulties in characterizing (...) so-called “soft” interventions. (shrink)